標(biāo)題: Titlebook: Exploring Pancreatic Metabolism and Malignancy; Ganji Purnachandra Nagaraju,Aramati BM Reddy Book 2019 Springer Nature Singapore Pte Ltd. [打印本頁(yè)] 作者: Harding 時(shí)間: 2025-3-21 16:29
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy影響因子(影響力)
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy被引頻次
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy被引頻次學(xué)科排名
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書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy讀者反饋
書(shū)目名稱(chēng)Exploring Pancreatic Metabolism and Malignancy讀者反饋學(xué)科排名
作者: 卜聞 時(shí)間: 2025-3-21 20:14
Pancreatitis: Clinical Aspects of Inflammatory Phenotypes,into the small intestine and attacks the pancreas. When these activated digestive enzymes are activated still in the pancreas, irritating the cells of our pancreas causes inflammation. These cause complications such as the formation of the pseudocyst, breathing problem, failure in the kidney, diabet作者: 檔案 時(shí)間: 2025-3-22 02:16 作者: Bumptious 時(shí)間: 2025-3-22 06:30 作者: Esophagus 時(shí)間: 2025-3-22 10:24 作者: SEMI 時(shí)間: 2025-3-22 16:44 作者: SEMI 時(shí)間: 2025-3-22 17:04
Diabetes with Pancreatic Ductal Adenocarcinoma,o conservative therapy. Diabetes is recently been correlated with mortality and morbidity from PDAC. The association between diabetes and PDAC stems from the structural association between the endocrine and exocrine pancreas and aberrant expression of hormones from islets. It can also result from ot作者: 清楚 時(shí)間: 2025-3-22 23:09 作者: Armory 時(shí)間: 2025-3-23 03:20 作者: companion 時(shí)間: 2025-3-23 06:59 作者: 不再流行 時(shí)間: 2025-3-23 11:51
Role of Heat Shock Protein 90 in Diabetes and Pancreatic Cancer Management,ly coordinating regulatory mechanisms in order to control their activity. One of the key regulators responsible for the upregulation of Hsp90 is heat shock factor (HSF1), whose primary role is to bind heat shock elements (HSEs) with Hsp90 promoters. HSF1 functions by interacting with the transcripti作者: Lyme-disease 時(shí)間: 2025-3-23 14:05 作者: 珊瑚 時(shí)間: 2025-3-23 21:24 作者: LATHE 時(shí)間: 2025-3-23 22:58
Exosomes: Mediators and Therapeutic Targets of Diabetes and Pancreatic Cancer, mediators and therapeutic targets of diabetes mellitus and pancreatic cancer owing to their role in controlling .-cell mass and insulin signaling. Early diagnosis of diabetes and pancreatic cancer can remarkably expand therapeutic approaches and overall survival. Further, identification of biomarke作者: Morose 時(shí)間: 2025-3-24 03:16 作者: 鐵砧 時(shí)間: 2025-3-24 09:49
Energy, Environment, and Sustainabilityated mitochondrial enzymes not only participate in the metabolic reprogramming of tumor cells but also modulate cellular processes involved in tumor proliferation and progression. In this review, we described the association between the deregulated mitochondrial enzymes and development of pancreatic cancer.作者: legislate 時(shí)間: 2025-3-24 14:07
https://doi.org/10.1007/978-3-642-36838-7rther understand the role this pathway can play in deadly diseases such as PC. In this chapter we will explore the role of the HIF-1α pathway in PC, and in metabolic disorders, such as diabetes mellitus, and studies that have been conducted in order to further holistically understand the role of HIF-1α.作者: lipoatrophy 時(shí)間: 2025-3-24 16:34 作者: 窩轉(zhuǎn)脊椎動(dòng)物 時(shí)間: 2025-3-24 20:59 作者: adj憂郁的 時(shí)間: 2025-3-24 23:56
Book 2019et definition with a view to drug discovery. Metabolic syndrome, which includes adnominal obesity, hypertension, dyslipidemia, and hyperglycemia, has recently been shown to play an important role in the etiology and progression of various cancers. Further, obesity and diabetes have been associated w作者: 沒(méi)血色 時(shí)間: 2025-3-25 04:56
Methods and Models in Exploring Pancreatic Functions,atic cancer. All have unique advantages and drawbacks, hence the need to identify novel models for evaluation of pancreatic dysfunctions with high accuracy, sensitivity, and output for the diagnosis of the disease.作者: FIR 時(shí)間: 2025-3-25 08:17
https://doi.org/10.1007/978-981-99-2295-6ince the survival of many diagnosed PC patients are limited to months, understanding the origins and basic cascades of these different cytokines could better help in identifying the targets and developing different therapeutic approaches in screening of the patients with this disease.作者: Eclampsia 時(shí)間: 2025-3-25 14:24
Measurement-Based Care in Mental Disorderstudies also reported that antidiabetic drugs decrease risk for PC and increase survival in diabetes patients. In this chapter we describe the link between insulin resistance and pancreatic cancer risk.作者: Devastate 時(shí)間: 2025-3-25 16:46
Role of Inflammatory Cytokines in the Initiation and Progression of Pancreatic Cancer,ince the survival of many diagnosed PC patients are limited to months, understanding the origins and basic cascades of these different cytokines could better help in identifying the targets and developing different therapeutic approaches in screening of the patients with this disease.作者: TSH582 時(shí)間: 2025-3-25 21:54
Insulin Resistance Is a Common Core Tethered to Diabetes and Pancreatic Cancer Risk,tudies also reported that antidiabetic drugs decrease risk for PC and increase survival in diabetes patients. In this chapter we describe the link between insulin resistance and pancreatic cancer risk.作者: 消息靈通 時(shí)間: 2025-3-26 00:51 作者: 外貌 時(shí)間: 2025-3-26 07:24
Raj Mani,Marco Romanelli,Vijay Shuklaatic cancer. All have unique advantages and drawbacks, hence the need to identify novel models for evaluation of pancreatic dysfunctions with high accuracy, sensitivity, and output for the diagnosis of the disease.作者: osteoclasts 時(shí)間: 2025-3-26 11:25
https://doi.org/10.1007/978-981-32-9393-9Metabolic syndrome; Diabetes; Pancreatic Cancer; Inflammation; Targeting molecules; metabolic disease作者: GONG 時(shí)間: 2025-3-26 16:21
978-981-32-9395-3Springer Nature Singapore Pte Ltd. 2019作者: AER 時(shí)間: 2025-3-26 19:40 作者: 膽大 時(shí)間: 2025-3-27 00:53
http://image.papertrans.cn/f/image/320351.jpg作者: 背叛者 時(shí)間: 2025-3-27 04:36 作者: Magnificent 時(shí)間: 2025-3-27 05:33 作者: fender 時(shí)間: 2025-3-27 12:09
Data Acquisition and Data Processing,es mellitus is a risk factor for developing pancreatic cancer. Supporting this, several studies have reported that four type 2 diabetes-associated polymorphisms, namely, rs8050136 of ., rs1387153 of ., rs11039149 of . and rs780094 of ., are associated risk factors for pancreatic cancer. Enhanced con作者: Cholesterol 時(shí)間: 2025-3-27 13:42 作者: 著名 時(shí)間: 2025-3-27 21:41 作者: ANIM 時(shí)間: 2025-3-27 22:58
Energy, Environment, and Sustainability), apoptosis, and cell cycle regulation. Over the past years, researchers have concentrated on the complexity of pancreatic cancer by mapping genetic mutations associated with it. In these efforts, the role of mitochondrion to the pathogenesis of pancreatic cancer has been neglected. However, recent作者: 針葉 時(shí)間: 2025-3-28 03:41
Practical Issues in Implementing IEEE 1588o conservative therapy. Diabetes is recently been correlated with mortality and morbidity from PDAC. The association between diabetes and PDAC stems from the structural association between the endocrine and exocrine pancreas and aberrant expression of hormones from islets. It can also result from ot作者: Adherent 時(shí)間: 2025-3-28 08:06 作者: 嬉耍 時(shí)間: 2025-3-28 11:19 作者: 打擊 時(shí)間: 2025-3-28 14:39 作者: temperate 時(shí)間: 2025-3-28 21:11
,On Ellis’ Theory of Quantities,ly coordinating regulatory mechanisms in order to control their activity. One of the key regulators responsible for the upregulation of Hsp90 is heat shock factor (HSF1), whose primary role is to bind heat shock elements (HSEs) with Hsp90 promoters. HSF1 functions by interacting with the transcripti作者: CAMP 時(shí)間: 2025-3-29 02:38
Measurement-Based Care in Mental Disordersabout 80% of PC patients have impaired glucose tolerance (IGT) or new onset of diabetes. Obesity, smoking, family history, genetic factors, insulin resistance (IR), and chronic pancreatitis are significant risk factors for PC. Several lines of evidences demonstrated that the production of insulin in作者: sed-rate 時(shí)間: 2025-3-29 06:01
Measurements in Musculoskeletal Radiologyn as well as the mechanisms underlying its resistance to therapy. However, the 5-year overall survival rates remain in the single digits for pancreatic cancer. In the last 20?years, there is a significant advance in the understanding of our basic immune system. In addition, the cancer community is a作者: perimenopause 時(shí)間: 2025-3-29 11:11 作者: harrow 時(shí)間: 2025-3-29 12:24 作者: 他姓手中拿著 時(shí)間: 2025-3-29 19:08
Biology of Pancreas and Possible Diseases,ate pancreas homeostasis. In this present chapter, we discuss the biology of the pancreas and its functions and the development of disorders and pathological conditions. We also examine how this accumulating knowledge is guiding the discovery of new diagnostic and therapeutic approaches to cure or p作者: delta-waves 時(shí)間: 2025-3-29 21:43
Pancreatitis: Clinical Aspects of Inflammatory Phenotypes, include hereditary, hyperlipidaemia, ., cystic fibrosis and usage of certain medicines such as oestrogen and .. The tests to detect pancreatitis include enzymes of the pancreas, liver and kidney. Signs of infection include fever/fatigue, anaemia and decreased electrolyte and calcium level. In acute作者: Foreshadow 時(shí)間: 2025-3-30 02:37 作者: Ventilator 時(shí)間: 2025-3-30 06:54 作者: 窩轉(zhuǎn)脊椎動(dòng)物 時(shí)間: 2025-3-30 11:22
Role of Mitochondria in Pancreatic Metabolism, Diabetes, and Cancer,dysfunction and multitude of diseases. Pancreas is one such organ that is frequently affected by mitochondrial perturbations. Over the past decade, significant amount of research has been done on mitochondria in pancreatic dysfunction. Heretofore, accumulating evidences suggest that pancreatitis, di作者: wangle 時(shí)間: 2025-3-30 16:08 作者: 上漲 時(shí)間: 2025-3-30 17:54 作者: 螢火蟲(chóng) 時(shí)間: 2025-3-30 22:48
