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標(biāo)題: Titlebook: Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer; Andrew F. Olshan (Professor and Chair) Book 2010 Springer-Verlag New Y [打印本頁(yè)]

作者: 寓言    時(shí)間: 2025-3-21 18:21
書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer影響因子(影響力)




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer影響因子(影響力)學(xué)科排名




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer網(wǎng)絡(luò)公開(kāi)度




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer被引頻次




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer被引頻次學(xué)科排名




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer年度引用




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer年度引用學(xué)科排名




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer讀者反饋




書目名稱Epidemiology, Pathogenesis, and Prevention of Head and Neck Cancer讀者反饋學(xué)科排名





作者: Monolithic    時(shí)間: 2025-3-21 23:32

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作者: surrogate    時(shí)間: 2025-3-22 06:30
https://doi.org/10.1007/978-3-0348-6056-7for approximately 5.2% of the global cancer burden [6] . According to the Centers for Disease Control, approximately 20,000 cancers in the United States were attributable to HPV infection each year during the period from 1998 through 2003 [4, 7] . HPV infection is therefore a major cause of morbidit
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作者: 業(yè)余愛(ài)好者    時(shí)間: 2025-3-22 20:41
https://doi.org/10.1007/978-1-4419-1472-9cancer; chemoprevention; classification; epidemiology; molecular epidemiology; prevention
作者: 有危險(xiǎn)    時(shí)間: 2025-3-23 01:15

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作者: scoliosis    時(shí)間: 2025-3-23 08:19
978-1-4899-8288-9Springer-Verlag New York 2010
作者: NOTCH    時(shí)間: 2025-3-23 11:44
Anna Fishbein,Stephen H. Sheldoned on the cell of origin (mucosal epithelium) and histologic features. However, distinctive phenotypic patterns and genotypic correlates increasingly suggest that it might be more accurately thought of as consisting of different entities. These observations have been evolving in the context of the m
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作者: 工作    時(shí)間: 2025-3-24 00:04
,Verbreitung und Wesen der Elektrizit?t,ted to the two risk factors [1, 2] . Each is a head and neck cancer risk factor independent of the other factor [3] . An interaction between tobacco and alcohol on the risk of head and neck cancers was reported in the 1970s and is a paradigm of interaction between two environmental factors in human
作者: 進(jìn)入    時(shí)間: 2025-3-24 03:07

作者: Anonymous    時(shí)間: 2025-3-24 07:18

作者: 巫婆    時(shí)間: 2025-3-24 14:13
arcinoma, SCCHN). The focus was on factors that have been examined in multiple epidemiologic studies, including occupational exposures, gastroesophageal reflux, and oral health. We consider the study design, conduct, and analytic approach and methods that may have influenced the interpretation of re
作者: PLE    時(shí)間: 2025-3-24 16:09
Historische Diskursanalyse der Literatureefourths of all oral and pharyngeal cancers in the U.S. and an even higher attributable fraction of laryngeal cancers [1] . Othe r exposures include human papillomavirus, dietary, occupational, medical, and other factors that may also contribute to the etiology of this disease; however, only a frac
作者: Obsessed    時(shí)間: 2025-3-24 20:12

作者: Frequency-Range    時(shí)間: 2025-3-25 02:46
Pathophysiology of Allergic Reactions,rs of the oral cavity, pharynx, and larynx in the United States, and approximately 8,140 men and 3,120 women are expected to die from these cancers [1] . This chapter reviews the descriptive patterns of squamous cell carcinomas of the oral cavity, pharynx, and larynx.
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Ens Necessarium und Ens Realissimum,nds to halt, reverse, or prevent the initial phase of carcinogenesis or the progression of neoplastic cells to cancer [1] . The following three key biologic features of malignant transformation support the development of chemopreventive strategies: it involves the multipath, multistep, and multifocal disruption of normal cellular function.
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Chemoprevention of Head and Neck Cancers,nds to halt, reverse, or prevent the initial phase of carcinogenesis or the progression of neoplastic cells to cancer [1] . The following three key biologic features of malignant transformation support the development of chemopreventive strategies: it involves the multipath, multistep, and multifocal disruption of normal cellular function.
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Classification, Clinical Features, and Molecular Genetic Models,atures. Taken together, clinicians seek to use new and traditional tumor features to categorize tumors, predict their potential clinical course, and select appropriate strategies for their detection and treatment.
作者: 違抗    時(shí)間: 2025-3-27 01:51
Host Susceptibility and Molecular Epidemiology, assessment process. This chapter explores some of the host factors that modulate susceptibility to epithelial carcinogenesis induced by tobacco and alcohol exposures and describes some relevant molecular epidemiology association studies of single nucleotide polymorphism (SNP) and risk of SCCHN.
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作者: Amylase    時(shí)間: 2025-3-27 18:56
n addition, it will consider approaches to prevention and pr.The purpose of this book is to provide a current perspective on the epidemiology head and neck cancer. Cancers of the oral cavity, pharynx, and larynx comprise an important group of tumors with diverse international patterns of incidence a
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Descriptive Epidemiology: U.S. Patterns,rs of the oral cavity, pharynx, and larynx in the United States, and approximately 8,140 men and 3,120 women are expected to die from these cancers [1] . This chapter reviews the descriptive patterns of squamous cell carcinomas of the oral cavity, pharynx, and larynx.
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作者: Melodrama    時(shí)間: 2025-3-28 15:59
Risk Factors: Tobacco and Alcohol,ted to the two risk factors [1, 2] . Each is a head and neck cancer risk factor independent of the other factor [3] . An interaction between tobacco and alcohol on the risk of head and neck cancers was reported in the 1970s and is a paradigm of interaction between two environmental factors in human
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