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標(biāo)題: Titlebook: Gasotransmitters in Organ Transplantation; A New Era in Transpl George J. Dugbartey Book 2024 The Editor(s) (if applicable) and The Author( [打印本頁(yè)]

作者: GERD847    時(shí)間: 2025-3-21 16:41
書(shū)目名稱Gasotransmitters in Organ Transplantation影響因子(影響力)




書(shū)目名稱Gasotransmitters in Organ Transplantation影響因子(影響力)學(xué)科排名




書(shū)目名稱Gasotransmitters in Organ Transplantation網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Gasotransmitters in Organ Transplantation網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Gasotransmitters in Organ Transplantation被引頻次




書(shū)目名稱Gasotransmitters in Organ Transplantation被引頻次學(xué)科排名




書(shū)目名稱Gasotransmitters in Organ Transplantation年度引用




書(shū)目名稱Gasotransmitters in Organ Transplantation年度引用學(xué)科排名




書(shū)目名稱Gasotransmitters in Organ Transplantation讀者反饋




書(shū)目名稱Gasotransmitters in Organ Transplantation讀者反饋學(xué)科排名





作者: NATTY    時(shí)間: 2025-3-21 21:41

作者: Myofibrils    時(shí)間: 2025-3-22 02:36

作者: jarring    時(shí)間: 2025-3-22 05:50
O. Bartels,M. Neidhardt,H. Schellbergerplay important roles in cellular homeostasis and impact physiological and pathophysiological conditions. Nitric oxide was the first gasotransmitter to be identified followed by carbon monoxide, while hydrogen sulfide was recently established as the third member of the gasotransmitter family. Althoug
作者: 無(wú)情    時(shí)間: 2025-3-22 11:16

作者: yohimbine    時(shí)間: 2025-3-22 16:17
https://doi.org/10.1007/978-3-031-52569-8llenge in solid organ transplantation (SOT), and represents a major factor that increases acute tubular necrosis, decreases graft survival, and delays graft function. This complicates graft quality, post-transplant patient care and organ transplantation outcomes, and therefore undermines the success
作者: yohimbine    時(shí)間: 2025-3-22 20:38
https://doi.org/10.1007/978-3-7091-6384-9ong-term hemodialysis, optimal kidney preservation prior to transplantation remains a major challenge, as the gold standard of renal graft preservation by static cold storage tion in University of Wisconsin (UW) solution on ice at 4?°C contributes to graft injury and long-term post-transplantation c
作者: semble    時(shí)間: 2025-3-23 00:02

作者: 騷動(dòng)    時(shí)間: 2025-3-23 02:49
https://doi.org/10.1007/978-3-7091-0646-4opment of carbon monoxide-releasing molecules (CO-RM) and utilization of heme oxygenase-1 inducers have shown CO to be a promising therapy in reducing renal ischemia-reperfusion injury (IRI) and other inflammatory diseases. In this chapter, we will discuss the developments and application of CO-RM i
作者: 骨    時(shí)間: 2025-3-23 09:35

作者: 比賽用背帶    時(shí)間: 2025-3-23 12:27

作者: 減震    時(shí)間: 2025-3-23 15:27
LVQ and Counter-Propagation Networksutrition is no longer possible. Over the past decade, this complex therapeutic procedure has received significant clinical attention, illustrating its evolution from an experimental origin in the mid-twentieth century to an established clinical practice today. Despite several recent advances made in
作者: profligate    時(shí)間: 2025-3-23 20:00
Electronic Word of Mouth and Social Commerce, who are extremely sensitive to insulin and lack counter-regulatory measures, and thereby increasing their risk of neuroglycopenia and hypoglycemia unawareness. Thus, pancreatic islet transplantation restores normoglycemia and insulin independence, and prevents long-term surgical complications asso
作者: MIRTH    時(shí)間: 2025-3-24 01:55
General conclusions and future work,t-transplant outcomes. At the cellular level, hypoxia due to ischemia disturbs cellular metabolism and decreases cellular bioenergetics through dysfunction of mitochondrial electron transport chain, causing a switch from cellular respiration to anaerobic metabolism, and subsequent cascades of events
作者: CUMB    時(shí)間: 2025-3-24 03:03

作者: 追逐    時(shí)間: 2025-3-24 08:15
Configuring Your Development Environment,utine life-saving intervention for a variety of end-stage pulmonary diseases refractory to medical management. Despite the success and continuous refinement in lung transplantation techniques, the widespread application of this important life-saving procedure is severely hampered by poor allograft q
作者: Afflict    時(shí)間: 2025-3-24 11:10
https://doi.org/10.1007/978-1-4842-6150-7 improved quality of life, nutrition and survival. Compared to other types of organ transplants, it is a relatively new advancement in the field of organ transplantation. Nevertheless, great advances have been made over the past few decades to the present era, including the use of ischemic precondit
作者: 一窩小鳥(niǎo)    時(shí)間: 2025-3-24 17:52

作者: anniversary    時(shí)間: 2025-3-24 22:32
https://doi.org/10.1007/978-3-7091-0646-4n renal IRI, and organ transplantation as well as the anti-inflammatory mechanisms of CO in respect to mitigating apoptosis, suppressing dendritic cell maturation and signalling, inhibiting toll-like receptor activation, promoting anti-inflammatory responses, and the effects on renal vasculature.
作者: 初學(xué)者    時(shí)間: 2025-3-25 00:48

作者: Fracture    時(shí)間: 2025-3-25 05:41
roach to organ preservation and transplantation.Discusses cl.This book covers recent pre-clinical and clinical developments in gasotransmitters (nitric oxide, carbon monoxide and hydrogen sulfide) in all transplantable solid organs – kidney, heart, lung, liver, pancreas and intestine. Gasotransmitte
作者: inflate    時(shí)間: 2025-3-25 09:39

作者: cardiac-arrest    時(shí)間: 2025-3-25 15:35

作者: 入伍儀式    時(shí)間: 2025-3-25 16:02

作者: Grandstand    時(shí)間: 2025-3-25 21:43
A. A. Frolov,D. Husek,P. Combe,V. Sná?eleleasing molecules (CO-RMs) in preclinical models of lung and liver transplantation, and underlying molecular mechanisms of cyto- and organ protection during organ procurement, preservation, implantation and post-transplant periods. In addition, a discussion of the future of CO in clinical organ transplantation is provided.
作者: 表被動(dòng)    時(shí)間: 2025-3-26 03:32
Carbon Monoxide as a New Pharmacological Tool to Refine Protocols for Lung and Liver Transplantationeleasing molecules (CO-RMs) in preclinical models of lung and liver transplantation, and underlying molecular mechanisms of cyto- and organ protection during organ procurement, preservation, implantation and post-transplant periods. In addition, a discussion of the future of CO in clinical organ transplantation is provided.
作者: gait-cycle    時(shí)間: 2025-3-26 08:15
r supplementation of standard preservation solution with gasotransmitters during organ graft preservation to mitigate transplant-induced IRI. The book is divided into three parts for easier access: .Hydrogen Su978-3-031-48069-0978-3-031-48067-6
作者: languor    時(shí)間: 2025-3-26 08:43

作者: 駭人    時(shí)間: 2025-3-26 14:17

作者: Cpap155    時(shí)間: 2025-3-26 20:51

作者: 走調(diào)    時(shí)間: 2025-3-27 01:01
Hydrogen Sulfide Therapy Versus the Transplant Surgeon’s Inexorable Enemyor molecules in SOT, and suggestions for clinical translation. The findings in this chapter showed that storage of donor organs in H.S-supplemented preservation solution or administration of H.S to organ donor prior to organ procurement and to recipient at the start and during reperfusion is a novel
作者: Gastric    時(shí)間: 2025-3-27 04:23

作者: 悄悄移動(dòng)    時(shí)間: 2025-3-27 06:28

作者: cyanosis    時(shí)間: 2025-3-27 11:25
Carbon Monoxide Preserves Cellular Integrity and Intestinal Mucosal Barrier Function After Intestinah potential to improve current intestinal transplantation protocol, reduce post-transplant complications and ensure long-term transplant recipient survival. Carbon monoxide (CO), a gas previously known for its toxicity and death at high concentrations, is quickly moving past its historic notoriety t
作者: Nebulous    時(shí)間: 2025-3-27 16:32

作者: 無(wú)禮回復(fù)    時(shí)間: 2025-3-27 19:42
Mechanisms of Cell Death in Ischemia-Reperfusion Injury in Organ Transplantation and Nitric Oxide asrate ischemia and reperfusion may result in cell dysfunction, which may not lead to cell death due to activation of recovery systems to control ROS production and to ensure cell survival. However, prolonged and severe ischemia and reperfusion induce cell death by apoptosis, necrosis, necroptosis, mi
作者: GULP    時(shí)間: 2025-3-27 23:51
Protective Role of Nitric Oxide in Kidney and Heart Transplantation significant detrimental role. In this chapter, the author presents endogenous production of NO as well as its exogenous sources, and then discusses protective effects of constitutive NOS-derived NO in kidney and heart allotransplantation from experimental and clinical perspectives. He also highligh
作者: 延期    時(shí)間: 2025-3-28 04:02

作者: 急急忙忙    時(shí)間: 2025-3-28 06:55

作者: 地名詞典    時(shí)間: 2025-3-28 10:26

作者: Kindle    時(shí)間: 2025-3-28 15:43
O. Bartels,M. Neidhardt,H. Schellbergerwith one another in compensatory and reciprocal fashion when the level of one is depleted under pathological conditions. In addition, the chapter also summarizes various therapeutic properties of these gases such as antioxidant, anti-inflammatory, anti-apoptotic and vasodilatory properties, enabling
作者: jocular    時(shí)間: 2025-3-28 21:33

作者: 種屬關(guān)系    時(shí)間: 2025-3-29 01:40
https://doi.org/10.1007/978-3-031-52569-8or molecules in SOT, and suggestions for clinical translation. The findings in this chapter showed that storage of donor organs in H.S-supplemented preservation solution or administration of H.S to organ donor prior to organ procurement and to recipient at the start and during reperfusion is a novel
作者: 銀版照相    時(shí)間: 2025-3-29 06:36

作者: –吃    時(shí)間: 2025-3-29 10:53

作者: Armory    時(shí)間: 2025-3-29 13:40

作者: PRE    時(shí)間: 2025-3-29 18:29

作者: 有特色    時(shí)間: 2025-3-29 21:20
General conclusions and future work,rate ischemia and reperfusion may result in cell dysfunction, which may not lead to cell death due to activation of recovery systems to control ROS production and to ensure cell survival. However, prolonged and severe ischemia and reperfusion induce cell death by apoptosis, necrosis, necroptosis, mi
作者: 小丑    時(shí)間: 2025-3-30 00:55

作者: Peak-Bone-Mass    時(shí)間: 2025-3-30 05:58
Configuring Your Development Environment, However, primary graft dysfunction (PGD), allograft rejection and other post-transplant complications arising from unavoidable ischemia-reperfusion injury (IRI) of transplanted lungs, increase morbidity and mortality of lung transplant recipients annually. In the light of this worrying trend, nitri
作者: 細(xì)微差別    時(shí)間: 2025-3-30 11:13
https://doi.org/10.1007/978-1-4842-6150-7rch for novel therapeutic avenues and improved transplantation protocols to prevent or attenuate intestinal IRI. Among the many candidate agents that are being investigated to combat IRI and its associated complications, nitric oxide, an endogenously produced gaseous signaling molecule, is emerging
作者: 皮薩    時(shí)間: 2025-3-30 16:25
Blood Oxygenators and Artificial Lungssplantation. Nitric oxide, a ubiquitous molecule and the first identified member of a family of gaseous signaling molecules, is emerging as a suitable candidate to combat IRI and protect against islet apoptosis, and prevent complications after whole-pancreas or islet transplantation. In this chapter
作者: BOOR    時(shí)間: 2025-3-30 17:40

作者: 最小    時(shí)間: 2025-3-31 00:42
Physiological Role of Hydrogen Sulfide in the Renal Systemcially among industrial workers. This is due to its ability to reversibly inhibit the activity of cytochrome . oxidase, a terminal enzyme of the mitochondrial electron transport chain. Recently, however, H.S has risen above its notorious public image and is now seen by researchers as an endogenously
作者: 使害羞    時(shí)間: 2025-3-31 02:46

作者: 卷發(fā)    時(shí)間: 2025-3-31 05:09

作者: 優(yōu)雅    時(shí)間: 2025-3-31 09:18
Organ Graft Protection Following Pre-treatment of Transplant Donors with Hydrogen Sulfides an inevitable consequence of solid organ transplantation. Current organ preservation strategies such as static cold storage are aimed at reducing IRI. However, prolonged SCS exacerbates IRI. Recent research has examined pre-treatment approaches to more effectively attenuate IRI. Hydrogen sulfide (




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