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Titlebook: Advances in Artificial Intelligence; 31st Canadian Confer Ebrahim Bagheri,Jackie C.K. Cheung Conference proceedings 2018 Springer Internati

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發(fā)表于 2025-3-21 18:58:16 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Advances in Artificial Intelligence
期刊簡(jiǎn)稱31st Canadian Confer
影響因子2023Ebrahim Bagheri,Jackie C.K. Cheung
視頻videohttp://file.papertrans.cn/147/146708/146708.mp4
學(xué)科分類Lecture Notes in Computer Science
圖書封面Titlebook: Advances in Artificial Intelligence; 31st Canadian Confer Ebrahim Bagheri,Jackie C.K. Cheung Conference proceedings 2018 Springer Internati
影響因子.This book constitutes the refereed proceedings of the 31th Canadian Conference on Artificial Intelligence, Canadian AI 2018, held in Toronto, ON, Canada, in May 2018...?.The 16 regular papers and 18 short papers presented together with 7 Graduate Student Symposium papers and 4 Industry Track papers were carefully reviewed and selected from 72 submissions. The focus of the conference was on artificial intelligence research and advanced information and communications technology..
Pindex Conference proceedings 2018
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Synthesizing Controllers: On the Correspondence Between LTL Synthesis and Non-deterministic Planningiew on recent progress in hepatology.Important source of ref.Signaling Pathways in Liver Diseases, 2nd edition focuses on signaling pathways which are particularly important in liver diseases. Recent progress brought hepatology to new frontiers. The increasing frequency of surgery on steatotic and c
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One Single Deep Bidirectional LSTM Network for Word Sense Disambiguation of Text Datah with practical clinical applications.Presents a consistent.Signal transduction pathways are at the core of most biological processes and are critical regulators of heart physiology and pathophysiology.? The heart is both a transmitter and dynamic receptor of a variety of intracellular and extracel
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Reranking Candidate Lists for Improved Lexical Inductionulement à redécouvrir. L’apoptose est une mort cellulaire programmée, active, mise en oeuvre en réponse à des signaux d’origine intracellulaire (lésions de l’ADN, anomalies de la mitose, stress oxydatif) ou extracellulaires (messages de mort provenant d’autres cellules) et aboutissant à l’activation
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