Titlebook: Acute Catabolic State; Arthur Revhaug Book 1996 Springer-Verlag Berlin Heidelberg 1996 Acute Catabolic State.Critical Illness.Trauma.organ

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Alessandro Saffiotti,Zbigniew WasikThe acute response to skin injury following burn is characterized by hypermetabolism, increased cardiac output, lipolysis, gluconeogenesis, and proteolysis. This review will describe the early responses to thermal trauma and potential methods for modulating deleterious consequences of these responses.

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Wolfram Burgard,Cyrill Stachniss,Dirk H?hnelneeds … of repair and maintenance”, the net loss far exceeding the loss due to immobilization [1] and being too extensive to come from the local area of damaged tissue alone [2]. From the relationship between the losses of nitrogen, sulfur, and phosphorus Cuthbertson postulated that the catabolized

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https://doi.org/10.1007/978-3-540-73422-2d [1], but for over 100 years neurochemical and physiological evidence supporting the role of the brain as the integrative station for neural and hormonal regulation of peripheral metabolism has been reported by several studies [2–9]. Primarily, the hypothalamus is considered as one of the main stru

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Jonghyun Kim,Seungjun Lee,Poong Hyun Seonggulatory actions in target cells. The word is derived from the Greek hormaein, meaning “to set in motion” or “spur on” and was first used in 1905 by Starling and Bayliss. Study of the organs whose function is to secrete these substances (the endocrine system) over the subsequent 90 years has allowed

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Gabriel L. Miller,Eric R. Wagnerolic state. This is true in the sence that muscular strength has no relevance in fighting infections or in improving oxygenation during critical illness. Nevertheless, skeletal muscle has two major roles for such patients: (1) to serve as a reservoire for amino acid substrates needed in other tissue

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Sensor Models and Multisensor Integrationmpathetic nervous system [1]. The heart and vascular systems are effectors in this stress response and adapt the organism to the stressful situation by a number of compensatory alterations. This adaptation is illustrated by the classic hypovolemic shock with tachycardia, increased stroke work, and e

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