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標題: Titlebook: Lipid Metabolism in the Healthy and Disease Heart; Ger J. Vusse,Hans Stam Book 1992 Springer Science+Business Media Dordrecht 1992 ATP.Lip [打印本頁]

作者: 拿著錫    時間: 2025-3-21 18:32
書目名稱Lipid Metabolism in the Healthy and Disease Heart影響因子(影響力)




書目名稱Lipid Metabolism in the Healthy and Disease Heart影響因子(影響力)學科排名




書目名稱Lipid Metabolism in the Healthy and Disease Heart網(wǎng)絡公開度




書目名稱Lipid Metabolism in the Healthy and Disease Heart網(wǎng)絡公開度學科排名




書目名稱Lipid Metabolism in the Healthy and Disease Heart被引頻次




書目名稱Lipid Metabolism in the Healthy and Disease Heart被引頻次學科排名




書目名稱Lipid Metabolism in the Healthy and Disease Heart年度引用




書目名稱Lipid Metabolism in the Healthy and Disease Heart年度引用學科排名




書目名稱Lipid Metabolism in the Healthy and Disease Heart讀者反饋




書目名稱Lipid Metabolism in the Healthy and Disease Heart讀者反饋學科排名





作者: 屈尊    時間: 2025-3-21 22:25

作者: BORE    時間: 2025-3-22 03:43

作者: Cabinet    時間: 2025-3-22 06:15
Myocardial fatty acid oxidation during ischemia and reperfusion,provide indirect evidence for the hypothesis that intracellular calcium transport may be involved in the mechanisms responsible for the high oxidative metabolic rate early after reperfusion. (Mol Cell Biochem .: 103–109, 1992)
作者: Radiculopathy    時間: 2025-3-22 09:37
978-1-4613-6559-4Springer Science+Business Media Dordrecht 1992
作者: 導師    時間: 2025-3-22 15:21
Overview: 978-1-4613-6559-4978-1-4615-3514-0
作者: Diluge    時間: 2025-3-22 20:20

作者: –scent    時間: 2025-3-23 00:19

作者: LAP    時間: 2025-3-23 04:32
https://doi.org/10.1007/978-1-4615-3514-0ATP; Lipid; Oxidation; biochemistry; cardiac function; cells; endothelium; enzymes; metabolism; smooth muscle
作者: 利用    時間: 2025-3-23 09:27

作者: Buttress    時間: 2025-3-23 10:43
G. W. Sloof,F. C. Visser,T. Teerlink,E. F. I. Comans,M. J. van Eenige,G. J. van der Vusse,F. F. Knapior.Includes an analysis of the literature on both suitabili.Investment Management and Mismanagement: History, Findings, and Analysis will present the reader with: (1) a brief overview of portfolio management and its historical evolution , (2) the findings of a substantial amount of academic researc
作者: 不適當    時間: 2025-3-23 15:26

作者: 付出    時間: 2025-3-23 21:23

作者: florid    時間: 2025-3-24 00:43

作者: CONE    時間: 2025-3-24 04:57

作者: 舞蹈編排    時間: 2025-3-24 07:17

作者: Override    時間: 2025-3-24 12:22

作者: MOAT    時間: 2025-3-24 18:00

作者: critic    時間: 2025-3-24 19:36
Paradoxical role of lipid metabolism in heart function and dysfunction,art muscle is controlled by the availability of exogenous fatty acids, the rate of acyl translocation across the mitochondrial membrane and the rate of acetyl-CoA oxidation by the citric acid cycle. Carnitine acyl-CoA tranferase appears to have an important function in coupling the fatty acid activa
作者: DEVIL    時間: 2025-3-25 00:18

作者: 愛社交    時間: 2025-3-25 03:21

作者: Facilities    時間: 2025-3-25 08:41
Long term Incubation of Cardiac Myocytes with Oleic Acid and Very-low Density Lipoprotein Reduces Hould be released into the medium when cardiac myocytes were incubated with heparin. Incubation of cardiac myocytes with VLDL, or the products of TG breakdown, oleic acid or 2-monoolein, did not increase LPL activity in the medium. However, incubation of cardiac myocytes with either VLDL or oleic aci
作者: 充滿人    時間: 2025-3-25 15:28
Carnitine Palmitoyltransferase in the Heart is Controlled by a Different Mechanism than the Hepaticaspect of the regulatory mechanism for . fatty acid oxidation. Inhibition of myocardial carnitine palmitoyltransferase by malonyl-CoA may play an important role in regulation of fatty acid oxidation in the heart, but there has been a discrepancy in data relating to changes in malonyl-CoA sensitivity
作者: 休戰(zhàn)    時間: 2025-3-25 16:47

作者: insurrection    時間: 2025-3-25 21:15

作者: 反叛者    時間: 2025-3-26 03:15
Occurrence and Functions of the Phosphatidylinositol Cycle in the Myocardium,ules and which involves the hydrolysis of a quantitatively minor phospholipid, phosphatidylinositol 4,5-bisphosphate (PtdIns(4,5)P.) by a Ptdlns-specific phospholipase C (PLC). The evidence for the existence of receptor-mediated GTP binding protein-coupled PLC in myocardium and its possible function
作者: 耕種    時間: 2025-3-26 06:35

作者: evanescent    時間: 2025-3-26 10:04

作者: 溺愛    時間: 2025-3-26 12:53

作者: fringe    時間: 2025-3-26 20:18

作者: 卡死偷電    時間: 2025-3-26 22:41

作者: Delectable    時間: 2025-3-27 01:42
Myocardial fatty acid oxidation during ischemia and reperfusion,tially toxic intermediates such as acylCoA and acylcarnitine. After reperfusion both myocardial oxygen consumption and fatty acid oxidation may rapidly recover to preischemic levels, even when contractile function remains depressed. The mechanisms underlying the apparent dissociation between contrac
作者: Vo2-Max    時間: 2025-3-27 08:09
The relative contribution of glucose and fatty acids to ATP production in hearts reperfused followiidation rates during reperfusion are low under these conditions, which can result in a decreased recovery of mechanical function. Stimulation of glucose oxidation with the carnitine palmitoyl transferase I inhibitor, Etomoxir, or by directly stimulating pyruvate dehydrogenase activity with dichloroa
作者: FICE    時間: 2025-3-27 11:56

作者: Modicum    時間: 2025-3-27 14:50
Carnitine requirement of vascular endothelial and smooth muscle cells in imminent ischemia,re vulnerable to oxidative stress than muscle cells and are prone to loose carnitine early during hypoperfusion. This has been suggested by two observations. The first is that incubation of isolated endothelial cells in a low carnitine medium leads to oleate oxidation, dependent upon carnitine addit
作者: 技術    時間: 2025-3-27 20:45

作者: 自然環(huán)境    時間: 2025-3-27 23:23
John S. Charnock,Peter L. McLennan,Mahinda Y Abeywardena
作者: 類似思想    時間: 2025-3-28 03:06
Brian Rodrigues,Michael R. Spooner,David L. Severson
作者: 才能    時間: 2025-3-28 06:59

作者: 抱負    時間: 2025-3-28 13:57

作者: STYX    時間: 2025-3-28 15:35
Jos M. J. Lamers,Dick H. W. Dekkers,Karel Bezstarosti,Johanna T. A. Meij,Han A. A. van Heugten
作者: debacle    時間: 2025-3-28 20:56

作者: HEW    時間: 2025-3-29 02:47
Paradoxical role of lipid metabolism in heart function and dysfunction,ciated with cardiac dysfunction and cell damage in both ischemic and diabetic hearts. Alterations in the composition of membrane phospholipids are also consiered to change the activities of various membrane bound enzymes and subsequently heart function under different pathophysiological conditions.
作者: 有助于    時間: 2025-3-29 05:40

作者: 四溢    時間: 2025-3-29 08:39
The substrate specificity of phosphoinositide- phospholipase C in rat heart sarcolemma,very low rate (0.36 ± 0.08 nmol mg. min.). Unlike PtdIns(4,5)P., PLC-dependent PtdIns4P and Ptdlns hydrolysis was not inhibited by Ca. concentrations over 1 mM. The possibility of distinct isozymes being responsible for the different hydrolytic activities is discussed. (Mol Cell Biochem .: 27–31, 19
作者: Cholagogue    時間: 2025-3-29 13:04

作者: LINES    時間: 2025-3-29 17:33
Myocardial cell vulnerability to exogenous phospholipase attack,difference was much more pronounced in the latter. Finally, the cellular contents of ATP were maintained both in TG-loaded and hypothermic rewarmed myocytes, while energy depleted myocytes contained only about 25% of the normal ATP level. These results demonstrate that attack from exogenously added
作者: 注意    時間: 2025-3-29 19:47
Phosphatidylcholine metabolism in ischemic and hypoxic hearts,emonstrate the possible rate-limiting role of choline kinase and reconfirm the regulatory role of the cytidylyltrans-ferase in the biosynthesis of phosphatidylcholine. (Mol Cell Biochem 116: 53–58, 1992)
作者: CLEFT    時間: 2025-3-30 00:47
Occurrence and Functions of the Phosphatidylinositol Cycle in the Myocardium,5) source other than PtdIns(4,5)P. contributing to the stimulated DAG, 6) role of the Ptdlns pathway in cardiomyocyte growth and gene expression during the hypertrophic response. (Mol Cell Biochem .: 59–67, 1992)
作者: PUT    時間: 2025-3-30 08:00

作者: 大雨    時間: 2025-3-30 08:34

作者: expound    時間: 2025-3-30 12:23

作者: 宣傳    時間: 2025-3-30 17:51

作者: 樂意    時間: 2025-3-30 22:23
The relative contribution of glucose and fatty acids to ATP production in hearts reperfused followifatty acids for β-oxidation. Using hearts in which the myocardial triglycerides were pre-labeled, the contribution of both endogenous and exogenous fatty acid oxidation to myocardial ATP production was determined in hearts perfused with 11 mM glucose, 1.2 mM palmitate and 500 μU/ml insulin. In heart
作者: BRINK    時間: 2025-3-31 03:19

作者: investigate    時間: 2025-3-31 06:09

作者: 憤怒歷史    時間: 2025-3-31 09:55
Alain Grynberg,Gilles Nalbone,Jeannie Leonardi,Huguette Lafont,Pierre Athiasurnals. This work should be of value to academic researchers as a convenient source of summarized studies in these areas, while practitioners will find value its content as an efficient reference for determining the benefits of asset management as well as potential pitfalls..978-1-4419-4152-7978-0-387-33830-9
作者: jet-lag    時間: 2025-3-31 17:10
G. W. Sloof,F. C. Visser,T. Teerlink,E. F. I. Comans,M. J. van Eenige,G. J. van der Vusse,F. F. Knapurnals. This work should be of value to academic researchers as a convenient source of summarized studies in these areas, while practitioners will find value its content as an efficient reference for determining the benefits of asset management as well as potential pitfalls..978-1-4419-4152-7978-0-387-33830-9




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