標題: Titlebook: Etiology and Morphogenesis of Congenital Heart Disease; From Gene Function a Toshio Nakanishi,Roger R. Markwald,Hiroyuki Yamagi Book‘‘‘‘‘‘‘ [打印本頁] 作者: arouse 時間: 2025-3-21 19:35
書目名稱Etiology and Morphogenesis of Congenital Heart Disease影響因子(影響力)
書目名稱Etiology and Morphogenesis of Congenital Heart Disease影響因子(影響力)學科排名
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書目名稱Etiology and Morphogenesis of Congenital Heart Disease網(wǎng)絡公開度學科排名
書目名稱Etiology and Morphogenesis of Congenital Heart Disease被引頻次
書目名稱Etiology and Morphogenesis of Congenital Heart Disease被引頻次學科排名
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書目名稱Etiology and Morphogenesis of Congenital Heart Disease年度引用學科排名
書目名稱Etiology and Morphogenesis of Congenital Heart Disease讀者反饋
書目名稱Etiology and Morphogenesis of Congenital Heart Disease讀者反饋學科排名
作者: 排名真古怪 時間: 2025-3-21 21:20
Working with/in Deleuzo-Guattarian Ideas,ription factors and small molecules that reprogram human fibroblasts into cardiomyocyte-like cells and are testing these in large animals. Knowledge regarding the early steps of cardiac differentiation in vivo has led to effective strategies to generate necessary cardiac cell types for regenerative 作者: condemn 時間: 2025-3-22 02:45 作者: intrigue 時間: 2025-3-22 08:28
Working with Photography Projects,different mechanisms of glucose and lactate metabolism. Based on these findings, we succeeded in metabolically selecting cardiomyocytes using glucose-free and lactate-supplemented medium, with up to 99?% purity and no teratoma formation. Using our aggregation technique, we also showed that >90?% of 作者: 易發(fā)怒 時間: 2025-3-22 10:41
Developing Property Rights Theoryto override biochemical or structural deficits of CHD associated with valve and septal signaling hubs or fibroblast/myocyte interactions. Preliminary tests of the model indicate remedial potential for cardiac injuries.作者: Glucose 時間: 2025-3-22 14:22
Dangeni,Rui He,Natthaphon Tripornchaisakunborn human fetal population. Clinically, congenital heart disease patients with ciliary dysfunction were found to have more respiratory symptoms and disease, a finding with significant clinical implications, as congenital heart disease patients undergoing surgical palliation often have respiratory作者: Glucose 時間: 2025-3-22 20:42 作者: deviate 時間: 2025-3-23 00:30
https://doi.org/10.1007/978-1-4302-0802-0ontal septum and, in the other taxa studied, into the folding part of the interventricular septum. The vertical septum, indistinct in most reptiles, arises in crocodilians and pythonids at the posterior ventricular wall. It is homologous to the inlet septum in mammals and birds. Eventually, the vari作者: Nutrient 時間: 2025-3-23 01:49 作者: Desert 時間: 2025-3-23 05:51
The Arterial Epicardium: A Developmental Approach to Cardiac Disease and Repaires dormant, losing the activity of many genes..In human cardiac diseases, both arterial and venous epicardium can be activated again into EPDCs. The epicardial reactivation observed after experimental myocardial infarction and during aneurysm formation of the ascending aorta provides clinical releva作者: Fecundity 時間: 2025-3-23 11:03
Future Treatment of Heart Failure Using Human iPSC-Derived Cardiomyocytesdifferent mechanisms of glucose and lactate metabolism. Based on these findings, we succeeded in metabolically selecting cardiomyocytes using glucose-free and lactate-supplemented medium, with up to 99?% purity and no teratoma formation. Using our aggregation technique, we also showed that >90?% of 作者: 合適 時間: 2025-3-23 14:43 作者: 熒光 時間: 2025-3-23 18:03
Role of Cilia and Left-Right Patterning in Congenital Heart Diseaseunborn human fetal population. Clinically, congenital heart disease patients with ciliary dysfunction were found to have more respiratory symptoms and disease, a finding with significant clinical implications, as congenital heart disease patients undergoing surgical palliation often have respiratory作者: employor 時間: 2025-3-23 23:53
Meis1 Regulates Postnatal Cardiomyocyte Cell Cycle Arresteis1 in cardiomyocytes decreased neonatal myocyte proliferation and inhibited neonatal heart regeneration. Finally, we show that Meis1 is required for transcriptional activation of the synergistic CDK inhibitors p15, p16, and p21. These results identify Meis1 as a critical transcriptional regulator 作者: perjury 時間: 2025-3-24 05:08 作者: innate 時間: 2025-3-24 08:38
Etiology and Morphogenesis of Congenital Heart DiseaseFrom Gene Function a作者: 邊緣帶來墨水 時間: 2025-3-24 13:31
Etiology and Morphogenesis of Congenital Heart Disease978-4-431-54628-3作者: 使堅硬 時間: 2025-3-24 15:20 作者: corpus-callosum 時間: 2025-3-24 21:28
Stewart Johnstone,Adrian Wilkinsonith impact on development, disease, and repair. The epicardium differentiates from the proepicardial organ located at the venous pole (vPEO). The differentiation capacities of the vPEO into epicardium-derived cells (EPDCs) have been extensively described. A hitherto escaped part of the epicardium de作者: Cacophonous 時間: 2025-3-25 01:39
Forest vitality and stress implications,Direct injection of bone marrow-derived cells and isolated skeletal myoblasts has already been used clinically as a method to improve cardiac function by regenerating cardiac muscle cells and blood vessels. The research on reconstructing functional three-dimensional (3D) cardiac grafts using tissue 作者: 跳脫衣舞的人 時間: 2025-3-25 03:48
Working with Photography Projects,em with this treatment strategy and the successful clinical application of regenerative medicine is eagerly awaited. To this end, we developed a novel method to generate human induced pluripotent stem cells (iPSCs) from circulating human T lymphocytes using Sendai virus containing Yamanaka factors. 作者: 不溶解 時間: 2025-3-25 08:10
Developing Property Rights Theoryich mutations disrupt fundamental cell biological functions in cardiac progenitor cells in a lineage-specific manner. Based on the frequency of heart defects seen in a clinical setting, we emphasize two signaling hubs – . (e.g., .) ., . (.). We discuss them in the context of valve and septal develop作者: manifestation 時間: 2025-3-25 12:48 作者: Audiometry 時間: 2025-3-25 18:18
The Future of Public Sector Leadership,mmetry breaking at the node, differential patterning of the lateral plate by a signaling molecule Nodal, and finally situs-specific organogenesis. Breaking of L-R symmetry in the mouse embryo takes place in the ventral node, where two types of cilia are found. Whereas centrally located motile cilia 作者: BOOR 時間: 2025-3-25 21:55
Dangeni,Rui He,Natthaphon Tripornchaisakng congenital heart disease. This is supported by human clinical studies, which showed a high prevalence of ciliary dysfunction and respiratory symptoms and disease in patients with congenital heart disease. Our mouse studies indicate this involves essential roles for both primary and motile cilia i作者: ACME 時間: 2025-3-26 04:00 作者: Sad570 時間: 2025-3-26 04:19
https://doi.org/10.1007/978-94-017-9950-8 (CPCs)”. It is believed that multipotent CPCs are specified among the most anterior part of lateral plate mesoderm as belonging to the two anatomical fields; the first heart field (FHF), which is the future left ventricle and atria, and the second heart field (SHF), which is the future right ventri作者: 過剩 時間: 2025-3-26 10:31
Alexis Ocampo,Fabio Bella,J¨rgen MJ¨nch1078–1080, 2011; Proc Natl Acad Sci U S A 110:187–92, 2013). However, this regenerative capacity is lost by postnatal day 7 and the mechanisms of cardiomyocyte cell cycle arrest remain unclear. The homeodomain transcription factor Meis1 is required for normal cardiac development but its role in card作者: CAMEO 時間: 2025-3-26 13:22
Developing Socio-Emotional Intelligence,r cardiac development may cause congenital heart disease (CHD), manifested in the newborn or in the adult. Notch is an ancient, highly conserved signaling pathway that communicates adjacent cells to regulate cell fate specification, differentiation, and tissue patterning. Mutations in Notch signalin作者: 滑稽 時間: 2025-3-26 18:11
https://doi.org/10.1007/978-1-4302-0802-0ed a novel function of the epicardium in species with partial or complete ventricular septation including reptiles, birds and mammals. Most reptiles have a complex ventricle with three cava, partially separated by the horizontal and vertical septa. Crocodilians, birds and mammals, however, have comp作者: EXULT 時間: 2025-3-26 21:17
Implement Caching for Solutionse endoderm and the yolk syncytial layer (YSL) to form cardiac tube. The endoderm functions as a foothold for CPCs as evidenced by the endodermal mutants (./., ., ., ./., and .) showing two hearts (cardia bifida) [1]. Furthermore, mutant zebra fish (.) lacking sphingosine-1-phosphate (S1P) transporte作者: Oratory 時間: 2025-3-27 03:50 作者: Inexorable 時間: 2025-3-27 06:45
Strategy Implementation and Management, high frequency of congenital cardiovascular malformations (CM), such as ventricular and atrial septal defects [1]. Some studies have demonstrated that these cases were due to gene mutations and neural crest abnormality. Infants with CH and CM have been shown to have significantly lower T. levels th作者: Leaven 時間: 2025-3-27 11:47
Toshio Nakanishi,Roger R. Markwald,Hiroyuki YamagiFocuses on the etiology and morphogenesis of congenital heart disease.Includes contributions by an international group of distinguished scientists and clinicians.Highlights the latest research topics 作者: largesse 時間: 2025-3-27 15:35 作者: 多嘴 時間: 2025-3-27 18:30 作者: 形容詞 時間: 2025-3-28 00:07
https://doi.org/10.1007/978-4-431-54628-3Cellular Interaction; Congenital Heart Disease; iPS cells; Gene functions; Growth factors; Transcription 作者: meretricious 時間: 2025-3-28 05:16
Reprogramming Approaches to Cardiovascular Disease: From Developmental Biology to Regenerative Medic networks that guide early differentiation of cardiac progenitors and later morphogenetic events during cardiogenesis. We found that networks of transcription factors and miRNAs function through intersecting positive and negative feedback loops to reinforce differentiation and proliferation decision作者: 雪上輕舟飛過 時間: 2025-3-28 09:02
The Arterial Epicardium: A Developmental Approach to Cardiac Disease and Repairith impact on development, disease, and repair. The epicardium differentiates from the proepicardial organ located at the venous pole (vPEO). The differentiation capacities of the vPEO into epicardium-derived cells (EPDCs) have been extensively described. A hitherto escaped part of the epicardium de作者: surmount 時間: 2025-3-28 11:34 作者: 暗諷 時間: 2025-3-28 16:13 作者: amorphous 時間: 2025-3-28 21:18 作者: 貪婪地吃 時間: 2025-3-28 23:33 作者: 鍍金 時間: 2025-3-29 06:34 作者: 高興去去 時間: 2025-3-29 09:58
Role of Cilia and Left-Right Patterning in Congenital Heart Diseaseng congenital heart disease. This is supported by human clinical studies, which showed a high prevalence of ciliary dysfunction and respiratory symptoms and disease in patients with congenital heart disease. Our mouse studies indicate this involves essential roles for both primary and motile cilia i作者: Acumen 時間: 2025-3-29 12:29
Pulmonary Arterial Hypertension in Patients with Heterotaxy/Polysplenia Syndromesystemic-to-pulmonary shunt. However, its etiology is uncertain and its management is not well established. There was only a Japanese report about PAH in consecutive patients with heterotaxy/polysplenia syndrome [1]. They seemed to develop pulmonary vascular obstructive disease earlier and more seve作者: FLOUR 時間: 2025-3-29 19:38 作者: Functional 時間: 2025-3-29 22:41
Meis1 Regulates Postnatal Cardiomyocyte Cell Cycle Arrest1078–1080, 2011; Proc Natl Acad Sci U S A 110:187–92, 2013). However, this regenerative capacity is lost by postnatal day 7 and the mechanisms of cardiomyocyte cell cycle arrest remain unclear. The homeodomain transcription factor Meis1 is required for normal cardiac development but its role in card作者: 得意牛 時間: 2025-3-30 02:21
Intercellular Signaling in Cardiac Development and Disease: The NOTCH pathwayr cardiac development may cause congenital heart disease (CHD), manifested in the newborn or in the adult. Notch is an ancient, highly conserved signaling pathway that communicates adjacent cells to regulate cell fate specification, differentiation, and tissue patterning. Mutations in Notch signalin作者: certitude 時間: 2025-3-30 04:41 作者: 譏諷 時間: 2025-3-30 11:07 作者: 阻止 時間: 2025-3-30 12:28 作者: 束以馬具 時間: 2025-3-30 17:54 作者: 積習已深 時間: 2025-3-30 21:00 作者: 防銹 時間: 2025-3-31 02:19 作者: definition 時間: 2025-3-31 06:43 作者: construct 時間: 2025-3-31 13:02 作者: abysmal 時間: 2025-3-31 15:01 作者: buoyant 時間: 2025-3-31 18:50
Narsamma Lingam,Govinda Ishwar Lingam in consecutive patients with heterotaxy/polysplenia syndrome [1]. They seemed to develop pulmonary vascular obstructive disease earlier and more severe than expected, even in cases with only pre-tricuspid systemic-to-pulmonary shunt although more detailed analysis is required.作者: 恃強凌弱的人 時間: 2025-4-1 01:10 作者: Kidney-Failure 時間: 2025-4-1 05:04
Roles of Motile and Immotile Cilia in Left-Right Symmetry Breakinggenerate a leftward fluid flow, peripherally located immotile cilia sense a flow-dependent signal. Although Ca. signaling is implicated in flow sensing, it is still not clear what triggers Ca. signaling, a determinant molecule transported by the flow or mechanical force induced by the flow.作者: 馬具 時間: 2025-4-1 06:05
S1P-S1p2 Signaling in Cardiac Precursor Cells Migrationr which is expressed in the YSL show two hearts [2], indicating the essential role for S1P-mediated signal in cardiac development. This is also supported by a S1p2 receptor mutant (.) which exhibits two hearts [3]. However, it is still unclear how S1P released from YSL regulates CPC migration.作者: CURT 時間: 2025-4-1 11:25 作者: 惡心 時間: 2025-4-1 16:14
