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標(biāo)題: Titlebook: Geriatric Oncology; Martine Extermann Reference work 2020 Springer Nature Switzerland AG 2020 Geriatric Oncology.Cancer.Elderly.Biology of [打印本頁(yè)]

作者: 烈酒    時(shí)間: 2025-3-21 16:06
書目名稱Geriatric Oncology影響因子(影響力)




書目名稱Geriatric Oncology影響因子(影響力)學(xué)科排名




書目名稱Geriatric Oncology網(wǎng)絡(luò)公開度




書目名稱Geriatric Oncology網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Geriatric Oncology被引頻次




書目名稱Geriatric Oncology被引頻次學(xué)科排名




書目名稱Geriatric Oncology年度引用




書目名稱Geriatric Oncology年度引用學(xué)科排名




書目名稱Geriatric Oncology讀者反饋




書目名稱Geriatric Oncology讀者反饋學(xué)科排名





作者: reptile    時(shí)間: 2025-3-21 23:04
Integrating Geriatric Oncology in Public Health Planningo or three decades. To deal with and anticipate this challenge, successive national Cancer Control Plans have included specific actions on the organization of the treatment of elderly cancer patients. The organizational framework is based on an oncologist-geriatrician partnership at the head of onco
作者: synovitis    時(shí)間: 2025-3-22 00:35

作者: 憤慨點(diǎn)吧    時(shí)間: 2025-3-22 06:50

作者: TEN    時(shí)間: 2025-3-22 10:19

作者: 皮薩    時(shí)間: 2025-3-22 14:19

作者: 皮薩    時(shí)間: 2025-3-22 19:15
Aging and Cancer Biologyrs in different organs and tissues. Aging may increase or decrease the susceptibility of various tissues to initiation of carcinogenesis and usually facilitates promotion and progression of carcinogenesis. Aging may predispose to cancer at least by two mechanisms: tissue accumulation of cells in lat
作者: Benzodiazepines    時(shí)間: 2025-3-22 23:13
Chronic Mechanistic Target of Rapamycin Inhibition: Preventing Cancer to Delay Aging or Vice Versa?us macaques). Caloric restriction also improves healthspan, including reducing cancer development in mice and Rhesus macaques. Nonetheless, chronic caloric restriction to prolong life or healthspan, including cancer prevention, is unlikely to be popular or widely adopted for human clinical use. As c
作者: AGONY    時(shí)間: 2025-3-23 03:09

作者: Immobilize    時(shí)間: 2025-3-23 07:43
The Biologic Interconnections Between Aging and Lymphoma-cell lymphoma (DLBCL) – increases with age. In addition, its histopathogenic subcategories develop an age-dependent repartition – with less frequent germinal center-derived DLBCL and more activated B cell ones. Finally, some specific entities have been specifically described in an aged population,
作者: 石墨    時(shí)間: 2025-3-23 12:19
Aging of Natural Killer Cells in Acute Myeloid Leukemia immune system function associated with aging. Natural killer (NK) cells are innate lymphoid cells specialized in killing tumor cells as well as virus-infected cells without the requirement of prior sensitization. NK cells are also involved in regulating immune function as they produce several cytok
作者: 斥責(zé)    時(shí)間: 2025-3-23 14:20
Hematopoietic Stem Cell Aging and Malignant Hemopathies extensively studied; cellular senescence, a protective mechanism in response to DNA damage, can induce permanent growth arrest and resistance to apoptosis. Chronological age also favors the accumulation of genetic and epigenetic changes that are important contributing factors in the complex pathoge
作者: glomeruli    時(shí)間: 2025-3-23 21:35
Mitochondria, Oxidative Stress, Cancer, and Aging take place. Although ROS are an inevitable by-products of respiration, they do not necessarily have detrimental effects; low doses of ROS can have beneficial effects on cells, and their production can be finely regulated in mitochondria. Increasing ROS levels and products of the oxidative stress, w
作者: OPINE    時(shí)間: 2025-3-24 01:58
Normal and Abnormal Aging (General Perspective for the Oncologist)nged state, at homeostasis, age-related decrements in function from these changes are few in part because reserves are used to maintain homeostasis. When challenged, whether by diseases, environment, medications, etc., those age-related changes become apparent. These changes lead to increased vulner
作者: facetious    時(shí)間: 2025-3-24 03:44
Respiratory Organ Aging and Cancerg aging may mimic obstructive and restrictive lung diseases. Lung parenchyma loses elasticity via alveolar wall and mesenchymal degradation and distortion, similar to emphysema. Muscles of respiration become sarcopenic and weaken, while the thorax contorts due to osteoporotic vertebral fractures, al
作者: wangle    時(shí)間: 2025-3-24 07:17
Digestive Organ Aging and Cancer incidence above 75?years, and diagnosis in the elderly is done at more advanced stages..Management is complex with intensive treatments combining all available anticancer modalities of treatment depending on stage: major surgery, radiotherapy, chemotherapy, radio-chemotherapy, targeted therapy, etc
作者: 歸功于    時(shí)間: 2025-3-24 13:13
https://doi.org/10.1007/978-3-319-57415-8Geriatric Oncology; Cancer; Elderly; Biology of Aging and Cancer; Comorbidity; Treatment Decision Making
作者: 強(qiáng)制令    時(shí)間: 2025-3-24 17:08

作者: 油氈    時(shí)間: 2025-3-24 22:59

作者: 發(fā)酵    時(shí)間: 2025-3-24 23:57

作者: 暴露他抗議    時(shí)間: 2025-3-25 03:29
https://doi.org/10.1007/978-3-322-95856-3rentiated cells. For a broad range of types of stem cells, such as embryonic, muscle, and hematopoietic stem cells, these mechanisms have already been described. Either a complete absence or corrupted activity of checkpoints such as the G1/S damage response and the decatenation checkpoint was found
作者: 我邪惡    時(shí)間: 2025-3-25 08:54
https://doi.org/10.1007/978-3-322-91643-3es. Thus, cellular senescence derives from stress-activated programs, as opposed to other cellular states of irreversible growth arrest, such as post-mitosis and terminal differentiation, which are a consequence of developmental-activated programs. Senescent cells are characterized by the expression
作者: 牛馬之尿    時(shí)間: 2025-3-25 12:54
Wie viel Staat braucht die Bildung?of the immune system may play a major role in waning defense against tumorigenesis. Thus, immunosenescence may contribute to the higher rate of occurrence of tumors in the elderly. However, exactly how these age-related changes in immunity translate to cancer development is not well defined and unde
作者: fructose    時(shí)間: 2025-3-25 17:06

作者: climax    時(shí)間: 2025-3-25 22:50
Peter Vogel,Ellen B?gemann-Gro?heimus macaques). Caloric restriction also improves healthspan, including reducing cancer development in mice and Rhesus macaques. Nonetheless, chronic caloric restriction to prolong life or healthspan, including cancer prevention, is unlikely to be popular or widely adopted for human clinical use. As c
作者: Ejaculate    時(shí)間: 2025-3-26 01:15
Erziehungswissenschaft: Lehre und Studium calpain-calpastatin system (CCS). Activity of the CCS is implicated in the processes of proliferation and apoptosis of many human cell types. We have demonstrated a necessity of resting activity of the CCS for adequate proliferative response of nonmalignant T lymphocytes, as well as the reduction o
作者: 鴕鳥    時(shí)間: 2025-3-26 06:50
P?dagogische Arbeit in Organisationen-cell lymphoma (DLBCL) – increases with age. In addition, its histopathogenic subcategories develop an age-dependent repartition – with less frequent germinal center-derived DLBCL and more activated B cell ones. Finally, some specific entities have been specifically described in an aged population,
作者: fiscal    時(shí)間: 2025-3-26 11:09

作者: Chandelier    時(shí)間: 2025-3-26 16:33
https://doi.org/10.1007/978-3-658-05203-4 extensively studied; cellular senescence, a protective mechanism in response to DNA damage, can induce permanent growth arrest and resistance to apoptosis. Chronological age also favors the accumulation of genetic and epigenetic changes that are important contributing factors in the complex pathoge
作者: 手銬    時(shí)間: 2025-3-26 17:32
Erziehungswissenschaftliche Jugendforschung take place. Although ROS are an inevitable by-products of respiration, they do not necessarily have detrimental effects; low doses of ROS can have beneficial effects on cells, and their production can be finely regulated in mitochondria. Increasing ROS levels and products of the oxidative stress, w
作者: FLAIL    時(shí)間: 2025-3-27 00:41

作者: Obituary    時(shí)間: 2025-3-27 01:33

作者: 無(wú)意    時(shí)間: 2025-3-27 06:30

作者: 羅盤    時(shí)間: 2025-3-27 11:15
Springer Nature Switzerland AG 2020
作者: 骨    時(shí)間: 2025-3-27 16:28

作者: 指數(shù)    時(shí)間: 2025-3-27 18:20

作者: 抵消    時(shí)間: 2025-3-27 23:36
https://doi.org/10.1007/978-3-322-85587-9ry 2016) and the territorial reform that identifies 13 regions in mainland France and five regions in overseas. It also has to take into account not only the therapeutic initial decision but also the patients pathway and their quality of life during and after the cancer treatment. The ability to ada
作者: 脫毛    時(shí)間: 2025-3-28 02:20

作者: GROUP    時(shí)間: 2025-3-28 09:09
https://doi.org/10.1007/978-3-322-95856-3r alterations in them significantly contribute to the transformation process and the development of diseases, such as MDS and leukemia. The following chapter will provide a general overview of cell cycle control, checkpoint activity, and DNA repair mechanisms in hematopoietic and other adult stem ce
作者: conformity    時(shí)間: 2025-3-28 12:59

作者: 摻假    時(shí)間: 2025-3-28 16:31
Wie viel Staat braucht die Bildung?ents obtaining clinical benefit in most trials, and it cannot yet be excluded that age may play a role in the failure of some therapies in some patients. Thus, although there are no reasons to refuse the elderly these treatments, appropriate clinical trials and not just anecdotal evidence are requir
作者: enmesh    時(shí)間: 2025-3-28 19:38
,Sozialp?dagogische Bildungsforschung,growth. The available data on the effects of environmental carcinogens on life span and the aging at different levels of integration are critically analyzed. The exposure to various mutagenic agents, i.e., 5-bromodeoxyuridine, alkylating substances, carcinogenic polycyclic aromatic hydrocarbons, nit
作者: 翅膀拍動(dòng)    時(shí)間: 2025-3-29 01:23
Peter Vogel,Ellen B?gemann-Gro?heimtive dubbed “rapalogues,” are considered too dangerous for chronic human use for longevity extension or cancer prevention. Nonetheless, these data raise the interesting possibility that a pharmacologic intervention to suppress mTOR, or more specifically mTORC1, could prolong lifespan or healthspan i
作者: 惰性氣體    時(shí)間: 2025-3-29 05:14
Rainer Kokemohr,Hans-Christoph Kollerdults with a median age at diagnosis usually over 65?years old. NK cells in AML patients show a reduced expression of several activating receptors that impair NK cell function. Low level of expression of activating receptors such as NKp46 has been correlated with disease progression and patient surv
作者: Psychogenic    時(shí)間: 2025-3-29 10:49

作者: 疏忽    時(shí)間: 2025-3-29 14:05
Empirische Forschung und Waldorfp?dagogikhallenge overwhelms them, that is frailty. Additionally, aging can modify the impact, presentation, and natural history of any illness. This is because the substrate, the aging person, more than the disease pathophysiology, has been modified. In any given person, the system where the reserves are th
作者: Parameter    時(shí)間: 2025-3-29 15:34
Erziehungsziel "Selbstst?ndigkeit"the leading cancer-related cause of death in the world. Most lung cancer in the USA is attributable to smoking. Globally, indoor air pollution is also a significant risk factor. Approximately 85% of lung cancer is non-small cell lung cancer (NSCLC), and adenocarcinoma is the predominant histologic t
作者: defuse    時(shí)間: 2025-3-29 22:06

作者: Stagger    時(shí)間: 2025-3-30 02:51
Population Trends in Aging and Cancernt over time while the percentage of persons dying from cancer decreased with time up to the age of 79?years but increased for those aged 80?years or more, in whom about a third of all cancer deaths occurred in 2012. There was an increase in the number of prevalent cancer cases aged 70?years or olde
作者: 允許    時(shí)間: 2025-3-30 06:22
Integrating Geriatric Oncology in Public Health Planningry 2016) and the territorial reform that identifies 13 regions in mainland France and five regions in overseas. It also has to take into account not only the therapeutic initial decision but also the patients pathway and their quality of life during and after the cancer treatment. The ability to ada
作者: 植物群    時(shí)間: 2025-3-30 10:15
Healthcare Informatics and Technology in Managing the Older Cancer Patient technology will require more senior adults to accept innovative approaches to healthcare. To bridge this digital divide, improvements must be made in terms of usability, data collection, privacy, speed, volume, and cost. With the aid of developing technologies, patient-reported outcomes may also be
作者: 名義上    時(shí)間: 2025-3-30 12:29
Role of Cell Cycle Control, Checkpoints, and DNA Repair Mechanisms in Stem Cells and Changes with Agr alterations in them significantly contribute to the transformation process and the development of diseases, such as MDS and leukemia. The following chapter will provide a general overview of cell cycle control, checkpoint activity, and DNA repair mechanisms in hematopoietic and other adult stem ce
作者: 詞匯表    時(shí)間: 2025-3-30 17:37

作者: Incompetent    時(shí)間: 2025-3-31 00:09

作者: epinephrine    時(shí)間: 2025-3-31 03:52

作者: pulse-pressure    時(shí)間: 2025-3-31 05:08
Chronic Mechanistic Target of Rapamycin Inhibition: Preventing Cancer to Delay Aging or Vice Versa?tive dubbed “rapalogues,” are considered too dangerous for chronic human use for longevity extension or cancer prevention. Nonetheless, these data raise the interesting possibility that a pharmacologic intervention to suppress mTOR, or more specifically mTORC1, could prolong lifespan or healthspan i
作者: 使隔離    時(shí)間: 2025-3-31 13:11
Aging of Natural Killer Cells in Acute Myeloid Leukemiadults with a median age at diagnosis usually over 65?years old. NK cells in AML patients show a reduced expression of several activating receptors that impair NK cell function. Low level of expression of activating receptors such as NKp46 has been correlated with disease progression and patient surv
作者: 他一致    時(shí)間: 2025-3-31 15:31
Hematopoietic Stem Cell Aging and Malignant Hemopathiesation. However, at present our understanding of the process of aging is far from complete, and many open questions are currently under investigation. This chapter will focus on the complex multistep interplay between aging and the higher incidence of malignant hemopathies.
作者: excrete    時(shí)間: 2025-3-31 17:31
Normal and Abnormal Aging (General Perspective for the Oncologist)hallenge overwhelms them, that is frailty. Additionally, aging can modify the impact, presentation, and natural history of any illness. This is because the substrate, the aging person, more than the disease pathophysiology, has been modified. In any given person, the system where the reserves are th
作者: artless    時(shí)間: 2025-3-31 21:41
Respiratory Organ Aging and Cancerthe leading cancer-related cause of death in the world. Most lung cancer in the USA is attributable to smoking. Globally, indoor air pollution is also a significant risk factor. Approximately 85% of lung cancer is non-small cell lung cancer (NSCLC), and adenocarcinoma is the predominant histologic t
作者: sleep-spindles    時(shí)間: 2025-4-1 04:42





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