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Titlebook: Whole Body Ultrasonography in the Critically Ill; Daniel A. Lichtenstein Book 2010 Springer-Verlag Berlin Heidelberg 2010 Critical Ultraso

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31#
發(fā)表于 2025-3-26 23:15:30 | 只看該作者
Lung and Interstitial Syndromes, demonstrating that the diagnosis of pleural effusion or alveolar disorders was just an appetizer. If lung ultrasound is a raison d’être of critical ultrasound, the potential of interstitial syndrome is the raison d’être of lung ultrasound. Based on artifact analysis, it changes the approach to th
32#
發(fā)表于 2025-3-27 04:49:58 | 只看該作者
33#
發(fā)表于 2025-3-27 07:26:12 | 只看該作者
Peritoneumas dealt with before the lung chapters, which standardize this semiotic of air. Meanwhile, one practical suggested label for designating the abdominal gas structures is the term “G-lines” – . for gut.
34#
發(fā)表于 2025-3-27 10:16:08 | 只看該作者
Liver probes. Their only use seems to be for accurate measurement of hepatic size, which is a tool of interest for the radiologist but not yet a tool for the intensivist. Unexpected uses may emerge from knowing the size of the liver, but this can be achieved using our probe.
35#
發(fā)表于 2025-3-27 17:13:53 | 只看該作者
36#
發(fā)表于 2025-3-27 20:53:46 | 只看該作者
in a variety of settings, including unexplained shock, management of hemodynamic instability, acute respiratory failure (the BLUE protocol), and the critically ill neonate, is carefully explained. Interventiona978-3-642-42307-9978-3-642-05328-3
37#
發(fā)表于 2025-3-27 23:49:32 | 只看該作者
Daniel A. Lichtensteinowledge regarding human β-cell replication. It seems clear that unlike in rodents, spontaneous replication of adult human β cells is uncommon. Further, standard manipulations, nutrients, and growth factors that induce rodent β-cell replication fail to do so in human β cells. In this chapter we focus
38#
發(fā)表于 2025-3-28 03:26:31 | 只看該作者
39#
發(fā)表于 2025-3-28 08:04:15 | 只看該作者
40#
發(fā)表于 2025-3-28 11:56:21 | 只看該作者
Daniel A. Lichtensteinst-translationally modified by deamidation in its loop domain in human liver tissues; deamidated Bcl-xL is a major form in normal liver tissues, whereas the level of deamidated Bcl-xL is lower than that of unmodified Bcl-xL in the majority of HCC tissues. As protein deamidation of Bcl-xL leads to a
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