書(shū)目名稱(chēng)Group B Coxsackieviruses影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Group B Coxsackieviruses網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Group B Coxsackieviruses網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Group B Coxsackieviruses被引頻次
書(shū)目名稱(chēng)Group B Coxsackieviruses被引頻次學(xué)科排名
書(shū)目名稱(chēng)Group B Coxsackieviruses年度引用
書(shū)目名稱(chēng)Group B Coxsackieviruses年度引用學(xué)科排名
書(shū)目名稱(chēng)Group B Coxsackieviruses讀者反饋
書(shū)目名稱(chēng)Group B Coxsackieviruses讀者反饋學(xué)科排名
作者: 條約 時(shí)間: 2025-3-21 22:59
https://doi.org/10.1007/978-3-319-14860-1The CVB have long been recognized as significant pathogens of infants and children. Although the major route for transmission of the CVB is fecal-oral, vertical transmission from mother to infant is also possible. This review will focus on the more common or clinically relevant CVB-related syndromes, their diagnosis, treatment, and prevention.作者: EPT 時(shí)間: 2025-3-22 02:41 作者: 老巫婆 時(shí)間: 2025-3-22 08:29 作者: 乏味 時(shí)間: 2025-3-22 12:16 作者: projectile 時(shí)間: 2025-3-22 13:20 作者: projectile 時(shí)間: 2025-3-22 18:45
Investigations of Anorectal Function,o explore the mechanisms by which CVB4 causes acute and chronic pancreatitis. The present review develops the idea that disease progression is a continuum, from acute inflammatory disease to chronic inflammatory disease to cancer, and that accompanying changes in gene expression are both quantitative and qualitative.作者: GUISE 時(shí)間: 2025-3-22 21:33
R. Uday Kiran,Masashi Toyoda,Koji Zettsucontained viral myocarditis to a pathogenic autoimmune response is made within hours after induction of infection and is characterized by production of a few key cytokines, including IL-1β and TNFα. Many of the lessons learned from study of these models are applicable to human myocarditis and dilated cardiomyopathy.作者: amplitude 時(shí)間: 2025-3-23 04:18
Comparative Genomics of the Coxsackie B Viruses and Related Enteroviruses by species (not by type), reflecting a high frequency of intertypic recombination within a species. Further genomic studies, accompanied by well-characterized clinical outcome/disease data, will facilitate fine-scale mapping of genetic determinants that contribute to virulence.作者: 商品 時(shí)間: 2025-3-23 09:18
CVB Translation: Lessons from the Poliovirusescation of translation factors to achieve host translation shutoff and promotion of viral translation are discussed. The translational bases for neurovirulent phenotypes and tissue specificity are also addressed.作者: 隱語(yǔ) 時(shí)間: 2025-3-23 13:19
Host Immune Responses to Coxsackievirus B3emonstrate the interplay between the virus and the cell, which ultimately determines both the type and strength of the adaptive immune response as well as whether autoimmunity will follow the infection.作者: A精確的 時(shí)間: 2025-3-23 17:05 作者: 寄生蟲(chóng) 時(shí)間: 2025-3-23 21:57
Autoimmunity in Coxsackievirus Infectioncontained viral myocarditis to a pathogenic autoimmune response is made within hours after induction of infection and is characterized by production of a few key cytokines, including IL-1β and TNFα. Many of the lessons learned from study of these models are applicable to human myocarditis and dilated cardiomyopathy.作者: 接觸 時(shí)間: 2025-3-23 22:28
https://doi.org/10.1007/978-3-642-81302-3ng them excellent viruses with which to engage the problem of how the host environment interacts with specific viral genetics to promote varying efficiencies of viral replication. It is not known how highly virulent CVB strains may arise but evidence suggests such strains are not the norm.作者: angiography 時(shí)間: 2025-3-24 04:16 作者: 芭蕾舞女演員 時(shí)間: 2025-3-24 09:28 作者: 移植 時(shí)間: 2025-3-24 13:43
https://doi.org/10.1007/978-1-4939-1118-9racterized alone, bound to the adenovirus fiber knob, and, in full-length CAR, docked in the canyon structure of the coxsackievirus capsid. Although the past decade has produced a burst of new knowledge about CAR, significant questions concerning its function in normal physiology and coxsackievirus-related pathology remain unanswered.作者: 溫和女人 時(shí)間: 2025-3-24 15:02
The Coxsackievirus and Adenovirus Receptorracterized alone, bound to the adenovirus fiber knob, and, in full-length CAR, docked in the canyon structure of the coxsackievirus capsid. Although the past decade has produced a burst of new knowledge about CAR, significant questions concerning its function in normal physiology and coxsackievirus-related pathology remain unanswered.作者: BILE 時(shí)間: 2025-3-24 21:42
Group B Coxsackievirus Virulenceng them excellent viruses with which to engage the problem of how the host environment interacts with specific viral genetics to promote varying efficiencies of viral replication. It is not known how highly virulent CVB strains may arise but evidence suggests such strains are not the norm.作者: vocation 時(shí)間: 2025-3-25 03:07
Preferential Coxsackievirus Replication in Proliferating/Activated Cells: Implications for Virus Tro have evolved to undergo productive infection in cells at the G . /S stage of the cell cycle, and to preferentially establish persistence/latent infection in quiescent cells, and we provide possible explanations for these outcomes. Finally, we consider the implications of these interactions for virus transmission and host pathology.作者: 輕觸 時(shí)間: 2025-3-25 03:21 作者: 錯(cuò)誤 時(shí)間: 2025-3-25 07:30 作者: 侵略主義 時(shí)間: 2025-3-25 13:23 作者: Postulate 時(shí)間: 2025-3-25 16:12
Group B Coxsackievirus Virulencer than through examination of naturally occurring virus strains. The CVB replicate well in a variety of different murine and human cell cultures, making them excellent viruses with which to engage the problem of how the host environment interacts with specific viral genetics to promote varying effic作者: 調(diào)味品 時(shí)間: 2025-3-25 23:22
The Coxsackievirus and Adenovirus Receptorulin superfamily protein with two extracellular Ig-like domains, a single membrane-spanning sequence, and a significant cytoplasmic domain. It is structurally and functionally similar to the junctional adhesion molecules. The amino terminal domain, distal from the membrane, has been structurally cha作者: 愛(ài)社交 時(shí)間: 2025-3-26 03:04
Coxsackievirus B RNA Replication: Lessons from Poliovirusion of enteroviruses, like coxsackievirus and poliovirus, are highly conserved. These processes require two steps of RNA amplification: (i) complete synthesis of the negative- strand RNA using input RNA as the template and (ii) synthesis of the positivestrand RNA using the intermediate negative-stra作者: 單獨(dú) 時(shí)間: 2025-3-26 06:48
CVB Translation: Lessons from the Poliovirusesiruses. Like poliovirus, coxsackievirus gene expression is controlled largely at the translation level and coxsackievirus infection results in profound changes in the profile of mRNAs with access to the protein synthesis machinery of the host cell. This review chronicles the advances in understandin作者: Heart-Attack 時(shí)間: 2025-3-26 12:01 作者: 慢慢沖刷 時(shí)間: 2025-3-26 15:38
The Impact of CVB3 Infection on Host Cell Biologyo understand the life cycle of CVB3 and its interactions with the host cell. Infection causes rapid death of host cardiomyocytes by altering normal cellular homeostasis for the efficient release of progeny virion. In this chapter, we will examine the impact that CVB3 replication has on host cell bio作者: Cleave 時(shí)間: 2025-3-26 17:13 作者: 館長(zhǎng) 時(shí)間: 2025-3-26 21:45
CVB-Induced Pancreatitis and Alterations in Gene Expressions of the pancreas and heart. Chronic inflammatory diseases are of major clinical concern, since they predispose affected individuals to cancer. For example, chronic pancreatitis, which can develop from acute pancreatitis, is a major risk factor for pancreatic cancer, a disease with an exceedingly po作者: Mangle 時(shí)間: 2025-3-27 02:06
The CVB and Etiology of Type 1 Diabetest environmental factors are involved in disease development. The most often cited environmental agents implicated as initiators of T1D are the human enteroviruses, in particular the group B coxsackieviruses (CVB). Although the connection between the CVB and T1D has not been firmly established, signi作者: 方舟 時(shí)間: 2025-3-27 08:47 作者: Monolithic 時(shí)間: 2025-3-27 13:25
Autoimmunity in Coxsackievirus Infectionve cross-reactions. Sometimes these virus-triggered immune responses progress to a pathogenic autoimmunity to form autoimmune disease. To delineate the mechanisms determining induction of autoimmune disease, we have investigated in detail a model of autoimmune myocarditis induced in genetically susc作者: collagen 時(shí)間: 2025-3-27 14:39 作者: hyperuricemia 時(shí)間: 2025-3-27 19:34
Mildred Trotter,Barbara B. Hixonand random drift acting on heterogeneous mutant spectra. Direct experimental evidence indicates that high mutation rates and complex mutant spectra can serve for the adaptation of enteroviruses to complex environments. Studies with the RNA-dependent RNA polymerase of picornaviruses suggest that mult作者: 等待 時(shí)間: 2025-3-27 22:34
Yusuf Cem Kaplan,Hilal Erol-Coskuncapsid sequence alignments and virion structures allows correlation of capsid diversity with surface features, such as loops, the receptor canyon, and antigenic sites. Pairwise sequence comparisons and phylogenetic analyses can be used to rapidly identify and classify enteroviruses. Enteroviruses ar作者: somnambulism 時(shí)間: 2025-3-28 02:57
https://doi.org/10.1007/978-3-642-81302-3r than through examination of naturally occurring virus strains. The CVB replicate well in a variety of different murine and human cell cultures, making them excellent viruses with which to engage the problem of how the host environment interacts with specific viral genetics to promote varying effic作者: craven 時(shí)間: 2025-3-28 06:59
https://doi.org/10.1007/978-1-4939-1118-9ulin superfamily protein with two extracellular Ig-like domains, a single membrane-spanning sequence, and a significant cytoplasmic domain. It is structurally and functionally similar to the junctional adhesion molecules. The amino terminal domain, distal from the membrane, has been structurally cha作者: expire 時(shí)間: 2025-3-28 13:59 作者: ALB 時(shí)間: 2025-3-28 17:40 作者: 集中營(yíng) 時(shí)間: 2025-3-28 19:48 作者: construct 時(shí)間: 2025-3-29 00:11
Protocol of Immunization of the Adult Womano understand the life cycle of CVB3 and its interactions with the host cell. Infection causes rapid death of host cardiomyocytes by altering normal cellular homeostasis for the efficient release of progeny virion. In this chapter, we will examine the impact that CVB3 replication has on host cell bio作者: 繼而發(fā)生 時(shí)間: 2025-3-29 07:08
Catherine Coyle,Victoria Lavin,Anthea Creergans including the heart, acinar and islet pancreas, liver, skeletal muscle, central nervous system, and testes. Damage to tissues occurs not only from the direct virus replication and infection of cells, but also from the host response to infection. However, without host immunity and response, the作者: Jingoism 時(shí)間: 2025-3-29 10:57
Investigations of Anorectal Function,s of the pancreas and heart. Chronic inflammatory diseases are of major clinical concern, since they predispose affected individuals to cancer. For example, chronic pancreatitis, which can develop from acute pancreatitis, is a major risk factor for pancreatic cancer, a disease with an exceedingly po作者: 我吃花盤(pán)旋 時(shí)間: 2025-3-29 12:06 作者: GLUE 時(shí)間: 2025-3-29 15:44
Periodic Orbits in Trigonometric Series,rovirus RNA at stages of disease after acute infection and correlation of enterovirus replication with worse clinical outcome suggests continued replication of the virus is involved in the progression of the disease. This finding is mirrored by the murine model of coxsackievirus B3 myocarditis, in w作者: parasite 時(shí)間: 2025-3-29 21:23
R. Uday Kiran,Masashi Toyoda,Koji Zettsuve cross-reactions. Sometimes these virus-triggered immune responses progress to a pathogenic autoimmunity to form autoimmune disease. To delineate the mechanisms determining induction of autoimmune disease, we have investigated in detail a model of autoimmune myocarditis induced in genetically susc作者: 入會(huì) 時(shí)間: 2025-3-30 03:33
https://doi.org/10.1007/978-81-322-1895-1thy. While considerable attention has focused on the role of the cellular and humoral, antigen-specific immune system in viral myocarditis, the interaction between the virus and the infected host myocyte is also important. Coxsackievirus has a relative tropism for the heart that is in part mediated 作者: progestin 時(shí)間: 2025-3-30 06:46
Steven Tracy,M. Steven Oberste,Kristen M. Drescher作者: Alopecia-Areata 時(shí)間: 2025-3-30 09:24
Coxsackieviruses and Quasispecies Theory: Evolution of Enteroviruseseroviruses is probably attributable to profound biological effects of some mutations that, because of their limited number, do not necessarily affect the phylogenetic position of the virus. The combination of highly dynamic mutant spectra with unpredictable alterations of biological behavior by mini作者: 小教堂 時(shí)間: 2025-3-30 15:16
Coxsackievirus B RNA Replication: Lessons from Poliovirus template. The RNA secondary structures at the 5′ and 3′ termini of the template position the RNA replication complex at the initiation site(s) for both negative- and positive-strand RNA synthesis, thus providing binding sites for viral and host proteins that may functionally circularize the genome 作者: PIZZA 時(shí)間: 2025-3-30 16:34 作者: 嘮叨 時(shí)間: 2025-3-30 22:58
Persistent Coxsackievirus Infection: Enterovirus Persistence in Chronic Myocarditis and Dilated Cardation of dilated cardiomyopathy, the role of virus persistence is likely to include direct effects of viral replication as well as induction of inflammation in the heart. Factors that control the extent of cardiac infection with terminally deleted enteroviruses and the relative roles of continued im
