標(biāo)題: Titlebook: Lung Stem Cells in the Epithelium and Vasculature; Amy Firth,Jason X.-J. Yuan Book 2015 Springer International Publishing Switzerland 2015 [打印本頁] 作者: otitis-externa 時間: 2025-3-21 17:17
書目名稱Lung Stem Cells in the Epithelium and Vasculature影響因子(影響力)
書目名稱Lung Stem Cells in the Epithelium and Vasculature影響因子(影響力)學(xué)科排名
書目名稱Lung Stem Cells in the Epithelium and Vasculature網(wǎng)絡(luò)公開度
書目名稱Lung Stem Cells in the Epithelium and Vasculature網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Lung Stem Cells in the Epithelium and Vasculature被引頻次
書目名稱Lung Stem Cells in the Epithelium and Vasculature被引頻次學(xué)科排名
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書目名稱Lung Stem Cells in the Epithelium and Vasculature讀者反饋
書目名稱Lung Stem Cells in the Epithelium and Vasculature讀者反饋學(xué)科排名
作者: angina-pectoris 時間: 2025-3-21 20:59
Type II Cells as Progenitors in Alveolar Repairal roles in mediating the gas exchange. Cuboidal type II cells occupy only 5 % of the area but have multiple functions including secreting surfactant, transporting ion and fluid, and modulating immunity. Type II cells also function as progenitor cells by self-renewal and differentiating into type I 作者: PIZZA 時間: 2025-3-22 02:10 作者: 一罵死割除 時間: 2025-3-22 08:38
Bronchioalveolar Stem Cells in Cancerfollowing injury, and in regeneration following tissue loss. In the highly branched and compartmentalized lung, homeostatic maintenance and responses to injury are driven by multiple, different, resident stem/progenitor cell populations. Consistent or repeated failure of lung stem/progenitor cell fu作者: GUILE 時間: 2025-3-22 12:07
Role of Progenitors in Pulmonary Fibrosis and Asthmanges that impair lung function. Lung injury elicits the recruitment of stem cells and other progenitor cells and fibrocytes to the airways. These cells proliferate and differentiate into fibroblasts and myofibrolasts that secrete collagen and other extracellular matrix components that damage the nor作者: ABOUT 時間: 2025-3-22 14:27
Challenges of Cell Therapy for Lung Diseases and Critical Illnesseseen increasing in the past decades. Moreover, the currently available treatments are not able to reverse the pathological damage or to restore normal organ function. Thus, there is an urgent search for new regenerative strategies. In parallel, while advances in supportive care have improved survival作者: Spirometry 時間: 2025-3-22 17:32
The Hope for iPSC in Lung Stem Cell Therapy and Disease Modelingly difficult; iPSC provide the potential for the generation of reproducible systems for investigating the cellular, molecular, and genetic mechanisms that lead to the development and pathogenesis of human lung disease. In the future, this technology holds great potential for the development of novel作者: 粗魯性質(zhì) 時間: 2025-3-22 22:06
Ex Vivo Lung Bioengineeringe therapeutic option. However, the number of suitable donor lungs is inadequate to meet current clinical demands and thus alternative options are desperately needed. There are a number of conceptually viable alternatives for generating functional pulmonary tissue ex vivo, including the use of either作者: 多節(jié) 時間: 2025-3-23 01:43
Endothelial Colony-Forming Cells in Pulmonary Arterial Hypertensionnd led to the discovery of a subset of cells with robust endothelial regenerative capacity. These so-called endothelial colony-forming cells (ECFCs) are phenotypically identical to cultured endothelial cells, but exhibit high proliferative potential, capacity for self-regeneration, and in vivo vascu作者: Coronary-Spasm 時間: 2025-3-23 07:20
Mesothelial Progenitors in Development, Lung Homeostasis, and Tissue Repairetic tools, lineage properties, and signals controlling migration and differentiation. We also discuss the potential role of this cell in endogenous injury responses, inflammation, fibrosis, and tissue regeneration.作者: lipoatrophy 時間: 2025-3-23 12:43 作者: Brain-Waves 時間: 2025-3-23 15:45 作者: 有節(jié)制 時間: 2025-3-23 21:42
Hematopoietic Stem Cells and Chronic Hypoxia-Induced Pulmonary Vascular Remodellingcan lead to severe and progressive pulmonary hypertension (PH) and right heart failure. Key transcriptional regulators are known to coordinate the cellular response to oxygen deprivation (i.e., early growth response (Egr)-1 and hypoxia-inducible factor (HIF)-1α and HIF-2α), but some of them are also作者: tattle 時間: 2025-3-24 01:51
Fibrocytes and Pulmonary Vascular Remodeling: The Good, the Bad, and the Progenitorsthemselves in the lung vasculature(s). The primary diseases associated with PVR are pulmonary edema and pulmonary hypertension (PH), which is a prevalent disease of many etiologies. The lymphatic system is important in maintaining tissue homeostasis and its vasculature provides a means of interface 作者: A保存的 時間: 2025-3-24 06:26 作者: 刺耳 時間: 2025-3-24 06:55
Pulmonary Arterial Hypertension: A Stem Cell Hypothesiss, also known as pulmonary vascular remodeling. While the muscularization of the small pulmonary arteries is a physiological and reversible response to chronic hypoxia, the vascular changes known as “angioproliferative” are irreversible and refractory to therapy. Our hypothesis of the “quasi-maligna作者: Esophagus 時間: 2025-3-24 12:59 作者: EXULT 時間: 2025-3-24 18:27 作者: Processes 時間: 2025-3-24 19:28
Amy L. Firth,Jason X.-J. Yuanrole of lateness and melancholy in European fiction at the t.This book is the first comparative study of novels by Patrick Modiano, W. G. Sebald, and Antonio Mu?oz Molina. Drawing on many literary figures, movements, and traditions, from the Spanish Golden Age, to German Romanticism, to French philo作者: Orgasm 時間: 2025-3-25 02:13
Quentin Felty,Seiijiro Sakao,Norbert F. Voelkeles, altering the membrane ionic ATP-dependent pumps which favours the entry of calcium, sodium and water to the cytoplasma. The catabolism of adenine nucleotides results in an accumulation of hypoxanthine. The supply of molecule oxygen during blood reperfusion generates oxygen free radicals which ar作者: 分期付款 時間: 2025-3-25 04:59
Glenn Marsboom,Min Zhang,Jalees Rehman,Asrar B. Malik Ph.D.strated by the profound immunodeficiency caused by the HIV virus which preferentially infects CD4+ cells. Indeed, CD4+ T cells are required for the induction of cell-mediated immune responses involving activated macrophages or cytotoxic CD8 + T cells as well as for the activation of B cells and thei作者: 托運 時間: 2025-3-25 11:09 作者: CYT 時間: 2025-3-25 11:44 作者: 細(xì)微的差異 時間: 2025-3-25 16:05
Lung Stem Cells in the Epithelium and Vasculature978-3-319-16232-4Series ISSN 2196-8985 Series E-ISSN 2196-8993 作者: 使堅硬 時間: 2025-3-25 23:27 作者: OFF 時間: 2025-3-26 03:13 作者: 極力證明 時間: 2025-3-26 07:03
Mesothelial Progenitors in Development, Lung Homeostasis, and Tissue Repairetic tools, lineage properties, and signals controlling migration and differentiation. We also discuss the potential role of this cell in endogenous injury responses, inflammation, fibrosis, and tissue regeneration.作者: 避開 時間: 2025-3-26 11:31 作者: Longitude 時間: 2025-3-26 14:59
The Role of Stem Cells in Vascular Remodeling in CTEPHe that attracts stem and progenitor cells exacerbating the associated pulmonary vascular remodeling. This chapter focuses on what is currently known about the presence and role of such stem and progenitor cells in the pathogenesis of CTEPH.作者: 親愛 時間: 2025-3-26 19:40 作者: 審問 時間: 2025-3-26 23:06 作者: phlegm 時間: 2025-3-27 03:53
Book 2015 it discusses the rapidly growing field of stem cells and cell therapy as it relates to lung biology and disease as well as .ex vivo .lung bioengineering. Such approaches may provide novel therapeutic approaches for lung diseases. Human pluripotent stem cell differentiation to model the pulmonary ep作者: 欲望小妹 時間: 2025-3-27 08:28
2196-8985 and vasculature from development through disease and repair.This book covers the identification and role of endogenous lung stem cells in health and disease, particularly the most recent advances. In addition, it discusses the rapidly growing field of stem cells and cell therapy as it relates to lu作者: 盲信者 時間: 2025-3-27 11:19
The Hope for iPSC in Lung Stem Cell Therapy and Disease Modelingthat lead to the development and pathogenesis of human lung disease. In the future, this technology holds great potential for the development of novel screening techniques and even cell-based therapeutic approaches for human lung disease. This chapter discusses how iPSC and gene editing technologies are being applied to study human lung disease.作者: 支架 時間: 2025-3-27 15:16 作者: 一罵死割除 時間: 2025-3-27 18:50 作者: Anguish 時間: 2025-3-27 23:02 作者: 吝嗇性 時間: 2025-3-28 05:48 作者: 喚起 時間: 2025-3-28 07:05
Stem Cell Niches in the Lungf submucosal glands (SMGs), basal cells in intercartilaginous regions of the proximal trachea and bronchi, variant club cells (formerly termed Clara cells) in the neuroendocrine bodies (NEBs) of bronchioles, bronchioalveolar stem cells (BASCs) at bronchioalveolar duct junctions (BADJs) in the respir作者: Limited 時間: 2025-3-28 11:22
Bronchioalveolar Stem Cells in Cancere of cell that incurred the first oncogenic mutation. Over the last two decades, multiple studies have postulated that stem cells themselves, or cells that have de-differentiated and acquired stem-like properties, may serve as the originating cancer cell. Like stem cells that drive development, tiss作者: separate 時間: 2025-3-28 18:38 作者: Ornithologist 時間: 2025-3-28 19:30
Challenges of Cell Therapy for Lung Diseases and Critical Illnessescal experimental models of pulmonary disorders and critical illnesses as well as in ongoing clinical trials will be reviewed. Relevant mechanisms of MSC actions and next steps in the translational pathway for MSC therapy for lung diseases will be discussed.作者: 無政府主義者 時間: 2025-3-28 22:56 作者: 無畏 時間: 2025-3-29 04:33
Pulmonary Arterial Hypertension: A Stem Cell Hypothesisistry indicates that cells which express stem cell markers are present in the lung vessels from rats that developed severe angio-obliterative PAH in the Sugen 5416/chronic hypoxia (Su/Hx) model and in the lungs from patients with severe PAH. Acceptance of a stem cell hypothesis of severe angio-oblit作者: 誘使 時間: 2025-3-29 09:50
Vascular Repair and Regeneration by Endothelial Progenitor Cellsssels after injury has been demonstrated in animal models of myocardial infarction, hind limb ischemia, and wound healing. Recent findings also indicate that EPCs could be involved in pulmonary vascular repair and in this chapter, we will discuss the experiments that have been performed to establish作者: Cantankerous 時間: 2025-3-29 13:31
Book 2015l Biology and Regenerative Medicine. series is essential reading for researchers and clinicians interested in stem cells, lung biology and regenerative medicine. It is also an invaluable resource for advanced students studying cell biology, regenerative medicine and lung physiology..作者: 運動吧 時間: 2025-3-29 15:43
Kelley L. Colvin,Ozus Lohani,Michael E. Yeager his hopes of confronting these supporters of the National Socialist?regime, his remark also offers an intriguing point of departure for reading Dora Bruder. It demonstrates a clear awareness on the part of the narrator of the late historical position from which he is writing, as well as of the nati作者: Nerve-Block 時間: 2025-3-29 21:34 作者: Amenable 時間: 2025-3-30 00:42
Quentin Felty,Seiijiro Sakao,Norbert F. Voelkelmorphonuclear cells become activated and produce oxygen free radicals via the respiratory burst, and proteases. The altered endothelium in ischemia-reperfusion injury results in an imbalance of the ratio between endothelin and nitric oxide that entails a vasoconstrictory state which impairs renal fu作者: 變白 時間: 2025-3-30 06:12 作者: arrhythmic 時間: 2025-3-30 09:53 作者: 四目在模仿 時間: 2025-3-30 13:23
Thomas J. Lynch,Xiaoming Liu,Jun Wei,John F. Engelhardt作者: 絕種 時間: 2025-3-30 17:38 作者: 苦笑 時間: 2025-3-30 22:58 作者: 衣服 時間: 2025-3-31 01:08 作者: 有雜色 時間: 2025-3-31 08:18
Darcy E. Wagner,Franziska E. Uhl,Melanie K?nigshoff,Daniel J. Weiss作者: 很是迷惑 時間: 2025-3-31 10:54
Kewal Asosingh Ph.D.,Jonathan Rose,Serpil Erzurum M.D.作者: 良心 時間: 2025-3-31 13:39 作者: –DOX 時間: 2025-3-31 18:53 作者: 表主動 時間: 2025-3-31 22:55 作者: 發(fā)出眩目光芒 時間: 2025-4-1 05:42
Equilibrium Studies of Electron Transfer Reactions, Locke in our laboratory has measured the gas-phase basicity of several amino acids, purines, pyrimidines and nucleosides using a direct insertion probe to admit these low volatility compounds to the ICR analyzer cell [3,4].
