派博傳思國際中心

標(biāo)題: Titlebook: Regulation of Signal Transduction in Human Cell Research; Nariyoshi Shinomiya,Hiroaki Kataoka,Qian Xie Book 2018 Springer Nature Singapore [打印本頁]

作者: 海市蜃樓    時(shí)間: 2025-3-21 16:03
書目名稱Regulation of Signal Transduction in Human Cell Research影響因子(影響力)




書目名稱Regulation of Signal Transduction in Human Cell Research影響因子(影響力)學(xué)科排名




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書目名稱Regulation of Signal Transduction in Human Cell Research網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Regulation of Signal Transduction in Human Cell Research被引頻次




書目名稱Regulation of Signal Transduction in Human Cell Research被引頻次學(xué)科排名




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書目名稱Regulation of Signal Transduction in Human Cell Research年度引用學(xué)科排名




書目名稱Regulation of Signal Transduction in Human Cell Research讀者反饋




書目名稱Regulation of Signal Transduction in Human Cell Research讀者反饋學(xué)科排名





作者: COW    時(shí)間: 2025-3-21 21:44
Zhendong A. Zhong,Nicole J. Ethen,Bart O. Williamscorrelate it with chronological age. Specific techniques employed in this process vary with the sample available for analysis as well as the general age of the individual (.,.). Some techniques are specific to particular bones or even parts of bones. Techniques that would be ideal to use to estimate
作者: overture    時(shí)間: 2025-3-22 01:50
Toshiaki Ishizukalysis, and ‘a(chǎn)rchaeological’ digging.Explores how ‘forensic’ This book describes and analyses a particular literary mode that challenges the aesthetics of testimony by approaching the past through detection, analysis, and ‘a(chǎn)rchaeological’ digging. How does forensic literature narrate the past in term
作者: 山頂可休息    時(shí)間: 2025-3-22 05:01

作者: Stagger    時(shí)間: 2025-3-22 09:49

作者: Feedback    時(shí)間: 2025-3-22 14:59

作者: Individual    時(shí)間: 2025-3-22 20:45
Brian M. Cartwright,Phillip R. Musich,Yue Zous a series of informative case studiesThis book offers up-to-date information and guidance on the application of metrology in legal proceedings, clarifying the limits of validity of scientific evidence and presenting an illuminating series of case studies in which measurement uncertainty has played
作者: Compassionate    時(shí)間: 2025-3-22 22:00
Nariyoshi Shinomiya,Qian Xie,George F. Vande Woudes a series of informative case studiesThis book offers up-to-date information and guidance on the application of metrology in legal proceedings, clarifying the limits of validity of scientific evidence and presenting an illuminating series of case studies in which measurement uncertainty has played
作者: chronicle    時(shí)間: 2025-3-23 04:58

作者: 發(fā)牢騷    時(shí)間: 2025-3-23 09:07

作者: Fester    時(shí)間: 2025-3-23 12:35
Atsushi Yoshimori,Sei-Ichi Tanumas a series of informative case studiesThis book offers up-to-date information and guidance on the application of metrology in legal proceedings, clarifying the limits of validity of scientific evidence and presenting an illuminating series of case studies in which measurement uncertainty has played
作者: 闖入    時(shí)間: 2025-3-23 16:54
Using Genetically Engineered Mouse Models to Study Wnt Signaling in Bone Development and Disease, pathway regulates skeletal development and postnatal homeostasis is of great value for human skeletal health. We will review how genetically engineered mouse models (GEMMs) have been and are being used to uncover the mechanisms and etiology of bone diseases in the context of Wnt signaling.
作者: FADE    時(shí)間: 2025-3-23 18:33

作者: 放大    時(shí)間: 2025-3-23 22:36

作者: 初次登臺(tái)    時(shí)間: 2025-3-24 04:59

作者: Gerontology    時(shí)間: 2025-3-24 09:15
The HGF/MET Signaling and Therapeutics in Cancer,valuated in clinical trials for the treatment of various cancers. In this chapter, we discuss the role of HGF/MET signaling in cancer development and progression, the strategies for targeting MET signaling, as well as the promises and challenges of MET-targeted therapeutics.
作者: LAVE    時(shí)間: 2025-3-24 11:47
Using Genetically Engineered Mouse Models to Study Wnt Signaling in Bone Development and Disease,between bone formation (by osteoblasts and osteocytes) and bone resorption (by osteoclasts) controls postnatal bone homeostasis. For the past decade, a vast amount of evidence has shown that Wnt signaling plays a pivotal role in regulating this balance. Therefore, understanding how the Wnt signaling
作者: inscribe    時(shí)間: 2025-3-24 16:14

作者: 鬧劇    時(shí)間: 2025-3-24 20:21

作者: 變態(tài)    時(shí)間: 2025-3-24 23:30
,Regulation of EMT by TGF-β Signaling in Cancer Cells,TGF-β can suppress tumorigenesis by inhibiting cell-cycle progression and stimulating apoptosis in the early stages of cancer, suggesting that it acts as a tumor suppressor during cancer initiation. However, TGF-β can also act as a tumor promoter at later stages of cancer progression. TGF-β plays fu
作者: Notorious    時(shí)間: 2025-3-25 07:06
,The Functional Interplay Between Pro-oncogenic RUNX2 and Hypoxia-Inducible Factor-1α (HIF-1α) Durinnerally O. concentration is less than 2%). The elevated level of hypoxia is tightly associated with genetic instability, tumor progression, drug resistance, and/or poor clinical outcome after treatment, indicating that hypoxia exerts a strong selection pressure for the survival of cancer stem cells
作者: STING    時(shí)間: 2025-3-25 10:56

作者: 改變立場(chǎng)    時(shí)間: 2025-3-25 13:02
Met Activation and Carcinogenesis,cer cells, MET is activated upon stimulation by its only ligand, hepatocyte growth factor/scatter factor (HGF/SF), or becomes active due to mutations or amplifications that produce constitutive activation of the MET kinase. The biological consequences of HGF/SF-MET signaling include cell proliferati
作者: landmark    時(shí)間: 2025-3-25 18:42
The HGF/MET Signaling and Therapeutics in Cancer,tivates MET to regulate diversified cellular and molecular activities such as proliferation, motility, differentiation, and survival. Aberration of HGF/MET signaling plays a proven role in promoting cancer initiation and malignant progression, providing a strong rationale for targeting the MET signa
作者: 減去    時(shí)間: 2025-3-25 20:55

作者: NEG    時(shí)間: 2025-3-26 04:04
Molecular Designing of Small-Molecule Inhibitors for Apoptosis Regulation,processes has been implicated in a wide variety of diseases, such as cancer, neurodegenerative disorders, and ischemic injury. To date, many kinds of key proteins in apoptotic processes have been identified and targeted for therapeutic strategies. Several effective small molecules have been designed
作者: 軍火    時(shí)間: 2025-3-26 05:22

作者: Asperity    時(shí)間: 2025-3-26 08:53

作者: CHURL    時(shí)間: 2025-3-26 13:37

作者: countenance    時(shí)間: 2025-3-26 18:36
https://doi.org/10.1007/978-981-10-7296-3Signal transduction; Carcinogenesis; Cell death; Molecular design; Human cell
作者: 狗窩    時(shí)間: 2025-3-26 22:28

作者: 沒血色    時(shí)間: 2025-3-27 04:46
Regulation of Signal Transduction in Human Cell Research
作者: 變形詞    時(shí)間: 2025-3-27 08:52
,Role of the?Receptor-Mediated Signaling Pathways on the?Proliferation and Differentiation of Pluripdent MEK/ERK and PI3K/Akt signaling pathways independent of FGF2..Activation of the Smad signaling pathway by bone morphogenetic proteins (BMPs) and activin/nodal has been shown to promote cardiovascular differentiation of mouse and human ES or iPS cells. In addition, treatment with isoproterenol (a
作者: GROUP    時(shí)間: 2025-3-27 11:07
,Regulation of microRNA Expression by?Growth Factors in Tumor Development and Progression,ng the translation of growth factor-induced functional molecules. There are a growing number of reports that describe the translational regulation of growth factors, their receptors, and intracellular signaling molecules by miRNAs in various tumors. However, less of the reports describe the regulati
作者: 錯(cuò)事    時(shí)間: 2025-3-27 16:10
,Regulation of EMT by TGF-β Signaling in Cancer Cells, are implicated in this process. Among them, TGF-β induces the EMT by propagating intracellular signals and activating transcription factors. This review describes new insights into the molecular mechanisms underlying induction of the EMT by TGF-β in cooperation with signals from growth factors and
作者: BLAND    時(shí)間: 2025-3-27 18:01

作者: 蘑菇    時(shí)間: 2025-3-27 22:00
DNA Damage: Cellular Responses, Repair, and Cancer Treatment,owever, have implicated that DNA-PK can regulate DDS through hindrance of ATM-DDS, giving rise to an evolving view in which all three PIKK family members are essential for regulation of DDS, but not its initiation. This chapter presents a discussion of the signaling within human systems induced by D
作者: 高度    時(shí)間: 2025-3-28 02:57
Met Activation and Carcinogenesis,tumors mediated by HGF/SF-MET signaling. Such tumors include carcinomas, sarcomas, and lymphomas, demonstrating the breadth of MET signaling as driving force of cancer. In this chapter, we will discuss the role of HGF/SF-MET signaling in carcinogenesis and the animal models used in developing therap
作者: 代理人    時(shí)間: 2025-3-28 08:27
Pericellular Activation of Peptide Growth Factors by Serine Proteases,receptor tyrosine kinase. There are two mechanisms for HGF activation .: serum activation and cellular activation. Type II transmembrane serine proteases are membrane-anchored proteases that are part of cellular HGF-activating machinery. In the past decade, evidence for the roles of these proteases
作者: Adrenal-Glands    時(shí)間: 2025-3-28 11:27

作者: Ptsd429    時(shí)間: 2025-3-28 16:10

作者: 難解    時(shí)間: 2025-3-28 18:48
Brian M. Cartwright,Phillip R. Musich,Yue Zouisition of accurate measurements. Against this background, Forensic Metrology will be of value for lawyers and judges in both civil and common law countries, as well as engineers and other scientists with an interest in the subject..978-3-031-14621-3978-3-031-14619-0Series ISSN 2198-7300 Series E-ISSN 2198-7319
作者: 沐浴    時(shí)間: 2025-3-29 00:59

作者: 一再遛    時(shí)間: 2025-3-29 03:05
Douglas P. Thewke,Jianqun Kou,Makenzie L. Fulmer,Qian Xieisition of accurate measurements. Against this background, Forensic Metrology will be of value for lawyers and judges in both civil and common law countries, as well as engineers and other scientists with an interest in the subject..978-3-031-14621-3978-3-031-14619-0Series ISSN 2198-7300 Series E-ISSN 2198-7319




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