Titlebook: Alcohol Intoxication and Withdrawal I; Experimental Studies Milton M. Gross Book 1973 The Editor(s) (if applicable) and The Author(s), unde

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Marko Grobelnik,Maja ?krjanc,Darko Zupani?e last few decades to produce a set of conditions which results in the oral overindulgence of alcohol has been rather disappointing. Transient large or sustained moderate intake increases have been demonstrated. However, no chronic, oral overindulgence model has emerged in which animals maintain hig

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A Role for Tetrahydroisoquinoline Alkaloids as False Adrenergic Neurotransmitters in Alcoholismese substances can then function as false adrenergic transmitters (Cohen and Collins, 1970). By interfering with adrenergic mechanisms in the brain and in the periphery, biosynthesized TIQ alkaloids may be capable of altering mood and behavior. In this way, they may play a role during alcohol intoxication and in post-intoxication states.

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Valerio Grossi,Dino Pedreschi,Franco Turinihe development of a state of addiction to ethanol. Such theories have been nurtured by a recognition of a common pathway of catabolism for the deaminated derivatives of the biogenic amines and acetaldehyde, the initial metabolite of ethanol.

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Marko Grobelnik,Maja ?krjanc,Darko Zupani?r sustained moderate intake increases have been demonstrated. However, no chronic, oral overindulgence model has emerged in which animals maintain high blood ethanol levels leading to physical dependence indices unequivocally attributable to the elevated ethanol intake.

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