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Titlebook: Advances in Dementia Research; K. Jellinger,R. Schmidt,M. Windisch Conference proceedings 2000 Springer-Verlag Wien 2000 Assessment.Nervou

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發(fā)表于 2025-3-21 17:10:39 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Advances in Dementia Research
影響因子2023K. Jellinger,R. Schmidt,M. Windisch
視頻videohttp://file.papertrans.cn/148/147762/147762.mp4
學(xué)科分類Journal of Neural Transmission. Supplementa
圖書封面Titlebook: Advances in Dementia Research;  K. Jellinger,R. Schmidt,M. Windisch Conference proceedings 2000 Springer-Verlag Wien 2000 Assessment.Nervou
影響因子Dementia is a major health problem that becomes increasingly common with advancing age. Despite recent progress in neurobiology and molecular genetics, the aetiology and pathogenesis of most dementia disorders are still poorly understood, and early exact diagnosis as a prerequisite of effective treatment needs to be improved. The present volume includes the contributions of renowned experts in the field of neurodegeneration presented at the International Symposium "Ageing and Dementia 1999”, September 24–26, 1999 at Graz. It focusses on genetics, epidemiology, new neuroimaging techniques, and the role of vascular, immunological and other mechanisms including oxidative stress and estrogens in the development of neurodegeneration and dementia. Recent developments in diagnosis and therapy of Alzheimer disease and related disorders are reviewed and future therapeutic approaches are discussed. Thus, this volume provides a timely overview of most recent developments in dementia research and treatment strategies of dementia disorders.
Pindex Conference proceedings 2000
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https://doi.org/10.1007/978-1-4471-1909-8linical predictors, and cognitive consequences of MRI white matter hyperintensity in elderly individuals without neuropsychiatric disease. Lesion progression was found in a total of 49 (17.9%) individuals. It was minor in 27 (9.9%) and marked in 22 (8.1%) participants. Diastolic blood pressure (odds
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Robert D. Goldman,Peter M. Steinertiation in defining the cognitive syndrome, in vascular etiologies, and allowable brain changes in current clinical definitions of VaD have resulted in variable estimates of prevalence, of groups of subjects, and of the types and distribution of putative causal brain lesions. Thus current criteria fo
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Intermediate Filament-Associated Proteinsnormalities consists of hyperintense foci in the deep and subcortical white matter probably related to microvascular disturbances and of signal hyperintensities around the lateral ventricles. It has also been suggested that these abnormalities may contribute to the development of cognitive impairmen
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