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Titlebook: Acute Lung Injury; John J. Marini,Timothy W. Evans Conference proceedings 1998 Springer-Verlag Berlin Heidelberg 1998 Sepsis.care.clinical

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樓主: antithetic
21#
發(fā)表于 2025-3-25 04:34:24 | 只看該作者
BAL Inflammatory Markers of Initiation and Resolution of ALIriculums need to address land and water degradation as well as rehabili- tion issues. These topics are becoming more important to society, and an increasing number of earth science students are pursuing 978-3-642-44609-2978-3-642-12419-8
22#
發(fā)表于 2025-3-25 09:43:42 | 只看該作者
23#
發(fā)表于 2025-3-25 13:45:14 | 只看該作者
Near—Optimal Control of Dynamic Flowshopsrome (ARDS), cytokines occupy a central place. Since the discovery of the role of these substances in inflammatory processes of different types and locations of the human body, a number of experimental and clinical investigations suggest that they play an important role in ARDS.
24#
發(fā)表于 2025-3-25 16:03:54 | 只看該作者
25#
發(fā)表于 2025-3-25 21:16:37 | 只看該作者
26#
發(fā)表于 2025-3-26 00:19:19 | 只看該作者
Near—Optimal Controls of Dynamic Jobshopse lungs with a low funcional residual capacity, and diffuse radiographic infiltrates due to increased lung microvascular permeability [1]. Since first described in 1967 [2], the syndrome has been associated with a high mortality, and it is disappointing that despite many clinical and laboratory inve
27#
發(fā)表于 2025-3-26 07:06:58 | 只看該作者
28#
發(fā)表于 2025-3-26 10:39:29 | 只看該作者
https://doi.org/10.1007/0-387-27615-7e of diffuse pulmonary infiltrates. The mechanisms that initiate, and then perpetuate the lung inflammation seen in ARDS remain poorly understood. Initially the syndrome was thought to involve only the lungs and to be the result of a single process; activation of the complement cascade. Subsequent i
29#
發(fā)表于 2025-3-26 14:55:42 | 只看該作者
30#
發(fā)表于 2025-3-26 16:49:54 | 只看該作者
Concept of Near—Optimal Control may directly affect lung parenchyma from the alveolar side (e.g. gastric acid aspiration), or — more classically — the lung vasculature may be the primary target site of circulating humoral or cellular mediators activated under conditions of systemic inflammatory events such as sepsis or severe pol
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