標(biāo)題: Titlebook: Etiopathogenesis; Yoshikuni Mizuno,Donald B. Calne (Director),R. Hor Book 1994 Birkh?user Boston 1994 cell.mortality.neurology.pathology.u [打印本頁] 作者: Traction 時(shí)間: 2025-3-21 17:55
書目名稱Etiopathogenesis影響因子(影響力)
書目名稱Etiopathogenesis影響因子(影響力)學(xué)科排名
書目名稱Etiopathogenesis網(wǎng)絡(luò)公開度
書目名稱Etiopathogenesis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Etiopathogenesis被引頻次
書目名稱Etiopathogenesis被引頻次學(xué)科排名
書目名稱Etiopathogenesis年度引用
書目名稱Etiopathogenesis年度引用學(xué)科排名
書目名稱Etiopathogenesis讀者反饋
書目名稱Etiopathogenesis讀者反饋學(xué)科排名
作者: 犬儒主義者 時(shí)間: 2025-3-21 21:14
Growth Factors in Parkinson’s Diseaseogical role and administration may have therapeutic effects (Hefti et al., 1989, 1990). Particular attention has been focused on Alzheimer’s disease (AD) and Parkinson’s disease (PD), the former because of the importance of nerve growth factor (NGF) in maintaining basal forebrain cholinergic neurons作者: 教唆 時(shí)間: 2025-3-22 01:39 作者: Onerous 時(shí)間: 2025-3-22 04:40
Neuroepidemiology of Parkinson’s Disease: Environmental InfluencesEtiological hypotheses need to be consistent with known epidemiologic factors, and descriptive epidemiologic observations may generate a new hypothesis. Analytic epidemiology seeks to identify risk factors that could lead to clues for causal associations with the disease.作者: Suggestions 時(shí)間: 2025-3-22 11:20
Discussion: Session 8 — 9 A.M. - 8 February 1993. Specifically he wondered how many cases of Alzheimer’s disease would fall into the category of genetically determined familial Alzheimer’s disease, how many were sporadic cases predominantly caused by environmental factors, and how many cases would fall in between these categories. George-Hyslop t作者: 協(xié)定 時(shí)間: 2025-3-22 13:39
Manganese-Induced Neurotoxicity is the twelfth most common element in the earth’s crust and the fourth most widely used metal in the world. Eight million tons of manganese metal are extracted annually, of which 94% is employed in the manufacture of steel. Electrolytic high-temperature reduction converts manganese oxide to ferroma作者: 協(xié)定 時(shí)間: 2025-3-22 18:06 作者: 晚間 時(shí)間: 2025-3-22 23:07 作者: 空洞 時(shí)間: 2025-3-23 04:53 作者: Jocose 時(shí)間: 2025-3-23 08:14
Mitochondrial Dysfunction, Aging, and Huntington’s Diseasezheimer’s disease is advancing age. Huntington’s disease typically has a onset in the thirties and forties. A number of studies show age-dependent increases in mitochondrial DNA deletions, oxidative damage to mitochondrial DNA, and reductions in activities of mitochondrial enzymes.作者: 儀式 時(shí)間: 2025-3-23 13:18
Energy Failure, Glutamate and Neuropathology: Relevance to Neurodegenerative DisordersA) receptors, allowing persistent activation of these receptors by endogenous glutamate is postulated as a mechanism to explain slow neuronal death in neurodegenerative disorders. Studies in rodents with mitochondrial respiratory chain toxins, aminoox-yacetic acid, 1-methyl-4-phenylpyridinium ion, a作者: essential-fats 時(shí)間: 2025-3-23 13:51 作者: OMIT 時(shí)間: 2025-3-23 18:39 作者: 怪物 時(shí)間: 2025-3-23 22:44
Discussion: 8—11 A.M. - February 9, 1993might be more widely distributed in the environment, and he further mentioned the disease in cattle called staffers which is found in the western part of North America and Canadian provinces and is caused by cattle being exposed to mildewed hay, or mildewed feed. Therefore, low-dose exposure may be 作者: 山頂可休息 時(shí)間: 2025-3-24 05:24 作者: Alpha-Cells 時(shí)間: 2025-3-24 07:02 作者: Pamphlet 時(shí)間: 2025-3-24 14:18
Deep Learning in Speaker Recognition,might be more widely distributed in the environment, and he further mentioned the disease in cattle called staffers which is found in the western part of North America and Canadian provinces and is caused by cattle being exposed to mildewed hay, or mildewed feed. Therefore, low-dose exposure may be possible under some circumstances.作者: deactivate 時(shí)間: 2025-3-24 18:38 作者: Nonthreatening 時(shí)間: 2025-3-24 20:00 作者: Focus-Words 時(shí)間: 2025-3-25 00:53 作者: 補(bǔ)角 時(shí)間: 2025-3-25 06:11
https://doi.org/10.1057/9781137331762After Olanow’s paper, Baimbridge (Vancouver) asked why manganese intoxication as a pallidal disease is not a hyperkinetic movement disorder. Olanow said that if the dorsal pallidum is involved, hypokinesia appears.作者: endocardium 時(shí)間: 2025-3-25 07:29
Discussion: Session 5 — 8 P.M. - 8 February 1993After Olanow’s paper, Baimbridge (Vancouver) asked why manganese intoxication as a pallidal disease is not a hyperkinetic movement disorder. Olanow said that if the dorsal pallidum is involved, hypokinesia appears.作者: observatory 時(shí)間: 2025-3-25 13:00 作者: 愛花花兒憤怒 時(shí)間: 2025-3-25 15:52
Advances in Research on Neurodegenerationhttp://image.papertrans.cn/e/image/316395.jpg作者: Aphorism 時(shí)間: 2025-3-25 21:23 作者: sebaceous-gland 時(shí)間: 2025-3-26 00:26 作者: 寬大 時(shí)間: 2025-3-26 07:18 作者: cliche 時(shí)間: 2025-3-26 11:08
Infrastructure and Connectivity,Etiological hypotheses need to be consistent with known epidemiologic factors, and descriptive epidemiologic observations may generate a new hypothesis. Analytic epidemiology seeks to identify risk factors that could lead to clues for causal associations with the disease.作者: headlong 時(shí)間: 2025-3-26 13:19
Life as a Humanitarian Practitioner. Specifically he wondered how many cases of Alzheimer’s disease would fall into the category of genetically determined familial Alzheimer’s disease, how many were sporadic cases predominantly caused by environmental factors, and how many cases would fall in between these categories. George-Hyslop t作者: Endemic 時(shí)間: 2025-3-26 18:05
Development Report on China’s New Media is the twelfth most common element in the earth’s crust and the fourth most widely used metal in the world. Eight million tons of manganese metal are extracted annually, of which 94% is employed in the manufacture of steel. Electrolytic high-temperature reduction converts manganese oxide to ferroma作者: 毗鄰 時(shí)間: 2025-3-26 21:08 作者: BILE 時(shí)間: 2025-3-27 04:34
William Ascher,Natalia Mirovitskayaalmer et al., 1988) (Fig. 1). Nitric oxide (NO) appears to be the biologically active nitrogen oxide released from the various nitric oxide synthase isoforms. In 1988, two laboratories using different methodology demonstrated that NO- is released from cytokine and lipopolysaccharide-treated murine m作者: 厚顏 時(shí)間: 2025-3-27 07:26 作者: 怎樣才咆哮 時(shí)間: 2025-3-27 10:04
zheimer’s disease is advancing age. Huntington’s disease typically has a onset in the thirties and forties. A number of studies show age-dependent increases in mitochondrial DNA deletions, oxidative damage to mitochondrial DNA, and reductions in activities of mitochondrial enzymes.作者: OUTRE 時(shí)間: 2025-3-27 16:50
Bert Hofman,Ella R. Gudwin,Kian Wie TheeA) receptors, allowing persistent activation of these receptors by endogenous glutamate is postulated as a mechanism to explain slow neuronal death in neurodegenerative disorders. Studies in rodents with mitochondrial respiratory chain toxins, aminoox-yacetic acid, 1-methyl-4-phenylpyridinium ion, a作者: Mnemonics 時(shí)間: 2025-3-27 19:18
https://doi.org/10.1057/9780230371675 of neurodegenerative diseases. While brain proteases have long been recognized for their importance to neuropeptide metabolism and protein turnover (Loh et al., 1984; Pope and Nixon, 1984), it is becoming increasingly clear that proteolysis plays a role in pathological neuronal degeneration as well作者: 性行為放縱者 時(shí)間: 2025-3-28 01:21 作者: 毀壞 時(shí)間: 2025-3-28 05:33
Deep Learning in Speaker Recognition,might be more widely distributed in the environment, and he further mentioned the disease in cattle called staffers which is found in the western part of North America and Canadian provinces and is caused by cattle being exposed to mildewed hay, or mildewed feed. Therefore, low-dose exposure may be 作者: harpsichord 時(shí)間: 2025-3-28 09:43
https://doi.org/10.1007/978-1-4684-9203-3cell; mortality; neurology; pathology; urology作者: SHRIK 時(shí)間: 2025-3-28 11:18
Book 1994iew the neurodegenerative disorders and to attempt to explore how progress might be made in this field, as the neurodegenerative disorders have been emerging to be one of the major causes of morbidity and mortality in modern societies. The first meeting took place in Seefeld, Austria, in February 19作者: 小步舞 時(shí)間: 2025-3-28 18:15 作者: Hearten 時(shí)間: 2025-3-28 19:37 作者: 友好 時(shí)間: 2025-3-29 00:50
William Ascher,Natalia Mirovitskayasoforms. In 1988, two laboratories using different methodology demonstrated that NO- is released from cytokine and lipopolysaccharide-treated murine macrophages (Hibbs et al., 1988; Marietta et al., 1988). These experiments unequivocally established NO- as a low molecular weight biosynthetic product of mammalian cells.作者: 抗原 時(shí)間: 2025-3-29 03:23 作者: 雜役 時(shí)間: 2025-3-29 10:10 作者: 中子 時(shí)間: 2025-3-29 13:37 作者: Exuberance 時(shí)間: 2025-3-29 18:52 作者: 吊胃口 時(shí)間: 2025-3-29 22:17 作者: 浸軟 時(shí)間: 2025-3-30 03:28
https://doi.org/10.1057/9780230371675r free calcium. Because the role of calpain 1 in neurodegenerative processes has been the subject of recent review (Seubert and Lynch, 1990; Siman, 1990; 1992), the present brief review is followed by a description of new studies directed at the localization, timing, and mode of activation of calpain 1 in animal models of neuronal damage.作者: 紅潤 時(shí)間: 2025-3-30 05:36 作者: ornithology 時(shí)間: 2025-3-30 11:19 作者: Contort 時(shí)間: 2025-3-30 13:58
Iron and Parkinson’s Diseasee et al. (1924), demonstrating a substantial increase of the iron content in Parkinsonian substantia nigra (SN). This has been confirmed repeatedly, for example, in a series of very recent studies which a variety of techniques including magnetic resonance imaging (MRI) for determination of iron have been used (see Youdim et al., 1993a).作者: 虛構(gòu)的東西 時(shí)間: 2025-3-30 17:17
Proteases and Pathological Neurodegenerationr free calcium. Because the role of calpain 1 in neurodegenerative processes has been the subject of recent review (Seubert and Lynch, 1990; Siman, 1990; 1992), the present brief review is followed by a description of new studies directed at the localization, timing, and mode of activation of calpain 1 in animal models of neuronal damage.
