標(biāo)題: Titlebook: Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2; Barry D. Nelkin Book 1996 Springer-Verlag Berlin Heidelberg 1996 biology.cell.g [打印本頁(yè)] 作者: obesity 時(shí)間: 2025-3-21 19:49
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2影響因子(影響力)
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2影響因子(影響力)學(xué)科排名
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2被引頻次
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2被引頻次學(xué)科排名
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2年度引用
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2年度引用學(xué)科排名
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2讀者反饋
書(shū)目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2讀者反饋學(xué)科排名
作者: WAIL 時(shí)間: 2025-3-21 23:56
Mutations in Ret in MEN 2,e. This genetic information coupled with a basic understanding of the ways in which the different types of mutation may affect the activity of the ret protein, has already provided interesting insights into the biology of receptor tyrosine kinases. A more detailed elucidation of the genotype: phenot作者: vitreous-humor 時(shí)間: 2025-3-22 03:02 作者: 大方一點(diǎn) 時(shí)間: 2025-3-22 07:21 作者: 切掉 時(shí)間: 2025-3-22 10:13
Origin of Adrenal Chromaffin Cells from the Neural Crest,istological techniques based on catecholamine biochemistry allowed early researchers to identify the precursors for adrenal chromaffin cells as they migrate from the primary sympathetic chains into the adrenal primordia. Subsequent production of antisera that recognize catecholamine-synthesizing enz作者: Liability 時(shí)間: 2025-3-22 14:31 作者: Liability 時(shí)間: 2025-3-22 19:00
Neuronal Properties of Thyroid C-Cell Tumor Lines,ntrast to the primarily endocrine nature of the parental C-cells. What can account for this transdifferentiation? In this chapter, we will try to answer that question by comparing the neuronal features of C-cell lines with those of normal C-cells treated with nerve growth factor. From that perspecti作者: Arthropathy 時(shí)間: 2025-3-22 23:51 作者: 颶風(fēng) 時(shí)間: 2025-3-23 03:14
Conclusion,w therapeutic targets for MTC, pheochromocytoma and biologically related cancers. Here, I would like to point out some of the questions which have been raised in the text, and which seem to me to be of special interest for future study. Of course, these choices reflect my bias; and others might emph作者: 不自然 時(shí)間: 2025-3-23 07:38
https://doi.org/10.1007/978-3-322-93658-5s has been reviewed extensively by Foulds.. Based on observations of karyotypic progression in hematopoietic and other neoplasms, Nowell. proposed a clonal evolution model for tumor progression. In this model, variant cell clones which possess some growth advantage overgrow the other cells in the tumor.作者: 胰臟 時(shí)間: 2025-3-23 10:46
,Der gro?e Wurf — die Grundsatzentscheidung,w therapeutic targets for MTC, pheochromocytoma and biologically related cancers. Here, I would like to point out some of the questions which have been raised in the text, and which seem to me to be of special interest for future study. Of course, these choices reflect my bias; and others might emphasize alternate lines of investigation.作者: 消滅 時(shí)間: 2025-3-23 17:11 作者: 就職 時(shí)間: 2025-3-23 19:46
Conclusion,w therapeutic targets for MTC, pheochromocytoma and biologically related cancers. Here, I would like to point out some of the questions which have been raised in the text, and which seem to me to be of special interest for future study. Of course, these choices reflect my bias; and others might emphasize alternate lines of investigation.作者: irritation 時(shí)間: 2025-3-24 02:08
https://doi.org/10.1007/978-3-662-21948-5biology; cell; gene; gene expression; genetics; molecular biology; mutation作者: 的事物 時(shí)間: 2025-3-24 03:28
978-3-662-21950-8Springer-Verlag Berlin Heidelberg 1996作者: 惰性氣體 時(shí)間: 2025-3-24 09:49
Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2978-3-662-21948-5Series ISSN 1080-3645 作者: 興奮過(guò)度 時(shí)間: 2025-3-24 11:43
Die Grundlagen der Tarifbildungelopmental and biochemical features, and their unique clinical associations. Elucidation of the genetic basis of the MEN 2 syndromes has spawned substantial improvements in diagnosis which in turn have led to more prompt and appropriate treatment of the associated endocrine tumors. In the foreseeabl作者: 輕推 時(shí)間: 2025-3-24 16:55 作者: Chameleon 時(shí)間: 2025-3-24 19:22 作者: grieve 時(shí)間: 2025-3-25 00:37 作者: NUDGE 時(shí)間: 2025-3-25 06:59
https://doi.org/10.1007/978-3-322-81441-8istological techniques based on catecholamine biochemistry allowed early researchers to identify the precursors for adrenal chromaffin cells as they migrate from the primary sympathetic chains into the adrenal primordia. Subsequent production of antisera that recognize catecholamine-synthesizing enz作者: gene-therapy 時(shí)間: 2025-3-25 10:36
,Rechtsgrundlagen der Korruptionspr?vention, medulla and thyroid C-cells. The 1961 report by Sipple,. then a surgery resident, of the more than coincidental association between pheochromocytoma and thyroid carcinoma focused attention on the entity now known as MEN 2A. Subsequent studies by Williams. established that the thyroid carcinomas wer作者: MULTI 時(shí)間: 2025-3-25 12:38
https://doi.org/10.1007/978-3-642-94182-5ntrast to the primarily endocrine nature of the parental C-cells. What can account for this transdifferentiation? In this chapter, we will try to answer that question by comparing the neuronal features of C-cell lines with those of normal C-cells treated with nerve growth factor. From that perspecti作者: Daily-Value 時(shí)間: 2025-3-25 15:48
https://doi.org/10.1007/978-3-322-93658-5s has been reviewed extensively by Foulds.. Based on observations of karyotypic progression in hematopoietic and other neoplasms, Nowell. proposed a clonal evolution model for tumor progression. In this model, variant cell clones which possess some growth advantage overgrow the other cells in the tu作者: 陪審團(tuán) 時(shí)間: 2025-3-25 21:49
,Der gro?e Wurf — die Grundsatzentscheidung,w therapeutic targets for MTC, pheochromocytoma and biologically related cancers. Here, I would like to point out some of the questions which have been raised in the text, and which seem to me to be of special interest for future study. Of course, these choices reflect my bias; and others might emph作者: Trigger-Point 時(shí)間: 2025-3-26 01:18 作者: 粗野 時(shí)間: 2025-3-26 05:46
Mutations in Ret in MEN 2, protein, has already provided interesting insights into the biology of receptor tyrosine kinases. A more detailed elucidation of the genotype: phenotype correlations in terms of different signaling pathways in different cell types provides a challenge for the future.作者: Corroborate 時(shí)間: 2025-3-26 08:44 作者: periodontitis 時(shí)間: 2025-3-26 15:28
1080-3645 ne expressions and signal transduction are placed in the context of their effects on the biology of the thyroid and adrenal cells which generate cancers in MEN2. Thus, this volume addresses the ret mutations, the effects these mutations may have on signal transduction, and developmental and neurobio作者: Altitude 時(shí)間: 2025-3-26 18:04 作者: institute 時(shí)間: 2025-3-26 21:31
Einleitung: Eine Zusammenfassung,the RTK gene superfamily were initially isolated as oncogenes that arose from mutations deregulating their kinase activity. Ret, a member of the RTK family, was first isolated as a transforming gene created by a recombination with the . gene during transfection of a T cell lymphoma DNA.作者: DENT 時(shí)間: 2025-3-27 03:20
Intracellular Signaling by the ret Tyrosine Kinase,the RTK gene superfamily were initially isolated as oncogenes that arose from mutations deregulating their kinase activity. Ret, a member of the RTK family, was first isolated as a transforming gene created by a recombination with the . gene during transfection of a T cell lymphoma DNA.作者: 創(chuàng)作 時(shí)間: 2025-3-27 08:45
Die Grundlagen der Tarifbildunge future, greater understanding of the molecular pathogenesis of these tumors is likely to allow a more rational targeting of anti-tumor therapy. This introductory chapter reviews the natural history and summarizes current approaches to diagnosis and management of patients with the MEN 2 syndromes.作者: Functional 時(shí)間: 2025-3-27 12:33 作者: 歡樂(lè)中國(guó) 時(shí)間: 2025-3-27 17:24
Clinical Manifestations of Multiple Endocrine Neoplasia Type 2,e future, greater understanding of the molecular pathogenesis of these tumors is likely to allow a more rational targeting of anti-tumor therapy. This introductory chapter reviews the natural history and summarizes current approaches to diagnosis and management of patients with the MEN 2 syndromes.作者: Accrue 時(shí)間: 2025-3-27 17:45 作者: Insubordinate 時(shí)間: 2025-3-27 22:51
Book 1996pects of the cells affected of MEN2, and provides the basic researcher - in genetics, signal transduction, cell transformation, developmental or neurobiology - with a better understanding of how the knowledge generated by each of these fields intercalates within the overall biology of MEN2.作者: ESO 時(shí)間: 2025-3-28 02:20
Molecular Events in the Development and Progression of Medullary Thyroid Cancer and PheochromocytomAlterations in several classes of genes can contribute to the development and progression of tumors. These abnormalities can be classified into three general groups: dominant oncogenes, tumor suppressor genes, and genetic instability genes. This chapter reviews what is known about abnormalities in these classes of genes in MTC and pheochromocytoma.作者: 致詞 時(shí)間: 2025-3-28 09:55 作者: 弄皺 時(shí)間: 2025-3-28 13:13
Mechanisms of Growth Factor-Mediated Signal Transduction in PC12 Cells,s dependency confounds the design of experiments to explore the mechanism of the growth factors actions. Moreover, the limited amount of material available for study and the absence of appropriate control populations of cells has impeded the progress in elucidating the cellular mechanisms responsibl作者: 盟軍 時(shí)間: 2025-3-28 15:10 作者: Vasodilation 時(shí)間: 2025-3-28 20:51
Neuronal Properties of Thyroid C-Cell Tumor Lines,tility of thyroid C-cell lines for identification of differentiation and growth genes will be exemplified by description of a novel splice product of the G.α transcript that is selectively expressed in a subset of neuroendocrine and neuronal cells. Finally, the applicability of C-cell lines for stud作者: incisive 時(shí)間: 2025-3-29 01:55 作者: 沉積物 時(shí)間: 2025-3-29 05:13
https://doi.org/10.1007/978-3-322-81441-8enal chromaffin cells in avian and mammalian embryos. I will then review the expression of catecholamine-synthesizing enzymes, neuronal markers and transcription factors in the sympathoadrenal lineage, and relate these patterns of differentiation to environmental cues encountered by pheochromocytes.作者: 護(hù)身符 時(shí)間: 2025-3-29 07:30
https://doi.org/10.1007/978-3-642-94182-5tility of thyroid C-cell lines for identification of differentiation and growth genes will be exemplified by description of a novel splice product of the G.α transcript that is selectively expressed in a subset of neuroendocrine and neuronal cells. Finally, the applicability of C-cell lines for stud
