標題: Titlebook: Oxidative Stress and Vascular Disease; John F. Keaney (Associate Professor of Medicine an Book 2000 Springer Science+Business Media New Yo [打印本頁] 作者: Callow 時間: 2025-3-21 16:55
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書目名稱Oxidative Stress and Vascular Disease讀者反饋學科排名
作者: Epithelium 時間: 2025-3-21 20:52 作者: 破裂 時間: 2025-3-22 04:10 作者: abolish 時間: 2025-3-22 06:56 作者: 過多 時間: 2025-3-22 09:48
Developments in Cardiovascular Medicinehttp://image.papertrans.cn/o/image/705319.jpg作者: debacle 時間: 2025-3-22 13:22
https://doi.org/10.1007/978-1-4615-4649-8atherosclerosis; cardiovascular; gene; gene expression; hypertension; oxidative stress; physiology; signal 作者: Deject 時間: 2025-3-22 20:02 作者: 為敵 時間: 2025-3-22 23:55 作者: 多產(chǎn)魚 時間: 2025-3-23 05:17 作者: modest 時間: 2025-3-23 09:21
Jay W. Heinecketic, prolymphoblastic and immunoblastic acute lymphoid leukaemias. Using computer analysis, we demonstrated that, among all the prognostic factors, i.e. type, age, tumour volume, leukocytosis..., the cytological type remains the only one which determines the other factors. The best therapeutic respo作者: scoliosis 時間: 2025-3-23 10:28
Roland Stockerthe immunoblastic one. Prognosis is best for the prolymphocytic and worst for the immunoblastic type. These data emphasize the diagnostic importance of cell typing in LLS as determined by cytological examination of smears in conjunction with the other above mentioned investigations.作者: 過濾 時間: 2025-3-23 17:40 作者: cloture 時間: 2025-3-23 21:54 作者: Diuretic 時間: 2025-3-24 00:08 作者: BLANK 時間: 2025-3-24 04:41
John F. Keaney Jr.ith the ciliate is unclear. The vestigial condition of . cytostome suggests that new algae cannot possibly be ingested in each ciliate generation, although they might be ingested at less frequent intervals during some unknown stage of . life cycle (Taylor et al., 1969, 1971).作者: Ethics 時間: 2025-3-24 10:31 作者: Exposition 時間: 2025-3-24 12:44
Oxidized Phospholipids as Mediators of Vascular Diseaseamage to the vessel wall initiated the disease. A refined version of this model recognizes that the early steps in atherogenesis need not necessarily be initiated by the cytotoxicity of oxidized LDL, but rather by activation of an inflammatory response in vascular tissue. Oxidation inappropriately s作者: ascetic 時間: 2025-3-24 15:25 作者: 老人病學 時間: 2025-3-24 20:45 作者: 高歌 時間: 2025-3-24 23:45
Book 2000est responses to hypercholesterolemia is the adhesion of monocytes to aortic endothelial cells followed by their penetration into the subendothelial space, where they differentiate into macrophages. These cells, and also medial smooth muscle cells that have migrated into the subendothelial space, th作者: 落葉劑 時間: 2025-3-25 06:02
Sean Davies,Thomas McIntyre,Stephen Prescott,Guy Zimmermanactors responsible for the human "disease" produced by uric acid in the joints and kidneys. The final four chapters provide a summary of therapeutic approaches 978-3-642-66869-2978-3-642-66867-8Series ISSN 0171-2004 Series E-ISSN 1865-0325 作者: 過時 時間: 2025-3-25 08:01
0166-9842 othelial space, where they differentiate into macrophages. These cells, and also medial smooth muscle cells that have migrated into the subendothelial space, th978-1-4613-7103-8978-1-4615-4649-8Series ISSN 0166-9842 作者: Fraudulent 時間: 2025-3-25 15:17
John F. Keaney (Associate Professor of Medicine an作者: Solace 時間: 2025-3-25 18:28 作者: aesthetician 時間: 2025-3-25 21:28
Sources of Vascular Oxidative Stresss an elevated concentration of low density lipoprotein (LDL), the major carrier of blood cholesterol (1). It is thus paradoxical that LDL often fails to exert atherogenic effects .. These observations led to the suggestion that LDL has to be modified to promote vascular disease (2,3). Subsequent stu作者: Inflated 時間: 2025-3-26 01:45
Antioxidant Defenses in the Vascular Walling on its extent and duration, and the type of oxidant(s) involved. Within the vascular wall both radicals (., one electron) and nucleophilic (two electron) oxidants are likely produced and contribute to oxidative stress (see Chapter 2). This chapter describes antioxidant defenses in the vascular w作者: 利用 時間: 2025-3-26 05:14 作者: GIST 時間: 2025-3-26 08:27 作者: 臭名昭著 時間: 2025-3-26 13:35 作者: nautical 時間: 2025-3-26 19:33 作者: 琺瑯 時間: 2025-3-26 23:26
Oxidation-Sensitive Transcription and Gene Expression in Atherosclerosis pulmonary emphysema, neurodegenerative disorders and atherosclerosis has focused on the putative role of ROS as cellular damaging agents through the potential toxic oxidative modification of macromolecules such as proteins, lipids and DNA and on the cytotoxicity of free radicals and their reaction 作者: BORE 時間: 2025-3-27 03:46
Atherosclerosis, Oxidative Stress, and Endothelial Functiontes alone, 5.8 million patients are discharged from the hospital annually with a diagnosis of cardiovascular disease (CVD) and in 1995 CVD claimed over 960,000 lives (1). This represents approximately 41% of all deaths in the United States and places cardiovascular disease as the number one cause of作者: 有毒 時間: 2025-3-27 08:05
Oxidants and Antioxidants in Platelet Functionrecise mechanisms that underlie these observations, however, are not clear. While the beneficial effects of antioxidants have been attributed to the prevention of oxidative modification of low-density lipoprotein (LDL) and the inhibition of atherogenesis, other factors may also be important. Platele作者: Adherent 時間: 2025-3-27 13:25
Antioxidants and Atherosclerosis: Animal Studiesd world, claiming over 960,000 lives annually in the United States alone (1). A wealth of data now links serum cholesterol, in particular low-density lipoprotein (LDL) cholesterol (2) to the development of atherosclerosis. Despite this causative association between LDL cholesterol and atherosclerosi作者: Polydipsia 時間: 2025-3-27 14:32
Human Studies of Antioxidants and Vascular Functiont suggest an association between increased intake of certain antioxidant vitamins and reduced risk for CVD. It is important to realize, however, that the putative mechanisms of any benefit remain uncertain. Investigations in animal models indicate that increased oxidative stress contributes to the a作者: glomeruli 時間: 2025-3-27 21:47 作者: Obstreperous 時間: 2025-3-27 23:38
Glycation and Glycoxidation in Diabetic Vascular Diseasee body (type I, insulin dependent diabetes mellitus (IDDM)) and when there is an excess of circulating insulin (type II, non-insulin dependent diabetes mellitus (NIDDM)). Type I diabetes is generally associated with a young or juvenile onset, while Type II diabetes typically occurs with older age an作者: 閑蕩 時間: 2025-3-28 02:19
Advanced Glycosylation Endproducts and Diabetic Vascular Diseaseretinopathy, nephropathy, neuropathy, and atherosclerotic vascular disease (1). Since diabetes mellitus afflicts at least 12 million individuals in the United States alone, the contribution of these complications to the overall morbidity and mortality of this disease is considerable (1,2). An import作者: 出汗 時間: 2025-3-28 09:14
Hyperglycemia and Diabetes — Induced Vascular Dysfunction: Role of Oxidative Stress lipid peroxides (., malondialdehyde) in plasma of experimental diabetic rodents (1–3) and in plasma of human type I and type II diabetes mellitus (2,4-7). More recently, F.-isoprostanes, nonenzymatic peroxidation products, were shown to be increased in plasma of diabetic patients (8). In addition t作者: ANT 時間: 2025-3-28 12:25 作者: osteopath 時間: 2025-3-28 17:49
dase and Periodic Acid Schiff (PAS) aid in differentiating acute lymphocytic leukemia (ALL) and nonlymphocytic leukemia. The following study examines the value of these established stains and a novel esterase reaction, Naphthol-AS-D-Acetate (NASDA) along with sodium fluoride inhibition (NASDA-F), in作者: peritonitis 時間: 2025-3-28 20:12
Helmut Siesathologie alterations of that cell and its progeny. All present research and discussions are centered around the leukemic cell. So is this symposium, which would not take place except for our primary interest in the leukemic cell. This does not preclude, of course, consideration of other definitions作者: Ophthalmologist 時間: 2025-3-29 01:24
Jay W. Heineckeh exists between the different varieties and types of AL and their therapeutic responsiveness and prognosis. AL is classically divided into three main types: acute lymphoid leukaemia (ALL), acute myeloid leukaemia (AML) and acute monocytoid leukaemia (AMoL) which are characterized by great differenc作者: 捐助 時間: 2025-3-29 06:58 作者: myriad 時間: 2025-3-29 07:59 作者: BROOK 時間: 2025-3-29 13:01 作者: 掃興 時間: 2025-3-29 18:53
Norbert Leitinger,Judith A. Berlinerace layer of the ocean which arises as a consequence of a particular wind field, the presence of a coastline, or other special conditions. Since deeper oceanic layers are usually enriched wi th nutrients, it is the permanent supply of nutrients which forms the basis for the high producti vi ty of up作者: Inscrutable 時間: 2025-3-29 19:50
Charles Kunsch,Russell M. Medfordace layer of the ocean which arises as a consequence of a particular wind field, the presence of a coastline, or other special conditions. Since deeper oceanic layers are usually enriched wi th nutrients, it is the permanent supply of nutrients which forms the basis for the high producti vi ty of up作者: 光滑 時間: 2025-3-30 02:50
John F. Keaney Jr.an, unlike most other ciliates, . and some members of the genera . and . (Blackbourn et al., 1973) contain chloroplasts. . and . contain chloroplasts, only, and occasionally have been found without them. ., however, also contains mitochondria in addition to the chloroplasts; furthermore, the organis作者: 男生戴手銬 時間: 2025-3-30 06:08 作者: 王得到 時間: 2025-3-30 10:38 作者: 高原 時間: 2025-3-30 15:50 作者: rheumatology 時間: 2025-3-30 20:23 作者: Eosinophils 時間: 2025-3-30 22:39
Human Studies of Antioxidants and Vascular Functiontherogenic process and to the development of vascular dysfunction. In such models, antioxidant treatment has often been shown to limit the extent of atherosclerosis and maintain normal vascular function. If applicable to humans, these effects could explain the clinical benefits of antioxidants.作者: Magnificent 時間: 2025-3-31 03:40 作者: 無所不知 時間: 2025-3-31 06:24
Book 2000in atherosclerosis; that hypercholesterolemia can be effectively treated; and that treatment significantly reduces not only coronary disease mortality but also all- cause mortality. Treatment to lower plasma levels of cholesterol - primarily low density lipoprotein (LDL) cholesterol - is now accepte
