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Titlebook: Adalat? in the Asian Pacific Region; David T. Kelly Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 Calcium-Antagoniste

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期刊全稱Adalat? in the Asian Pacific Region
影響因子2023David T. Kelly
視頻videohttp://file.papertrans.cn/145/144493/144493.mp4
圖書封面Titlebook: Adalat? in the Asian Pacific Region;  David T. Kelly Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 Calcium-Antagoniste
影響因子These are the proceedings of the third Asian Pacific Adalat Symposium. The first was held in Tokyo in 1982 and the second in Sydney in 1985. We were honoured that the late Dr. Sukaman and his colleagues were able to host this third symposium in Jakarta. This meeting was designed to contribute to and to promote international collaboration in cardiovascular research, and to help promote further knowledge and understanding about the treatment of vascular disease in the Asian Pacific area. Nifedipine was originally investigated in the mid-1960s by Prof. Fleckenstein. His first results showed this drug was a powerful calcium antagonist which altered excitation contraction. At this time, there was a rapid development of knowledge about the pathophysiology of ischaemic heart disease, and thus the important pharmacological work on calcium antagonists such as nifedipine paralleled and complimented the newer understanding of coronary physiology in man. In a short time most therapy was designed to reduce myocardial oxygen consumption and demand.
Pindex Conference proceedings 1989
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https://doi.org/10.1007/978-3-031-68617-7ct “watershed” between angina (a potentially reversible form of ischemia) and myocardial infarction (after 30 min an irreversible process; Table 1). Deciding in which state of ischemia a patient finds himself belongs to the most difficult decisions to be made in clinical medicine.
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Application of Machine Learning with R,84; Tiefenbrunn et al. 1983). Although it has been reported that nifedipine can improve diastolic properties of hypertrophic cardiomyopathy (Lorell et al. 1982), whether it can modify relaxation and ventricular filling abnormalities during ischemia is not yet know. Since left ventricular relaxation
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Artificial Intelligence and Human Rights,o suppress atherosclerosis in cholesterol-fed rabbits without lowering serum cholesterol levels. Kramsch et al. (1980, 1981) reported that lanthanum, an inorganic calcium antagonist, suppressed atherosclerosis in cholesterol- fed rabbits and monkeys, and Henry and Bentley (1981) demonstrated that ni
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https://doi.org/10.1007/978-3-031-63451-2istance and arterial pressure (Guazzi et al. 1977; Robinson et al. 1980). A satisfactory chronic reduction in arterial pressure following the administration of this drug has been demonstrated in hypertensive subjects (Kiowski et al. 1983; Murphy et al. 1983). However, the explanations for the mechan
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