派博傳思國際中心

標(biāo)題: Titlebook: Genetic Predisposition to Cancer; Rosalind A. Eeles (Senior Lecturer and Honorary Co Book 1996 Springer Science+Business Media Dordrecht 1 [打印本頁]

作者: 去是公開    時(shí)間: 2025-3-21 19:33
書目名稱Genetic Predisposition to Cancer影響因子(影響力)




書目名稱Genetic Predisposition to Cancer影響因子(影響力)學(xué)科排名




書目名稱Genetic Predisposition to Cancer網(wǎng)絡(luò)公開度




書目名稱Genetic Predisposition to Cancer網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Genetic Predisposition to Cancer被引頻次




書目名稱Genetic Predisposition to Cancer被引頻次學(xué)科排名




書目名稱Genetic Predisposition to Cancer年度引用




書目名稱Genetic Predisposition to Cancer年度引用學(xué)科排名




書目名稱Genetic Predisposition to Cancer讀者反饋




書目名稱Genetic Predisposition to Cancer讀者反饋學(xué)科排名





作者: sclera    時(shí)間: 2025-3-21 21:44
Das Wachstum industrieller Unternehmenagnosis; congenital mesoblastic nephroma is most common in the first 3 months of life, followed by a predominance of Wilms tumour (nephroblastoma) in early childhood which is overtaken by renal cell carcinoma in adult life. Kidney cancer is extremely rare between the ages of 7 and 40 years (see Figure 7.1).
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作者: cochlea    時(shí)間: 2025-3-22 12:09

作者: 閃光你我    時(shí)間: 2025-3-22 12:55
Malignant disease and variations in radiosensitivity in ataxia telangiectasia patientspisodes of severe infection. In addition, patients show thymic hypoplasia, hypogonadism, a high level of serum alphafetoprotein, growth retardation and an abnormality of blood vessels (telangiectasia) [3]. The . gene also confers an increased radiosensitivity which can be observed both in patients and in cultured cells from patients [4, 5].
作者: 閃光你我    時(shí)間: 2025-3-22 19:52

作者: colostrum    時(shí)間: 2025-3-22 22:59
Walther G. Hoffmann (O. Professor) called acoustic neuromas), meningiomas, schwannomas and spinal ependymomas. Although the majority of these tumours are not malignant, curative surgery is often difficult and NF2 is associated with significant disease-related morbidity and mortality.
作者: 我邪惡    時(shí)間: 2025-3-23 04:13

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作者: 手術(shù)刀    時(shí)間: 2025-3-23 21:59

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作者: 噴出    時(shí)間: 2025-3-24 14:21
https://doi.org/10.1007/978-1-4899-4501-3cancer; genetics; human; internal medicine; oncology; Redis
作者: Inexorable    時(shí)間: 2025-3-24 16:43

作者: 炸壞    時(shí)間: 2025-3-24 19:29
Overview: 978-0-412-56580-9978-1-4899-4501-3
作者: 惡意    時(shí)間: 2025-3-25 00:21

作者: abduction    時(shí)間: 2025-3-25 07:19

作者: 詞根詞綴法    時(shí)間: 2025-3-25 09:30
Wahl wirtschaftlicher Schnittbedingungen,ses it has been shown that loss of function of both alleles of these genes is required for tumour initiation. Because at least one functional copy of the gene product is required to prevent tumour initiation these genes have collectively been called ‘tumour suppressor genes’. Since their normal func
作者: 使饑餓    時(shí)間: 2025-3-25 14:33
Walther G. Hoffmann (O. Professor)sult in cancer development in the majority of affected individuals. In NF1, the tumours which develop in the majority of patients are benign cutaneous neurofibromas. People with .1 have a small but significant risk of specific cancers which include peripheral nerve sarcomas, rhabdomyosarcomas, atypi
作者: 宿醉    時(shí)間: 2025-3-25 16:06
Das Wachstum industrieller Unternehmen important aetiological factor thus far identified in this group of tumours. The histological type of tumour depends very much on the age at tumour diagnosis; congenital mesoblastic nephroma is most common in the first 3 months of life, followed by a predominance of Wilms tumour (nephroblastoma) in
作者: Baffle    時(shí)間: 2025-3-25 21:38
Das Wachstum von Verwaltungsorganisationenrt of a survey of nearly 650 children with rhabdomyosarcoma in the United States, Li and Fraumeni identified three pairs of affected siblings when less than one would have been expected by chance. In addition one pair of affected cousins was identified. Li and Fraumeni obtained information by interv
作者: 銼屑    時(shí)間: 2025-3-26 02:24
https://doi.org/10.1007/978-3-663-08341-2 be a Mendelian recessive disorder there is some evidence that this may not always be the case [2]. The major neurological features include progressive cerebellar ataxia presenting in infancy, oculomotor dyspraxia and dysarthria. Immunodeficiency is an important feature of this disorder although it
作者: gene-therapy    時(shí)間: 2025-3-26 07:08
https://doi.org/10.1007/978-3-642-91733-2ion to leukaemia. A comprehensive review of clinical and scientific aspects of FA has been published [1], and several shorter and more recent reviews on clinical and molecular findings are available [2–5]. This review will focus on cancer predisposition in FA, and on progress in identifying the Fanc
作者: Herpetologist    時(shí)間: 2025-3-26 09:27
https://doi.org/10.1007/978-3-7091-8204-8recognized features. The most frequent and important components of the syndrome are nevoid basal cell carcinomas and odontogenic keratocysts. Non-progressive skeletal anomalies are present in a high proportion of cases and are helpful diagnostically, as is the presence of ectopic calcification. Cong
作者: Malleable    時(shí)間: 2025-3-26 14:44
Allgemeine und spezielle Semantik,generated an intellectually satisfying and simple association between defects in DNA repair, increased mutability and cancer proneness. As the study of XP patients has proceeded, however, interesting anomalies and unanticipated complexities have been uncovered. In particular, as a consequence of usi
作者: abstemious    時(shí)間: 2025-3-26 19:57

作者: 土產(chǎn)    時(shí)間: 2025-3-27 00:00
,Die Entstehung des Warenhauses (1860–1880),e attempted to quantify the risks of breast cancer associated with a positive family history. Attempts have also been made to examine whether the pattern of related individuals with breast cancer are consistent with the effects of a single gene of large effect, shared environmental effects, many gen
作者: atrophy    時(shí)間: 2025-3-27 03:30
https://doi.org/10.1007/978-3-642-91200-9e of a high risk gene. Over the past 5 years a number of the genes responsible for inherited predisposition to breast and/or ovarian cancer have been identified or localized. In particular, .1 [1], .2 [2], the ataxia-telangiectasia gene [3,4], the .53 gene [5,6] and the androgen receptor gene [7] ar
作者: nutrition    時(shí)間: 2025-3-27 07:55

作者: 大都市    時(shí)間: 2025-3-27 11:54
https://doi.org/10.1007/978-3-642-92212-1The multiple endocrine neoplasia (MEN) syndromes comprise dominantly inherited predisposition to tumours of specific endocrine glands. Two distinct syndromes are recognized:
作者: penance    時(shí)間: 2025-3-27 16:27
Genetic predisposition to cancer: an introductionEvidence that inherited susceptibility plays a role in the risk of malignancy comes from three separate sources. These observations are that:
作者: gruelling    時(shí)間: 2025-3-27 18:24
Multiple endocrine neoplasiaThe multiple endocrine neoplasia (MEN) syndromes comprise dominantly inherited predisposition to tumours of specific endocrine glands. Two distinct syndromes are recognized:
作者: BILE    時(shí)間: 2025-3-27 23:50
From families to chromosomes: genetic linkage, and other methods for finding cancer-predisposition gd since then genes for all the major ‘inherited cancer syndromes’ (that is, those rare syndromes where evidence for Mendelian inheritance was apparent from clinical studies) have now been either identified or at least mapped precisely within the human genome (see Chapter 1). Genetic linkage analysis
作者: 客觀    時(shí)間: 2025-3-28 05:25
The Li-Fraumeni syndrome and the role of ,53 mutations in predisposition to cancer were due to inherited predisposition to the observed cancers [1]. In a more detailed report, a second pair of cousins with childhood soft tissue sarcoma were identified, and the finding of adrenocortical carcinoma and brain tumours in first-degree relatives of children with soft tissue sarcoma sugg
作者: 陰郁    時(shí)間: 2025-3-28 06:22
Xeroderma pigmentosum, Cockayne syndrome and trichothiodystrophy: sun sensitivity, DNA repair defectiduals with XP alone, XP with CS, or TTD alone. In order to understand the relationship between DNA damage/repair and cancer revealed in XP, it is necessary to study all three conditions at the clinical, cellular and molecular levels.
作者: 令人作嘔    時(shí)間: 2025-3-28 12:12

作者: 松果    時(shí)間: 2025-3-28 17:59
Familial breast canceren extensively studied, it is the subject of a separate chapter (Chapter 15) in this book. However, the .1 gene accounts for, at most, half (and probably less) of all familial breast cancer. In this chapter, we will begin by examining the epidemiological evidence for familial risks in breast cancer,
作者: 冰河期    時(shí)間: 2025-3-28 20:20

作者: 前奏曲    時(shí)間: 2025-3-29 00:24

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作者: 橢圓    時(shí)間: 2025-3-29 12:04
,Die Entstehung des Warenhauses (1860–1880),en extensively studied, it is the subject of a separate chapter (Chapter 15) in this book. However, the .1 gene accounts for, at most, half (and probably less) of all familial breast cancer. In this chapter, we will begin by examining the epidemiological evidence for familial risks in breast cancer,
作者: Chandelier    時(shí)間: 2025-3-29 19:26
Rosalind A. Eeles (Senior Lecturer and Honorary Co
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作者: Concrete    時(shí)間: 2025-3-30 07:19
Retinoblastoma: the paradigm for a genetically inherited cancer syndromeses it has been shown that loss of function of both alleles of these genes is required for tumour initiation. Because at least one functional copy of the gene product is required to prevent tumour initiation these genes have collectively been called ‘tumour suppressor genes’. Since their normal func
作者: Filibuster    時(shí)間: 2025-3-30 08:39
Neurofibromatosis (NF) types 1 and 2sult in cancer development in the majority of affected individuals. In NF1, the tumours which develop in the majority of patients are benign cutaneous neurofibromas. People with .1 have a small but significant risk of specific cancers which include peripheral nerve sarcomas, rhabdomyosarcomas, atypi
作者: gonioscopy    時(shí)間: 2025-3-30 16:21





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