書目名稱Maturation Phenomenon in Cerebral Ischemia II影響因子(影響力)學(xué)科排名
書目名稱Maturation Phenomenon in Cerebral Ischemia II網(wǎng)絡(luò)公開(kāi)度
書目名稱Maturation Phenomenon in Cerebral Ischemia II網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書目名稱Maturation Phenomenon in Cerebral Ischemia II被引頻次
書目名稱Maturation Phenomenon in Cerebral Ischemia II被引頻次學(xué)科排名
書目名稱Maturation Phenomenon in Cerebral Ischemia II年度引用
書目名稱Maturation Phenomenon in Cerebral Ischemia II年度引用學(xué)科排名
書目名稱Maturation Phenomenon in Cerebral Ischemia II讀者反饋
書目名稱Maturation Phenomenon in Cerebral Ischemia II讀者反饋學(xué)科排名
作者: 著名 時(shí)間: 2025-3-21 23:06
http://image.papertrans.cn/m/image/627817.jpg作者: 6Applepolish 時(shí)間: 2025-3-22 03:40
978-3-540-61673-3Springer-Verlag Berlin Heidelberg 1997作者: 溫和女人 時(shí)間: 2025-3-22 07:48
th analysis and discourse.Consistent style and level of coveNutrients, Dietary Supplements, and Nutriceuticals: Cost Analysis Versus Clinical Benefitsprovides the most current, concise, scientific appraisal and economic analysis (costs vs. benefit) of nutritional supplements and bioactive components作者: HALO 時(shí)間: 2025-3-22 10:50
th analysis and discourse.Consistent style and level of coveNutrients, Dietary Supplements, and Nutriceuticals: Cost Analysis Versus Clinical Benefitsprovides the most current, concise, scientific appraisal and economic analysis (costs vs. benefit) of nutritional supplements and bioactive components作者: Intuitive 時(shí)間: 2025-3-22 16:11 作者: 切掉 時(shí)間: 2025-3-22 20:36 作者: jarring 時(shí)間: 2025-3-23 01:04
N. G. Bazanth analysis and discourse.Consistent style and level of coveNutrients, Dietary Supplements, and Nutriceuticals: Cost Analysis Versus Clinical Benefitsprovides the most current, concise, scientific appraisal and economic analysis (costs vs. benefit) of nutritional supplements and bioactive components作者: 損壞 時(shí)間: 2025-3-23 02:03
K. Yamada,A. Masago,A. Iwata,E. Kohmura,T. Yuguchi,T. Sakaki,T. Hayakawath analysis and discourse.Consistent style and level of coveNutrients, Dietary Supplements, and Nutriceuticals: Cost Analysis Versus Clinical Benefitsprovides the most current, concise, scientific appraisal and economic analysis (costs vs. benefit) of nutritional supplements and bioactive components作者: exostosis 時(shí)間: 2025-3-23 06:13
J. Ikeda,S. Terakawa,K. Asai,I. Morita,S. Murota,K. Hirakawath analysis and discourse.Consistent style and level of coveNutrients, Dietary Supplements, and Nutriceuticals: Cost Analysis Versus Clinical Benefitsprovides the most current, concise, scientific appraisal and economic analysis (costs vs. benefit) of nutritional supplements and bioactive components作者: Dna262 時(shí)間: 2025-3-23 09:52 作者: 迅速成長(zhǎng) 時(shí)間: 2025-3-23 17:05 作者: 橫截,橫斷 時(shí)間: 2025-3-23 19:17 作者: transdermal 時(shí)間: 2025-3-23 23:38
J. Krieglstein,I. Semkova,C. Culmsee,P. Wolzlye posited that the body shows a common, integrated set of responses in an attempt to adapt to many different kinds of stress. Until his work, many scientists viewed different kinds of stress as having different effects on the body; for example, the effect of exposure to severe cold was thought to 作者: Vo2-Max 時(shí)間: 2025-3-24 03:09 作者: Coronary-Spasm 時(shí)間: 2025-3-24 07:17 作者: 陰謀小團(tuán)體 時(shí)間: 2025-3-24 11:11
K. Kawai,T. Nakagomi,T. Kirino,A. Tamura,N. Kawaie parvocellular neurons of the hypothalamic paraventricular nucleus (PVN) is triggered. CRH activates, in the specific context of the stressor, the sympathetic nervous system and the hypothalamic-pituitary-adrenal (HPA) axis, which promote a series of physiological and behavioral adaptations in orde作者: sperse 時(shí)間: 2025-3-24 17:52
T. Ide,T. Morimoto,T. Kirinometabolism in organs such as liver, muscle and fat. Worldwide, the prevalence of T2D is rapidly increasing, which, when not properly treated, ultimately leads to reduced life expectancy due to microvascular (retinopathy, nephropathy and neuropathy) and macrovascular (heart disease and stroke (Chap. 作者: Yourself 時(shí)間: 2025-3-24 20:40
N. Kawahara,C. A. Ruetzler,G. Mies,S. D. Groll,S. J. Wiegand,I. Klatzo very dynamic and modulated by environmental stimuli, such as dietary molecules. It is expected that the missing classical genetic heritability of the susceptibility for complex diseases and traits might be eventually explainable via epigenetics. For example, persons that are born with low birth wei作者: 傳授知識(shí) 時(shí)間: 2025-3-25 00:37 作者: 隼鷹 時(shí)間: 2025-3-25 05:41 作者: 健忘癥 時(shí)間: 2025-3-25 10:31 作者: 改良 時(shí)間: 2025-3-25 13:01
T. Kogure,Y. Yamasaki,Y. Matsuo,K. Kogureod pressure, ., hypertension, increases the risk of ischemic heart disease, stroke, peripheral vascular disease and other CVDs. .. The low intake of fruit and vegetables and whole grain fiber and high consumption of saturated fat and high-cholesterol diets can lead to hypercholesterolemia and athero作者: vector 時(shí)間: 2025-3-25 16:40
Molecular Approaches to Neuronal Functions and Neuronal Cell Deathis hoped that some of them may contribute to a molecular understanding of neuronal functions and neuronal cell death, and also to the development of new clinical methods for rescuing brain damage lesions..Some of our data on molecular and cellular biology of neuronal functions and neuronal cell deat作者: 欺騙世家 時(shí)間: 2025-3-25 23:58 作者: 中世紀(jì) 時(shí)間: 2025-3-26 02:56 作者: reflection 時(shí)間: 2025-3-26 07:25
Ischemic Neuronal Injury and Gene Expression of Facilitative and Inhibitory Growth Factorsitory factor (GIF) in ischemic brain tissue. The mRNA expression of these growth factors was studied using the in situ hybridization method and compared with the expression of c-., c-., and heat shock protein(HSP) 70 mRNA in the serial sections of the brain tissue during the recirculation period aft作者: B-cell 時(shí)間: 2025-3-26 11:06
Is Apoptosis Involved in Glutamate Neurotoxicity? the possible involvement of apoptosis in such death..In the first part of the study we employed Nomarski optics combined with a digital image processor to examine the acute type of glutamate neurotoxicity, and found a rapid change in the nucleus followed by cellular swelling. These changes could be作者: TRUST 時(shí)間: 2025-3-26 16:01
Glutamate Enhances Brain Damage from Ischemia and Traumaemia or other adverse conditions. Intensive research of the role of mediator compounds is clinically worthwile as more specific forms of treatment may emerge for the benefit of afflicted patients. In view of the great number of factors, cytokines, etc., which could play a role, it is mandatory that 作者: 鋼筆尖 時(shí)間: 2025-3-26 17:21 作者: 無(wú)關(guān)緊要 時(shí)間: 2025-3-26 22:12 作者: 高爾夫 時(shí)間: 2025-3-27 02:49
Stress Response and Ischemic Tolerance in the Brainhe cultured astrocytes exposed to hypoxia—reoxygenation (H—R). In a separate in vivo study, we examined the inducibility of ischemic tolerance and heat shock protein 72 (HSP72) in moderately symptomatic gerbils by unilateral preconditioning ischemia. In the former studies, we first attempted to clar作者: Minutes 時(shí)間: 2025-3-27 07:28
Preconditioning Depresses Excitatory Cell Signaling Following the Second Ischemic Insult3 days of interischemic interval protected the brain from a subsequent period of lethal ischemia of 10–15 min duration. Calcium-calmodulin protein kinase II (CaMKII) is translocated from its cytosolic form to the membrane-bound form during ischemia. In the CA1 region the translocated protein is boun作者: GNAW 時(shí)間: 2025-3-27 11:07
Electrophysiological Evaluation of Ischemic Tolerance Phenomenonice preparations. Firstly, the capacity for long-term potentiation triggered by tetanic stimulation, which is a putative synaptic background of memory, was examined in hippocampal slices from gerbils subjected to double ischemia. Capacity for long-term potentiation was transiently inhibited 2 days f作者: 廢除 時(shí)間: 2025-3-27 14:20
Transient Ischemia Depletes Free Ubiquitin in the Gerbil Hippocampal CA1 Neuronsippocampus was strongly dependent on the antibodies used. Using rabbit polyclonal antibody U-5379, immunoreactivity disappeared from the hippocampus in the early reperfusion period, and reappeared in the dentate granule cells and CA3 pyramidal cells, but never in the CA1 pyramidal cells. In contrast作者: 沉積物 時(shí)間: 2025-3-27 21:31
Insight into the Protective Mechanisms of Cortical Spreading Depression in Cardiac Arrest Cerebral Ige if elicited prior to ischemia. The present study was undertaken to investigate pathomechanisms of neuronal protection conferred by cortical spreading depression (CSD) elicited by the topical application of 5. KCl to the occipital cortex in Sprague-Dawley rats. Autoradiographic analysis on brain t作者: nostrum 時(shí)間: 2025-3-28 00:08 作者: 誰(shuí)在削木頭 時(shí)間: 2025-3-28 02:37
Features and Threshold of Infarct Development in Ischemic Maturation Phenomenonnecrosis in the cerebral cortex without inducing focal infarction. Furthermore, we investigated the threshold for induction of focal infarction in the cerebral cortex by changing the number and interval between repetitive ischemic insults. We found that with an increase in number of these insults an作者: irreparable 時(shí)間: 2025-3-28 07:02
Impending Cerebral Infarction and Inflammatory Response: A Possible Target for Therapeutic Interventee of ischemia, the changes become irreversible and the tissues undergo pan-necrosis. This activates a cascade of inflammatory response which scavenges the damaged cells in areas of impending infarction through liquification and phagocytosis. Induction of the immune response further accelerates disi作者: 雀斑 時(shí)間: 2025-3-28 13:30 作者: AROMA 時(shí)間: 2025-3-28 15:02 作者: tangle 時(shí)間: 2025-3-28 19:50 作者: 方舟 時(shí)間: 2025-3-29 02:53 作者: 眨眼 時(shí)間: 2025-3-29 05:58
Neuroprotection by Drug-Induced Neurotrophic Factorsl/glial hippocampal cultures to clenbuterol (1–100.) and selegiline (10p.-1.) significantly enhanced the content of NGF measured in the culture medium by two-site enzyme linked immunosorbent assay (ELISA). The excitotoxic injury was induced in the same type of cells after 14 days in vitro by exposur作者: 使迷醉 時(shí)間: 2025-3-29 08:49
Stress Response and Ischemic Tolerance in the Brainunder the stress of H—R. Another unique stress protein, oxygen-regulated protein 150 (ORP150), was purified and through several experimental steps we cloned the ORP150 gene successfully. These stress proteins may contribute to the adaptive response of astrocytes to ischemic stress, thereby ultimatel作者: 舊石器時(shí)代 時(shí)間: 2025-3-29 14:39
Preconditioning Depresses Excitatory Cell Signaling Following the Second Ischemic Insults are markedly diminished after 24h of recovery. We propose that during the preconditioning, protective factors (growth factors and cytokines) are induced in neurons and/or glia cells, which in turn stimulate intracellular neuroprotective mechanisms such as enhancers of proteins synthesis or bcl-2 e作者: Conspiracy 時(shí)間: 2025-3-29 15:57 作者: Melanoma 時(shí)間: 2025-3-29 22:42 作者: Distribution 時(shí)間: 2025-3-30 02:34 作者: 我們的面粉 時(shí)間: 2025-3-30 06:23 作者: Formidable 時(shí)間: 2025-3-30 08:24 作者: hermitage 時(shí)間: 2025-3-30 15:34 作者: Defiance 時(shí)間: 2025-3-30 17:40 作者: GREEN 時(shí)間: 2025-3-31 00:37 作者: GLUE 時(shí)間: 2025-3-31 02:18
F. R. Sharp,J. Honkaneimi,B. States,S. M. Massa physicians, general practitioners as well as those interested in diet and nutrition in disease resistance via immune regulation.978-1-4939-6184-9978-1-60761-308-4Series ISSN 2628-197X Series E-ISSN 2628-1961 作者: pacific 時(shí)間: 2025-3-31 08:06
N. G. Bazan physicians, general practitioners as well as those interested in diet and nutrition in disease resistance via immune regulation.978-1-4939-6184-9978-1-60761-308-4Series ISSN 2628-197X Series E-ISSN 2628-1961
