標(biāo)題: Titlebook: endoCANNABINOIDS; Actions at Non-CB1/C Mary E. Abood,Roger G Sorensen,Nephi Stella Book 2013 Springer Science+Business Media New York 2013 [打印本頁] 作者: 阿諛奉承 時間: 2025-3-21 18:26
書目名稱endoCANNABINOIDS影響因子(影響力)
書目名稱endoCANNABINOIDS影響因子(影響力)學(xué)科排名
書目名稱endoCANNABINOIDS網(wǎng)絡(luò)公開度
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書目名稱endoCANNABINOIDS被引頻次學(xué)科排名
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書目名稱endoCANNABINOIDS讀者反饋學(xué)科排名
作者: 公式 時間: 2025-3-21 21:24 作者: FRAX-tool 時間: 2025-3-22 01:57 作者: 誓言 時間: 2025-3-22 07:13 作者: nutrition 時間: 2025-3-22 11:31
GPR18 and NAGly Signaling: New Members of the Endocannabinoid Family or Distant Cousins?C) and .arachidonoyl glycine (NAGly) by an oxygenated metabolite of the endogenous cannabinoid anandamide (AEA). The review will outline what is known about this receptor, the ligands that activate and block the receptor, and how these interactions fit within cannabinoid/endocannabinoid signaling.作者: 吃掉 時間: 2025-3-22 16:36
978-1-4899-8846-1Springer Science+Business Media New York 2013作者: 吃掉 時間: 2025-3-22 17:52 作者: 異端 時間: 2025-3-22 21:56
Nanostructure Science and Technologyptors, G protein-coupled receptors (GPCRs) identified by molecular cloning in the early 1990s. Since that time, there has been a growing body of pharmacological evidence indicating the existence of additional molecular targets for cannabinoids. This chapter overviews the nonclassical targets thought作者: 小淡水魚 時間: 2025-3-23 02:42
https://doi.org/10.1007/978-94-011-2538-3ein-coupled receptors (GPCRs) that bind constituents of ., such as the classical cannabinoid agonist (?)-.-delta-9-tetrahydrocannabinol [(?)-Δ.-THC (.)]. CB.R and CB.R bind four other structural classes of ligands (see Fig. 2.1): nonclassical cannabinoid agonists typified by 2-[5-Hydroxy-2-(3-hydrox作者: LAVE 時間: 2025-3-23 08:14 作者: CLEFT 時間: 2025-3-23 10:15
https://doi.org/10.1007/978-1-4757-5876-4aims to summarise the work that became the first study to demonstrate a non-neuronal physiological role for GPR55 in vivo. In summary, male mice lacking GPR55 develop a high bone mass phenotype due to impairment in osteoclast function—consistent with GPR55 agonists O-1602 and LPI stimulating osteocl作者: Needlework 時間: 2025-3-23 16:07 作者: 竊喜 時間: 2025-3-23 19:36 作者: Synovial-Fluid 時間: 2025-3-23 22:41
https://doi.org/10.1007/978-3-642-84850-6odegenerative effects in various experimental models. Here we review the interaction of cannabinoids (plant, endogenous or synthetic) with microglia, which are considered to be the immune cells of the brain. We describe the functional endocannabinoid system (ligands, receptors, and enzymes) in micro作者: bypass 時間: 2025-3-24 04:24 作者: 上漲 時間: 2025-3-24 08:45
https://doi.org/10.1007/978-1-4684-7284-4can mediate fast inhibitory synaptic transmission, the 5-HT. receptors are involved in both excitatory and inhibitory synaptic transmission in the central and peripheral nervous system. These receptors play important roles in several physiological and pathological processes such as vomiting reflex, 作者: 深陷 時間: 2025-3-24 12:33
https://doi.org/10.1007/978-981-10-7371-7gene transcription. There are three peroxisome proliferator-activated receptor (PPAR) isoforms; α, δ (also known as β), and γ, which are activated by a numerous ligands including endocannabinoids. Activation of these receptors has been shown to modulate inflammation, in a number of different animal 作者: dandruff 時間: 2025-3-24 16:20
https://doi.org/10.1007/978-0-387-39975-1ome proliferator-activated alpha nuclear receptor (PPARα), which regulates genes involved in lipid metabolism and inflammatory responses, is a viable target for treating nicotine dependence. The endogenous ligands for PPARα, oleoylethanolamide and palmitoylethanolamide, are structurally similar to a作者: Adherent 時間: 2025-3-24 20:13 作者: 材料等 時間: 2025-3-25 02:24 作者: Hemiplegia 時間: 2025-3-25 05:38 作者: Allege 時間: 2025-3-25 08:33
GPR55 in the CNSme of the non-CB./CB./TRPV1 effects of cannabinoid compounds. However, it rapidly became clear that the pharmacology and signaling of GPR55 were quite complicated. Work over the last few years has found that lysophosphatidyl inositol is a major endogenous ligand for GPR55, with phytocannabinoids and作者: linguistics 時間: 2025-3-25 14:55
The Role of GPR55 in Bone Biologyaims to summarise the work that became the first study to demonstrate a non-neuronal physiological role for GPR55 in vivo. In summary, male mice lacking GPR55 develop a high bone mass phenotype due to impairment in osteoclast function—consistent with GPR55 agonists O-1602 and LPI stimulating osteocl作者: ALTER 時間: 2025-3-25 16:30
The Role of GPR55 in Cancervior. As will be described in this chapter, this receptor has been directly or indirectly related to the basic alterations that drive malignant growth: uncontrolled cancer cell proliferation, sustained angiogenesis, and metastatic capability. GPR55 activation promotes cell proliferation, produces pr作者: choroid 時間: 2025-3-25 21:53 作者: 發(fā)源 時間: 2025-3-26 03:55 作者: Cantankerous 時間: 2025-3-26 07:05 作者: ablate 時間: 2025-3-26 10:56 作者: Hdl348 時間: 2025-3-26 14:32 作者: 過于平凡 時間: 2025-3-26 17:59
Peroxisome Proliferator-Activated Nuclear Receptors and Drug Addictionome proliferator-activated alpha nuclear receptor (PPARα), which regulates genes involved in lipid metabolism and inflammatory responses, is a viable target for treating nicotine dependence. The endogenous ligands for PPARα, oleoylethanolamide and palmitoylethanolamide, are structurally similar to a作者: Distribution 時間: 2025-3-26 23:41 作者: Gene408 時間: 2025-3-27 02:08
https://doi.org/10.1007/978-94-007-5267-2 those devoid of significant activity at cannabinoid CB1 and CB2 receptors. Such evidence, and its possible implications in the potential therapeutic applications of endogenous, plant and synthetic cannabimimetic compounds, is reviewed in this chapter.作者: 大范圍流行 時間: 2025-3-27 08:40
https://doi.org/10.1007/b105867and chemical characterization of ?.-tetrahydrocannabinol (THC) in 1964 as the first bioactive ingredient produced by . that revived the scientific community’s interest in further understanding and optimizing the unique therapeutic properties of phytocannabinoids (phyto-CB) (Mechoulam and Gaoni 1967).作者: Myocarditis 時間: 2025-3-27 09:53 作者: 玩忽職守 時間: 2025-3-27 17:08
Conclusions: Therapeutic Potential of Novel Cannabinoid Receptorsand chemical characterization of ?.-tetrahydrocannabinol (THC) in 1964 as the first bioactive ingredient produced by . that revived the scientific community’s interest in further understanding and optimizing the unique therapeutic properties of phytocannabinoids (phyto-CB) (Mechoulam and Gaoni 1967).作者: 機(jī)制 時間: 2025-3-27 20:27
GPR55 in the CNS complicated. Work over the last few years has found that lysophosphatidyl inositol is a major endogenous ligand for GPR55, with phytocannabinoids and endocannabinoids having a lesser role. This review briefly summarizes our current understanding of GPR55 signaling and its role in the central nervous system.作者: 功多汁水 時間: 2025-3-28 00:05
The Role of GPR55 in Bone Biologyng GPR55 develop a high bone mass phenotype due to impairment in osteoclast function—consistent with GPR55 agonists O-1602 and LPI stimulating osteoclast function. These studies advocate the development of GPR55 antagonists for the treatment of diseases associated with excessive osteoclast activity such as osteoporosis.作者: 性行為放縱者 時間: 2025-3-28 02:40
Book 2013ield of cannabinoid research who have an interest in learning about these compounds and their atypical cannabinoid signalling. This book provides insight into the potential medical application of cannabinoids and their therapeutic development for the treatment of human disease.作者: Ferritin 時間: 2025-3-28 09:08
Nanostructure Science and Technology to be present in vascular endothelium, central nervous system and, possibly, other tissues, with emphasis on GPCRs. The possible relationship with orphan GPCR is discussed in greater detail, particularly GPR18, GPR55, GPR92, GPR119, and other GPCRs, which are reportedly activated by various endogenous, plant-derived, and synthetic cannabinoids.作者: 產(chǎn)生 時間: 2025-3-28 10:43
https://doi.org/10.1007/978-3-642-84850-6glial cell models. In addition, we review the activity of cannabinoid ligands at non-CB./non-CB. GPCR targets, mainly GPR55 and GPR18, with focus on microglia. Finally, we discuss non-CB./non-CB.-mediated effects of cannabinoid ligands on microglial migration, transcriptional regulation, and anti-inflammation in a multiple sclerosis-like model.作者: forebear 時間: 2025-3-28 15:20 作者: Consensus 時間: 2025-3-28 19:02
Overview of Nonclassical Cannabinoid Receptors to be present in vascular endothelium, central nervous system and, possibly, other tissues, with emphasis on GPCRs. The possible relationship with orphan GPCR is discussed in greater detail, particularly GPR18, GPR55, GPR92, GPR119, and other GPCRs, which are reportedly activated by various endogenous, plant-derived, and synthetic cannabinoids.作者: limber 時間: 2025-3-29 01:16 作者: fluffy 時間: 2025-3-29 03:40 作者: 起皺紋 時間: 2025-3-29 08:56
1048-6909 eir atypical cannabinoid signalling. This book provides insight into the potential medical application of cannabinoids and their therapeutic development for the treatment of human disease.978-1-4899-8846-1978-1-4614-4669-9Series ISSN 1048-6909 Series E-ISSN 2524-6488 作者: 不整齊 時間: 2025-3-29 13:24 作者: 治愈 時間: 2025-3-29 19:02
The Role of GPR55 in Canceression correlates with tumor malignancy. Together, these data indicate that GPR55 plays a relevant role in cancer and opens the possibility of considering this orphan receptor as a new therapeutic target and potential biomarker in oncology.作者: Amplify 時間: 2025-3-29 19:46 作者: 不能逃避 時間: 2025-3-30 02:32
https://doi.org/10.1007/978-3-0348-8946-9 complicated. Work over the last few years has found that lysophosphatidyl inositol is a major endogenous ligand for GPR55, with phytocannabinoids and endocannabinoids having a lesser role. This review briefly summarizes our current understanding of GPR55 signaling and its role in the central nervous system.作者: Consensus 時間: 2025-3-30 08:01 作者: 宏偉 時間: 2025-3-30 11:48
Book 2013 in learning about future directions in cannabinoid research. Additionally, this book may be of value to investigators currently working outside the field of cannabinoid research who have an interest in learning about these compounds and their atypical cannabinoid signalling. This book provides insi
