標(biāo)題: Titlebook: Genetic Approaches to Microbial Pathogenicity; W. Goebel Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive licens [打印本頁(yè)] 作者: STRI 時(shí)間: 2025-3-21 18:56
書目名稱Genetic Approaches to Microbial Pathogenicity影響因子(影響力)
書目名稱Genetic Approaches to Microbial Pathogenicity影響因子(影響力)學(xué)科排名
書目名稱Genetic Approaches to Microbial Pathogenicity網(wǎng)絡(luò)公開度
書目名稱Genetic Approaches to Microbial Pathogenicity網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Genetic Approaches to Microbial Pathogenicity被引頻次
書目名稱Genetic Approaches to Microbial Pathogenicity被引頻次學(xué)科排名
書目名稱Genetic Approaches to Microbial Pathogenicity年度引用
書目名稱Genetic Approaches to Microbial Pathogenicity年度引用學(xué)科排名
書目名稱Genetic Approaches to Microbial Pathogenicity讀者反饋
書目名稱Genetic Approaches to Microbial Pathogenicity讀者反饋學(xué)科排名
作者: Oratory 時(shí)間: 2025-3-21 21:32
Thomas M. Chen,Lee Jarvis,Stuart Macdonaldof human origin. Some characteristics of these fimbrial adhesins are shown in Table 1. It should be noted that this list is not complete and that the number of discovered fimbrial adhesins is rapidly increasing.作者: Processes 時(shí)間: 2025-3-22 00:40 作者: absolve 時(shí)間: 2025-3-22 06:06
mutagenesis, DNA sequencing, and other procedures, individual gene products are identified and their functions assessed. Studies of regulatory features help our understanding of what molecular mechanisms determine expression of the virulence function.作者: Mirage 時(shí)間: 2025-3-22 09:43
https://doi.org/10.1057/9780230109773diphosphate (ADP) ribosyl-transferase activity of fragment A; and (ii) those that carried a nonsense mutation in the fragment B encoding portion of the structural gene which resulted in the expression of a truncated toxin-related molecule that is devoid of the eukaryotic cell receptor binding domain作者: Assemble 時(shí)間: 2025-3-22 16:34 作者: Assemble 時(shí)間: 2025-3-22 19:21 作者: 凈禮 時(shí)間: 2025-3-22 22:23
Genetics of , Hemolysin,ing from . . 1971 b). Forming another subclass of the “Nonenzymatic” proteins are the sulfhydryl .)-activated hemolysins such as streptolysin .of ., pneumolysin of ., the θ-toxin of., as well as cereolysin and listeriolysin of . and species of . (for review see . and . and .. These toxins are assume作者: Servile 時(shí)間: 2025-3-23 02:28
Genes Determining Adhesin Formation in Uropathogenic ,, mutagenesis, DNA sequencing, and other procedures, individual gene products are identified and their functions assessed. Studies of regulatory features help our understanding of what molecular mechanisms determine expression of the virulence function.作者: seroma 時(shí)間: 2025-3-23 08:52 作者: dry-eye 時(shí)間: 2025-3-23 10:31 作者: 群居男女 時(shí)間: 2025-3-23 16:53
Genetic Analysis of , Virulence, infection with streptococci of the type originally described by C.. It was subsequently demonstrated that . is the principal etiological agent of dental caries in humans (see N. 1978). . only colonizes humans following tooth eruption during the 1st year of life, with most humans being colonized during early childhood (S. et al. 1976).作者: OMIT 時(shí)間: 2025-3-23 21:53 作者: 退出可食用 時(shí)間: 2025-3-24 01:10
Genetic Determinants of Virulence in , and Dysenteric Strains of ,: Their Involvement in the Pathogecation of blood and mucus. Primates are considered to be the only natural hosts susceptible to. infection, a consideration that has seriously hindered the development of inexpensive animal models for study of the disease.作者: ACME 時(shí)間: 2025-3-24 03:14 作者: amyloid 時(shí)間: 2025-3-24 07:56 作者: 擔(dān)心 時(shí)間: 2025-3-24 11:44 作者: arthrodesis 時(shí)間: 2025-3-24 17:23 作者: Ordeal 時(shí)間: 2025-3-24 20:06
Genetic Analysis of Bacterial Virulence Determinants in , and the Pathogenic ,,anding better the molecular epidemiology of disease agents (H.et al. 1984). We suppose that in the near future most virulence factors defined by classic studies in medical microbiology will be analyzed at the genetic level in some manner or other.作者: 帶子 時(shí)間: 2025-3-24 23:33
Gonococcal Pilus: Genetics and Structure, variation are closely linked phenomena; that the regulation of pilus expression is reminiscent of yeast mating type interconversion and of immunoglobulin gene rearrangement; and that the gonococcal pilin belongs to a class or family of bacterial pilins with a common subunit structure.作者: 腐爛 時(shí)間: 2025-3-25 04:02 作者: critique 時(shí)間: 2025-3-25 09:17 作者: 花爭(zhēng)吵 時(shí)間: 2025-3-25 11:40
Genetic Analysis of Bacterial Virulence Determinants in , and the Pathogenic ,,eview, see Macrina 1984). This has been especially true in the analysis of enterotoxin structure and function (So et al. 1976, 1978; M.and F.1980; P.and M.1982) as well as in the evaluation of bacterial pili and hemolysins in gastrointestinal or extraintestinal infections (S. and H. 1978; W. etal. 1作者: 口味 時(shí)間: 2025-3-25 16:10
Gonococcal Pilus: Genetics and Structure,(e.g., influenza virus), parasites (e.g., .), and bacteria (e.g., .). Recent work has shown that two surface proteins of . undergo antigenic variation as well. These two virulence factors are the pilus and the opacity (PII) proteins, both of which promote gonococcal binding to host (human) epithelia作者: 一個(gè)姐姐 時(shí)間: 2025-3-25 23:57 作者: 思考才皺眉 時(shí)間: 2025-3-26 01:40 作者: 欺騙世家 時(shí)間: 2025-3-26 07:56
Genetic Determinants of Virulence in , and Dysenteric Strains of ,: Their Involvement in the Pathogldwide. Recognized as a clinical disease entity since the time of Hippocrates, dysentery is characterized by painful abdominal cramps and frequent defecation of blood and mucus. Primates are considered to be the only natural hosts susceptible to. infection, a consideration that has seriously hindere作者: LEERY 時(shí)間: 2025-3-26 11:20
Cholera Toxin: Genetic Analysis, Regulation, and Role in Pathogenesis,early involved in the pathogenesis of a wide variety of bacterial diseases affecting mankind. These include cholera, diphtheria, pertussis, anthrax, dysentery, tetanus, gas gangrene, and opportunistic infections caused by ., and .. Even . does on occasion possess the necessary genetic information fo作者: COLON 時(shí)間: 2025-3-26 13:23
Molecular Biology of Fimbriae of Enterotoxigenic ,,toxigenic . (ETEC) strains to adhere to the intestinal epithelium is required as an initial step in establishing diarrheal disease. The bacterial cell surface structures that are responsible for adherence have been called adhesins or colonization factors (see also G. and D. G. 1982, for a recent rev作者: 寵愛 時(shí)間: 2025-3-26 19:01
Genetics of , Hemolysin,cter varies from organism to organism. Extracellular enzymes have been shown to be responsible for hemolysis by some bacteria, e.g., the phospholipases of . . et al. 1982) and . (also called a-toxin; . 1979) and also the sphingiomyelinase C produced by. (β-toxin; . and . 1983). “Nonenzymatic” protei作者: Assault 時(shí)間: 2025-3-27 00:36
Genes Determining Adhesin Formation in Uropathogenic ,, there is evidence that filamentous surface structures on the bacteria, called pili or fimbriae, are the adhesins that bind to epithelial cells of the host. The adhesins interact with complementary receptors which are carbohydrate-containing polymers on the target cells. The importance of adhesion i作者: FIN 時(shí)間: 2025-3-27 01:49
Genetic Basis of Iron Assimilation in Pathogenic ,,ope. In these societies the common gram-negative bacilli now isolated in the clinic are those which are prominently associated with man, such as ., or are, like ., those which are equipped with special devices to take advantage of the compromised host (Y. et al. 1982).作者: Adornment 時(shí)間: 2025-3-27 07:33 作者: squander 時(shí)間: 2025-3-27 12:06
Molecular Cloning of Genes Encoding Gram-Positive Virulence Factors,f cloned virulence determinants can make to advancing an understanding of infectious disease. Establishment of an infectious disease is the result of an imbalance in the steady-state “host-parasite” relationship. As the host defense mechanisms are many and interrelated, the ability of a bacterium to作者: Femine 時(shí)間: 2025-3-27 14:52 作者: 四海為家的人 時(shí)間: 2025-3-27 19:08 作者: 露天歷史劇 時(shí)間: 2025-3-27 22:17 作者: 有斑點(diǎn) 時(shí)間: 2025-3-28 04:16 作者: 展覽 時(shí)間: 2025-3-28 07:15
0070-217X Overview: 978-3-642-70588-5978-3-642-70586-1Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: 等級(jí)的上升 時(shí)間: 2025-3-28 13:26 作者: Altitude 時(shí)間: 2025-3-28 14:52 作者: 不能平靜 時(shí)間: 2025-3-28 19:37 作者: 凝視 時(shí)間: 2025-3-29 00:23
Cyberspace and International Relationsnal flora of mammals. Of these only a handful can cause infections in human hosts, and only . has been identified as a potentially life-threatening pathogen. This organism is usually associated with patients with an underlying disease who are subjected to extensive chemotherapy and hospitalization.作者: 調(diào)整校對(duì) 時(shí)間: 2025-3-29 04:49
Folgen der Telekommunikationsentwicklung,ldwide. Recognized as a clinical disease entity since the time of Hippocrates, dysentery is characterized by painful abdominal cramps and frequent defecation of blood and mucus. Primates are considered to be the only natural hosts susceptible to. infection, a consideration that has seriously hindere作者: 過(guò)時(shí) 時(shí)間: 2025-3-29 09:40 作者: 尊嚴(yán) 時(shí)間: 2025-3-29 11:44
Thomas M. Chen,Lee Jarvis,Stuart Macdonaldtoxigenic . (ETEC) strains to adhere to the intestinal epithelium is required as an initial step in establishing diarrheal disease. The bacterial cell surface structures that are responsible for adherence have been called adhesins or colonization factors (see also G. and D. G. 1982, for a recent rev作者: 頂點(diǎn) 時(shí)間: 2025-3-29 17:43 作者: incontinence 時(shí)間: 2025-3-29 20:50
there is evidence that filamentous surface structures on the bacteria, called pili or fimbriae, are the adhesins that bind to epithelial cells of the host. The adhesins interact with complementary receptors which are carbohydrate-containing polymers on the target cells. The importance of adhesion i作者: 噱頭 時(shí)間: 2025-3-30 01:16
https://doi.org/10.1007/978-4-431-67941-7ope. In these societies the common gram-negative bacilli now isolated in the clinic are those which are prominently associated with man, such as ., or are, like ., those which are equipped with special devices to take advantage of the compromised host (Y. et al. 1982).
