標(biāo)題: Titlebook: Genomic Instability and Cancer Metastasis; Mechanisms, Emerging Chris Maxwell,Cal Roskelley Book 2015 Springer International Publishing Swi [打印本頁] 作者: CLIP 時(shí)間: 2025-3-21 18:38
書目名稱Genomic Instability and Cancer Metastasis影響因子(影響力)
書目名稱Genomic Instability and Cancer Metastasis影響因子(影響力)學(xué)科排名
書目名稱Genomic Instability and Cancer Metastasis網(wǎng)絡(luò)公開度
書目名稱Genomic Instability and Cancer Metastasis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Genomic Instability and Cancer Metastasis被引頻次
書目名稱Genomic Instability and Cancer Metastasis被引頻次學(xué)科排名
書目名稱Genomic Instability and Cancer Metastasis年度引用
書目名稱Genomic Instability and Cancer Metastasis年度引用學(xué)科排名
書目名稱Genomic Instability and Cancer Metastasis讀者反饋
書目名稱Genomic Instability and Cancer Metastasis讀者反饋學(xué)科排名
作者: 季雨 時(shí)間: 2025-3-21 22:35 作者: 反省 時(shí)間: 2025-3-22 04:15 作者: 織物 時(shí)間: 2025-3-22 06:28
Karl-Heinz Huhnstock,Werner Kutschaage, the DNA double strand break (DSB), and how it relates to cancer. DSB sensor proteins in the nucleus detect DNA breaks within minutes following damage. These proteins are now routinely labeled by immunocytochemistry, and access to high throughput fluorescence microscopy and robotics open the doo作者: 肉身 時(shí)間: 2025-3-22 10:46
Knochen- und Gelenkkrankheiten,e metastatic cascade are made up of interactions between metastatic tumour cells, the solid tumour microenvironment, host normal cells, and host tissue. While our understanding of tumour cell migration and invasion has been greatly improved using a variety of . cell culture systems, the inherent com作者: 散布 時(shí)間: 2025-3-22 13:54 作者: 散布 時(shí)間: 2025-3-22 18:32 作者: Medicare 時(shí)間: 2025-3-23 00:46 作者: 淘氣 時(shí)間: 2025-3-23 04:24 作者: GLIB 時(shí)間: 2025-3-23 07:54 作者: Mets552 時(shí)間: 2025-3-23 12:45
https://doi.org/10.1007/978-3-319-12136-9Cancer Metastasis; DNA damage response; Genomic instability; Therapeutic approaches; Tumor heterogeneity作者: amenity 時(shí)間: 2025-3-23 14:47 作者: 違法事實(shí) 時(shí)間: 2025-3-23 19:17
Genomic Instability and Cancer Metastasis978-3-319-12136-9Series ISSN 1568-2102 Series E-ISSN 2215-1648 作者: DOLT 時(shí)間: 2025-3-24 02:02
Chris Maxwell,Cal RoskelleyConnects cancer metastasis with genomic instability in a comprehensive manner.Outlines the fundamental mechanisms responsible for these cellular and tissue phenotypes.Reviews emerging themes and highl作者: 煩躁的女人 時(shí)間: 2025-3-24 04:50
Gonorrhoische Erkrankungen der Haut,efect in DNA repair, and secondary cancers developed following treatment with chemotherapeutic drugs that damage the DNA. We will focus on four major repair pathways with particular attention to the exploitation of recent knowledge to improve cancer therapy.作者: Polydipsia 時(shí)間: 2025-3-24 08:38 作者: opprobrious 時(shí)間: 2025-3-24 14:38 作者: 羊欄 時(shí)間: 2025-3-24 18:34 作者: gustation 時(shí)間: 2025-3-24 21:16
Microenvironmental Control of Metastatic Progression,ental alterations can have profound effects on the genome and epigenome of tumor cells as well as their signal transduction pathways, both biochemical and mechanical. All of these effects contribute to the invasion and progression of the metastatic tumor organ.作者: 腐敗 時(shí)間: 2025-3-25 02:51 作者: insipid 時(shí)間: 2025-3-25 05:13
Knochen- und Gelenkkrankheiten,cal forces or defects in mechanosensors can result in misregulation of signaling pathways inside the cell, and ultimately lead to malignancy. Here, we discuss the ways in which physical attributes of the tumor microenvironment can promote metastasis and genomic instability, two hallmark features of cancer.作者: Flinch 時(shí)間: 2025-3-25 08:21 作者: 帶來墨水 時(shí)間: 2025-3-25 12:37 作者: Conjuction 時(shí)間: 2025-3-25 16:36
1568-2102 ular and tissue phenotypes.Reviews emerging themes and highlMetastasis is the primary cause of mortality associated with cancer, and tumor genomic heterogeneity is a likely source for the cells that support cancer progression, resistance to therapy, and disease relapse. This book connects cancer met作者: perpetual 時(shí)間: 2025-3-25 20:57
Cancer Metastasis: Tracking and Attacking a Moving Target,ion and the challenge of aiming clinical therapies at a seemingly unpredictable moving target. From an evolutionary perspective, metastasis can be considered as a process during which novel cell populations are generated that can exploit the unique tissue environments they encounter at a secondary t作者: 裝勇敢地做 時(shí)間: 2025-3-26 00:42 作者: Morphine 時(shí)間: 2025-3-26 05:58 作者: 裝飾 時(shí)間: 2025-3-26 10:23
Mathematical Modeling for DNA Repair, Carcinogenesis and Cancer Detection,age, the DNA double strand break (DSB), and how it relates to cancer. DSB sensor proteins in the nucleus detect DNA breaks within minutes following damage. These proteins are now routinely labeled by immunocytochemistry, and access to high throughput fluorescence microscopy and robotics open the doo作者: 不如樂死去 時(shí)間: 2025-3-26 14:11
Animal Models of Metastasis,e metastatic cascade are made up of interactions between metastatic tumour cells, the solid tumour microenvironment, host normal cells, and host tissue. While our understanding of tumour cell migration and invasion has been greatly improved using a variety of . cell culture systems, the inherent com作者: Inculcate 時(shí)間: 2025-3-26 17:55
Microenvironmental Control of Metastatic Progression,dent and some newly recruited, that are often activated due to factors released by the tumor cells themselves. These activated cells then release soluble factors that feed forward on the tumour cells in a symbiotic manner. Activated cells also alter the deposition and processing of extracellular mat作者: 贊成你 時(shí)間: 2025-3-26 22:29 作者: 現(xiàn)暈光 時(shí)間: 2025-3-27 03:31 作者: 謙虛的人 時(shí)間: 2025-3-27 08:18 作者: Axon895 時(shí)間: 2025-3-27 13:04
,Nanomedicine—Nanoparticles in Cancer Imaging and Therapy,nanotechnology is a quickly evolving field of interdisciplinary research that involves the biomedical application of nanoparticles, which are nanoscale devices that are able to overcome biological barriers, specifically recognize a single type of cancer cell, and accumulate preferentially in tumors.作者: 大氣層 時(shí)間: 2025-3-27 17:19 作者: intercede 時(shí)間: 2025-3-27 21:32
The Generation, Detection, and Prevention of Genomic Instability During Cancer Progression and Metaffers selective advantages to the tumor, the tumor-specific loss of these pathways may provide therapeutic opportunities, which could be personalized through knowledge of the specific types of genomic instability that characterize an individual’s tumor.作者: albuminuria 時(shí)間: 2025-3-28 00:15 作者: disparage 時(shí)間: 2025-3-28 02:13
Animal Models of Metastasis,to study mechanisms underlying metastatic tumour cell dissemination. We then discuss selected genetically engineered mouse models of metastatic cancer, which are powerful tools to elucidate the oncogenic potential of genes of interest. Murine tumour xenografts are also considered as the traditional 作者: 殺蟲劑 時(shí)間: 2025-3-28 09:52 作者: 流眼淚 時(shí)間: 2025-3-28 11:01
,Nanomedicine—Nanoparticles in Cancer Imaging and Therapy,rugs can be integrated into their design for more efficient imaging and treatment of the tumor with fewer side effects. Recent advances in nanomedicine raise exciting possibilities for future nanoparticle applications in personalized cancer therapy.作者: 排斥 時(shí)間: 2025-3-28 14:50 作者: 聚集 時(shí)間: 2025-3-28 21:09
,Otologische R?ntgendiagnostik,tumor. These drivers of evolution can generate successful metastatic cells that either survive as dormant cells, or thrive as secondary tumors during the time course of the disease. The metastatic target is thus dynamic, requiring a dynamic approach to treatment. Here we will discuss the growing inf作者: Calibrate 時(shí)間: 2025-3-29 00:02
Therapie der depressiven Zustandsbilder,ffers selective advantages to the tumor, the tumor-specific loss of these pathways may provide therapeutic opportunities, which could be personalized through knowledge of the specific types of genomic instability that characterize an individual’s tumor.作者: 征服 時(shí)間: 2025-3-29 05:29
Karl-Heinz Huhnstock,Werner Kutschafluencing these levels. We emphasize ionizing radiation, which is the principal environmental factor that increases DSB levels. Mathematical models associating a mutation probability for each DSB have been used to explain the dose dependence of cancer incidence observed after exposure to high doses 作者: HEDGE 時(shí)間: 2025-3-29 08:06 作者: peritonitis 時(shí)間: 2025-3-29 12:03
https://doi.org/10.1007/978-3-663-14645-2ition, these approaches are highly specific and restricted to tumor cells, and are expected to decrease side effects associated with current strategies. Both synthetic genetic approaches have been applied extensively in pre-clinical studies and numerous candidate drug targets have been identified, i作者: 配偶 時(shí)間: 2025-3-29 17:43 作者: grudging 時(shí)間: 2025-3-29 23:41
1568-2102 s of medicine, oncology, and cancer biology as well as for the content expert searching for thorough reviews of current knowledge in these areas.978-3-319-35295-4978-3-319-12136-9Series ISSN 1568-2102 Series E-ISSN 2215-1648
