標題: Titlebook: New Aspects in the Treatment of Failing Heart; Hisakazu Yasuda (Emeritus Professor),Hideaki Kawag Conference proceedings 1992 Springer-Ver [打印本頁] 作者: Pierce 時間: 2025-3-21 18:42
書目名稱New Aspects in the Treatment of Failing Heart影響因子(影響力)
書目名稱New Aspects in the Treatment of Failing Heart影響因子(影響力)學科排名
書目名稱New Aspects in the Treatment of Failing Heart網(wǎng)絡(luò)公開度
書目名稱New Aspects in the Treatment of Failing Heart網(wǎng)絡(luò)公開度學科排名
書目名稱New Aspects in the Treatment of Failing Heart被引頻次
書目名稱New Aspects in the Treatment of Failing Heart被引頻次學科排名
書目名稱New Aspects in the Treatment of Failing Heart年度引用
書目名稱New Aspects in the Treatment of Failing Heart年度引用學科排名
書目名稱New Aspects in the Treatment of Failing Heart讀者反饋
書目名稱New Aspects in the Treatment of Failing Heart讀者反饋學科排名
作者: 驚奇 時間: 2025-3-21 20:15
nts. The fifty-one selected contributions in this volume based on the 14th International Society of Heart Research (ISHR) Meeting in 1992 document the discoveries being made at the frontiers of research with cardiac molecular and genetic biology and technology. The contents may be divided into five 作者: Multiple 時間: 2025-3-22 00:34 作者: 口音在加重 時間: 2025-3-22 06:36 作者: aneurysm 時間: 2025-3-22 12:47 作者: 蛙鳴聲 時間: 2025-3-22 13:54
Conference proceedings 1992ocardial cells, 4) electrophysiological abnormalities of the failing heart, and 5) recent concepts in drug therapy. The book builds a bridge for the flow of up-to-date information from active veteran researchers to young talented investigators who offer great promise for the future.作者: ARBOR 時間: 2025-3-22 20:52
iseased myocardial cells, 4) electrophysiological abnormalities of the failing heart, and 5) recent concepts in drug therapy. The book builds a bridge for the flow of up-to-date information from active veteran researchers to young talented investigators who offer great promise for the future.978-4-431-68221-9978-4-431-68219-6作者: seruting 時間: 2025-3-22 22:52 作者: 我不明白 時間: 2025-3-23 02:03 作者: 閑逛 時間: 2025-3-23 06:39 作者: arcane 時間: 2025-3-23 11:22
978-4-431-68221-9Springer-Verlag Tokyo 1992作者: 鄙視讀作 時間: 2025-3-23 15:45
http://image.papertrans.cn/n/image/664842.jpg作者: 出來 時間: 2025-3-23 18:56
https://doi.org/10.1007/978-4-431-68219-6Angiotensin II; angina pectoris; arrhythmia; cardiovascular; cardiovascular system; drugs; heart; heart dis作者: 切割 時間: 2025-3-24 01:41
Hisakazu Yasuda (Emeritus Professor),Hideaki Kawag作者: 災(zāi)禍 時間: 2025-3-24 05:13 作者: Irritate 時間: 2025-3-24 10:19
erage run length concept that can be used to quantify what a particular process-monitoring scheme can be expected to provide in Sect.?3.5. Finally, in Sect.?3.6?the chapter closes with a discussion clarifying the relationship between “statistical process control”?and “engineering control”?and presen作者: VOC 時間: 2025-3-24 11:13 作者: atopic-rhinitis 時間: 2025-3-24 18:23 作者: nonsensical 時間: 2025-3-24 22:02 作者: 蒙太奇 時間: 2025-3-25 01:22
Kazuo Ichiharafrom such scales, even though the scales themselves are quite reliable, precise, and sensitive to change. Without meaningful approaches to defining clinical significance, such scales are unlikely to become acceptable in clinical trials except, perhaps, as exploratory endpoints. This paper discusses 作者: 利用 時間: 2025-3-25 05:38 作者: 舉止粗野的人 時間: 2025-3-25 10:42 作者: 苦笑 時間: 2025-3-25 12:54 作者: 地名詞典 時間: 2025-3-25 17:06
Nicholas Sperelakis,Noritsugu Tohse,Hiroshi Masuda,János Mészárosem which is much more robust to speech recognition errors and for which a dialogue strategy can be learned automatically using reinforcement learning. The thesis describes both the architecture, the algorithms needed for fast real-time inference over very large networks, model parameter estimation a作者: bibliophile 時間: 2025-3-25 21:27 作者: 五行打油詩 時間: 2025-3-26 03:41
Role of Protein Kinase System in the Signal Transduction of Stretch-mediated Myocyte Growthe observed the activation of MAP kinase by myocytes stretching. This result suggest that MAP kinase activation might increase in efficiency of protein synthesis on ribosomes induced by mechanical stimuli.作者: 手工藝品 時間: 2025-3-26 07:53
Mitochondrial DNA Mutations in CardiomyopathyA. We review: (1) mtDNA mutations in cardiomyocytes of patients with dilated or hypertrophic cardiomyopathy, (2) a patient with hypertrophic cardiomyopathy associated with left ventricular dilatation, (3) a patient with mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episode作者: 斑駁 時間: 2025-3-26 10:35 作者: 津貼 時間: 2025-3-26 13:27
GTP-binding Proteins and Bacterial Toxin-catalyzed ADP-ribosylationine which is four amino acid residues away from the carboxyl terminus of the α-subunits. Since the carboxyl terminus region is responsible for the interaction with receptors, the ADP-ribosylation results in an uncoupling of the G proteins from receptors[4–5]; only receptor-mediated signalings are se作者: 領(lǐng)導權(quán) 時間: 2025-3-26 19:57 作者: 對待 時間: 2025-3-27 01:02
Alterations in α-Adrenergic Receptors in Response to Myocardial Ischemia and their Role in Arrhythmo nonreentrant mechanisms contribute to the development of arrhythmias during early myocardial ischemia [5,6]. Thus, approaches to evaluate the electrophysiologic mechanisms underlying the arrhythmogenic effects of catecholamines should not only evaluate potential influences on electrophysiologic ind作者: Occupation 時間: 2025-3-27 02:53
Enzyme-Specific Desensitization of Adenylyl Cyclase in Hypertrophied Heart in Monocrotaline-Treated n experimental model of right ventricular failure. Although the phenomenon of desensitization, homologous or heterologous, has been recognized at the in vitro cultured cells, few studies have examined this phenomenon in the in vivo model of heart failure.作者: START 時間: 2025-3-27 09:22 作者: abstemious 時間: 2025-3-27 11:17 作者: Antigen 時間: 2025-3-27 15:30 作者: 糾纏,纏繞 時間: 2025-3-27 21:07 作者: 制定法律 時間: 2025-3-27 22:11
Toshiaki Katada,Yoshiharu Ohoka,Katsunobu Takahashi,Taroh Iiri Regression model and local indicators of spatial association; all of these tools provide the policy-maker with a valuable support to policy development..978-88-470-5607-7978-88-470-2751-0Series ISSN 1431-1968 Series E-ISSN 2628-8966 作者: TIGER 時間: 2025-3-28 04:16 作者: 多山 時間: 2025-3-28 09:38 作者: Rankle 時間: 2025-3-28 13:05
Katsuyuki Tobise,Hiromitsu Yoshie,Kazumi Uekita,Masaaki Fujita,Sokichi Onodera Regression model and local indicators of spatial association; all of these tools provide the policy-maker with a valuable support to policy development..978-88-470-5607-7978-88-470-2751-0Series ISSN 1431-1968 Series E-ISSN 2628-8966 作者: 刀鋒 時間: 2025-3-28 17:53 作者: SPER 時間: 2025-3-28 21:28
Christophe Leclercq,Jean-Marie Moalic,Daniéle Charlemagne,Pascale Mansier,Claude Sainte-Beuve,Franco作者: addition 時間: 2025-3-29 02:40
Hideaki Kawaguchi,Mikako Shoki,Hitoshi Sano,Toshiyuki Kudo,Hirofumi Sawa,Hiroshi Okamoto,Naoki Mochi作者: 從屬 時間: 2025-3-29 04:44
Postischemic Myocardial Dysfunction (Myocardial “Stunning”): Role of Oxidative StressIn addition, coronary reperfusion by means of thrombolytic therapy, percutaneous transluminal angioplasty or bypass surgery is rapidly emerging as the fundamental strategy in the management of acute ischemic syndromes. The recent appreciation of the role of reperfusion in the natural history of coro作者: 未完成 時間: 2025-3-29 07:26
Degradation of Membrane Phospholipids in the Ischemic and Reperfused Heartlactate to fulfil their energy requirements. Contraction and relaxation of the myofibrils consume over 70 of ATP produced by mitochondrial activity, the remaining part is used for transport of ions across membranes, and maintenance of cellular integrity. The plasmalemma, the membrane enclosing the c作者: 親屬 時間: 2025-3-29 12:59 作者: 音樂會 時間: 2025-3-29 15:35 作者: 廢除 時間: 2025-3-29 23:31
Role of Protein Kinase System in the Signal Transduction of Stretch-mediated Myocyte Growthshes and imposed an in vitro mechanical load by stretching the adherent cells. Myocyte stretching increased total cell RNA content and mRNA levels of c-fos and skeletal α-actin followed by activation of protein synthesis. CAT assay indicated that sequences containing a serum response element were re作者: 翅膀拍動 時間: 2025-3-30 02:37 作者: VERT 時間: 2025-3-30 06:51
PI Response and Calcium Overload in Cardiomyopathic Hamster Heart Cellosphatidylinositol (PI) turnover pathway generates two second messengers, inositol-(l,4,5)-trisphosphate (IP.) and sn-1,2-diacylglycerol (DAG) (5,6). DAG stimulates membrane-bound phospholipid-dependent, Ca.- dependent protein kinase C (7), while IP. releases Ca. from stores in the sarcoplasmic reti作者: languid 時間: 2025-3-30 08:16
Mitochondrial DNA Mutations in Cardiomyopathy, 22 tRNAs required for translation of their mRNA, and two rRNA [1]. The mutation rate in mitochondrial DNA (mtDNA) is more than 10 times higher than that of nuclear DNA [2]. This is due to absence of introns, lack of DNA-binding proteins such as histones, simple DNA repair mechanism, and constant e作者: 人類學家 時間: 2025-3-30 15:53 作者: 反感 時間: 2025-3-30 20:29 作者: 主講人 時間: 2025-3-30 23:09
Regulation of the Gs Protein in the Heartones and neurotransmitters that regulate the heart and vascular system have been shown to produce their effects by the activation of such proteins (1). Acetylcholine released by post?ganglionic parasympathetic (vagal) neurons and norepinephrine released at postganglionic sympathetic neurons are the 作者: 違法事實 時間: 2025-3-31 03:19
Alterations in α-Adrenergic Receptors in Response to Myocardial Ischemia and their Role in Arrhythmohythmias during early myocardial ischemia [1–3]. There are at least two major mechanisms which underly the release of catecholamines during ischemia, the most prominent of which is local release from sympathetic nerve endings directly in the absence of any sympathetic neural activation [4]. In addit作者: Hamper 時間: 2025-3-31 06:23
Enzyme-Specific Desensitization of Adenylyl Cyclase in Hypertrophied Heart in Monocrotaline-Treated results in pulmonary arterial hypertension, leading to a severe right ventricular hypertrophy and eventually to failure [1]. As reported in patients with heart failure caused by primary pulmonary hypertension, a chamber-specific decrease in the myocardial β-adrenoceptors was shown mostly in the fai作者: 兒童 時間: 2025-3-31 12:46
A Mechanism of Catecholamine Tolerance In Congestive Heart Failureiac dysfunction observed in CHF. However, CHF patients (or animals) share several similar features: reduced cardiac contractility, increased sympathetic nerve activity, and activation of several hormonal systems (e.g., renin-angiotensin system, vasopressin, and atrial natriuretic factor) to compensa
