標題: Titlebook: HCV Infection and Cryoglobulinemia; Franco Dammacco Book 20121st edition Springer-Verlag Italia 2012 Autoimmunity.Cryoglobulinemia.HCV.Imm [打印本頁] 作者: CILIA 時間: 2025-3-21 19:16
書目名稱HCV Infection and Cryoglobulinemia影響因子(影響力)
書目名稱HCV Infection and Cryoglobulinemia影響因子(影響力)學科排名
書目名稱HCV Infection and Cryoglobulinemia網(wǎng)絡(luò)公開度
書目名稱HCV Infection and Cryoglobulinemia網(wǎng)絡(luò)公開度學科排名
書目名稱HCV Infection and Cryoglobulinemia被引頻次
書目名稱HCV Infection and Cryoglobulinemia被引頻次學科排名
書目名稱HCV Infection and Cryoglobulinemia年度引用
書目名稱HCV Infection and Cryoglobulinemia年度引用學科排名
書目名稱HCV Infection and Cryoglobulinemia讀者反饋
書目名稱HCV Infection and Cryoglobulinemia讀者反饋學科排名
作者: 未成熟 時間: 2025-3-21 22:51
https://doi.org/10.1007/978-88-470-1705-4Autoimmunity; Cryoglobulinemia; HCV; Immune Complex Diseases; Malignant Lymphoma; hepatology; infectious d作者: TIA742 時間: 2025-3-22 03:26
Franco DammaccoComprehensive review of cryoglobulinemia, for scientists and specialists involved in several disciplines, including internal medicine, hepatology, hematology, oncology, infectious diseases, rheumatolo作者: 褪色 時間: 2025-3-22 08:03
http://image.papertrans.cn/h/image/420171.jpg作者: apiary 時間: 2025-3-22 09:42 作者: gerontocracy 時間: 2025-3-22 13:22
https://doi.org/10.1007/978-1-349-26543-5nly the introduction of a new nomenclature (largely removing the designation “essential”), but studies of the relationship between this viral disease and the emergence of pathological clones of B cells.作者: dendrites 時間: 2025-3-22 18:43 作者: Tinea-Capitis 時間: 2025-3-22 21:31
https://doi.org/10.1007/978-3-030-14533-0racellular pathways. Higher serum levels of gC1qR in mixed cryoglobulinemia indicate that this protein plays a relevant role in the pathogenesis of cryoglobulin-related damage, including immune complex formation and cryoprecipitation.作者: Pigeon 時間: 2025-3-23 02:28
Cryoglobulinemia and Chronic HCV Infection: An Evolving Storynly the introduction of a new nomenclature (largely removing the designation “essential”), but studies of the relationship between this viral disease and the emergence of pathological clones of B cells.作者: 細菌等 時間: 2025-3-23 05:49
The Complement System in Cryoglobulinemiaonents in serum is paralleled by the deposition of complement in affected tissues such as the kidney. Experimental data suggest that complement activation is a major factor explaining the inflammatory organ damage seen in cryoglobulinemia in vivo. However, more studies are required to clarify the pathogenic role of complement in cryoglobulinemia.作者: ADORE 時間: 2025-3-23 11:16
The Pivotal Role of C1qR in Mixed Cryoglobulinemiaracellular pathways. Higher serum levels of gC1qR in mixed cryoglobulinemia indicate that this protein plays a relevant role in the pathogenesis of cryoglobulin-related damage, including immune complex formation and cryoprecipitation.作者: 相符 時間: 2025-3-23 15:03 作者: mitral-valve 時間: 2025-3-23 20:57
https://doi.org/10.1007/978-3-030-80367-4ologically mediated pathology. HCV-specific antibodies can limit but not completely ablate HCV infection of new target cells. As for T cell responses, the efficacy of antibody responses may be limited by viral evolution. Interferons and some soon-to-be available antiviral drugs may enhance innate and adaptive immune mechanisms.作者: 永久 時間: 2025-3-24 01:06
Michael Kaeding,Johannes Pollak,Paul Schmidt be fully elucidated. In this chapter, we consider the main organ-specific autoimmune disorders directly related to HCV infection or indirectly induced by treatment for HCV infection, and interferons in particular.作者: exceed 時間: 2025-3-24 03:15
https://doi.org/10.1007/978-1-349-13095-5n in patients with chronic hepatitis C and in those with low-level, essentially asymptomatic HCV infection. We also discuss the methods and pitfalls of HCV detection in immune cells and review the data on in vitro infection of lymphoid cells with wild-type HCV isolates.作者: 排出 時間: 2025-3-24 06:32 作者: COMA 時間: 2025-3-24 12:49 作者: 壕溝 時間: 2025-3-24 17:51 作者: 疼死我了 時間: 2025-3-24 19:20 作者: 合適 時間: 2025-3-25 02:03 作者: 放肆的我 時間: 2025-3-25 03:43
https://doi.org/10.1007/978-3-031-55935-8 cell interaction and define the nature of the HCV antigen, driving B cell disorders. Starting from a brief review of the pathophysiology of B cell activation, we discuss in detail functional and molecular evidences supporting or arguing against current directions in B cell biology during chronic HCV infection.作者: Anthrp 時間: 2025-3-25 10:02 作者: 一致性 時間: 2025-3-25 12:32 作者: OFF 時間: 2025-3-25 17:14
Molecular Insights into the Disease Mechanisms of Type II Mixed Cryoglobulinemia several findings from studies of cryoimmunoglobulin glycosylation together with the characterization of monoclonal IgM rheumatoid factor activity/recognition may provide a better understanding of the pathologic process linking cryoprecipitation with hepatitis C virus infection.作者: entice 時間: 2025-3-25 20:05
reported that a high proportion of ‘essential’ cryoglobulinemias are in fact associated with HCV infection. This hallmark has resulted in a new impetus and great enthusiasm in carrying out new researches made p978-88-470-5581-0978-88-470-1705-4作者: semble 時間: 2025-3-26 00:23 作者: 聽覺 時間: 2025-3-26 06:42
The Future of Employment Relationsferative disorder. Some individuals develop B cell non-Hodgkin lymphoma, which is usually of low-grade, although more aggressive forms have been reported. Mixed cryoglobulinemia causes a small-vessel vasculitis, manifesting as palpable purpura, arthritis, membranoproliferative glomerulonephritis, an作者: Notify 時間: 2025-3-26 09:49
https://doi.org/10.1007/978-94-017-3331-1cells relative to the number of HCV genomes detected. B cells, like other blood cells, do not express all of the cellular factors believed to be required for HCV entry or productive infection. Thus, a number of significant questions must be addressed before we understand whether, and how, infection 作者: 產(chǎn)生 時間: 2025-3-26 13:01 作者: 交響樂 時間: 2025-3-26 17:11
The Future of Financial Systems and Servicesypes may be expanded on specific B-cell subsets or may provide a marker for coexistent lymphoproliferative disease, both of which may regress with clearance of the virus. In other instances, MC CRI and cryoglobulinemia may persist even after successful antiviral therapy. The full utility of antibodi作者: 最初 時間: 2025-3-26 21:32 作者: 亂砍 時間: 2025-3-27 03:26 作者: tinnitus 時間: 2025-3-27 08:51 作者: mitral-valve 時間: 2025-3-27 13:00
HCV and Blood Cells: How Can We Distinguish Infection from Association?cells relative to the number of HCV genomes detected. B cells, like other blood cells, do not express all of the cellular factors believed to be required for HCV entry or productive infection. Thus, a number of significant questions must be addressed before we understand whether, and how, infection 作者: 陶醉 時間: 2025-3-27 16:27 作者: Comedienne 時間: 2025-3-27 19:00 作者: Decibel 時間: 2025-3-28 00:13 作者: 損壞 時間: 2025-3-28 04:04 作者: 欲望 時間: 2025-3-28 06:33
https://doi.org/10.1007/978-94-6209-512-0nstruction of the main advances recorded in the study of mixed cryoglobulinemia (MC). Detailed accounts of its clinical features and structural heterogeneity appeared in the 1960s and in 1970s. These reports defined cryoglobulins as immune complexes most frequently consisting of a monoclonal IgM kap作者: insidious 時間: 2025-3-28 14:00
The Future of Employment Relationsnfected. HCV is a major cause of liver-related morbidity and mortality, and it frequently causes extrahepatic manifestations of disease. This chapter considers the natural history, pathogenesis, and prevention of HCV. This positive strand RNA virus is transmitted by percutaneous or, in very rare cir作者: scotoma 時間: 2025-3-28 16:30 作者: 潔凈 時間: 2025-3-28 20:26
https://doi.org/10.1007/978-3-031-55935-8ing that B cells are not direct targets for productive virus replication and that viral RNA sequences cannot be integrated in the host genome. The current speculation is that protracted stimulation either by the virus alone or by HCV-containing immune complexes expands clones of B cells, which may u作者: 記憶法 時間: 2025-3-28 23:41 作者: Incommensurate 時間: 2025-3-29 05:07 作者: coagulation 時間: 2025-3-29 08:23
Martin Holland (Senior Lecturer)cular virology are greatly contributing to the advancement of knowledge in this field. Model systems for the study of HCV cell entry include HCV-like particles, HCV pseudoparticles, cell-culture-produced HCV, and the tupaia animal model. Following the identification of tetraspanin CD81 with the use 作者: ingenue 時間: 2025-3-29 14:49
https://doi.org/10.1007/978-1-349-13095-5 also invades and persists in cells of the immune system. The functional and pathological consequences of HCV lymphotropism have become increasingly recognized. Studies on HCV compartmentalization in patients with symptomatic or clinically silent (occult) HCV infections demonstrated viral replicatio作者: 單獨 時間: 2025-3-29 18:56
https://doi.org/10.1007/978-1-349-26543-5 cold with diseases. One major point was that there was no evidence for a known disease in many patients with “mixed” cryoglobulins. Thus, the terminology “mixed essential cryoglobulinemia” became accepted. The discovery of HCV as the causative agent of mixed essential cryoglobulinemia allowed not o作者: ALLEY 時間: 2025-3-29 20:32
https://doi.org/10.1057/9781137376374l activities, complement mediates the disposal of immune complexes and the products of inflammatory injury. Accordingly, low levels of complement suggesting ongoing complement activation and consumption are a well-known phenomenon in patients with cryoglobulinemia. The consumption of complement comp作者: 寬度 時間: 2025-3-30 01:02 作者: 光滑 時間: 2025-3-30 06:46 作者: BRAND 時間: 2025-3-30 10:00
https://doi.org/10.1057/9780230620858ongly associated with MC-II is the presence of cryoprecipitates, thought to be involved in the maintenance and perpetuation of the disease. The biological characteristics of MC-II cryoglobulins are extremely relevant to the etiopathogenetic mechanisms linking hepatitis C virus antigen stimulation to作者: 保存 時間: 2025-3-30 15:53
