標(biāo)題: Titlebook: Epigenetics and Neuroendocrinology; Clinical Focus on Ps Dietmar Spengler,Elisabeth Binder Book 2016 Springer International Publishing Swit [打印本頁(yè)] 作者: retort 時(shí)間: 2025-3-21 18:36
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology影響因子(影響力)
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology被引頻次
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology被引頻次學(xué)科排名
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology年度引用
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology年度引用學(xué)科排名
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology讀者反饋
書(shū)目名稱(chēng)Epigenetics and Neuroendocrinology讀者反饋學(xué)科排名
作者: 高射炮 時(shí)間: 2025-3-21 20:30 作者: critique 時(shí)間: 2025-3-22 00:51
2191-2262 role in the development of the neuroendocrine axes as well as in the mediation of gene-environment interactions in stress-related psychiatry disorders. ??.978-3-319-80681-5978-3-319-29901-3Series ISSN 2191-2262 Series E-ISSN 2191-2270 作者: lipoatrophy 時(shí)間: 2025-3-22 05:39
A Marxist Theory of International Relations,sms of . could also be linked to epigenetic changes evoked in response to stress exposure, giving rise to “epialleles.” Here we present and discuss . as stress reactivity gene describing the molecular genetics and epigenetics of . and illustrate FKBP5 as a model at the interface of genetics, epigenetics, and neuroendocrinology.作者: abracadabra 時(shí)間: 2025-3-22 09:10 作者: IRS 時(shí)間: 2025-3-22 15:02
Book 2016al interactions between the central nervous system (CNS) and endocrine systems in the coordination of homeostasis and various physiological responses from adaptation to disease. Most recently, epigenetic mechanisms were recognized for their role in the development of the neuroendocrine axes as well 作者: IRS 時(shí)間: 2025-3-22 20:46 作者: 千篇一律 時(shí)間: 2025-3-23 00:02
https://doi.org/10.1057/9780230107755atterns. Impairment of the epigenetic control of . expression may contribute to human pathological conditions including substance dependence, depression, and chronic pain. Epigenetic and environmental factors may mechanistically converge on the . CpG-SNPs associated with a risk for alcohol dependenc作者: 阻塞 時(shí)間: 2025-3-23 03:43 作者: intrude 時(shí)間: 2025-3-23 09:24 作者: paleolithic 時(shí)間: 2025-3-23 10:26 作者: Harass 時(shí)間: 2025-3-23 16:44 作者: degradation 時(shí)間: 2025-3-23 21:42
Continued Decline of Indian Languages,een early social environment, epigenetics, and long-term outcomes. The implications of maternal care on DNA methylation as well as the importance of maternal well-being during pregnancy to prevent future health problems are considered.作者: licence 時(shí)間: 2025-3-24 00:39
Queenston and Kelley Preschools,tion in the blood of BPD subjects. Especially, GR/NR3C1 methylation was positively associated with childhood maltreatment and clinical severity in BPD. These data may provide new insights into epigenetic mechanisms underlying the pathogenesis of borderline personality disorder.作者: 諄諄教誨 時(shí)間: 2025-3-24 05:20
https://doi.org/10.1007/978-1-349-25568-9ing gene expression. Here, we provide an overview of the functional organization and epigenetic regulation of the murine and human oxytocin receptor gene..Studies in mice have shown that the oxytocin receptor gene (.) is epigenetically regulated by DNA methylation with experience- and tissue-specifi作者: 規(guī)章 時(shí)間: 2025-3-24 06:57 作者: 并置 時(shí)間: 2025-3-24 11:14 作者: capsaicin 時(shí)間: 2025-3-24 16:01 作者: LATE 時(shí)間: 2025-3-24 19:20 作者: Affection 時(shí)間: 2025-3-25 01:21
Epigenetic Alterations to , and , and the Developmental Origins of Mental Disease Riskter reviews the evidence linking (1) prenatal exposures to epigenetic modification of genes involved in the regulation of cortisol (. and .) and (2) how such epigenetic alterations can in turn lead to changes in offspring mental disease risk.作者: 鞏固 時(shí)間: 2025-3-25 03:57
Intergenerational Effects of PTSD on Offspring Glucocorticoid Receptor Methylationies have prompted investigation into methylation of the . promoter, which regulates transcription of the glucocorticoid receptor gene. Here, we review evidence of . methylation alterations in offspring of parents exposed to extreme stress and offspring of parents who developed PTSD in response to ex作者: 建筑師 時(shí)間: 2025-3-25 07:55
Epigenetic Biomarkers for Early-Life Adversityment and increase the vulnerability to psychopathology epigenetic regulation. This chapter will review the findings from human studies that support this hypothesis. We will describe epigenetic changes associated with early-life adversity and will discuss their potential as biomarkers for early-life 作者: Concomitant 時(shí)間: 2025-3-25 12:39
Epigenetic Biomarkers of Prenatal Maternal Stresseen early social environment, epigenetics, and long-term outcomes. The implications of maternal care on DNA methylation as well as the importance of maternal well-being during pregnancy to prevent future health problems are considered.作者: Entirety 時(shí)間: 2025-3-25 19:10 作者: 笨拙的我 時(shí)間: 2025-3-25 22:58
Oxytocin Receptors and Neurobehavioring gene expression. Here, we provide an overview of the functional organization and epigenetic regulation of the murine and human oxytocin receptor gene..Studies in mice have shown that the oxytocin receptor gene (.) is epigenetically regulated by DNA methylation with experience- and tissue-specifi作者: 愚笨 時(shí)間: 2025-3-26 01:57 作者: 后退 時(shí)間: 2025-3-26 04:41
Epialleles glucocorticoid receptor (GR) takes the central stage in balancing the activity of the HPA axis through its negative feedback on the peptides corticotrophin-releasing hormone and adrenocorticotropic hormone. The activity of GR, in turn, is also controlled by several cofactors including FK506-binding作者: recession 時(shí)間: 2025-3-26 09:46 作者: 預(yù)知 時(shí)間: 2025-3-26 14:27 作者: STENT 時(shí)間: 2025-3-26 19:26
Epigenetic Risk Factors for Diseases: A Transgenerational Perspectivevelopment and adulthood. While genetic features are embedded in the DNA sequence inherited from parents, non-genetic features (which include epigenetic modifications) are acquired through experiences and environmental exposure across life. However, it is now acknowledged that non-genetic features ca作者: ITCH 時(shí)間: 2025-3-26 23:03
Epigenetic Alterations to , and , and the Developmental Origins of Mental Disease Riskpring over the life course and contribute to the intergenerational transmission of disease risk. Many psychiatric conditions are now thought to originate in part .. As such, a myriad of gestational exposures may contribute to the biological embedding of poor mental health for the next generation. In作者: AWL 時(shí)間: 2025-3-27 02:16
Intergenerational Effects of PTSD on Offspring Glucocorticoid Receptor Methylation parents confers vulnerability to PTSD in offspring. This is underlined by the observation that offspring born to individuals with PTSD exhibit low baseline cortisol levels, a neuroendocrine risk factor for PTSD. Several mechanisms have been proposed to mediate intergenerational inheritance of PTSD,作者: 鎮(zhèn)痛劑 時(shí)間: 2025-3-27 06:22
Epigenetic Biomarkers for Early-Life Adversityl periods in childhood is associated with an increased risk for psychopathology in adulthood, such as mood disorders and mental illness. We have only recently started to uncover some of the mechanisms that may contribute to behavioral changes induced by early-life adversity. Epigenetics modification作者: 抗體 時(shí)間: 2025-3-27 13:22 作者: 延期 時(shí)間: 2025-3-27 13:38 作者: 衍生 時(shí)間: 2025-3-27 21:48 作者: Seminar 時(shí)間: 2025-3-27 23:11 作者: humectant 時(shí)間: 2025-3-28 05:37
https://doi.org/10.1007/978-3-319-29901-3DNA methylation; epigenetic biomarkers; gene-environmental interactions; neurendocrine differentiation; 作者: Cpap155 時(shí)間: 2025-3-28 07:50
A Marxist Theory of International Relations, glucocorticoid receptor (GR) takes the central stage in balancing the activity of the HPA axis through its negative feedback on the peptides corticotrophin-releasing hormone and adrenocorticotropic hormone. The activity of GR, in turn, is also controlled by several cofactors including FK506-binding作者: subacute 時(shí)間: 2025-3-28 11:31
American Hegemony after the Great Recessionncluding unipolar and bipolar depression and posttraumatic stress disorder (PTSD). Consequently, irregularities in the mRNA expression or protein levels of neuropeptide hormones and receptors involved in related stress pathways can trigger these neurological disorders. As a critical modulator of the作者: PLIC 時(shí)間: 2025-3-28 18:12 作者: EXPEL 時(shí)間: 2025-3-28 21:41
Stephen King: Powers of Horror,velopment and adulthood. While genetic features are embedded in the DNA sequence inherited from parents, non-genetic features (which include epigenetic modifications) are acquired through experiences and environmental exposure across life. However, it is now acknowledged that non-genetic features ca作者: alabaster 時(shí)間: 2025-3-28 23:47 作者: Comedienne 時(shí)間: 2025-3-29 06:51 作者: paltry 時(shí)間: 2025-3-29 10:06
Ethnicity and American Popular Culture,l periods in childhood is associated with an increased risk for psychopathology in adulthood, such as mood disorders and mental illness. We have only recently started to uncover some of the mechanisms that may contribute to behavioral changes induced by early-life adversity. Epigenetics modification作者: Nausea 時(shí)間: 2025-3-29 13:26
Continued Decline of Indian Languages,tion in response to prenatal stress has been shown in animal model studies. Although prenatal time represents a sensitive period of development, little is known about the impact of maternal stress during pregnancy on DNA methylation during the life span in humans. In this review, we provide a brief 作者: 呼吸 時(shí)間: 2025-3-29 19:25
Queenston and Kelley Preschools, well established, little is known on the pathogenesis of BPD. Epigenetic alterations are a hallmark for abnormal gene expression and could be involved in the etiology of BPD. These epigenetic changes are mainly classified into altered histone modifications and DNA methylation. Here, we summarize re
