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標(biāo)題: Titlebook: Interaction Between Neurons and Glia in Aging and Disease; Jo?o O. Malva,Ana Cristina Rego,Catarina R. Olivei Book 2007 Springer-Verlag US [打印本頁(yè)]

作者: 召集會(huì)議    時(shí)間: 2025-3-21 17:46
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作者: MEET    時(shí)間: 2025-3-22 00:03

作者: inchoate    時(shí)間: 2025-3-22 03:31
Maria Jo?o Saraivanktionalen Arbeitsteilung eine neue Sichtweise dar. Der Grundgedanke der funktionalen Arbeitsteilung besteht darin, dass Stellen mit gleichen Aufgaben zu spezialisierten Abteilungen zusammengefasst werden. Als Klassiker dieses Top-Down-Ansatzes des Organisierens kann das Werk von Koslos bezeichnet w
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Paul A. Adlard,Ashley I. Bushnktionalen Arbeitsteilung eine neue Sichtweise dar. Der Grundgedanke der funktionalen Arbeitsteilung besteht darin, dass Stellen mit gleichen Aufgaben zu spezialisierten Abteilungen zusammengefasst werden. Als Klassiker dieses Top-Down-Ansatzes des Organisierens kann das Werk von Koslos bezeichnet w
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作者: commune    時(shí)間: 2025-3-23 10:05
Subventricular Zone Cells as a Tool for Brain Repairal progenitors, offering promising perspectives for future cell-based therapies. Many factors able to modulate neuronal production have been identified but efficient differentiation into specific neuronal phenotypes is still a challenge. Animal models of brain diseases have been used in neuronal rep
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作者: Pathogen    時(shí)間: 2025-3-23 23:49
Neurotrophin Signaling and Cell Survivalcal functions are mediated by two distinct families of receptors, the Trk receptor tyrosine kinases (TrkA, TrkB and TrkC) and the p75 neurotrophin receptors (p75NTR with different signaling activity. Both receptor types convey trophic signals, but p75NTR activation may also cause apoptotic cell deat
作者: AGATE    時(shí)間: 2025-3-24 04:12
GDNF: a Key Player in Neuron-Glia Crosstalk and Survival of Nigrostriatal Dopaminergic Neuronsn Parkinson’s disease (PD). In animal models of PD, GDNF delivery has been shown to both protect dopaminergic neurons against toxin-induced injury and to rescue damaged neurons, promoting recovery of the motor deficit. GDNF may act both as a target-derived neurotrophic factor in the striatum, and as
作者: BROTH    時(shí)間: 2025-3-24 09:42
Molecular Pathways of Mitochondrial Dysfunction in Neurodegeneration: the Paradigms of Parkinson’s aracellular calcium homeostasis, and producers of endogenous reactive oxygen species (ROS). Mitochondria also regulate cell death, marking the point of no return in necrosis and apoptosis. Many evidences have been raised implicating mitochondria defects as crucial mechanisms in the pathogenesis of se
作者: 擦試不掉    時(shí)間: 2025-3-24 11:28
Zinc Homeostasis and Brain Injurye brain is regulated through highly dynamic pathways and is deeply connected with other major signaling pathways, such as NO- and MAP kinase-dependent systems. Zn2+ signaling in neurons and glia also interplays with proton and Ca2+ homeostasis. Zn2+ appears to promote injury with greater potency com
作者: 平庸的人或物    時(shí)間: 2025-3-24 18:30
Aging and Cognitive Decline: Neuroprotective Strategiesable to reverse or prevent neuronal damage. Neuroprotective strategies have been classified according to the mechanisms of neuronal death, and this chapter will deal mainly with neuroinflammation and related processes that occur in aging, cognitive decline, and dementia. The evidence for inflammator
作者: Chameleon    時(shí)間: 2025-3-24 23:04

作者: glamor    時(shí)間: 2025-3-25 01:07
Neuroinflammation and Excitotoxicity in Neurobiology of HIV-1 Infection and AIDS: Targets for Neuropfected peripheral immune-competent cells, in particular macrophages, appear to infiltrate the central nervous system (CNS) and provoke a neuropathological and inflammatory response involving all cell types in the brain. In fact, HIV-1 seems to enter the brain very soon after infection and can subseq
作者: 粘連    時(shí)間: 2025-3-25 05:49
Neuroinflammation and Mitochondrial Dysfunction in Alzheimer’s and Prion’s Diseasesmyloidogenic proteins, amyloid-beta (A ), and pathogenic scrapie prion (PrPSc), respectively. In both disorders, cerebral amyloid deposits are associated with a local inflammatory response, which is initiated by the activation of microglia and recruitment of astrocytes. Activated microglia, particul
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作者: 修飾語(yǔ)    時(shí)間: 2025-3-25 17:36
athology.Includes supplementary material: .Interactions Between Neurons and Glia in Aging and Disease is a source of "reference reviews" and "concise overviews" of the functional cross-talk between neurons and glial cells, with a focus on microglia and inflammation in the aging brain and in neurolog
作者: Induction    時(shí)間: 2025-3-25 22:48
Book 2007tween neurons and glial cells, with a focus on microglia and inflammation in the aging brain and in neurological disorders. The scientific area covered by this book may be considered of key interest, and it is recognized, by the scientific community, as a major competitive area with critical implica
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作者: phase-2-enzyme    時(shí)間: 2025-3-26 06:37

作者: 嬰兒    時(shí)間: 2025-3-26 10:39
Aging and Cognitive Decline: Neuroprotective Strategiesy phenomena in dementia, especially in Alzheimer’s disease (AD), is impressive, and the role of inflammatory cells and mediators has been extensively studied. Furthermore, anti-inflammatory drugs (specially the nonsteroidal anti-inflammatory drugs (NSAIDs)) have been shown to attenuate these phenomena in preclinical studies.
作者: 虛度    時(shí)間: 2025-3-26 13:01
Neuroinflammation and Mitochondrial Dysfunction in Alzheimer’s and Prion’s Diseasesarly those in the vicinity of amyloid deposits can produce and release proinflammatory cytokines, chemokines, complement proteins, acute-phase proteins, and reactive oxygen and nitrogen species that can damage the neighboring neurons.
作者: EXUDE    時(shí)間: 2025-3-26 20:34
The Impact of an Imbalance Between Proinflammatory and Anti-inflammatory Influences on Synaptic funct it is an imbalance between proinflammatory and anti-inflammatory cytokines that leads to age-related deficits in synaptic function and that strategies that restore the balance are likely to be beneficial in reducing the deterioration of function that accompanies age.
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作者: GLIDE    時(shí)間: 2025-3-27 12:49
Opposite Modulation of Peripheral Inflammation and Neuroinflammation by Adenosine A2A Receptorsview the evidences suggesting that A2A receptor control neuroinflammation and discuss the possible mechanisms underlying the opposite modulation by A2A receptors of peripheral inflammation and neuroinflammation.
作者: OTHER    時(shí)間: 2025-3-27 16:55

作者: 中子    時(shí)間: 2025-3-27 17:54
Inflammation and Neuronal Susceptibility to Excitotoxic Cell Deathlight into the cellular and molecular mechanisms of neurodegeneration. The response of microglia and astrocytes to injury, releasing signaling molecules (specially, cytokines, chemokines, growth factors, reactive oxygen and nitrogen species), is central in neuroinflammation and in neuron death/survival.
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作者: ROOF    時(shí)間: 2025-3-28 14:00
Neuroinflammation and Excitotoxicity in Neurobiology of HIV-1 Infection and AIDS: Targets for Neuropical and inflammatory response involving all cell types in the brain. In fact, HIV-1 seems to enter the brain very soon after infection and can subsequently induce severe and debilitating neurological problems that range from mild behavioral abnormalities and motor dysfunction to frank dementia.
作者: A精確的    時(shí)間: 2025-3-28 15:07
Epilepsy, Brain Injury and Cell Deathx. Within the hippocampus, pyramidal cells of the areas CA1 and CA3 and interneurons of the dentate hilus are most vulnerable, whereas granule cells of the dentate gyrus, CA2 pyramidal cells and the subiculum are comparatively preserved.
作者: Anterior    時(shí)間: 2025-3-28 21:09
Jo?o O. Malva,Ana Cristina Rego,Catarina R. OliveiThe book is broken into three main sections to help guide the reader from the basic functional cross-talk between neuron and glia to brain pathology.Includes supplementary material:
作者: 慢跑    時(shí)間: 2025-3-29 00:48
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作者: 連接    時(shí)間: 2025-3-29 13:28
s from the basic functional cross-talk between neuron and glia to brain pathology. The specific proposed sections are: 1) Neuroimmunity and Neuroinflammation; 2) Signalling and inflammation in aging; 3) Neurodegeneration and inflammation in age related diseases..978-1-4419-4360-6978-0-387-70830-0
作者: 疾馳    時(shí)間: 2025-3-29 17:42
Frederico Sim?es do Couto,Alexandre de Mendon?aür die sich BPR zu einem ?u?erst lukrativen Produkt entwickelt hat. Zwar konnten die hohen an BPR geknüpften Erwartungen nur bei einem Teil der bisherigen Projekte realisiert werden, ungeachtet dessen erfreut sich BPR in der Managerliteratur weiterhin gro?er Popularit?t. Auf wissenschaftlicher Seite
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作者: 付出    時(shí)間: 2025-3-30 07:36
Marcus Kaul,Stuart A. Liptonng erfüllt wird, weshalb dies der Ausgangspunkt des prozessualen Gestaltens sei. Mit dem Begriff der Prozessorganisation wurde diese Idee von . 1983 aktualisiert.: Die wirkliche Struktur eines Betriebes ist die eines Stromes. Der Betriebsprozess stellt eine Leistungskette dar, die solange zu untergl
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Paul A. Adlard,Ashley I. Bushng erfüllt wird, weshalb dies der Ausgangspunkt des prozessualen Gestaltens sei. Mit dem Begriff der Prozessorganisation wurde diese Idee von . 1983 aktualisiert.: Die wirkliche Struktur eines Betriebes ist die eines Stromes. Der Betriebsprozess stellt eine Leistungskette dar, die solange zu untergl
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Microglial Cell Population Expansion Following Acute Neural Injurymechanisms. Increased insight into basic microglial cell biology may improve diagnostic and therapeutic possibilities for patients suffering from conditions that are currently without real treatment options.
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M. Wirenfeldt,L. Dissing- Olesen,A. A. Babcock,R. Ladeby,M. B. Jensen,T. Owens,Bente Finsen
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Ana Cristina Rego,Sandra Morais Cardoso,Catarina R. Oliveira
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