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Titlebook: Adrenergic Mechanisms in Myocardial Ischemia; Gerd Heusch,John Ross Conference proceedings 1991 Springer-Verlag Berlin Heidelberg 1991 arr

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樓主: Taylor
11#
發(fā)表于 2025-3-23 11:17:34 | 只看該作者
Overview: 978-3-662-11040-9978-3-662-11038-6
12#
發(fā)表于 2025-3-23 15:27:10 | 只看該作者
Installing DB2 for Linux and Windowsf these pathways and how to selectively activate or eliminate them allows for the production of unique animal models for further investigation about the interaction between the cardiac sympathetics and the heart during ischemia.
13#
發(fā)表于 2025-3-23 18:43:56 | 只看該作者
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發(fā)表于 2025-3-23 22:54:12 | 只看該作者
978-3-662-11040-9Springer-Verlag Berlin Heidelberg 1991
15#
發(fā)表于 2025-3-24 05:47:13 | 只看該作者
16#
發(fā)表于 2025-3-24 07:04:24 | 只看該作者
Installing DB2 for Linux and Windowsf these pathways and how to selectively activate or eliminate them allows for the production of unique animal models for further investigation about the interaction between the cardiac sympathetics and the heart during ischemia.
17#
發(fā)表于 2025-3-24 14:13:59 | 只看該作者
https://doi.org/10.1007/978-1-4302-0548-7s. Adrenergic stimulation of the ischemic myocardium is predominantly due to increased local noradrenaline concentrations in the heart, whereas plasma catecholamine levels are of minor relevance. During the first few minutes of ischemia, efferent sympathetic nerves are activated. Excessive accumulat
18#
發(fā)表于 2025-3-24 17:35:57 | 只看該作者
What Is DB2 and Where Can I Get It?cloned receptor, it appears to possess characteristics analogous to other membrane-bound receptors including seven membrane spanning domains, three extracellular, and three intracellular loops, with extensive glycosylation near the extracellular amino terminus. Although the receptor is coupled to ph
19#
發(fā)表于 2025-3-24 22:30:33 | 只看該作者
20#
發(fā)表于 2025-3-24 23:27:11 | 只看該作者
What Is DB2 and Where Can I Get It?er these conditions, treatment with β-adrenoceptor agonists seems to be of limited value as it might further down-regulate cardiac β-adrenoceptors, resulting, finally, in a loss of therapeutic efficacy. However, β-adrenoceptor antagonists might have beneficial effects, because they can protect the m
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