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標(biāo)題: Titlebook: Genetic Instability and Tumorigenesis; Michael B. Kastan Book 1997 The Editor(s) (if applicable) and The Author(s), under exclusive licens [打印本頁]

作者: panache    時間: 2025-3-21 19:37
書目名稱Genetic Instability and Tumorigenesis影響因子(影響力)




書目名稱Genetic Instability and Tumorigenesis影響因子(影響力)學(xué)科排名




書目名稱Genetic Instability and Tumorigenesis網(wǎng)絡(luò)公開度




書目名稱Genetic Instability and Tumorigenesis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Genetic Instability and Tumorigenesis被引頻次




書目名稱Genetic Instability and Tumorigenesis被引頻次學(xué)科排名




書目名稱Genetic Instability and Tumorigenesis年度引用




書目名稱Genetic Instability and Tumorigenesis年度引用學(xué)科排名




書目名稱Genetic Instability and Tumorigenesis讀者反饋




書目名稱Genetic Instability and Tumorigenesis讀者反饋學(xué)科排名





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0070-217X c cells which con- tribute to the transformed phenotype arise from DNA damage caused by such exposures. Therefore, understanding how cells respond to DNA-damagi978-3-642-64434-4978-3-642-60505-5Series ISSN 0070-217X Series E-ISSN 2196-9965
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Pathogenese des postthrombotischen Syndroms,w will focus on recent advances in cell cycle control and genetic instability in the budding yeast ., with emphasis on experimental topics in which loss of cell cycle progression control decreases the fidelity of chromosome transmission to daughter cells. Viable single gene mutations that perturb bo
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Genomic Instability and Its Role in Neoplasia,ade surrounding tissues, enter the vasculature, extravasate, and colonize a secondary site. Proficiency at each step is necessary for a tumor cell to become fully metastatic. Genetic alterations are the basis for this acquired variation (. 1987; . et al. 1987). The emergence of drug-resistant or rad
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https://doi.org/10.1007/978-3-531-90750-5 diseases, we think about inheriting one or two abnormal genes from our parents and these gene abnormalities confer the disease phenotype. In contrast, in the majority of cancers, no such inherited gene abnormalities can be identified (which does not mean that they do not exist) and there is no obvi
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Utho Creusen,Gordon Müller-Seitzon. New concepts make us realize the magnitude of the challenge of duplicating and distributing an accurate copy of the genome each time a cell divides. For each of the 10. somatic cell divisions that occur in a human lifetime, each copy of the genome must contain an intact sequence of some 3 billio
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https://doi.org/10.1007/978-3-662-26487-4cs, genetic epidemiology, tumor virology, chemical carcinogenesis, and others, have converged over the last few decades to provide many fundamental insights into the genetic basis of cancer. These studies have shown that the genesis of malignant tumors is dependent on the accumulation of a series of
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978-3-642-64434-4The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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Genomic Stability and Instability: A Working Paradigm,on. New concepts make us realize the magnitude of the challenge of duplicating and distributing an accurate copy of the genome each time a cell divides. For each of the 10. somatic cell divisions that occur in a human lifetime, each copy of the genome must contain an intact sequence of some 3 billio
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Genomic Instability and Its Role in Neoplasia,s to therapy. Genomic instability has been hypothesized to be a driving force behind multistep carcinogenesis (. 1976). A number of genetic changes are required for a normal cell to become tumorigenic (. 1959; . and . 1990). If genomic instability increases the rate at which these alterations occur,
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Role of DNA Excision Repair Gene Defects in the Etiology of Cancer, of human cancers. Mutations in genes important for normal cellular functions and growth properties, including many proto-oncogenes and tumor suppressor genes, may result from spontaneous or environmentally induced alterations in DNA and contribute directly to the multistage process leading to malig
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Utho Creusen,Gordon Müller-Seitzn nucleotide base pairs, and each daughter cell must receive the correct number of the 46 chromosomes into which the instruction book of life is parceled. The challenging task of accurate DNA replication and distribution must be accomplished while the cell is under constant attack by forces that damage DNA.
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Book 1997k about genetic diseases, we think about inheriting one or two abnormal genes from our parents and these gene abnormalities confer the disease phenotype. In contrast, in the majority of cancers, no such inherited gene abnormalities can be identified (which does not mean that they do not exist) and t
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Auction Design with a Revenue Targetns for many buyers, again in the context of a revenue target. For ., we provide a FPTAS to compute the optimal posted prices for a given sequence of buyers. For ., we apply the results of?[.] on sparse covers of distributions to obtain a PTAS in a setting where the seller has a constraint on discrim
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F. Bo?njakovi?ssential resource for students and teachers in philosophy of science. It is also ideal for anyone interested in the philosophical influences on contemporary science...?.978-3-031-56048-4978-3-031-56049-1Series ISSN 2947-9339 Series E-ISSN 2947-9347
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