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標題: Titlebook: HDL Deficiency and Atherosclerosis; G. Assmann Book 1995 Kluwer Academic Publishers 1995 Lipoprotein.atherosclerosis.cardiovascular.corona [打印本頁]

作者: 宣告無效    時間: 2025-3-21 16:16
書目名稱HDL Deficiency and Atherosclerosis影響因子(影響力)




書目名稱HDL Deficiency and Atherosclerosis影響因子(影響力)學科排名




書目名稱HDL Deficiency and Atherosclerosis網(wǎng)絡公開度




書目名稱HDL Deficiency and Atherosclerosis網(wǎng)絡公開度學科排名




書目名稱HDL Deficiency and Atherosclerosis被引頻次




書目名稱HDL Deficiency and Atherosclerosis被引頻次學科排名




書目名稱HDL Deficiency and Atherosclerosis年度引用




書目名稱HDL Deficiency and Atherosclerosis年度引用學科排名




書目名稱HDL Deficiency and Atherosclerosis讀者反饋




書目名稱HDL Deficiency and Atherosclerosis讀者反饋學科排名





作者: Pageant    時間: 2025-3-22 00:06
Role of high density lipoprotein subclasses in reverse cholesterol transport,DL) cholesterol and the risk of myocardial infarction (reviewed in ref. 1). The ability of HDL to protect the vessel wall from atherosclerosis has usually been explained by the reverse cholesterol transport model (reviewed in ref. 2) in which HDL mediates the flux of excess cholesterol from peripher
作者: 發(fā)起    時間: 2025-3-22 03:50

作者: amplitude    時間: 2025-3-22 07:45

作者: finite    時間: 2025-3-22 09:31
Intracellular cholesterol transport,nding the mechanisms involved in regulated, target-specific intracellular cholesterol transport is of great impact for understanding the pathogenesis of atherosclerosis and a number of inherited lipid storage disorders, such as Niemann-Pick disease (type C), Zellweger disease and Tangier disease. Si
作者: 愛好    時間: 2025-3-22 14:15

作者: lipoatrophy    時間: 2025-3-22 21:03

作者: pus840    時間: 2025-3-22 23:13
Gene therapy for the genetic dyslipoproteinaemias,idated. This new information provides a conceptual framework for understanding lipoprotein metabolism in normal subjects and patients with the genetic dyslipoproteinaemias. Schematic overviews of our current concepts of lipoprotein metabolism are illustrated in Figs. 1 and 2.
作者: 鼓掌    時間: 2025-3-23 03:07

作者: 罵人有污點    時間: 2025-3-23 08:12

作者: concise    時間: 2025-3-23 12:33
The Future of Predictive Safety Evaluationary artery disease (CAD).. HDL are highly heterogeneous with respect to the hydrated density, size and composition of the particles. The major apolipoprotein (apo) in HDL, apoA-I, has been shown to have a strong inverse correlation with risk of CAD., whereas an inverse correlation with apoA-II level
作者: 鍵琴    時間: 2025-3-23 13:52

作者: Epidural-Space    時間: 2025-3-23 22:06

作者: Arable    時間: 2025-3-23 23:27

作者: 成份    時間: 2025-3-24 05:21

作者: 領巾    時間: 2025-3-24 09:55

作者: 狂熱文化    時間: 2025-3-24 12:42

作者: Comedienne    時間: 2025-3-24 15:26
HDL metabolism and atherogenesis in genetically modified mice,he role of discrete genetic factors in atherogenesis. It allows study not only of the effects in mice of candidate mutations found in humans, but also through genetic synthesis it allows study of the effects of various combinations of the mutations.
作者: 認為    時間: 2025-3-24 20:55

作者: Exuberance    時間: 2025-3-25 02:05
https://doi.org/10.1007/978-3-030-72815-1support the concepts that HDL-related measures are as important as LDL-related ones in predicting vascular events. Furthermore, pharmacological or genetic modification of certain plasma HDL subfractions appears to hold promise for clinical benefits that are comparable to, or perhaps exceed, those of LDLC reduction.
作者: MAG    時間: 2025-3-25 03:52

作者: MAG    時間: 2025-3-25 09:33
Connection between cholesterol efflux and atherosclerosis, The two major subclasses include particles that contain both apoA-I and apoA-II (termed LpA-I, A-II) and those that contain apoA-I but not apoA-II (termed LpA-I). Apo-specific particles can be isolated by use of immunoaffinity columns with anti-apoA-I or anti-apoA-II antibody..
作者: 籠子    時間: 2025-3-25 14:06
https://doi.org/10.1057/9781137295866 possesses arginine. The apoE2 isoform, associated with type III hyperlipoproteinaemia., differs from apoE3 only at residue 158, where it possesses cysteine. Apolipoproteins E3 and E4 bind normally to the low density lipoprotein (LDL) receptor, whereas apoE2 does not.
作者: 角斗士    時間: 2025-3-25 15:50

作者: essential-fats    時間: 2025-3-25 23:18
Book 1995on of massive scientific research, continuous transfer of the results to medical practice, community actions and population aware- ness. The death rate from coronary heart disease in the United States rose 20% from 1950 to 1963, when a dramatic and steady downward inflection began, arriving today at
作者: Entreaty    時間: 2025-3-26 01:16
HDLC as a therapeutic target in coronary disease: current concepts and future directions,support the concepts that HDL-related measures are as important as LDL-related ones in predicting vascular events. Furthermore, pharmacological or genetic modification of certain plasma HDL subfractions appears to hold promise for clinical benefits that are comparable to, or perhaps exceed, those of LDLC reduction.
作者: Munificent    時間: 2025-3-26 05:08

作者: 織物    時間: 2025-3-26 09:51
HDL Deficiency and Atherosclerosis978-94-011-6585-3Series ISSN 0166-9842
作者: 監(jiān)禁    時間: 2025-3-26 13:04

作者: Adrenaline    時間: 2025-3-26 17:31

作者: nugatory    時間: 2025-3-26 21:28

作者: 生命層    時間: 2025-3-27 03:31

作者: 詢問    時間: 2025-3-27 09:05
Gene therapy for the genetic dyslipoproteinaemias,idated. This new information provides a conceptual framework for understanding lipoprotein metabolism in normal subjects and patients with the genetic dyslipoproteinaemias. Schematic overviews of our current concepts of lipoprotein metabolism are illustrated in Figs. 1 and 2.
作者: Progesterone    時間: 2025-3-27 10:41
https://doi.org/10.1007/978-94-011-6585-3Lipoprotein; atherosclerosis; cardiovascular; coronary heart disease; gene therapy; genetics; heart; heart
作者: 最低點    時間: 2025-3-27 16:15

作者: 健談    時間: 2025-3-27 18:58

作者: 騷動    時間: 2025-3-27 22:09

作者: 絆住    時間: 2025-3-28 03:01

作者: armistice    時間: 2025-3-28 07:35
The Future of Predictive Safety EvaluationapoA-I deficiency, LCAT deficiency, and fish-eye disease, but also patients with Tangier disease or unclassified forms of HDL deficiency did not present with premature atherosclerosis.. Family histories of these patients did not indicate any increased prevalence of coronary heart disease (CHD) event
作者: SLUMP    時間: 2025-3-28 10:52
https://doi.org/10.1057/9780230606906 a target-specific flux of cholesterol from intracellular pools to the plasma membrane are only poorly understood at present. It is generally accepted that intracellular trafficking of cholesterol and other sterols is not mediated by non-specific diffusion but requires target-specific transport mech
作者: Hyperplasia    時間: 2025-3-28 17:34
Die Typhuserkrankungen in Salza in dem Zeitraum 1901–1922978-3-662-28881-8
作者: 變量    時間: 2025-3-28 21:46
New York. All back issues can also be ordered from Plenum. As we embark on this new partnership with Plenum, we also initiate a new venture in that this important annual reference work now covers Canadian universities as well as those in the United States. We are sure that this broader base will gre
作者: 微生物    時間: 2025-3-29 01:36
ner. Topics and features: ideal for self-study, offering many pedagogical features such as chapter-opening key topics, chapter introductions and summaries, exercises, and a glossary; presents detailed information on major figures in computing, such as Boole, Babbage, Shannon, Turing, Zuse and Von Ne




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