作者: Geyser 時(shí)間: 2025-3-21 23:04
Physiology and Cardioprotection of the Epicardial Adipose Tissue, the heart is complex and not completely understood. EAT functions can be distinguished in (1) nutritional, (2) metabolic, (3) thermogenic, (4) regulatory, and (5) mechanical. Under normal physiological EAT serves as a buffer, absorbing fatty acids and protecting the heart against high fatty acids l作者: Fabric 時(shí)間: 2025-3-22 03:39 作者: Palatial 時(shí)間: 2025-3-22 05:35
Pathology and Cardiotoxicity of the Epicardial Adipose Tissue,eart and may cause a metabolic cardiomyopathy. Epicardial fat (EAT) can be considered ectopic fat accumulation of the heart. Epicardial fat and intra-myocardial triglycerides content are related. Excessive EAT can produce lipotoxic effects throughout an abnormal lipid deposition and fatty infiltrati作者: 道學(xué)氣 時(shí)間: 2025-3-22 09:38
Echocardiographic Imaging of the Epicardial Adipose Tissue,ws permit the most accurate measurement of EAT thickness overlying the right ventricle. EAT thickness is generally identified as the echo-free space between the outer wall of the myocardium and the visceral layer of pericardium and is measured perpendicularly on the free wall of the right ventricle 作者: Assemble 時(shí)間: 2025-3-22 15:41
Computed Tomography Imaging of Epicardial Adipose Tissue,tomated three-dimensional quantification, non-contrast- or contrast-enhanced cardiac CT imaging allows for reliable assessment of both epicardial adipose tissue volume and attenuation. Once, CT imaging is performed, information of epicardial fat is readily available. There is overwhelming data on th作者: Assemble 時(shí)間: 2025-3-22 20:05
Magnetic Resonance Imaging of the Epicardial Adipose Tissue,gold standard technique to measure the adipose tissue, also because it does not require ionizing radiations. Dedicated cardiac magnetic resonance (CMR) imaging protocols have been developed to assess EAT volume. CMR provides a precise and noninvasive assessment of both EAT thickness and volume. As E作者: 肥料 時(shí)間: 2025-3-23 00:08
Coronary Artery Disease and Epicardial Adipose Tissue,tery disease (CAD), as emerged only recently. The mechanisms through which epicardial fat can cause atherosclerosis are complex and multifactorial. Its anatomical proximity to the heart, the unique transcriptome, and intense proteasome are the major atherogenic factors of the epicardial adipose tiss作者: GRE 時(shí)間: 2025-3-23 03:58
Perivascular Adipose Tissue and Atherosclerosis,nd gaseous messengers, which affect the cardiovascular system in both endocrine and paracrine manners. The diversity of adipose tissue biology has led to a shift in our perception of fat as an entity, highlighting the importance of regional variability. In that context, perivascular adipose tissue, 作者: NOVA 時(shí)間: 2025-3-23 08:20 作者: 抱狗不敢前 時(shí)間: 2025-3-23 12:09
Heart Failure and Epicardial Adipose Tissue,le, although results are not univocal. Experimental studies suggest a potential protective role of the brown fat features of EAT against the systolic heart failure. A function of EAT p53 and adiponectin has been also suggested in patients with heart failure. A neuromodulatory role of EAT in heart fa作者: 暖昧關(guān)系 時(shí)間: 2025-3-23 16:33 作者: anus928 時(shí)間: 2025-3-23 21:18 作者: Permanent 時(shí)間: 2025-3-24 00:16
Adrenal Secretome and Epicardial Adipose Tissue,aldosterone can lead to accumulation of epicardial adipose tissue (EAT), interfering with functional state. In fact, increased mineralocorticoid signaling is critically involved in the secretion of proinflammatory cytokines by EAT with consequent profibrotic cascade activation in the myocardium. In 作者: HARD 時(shí)間: 2025-3-24 03:38
Targeting the Epicardial Adipose Tissue,icardial fat can serve as a target for pharmaceutical agents targeting the adipose tissue. EAT has shown to significantly respond to thiazolidinediones (TZD), glucagon-like peptide-1 receptor (GLP-1R) agonists, sodium-glucose cotransporter 2 inhibitors (SGLT2i), dipeptidyl peptidase-4 inhibitors (DP作者: Nomogram 時(shí)間: 2025-3-24 10:14 作者: Thrombolysis 時(shí)間: 2025-3-24 13:36 作者: 名義上 時(shí)間: 2025-3-24 15:18
Pathology and Cardiotoxicity of the Epicardial Adipose Tissue,um, EAT can affect the morphology and function of all of the heart chambers. Increased epicardial fat has been largely associated with increased left ventricular mass, abnormal geometry, enlarged atria, and diastolic dysfunction. Multifactorial physical and biomolecular mechanisms can explain the effects of excessive EAT on the heart.作者: 諷刺滑稽戲劇 時(shí)間: 2025-3-24 22:17
Cardiometabolic Risk and Epicardial Adipose Tissue,Imaging of EAT can stratify the cardiovascular risk in subjects with type 2 diabetes and pre-diabetes. EAT increases the risk of cardiovascular diseases in patients with HIV, psoriasis, and postmenopausal women. EAT measurement can serve as a diagnostic tool in assessing cardiovascular and metabolic risk.作者: 反應(yīng) 時(shí)間: 2025-3-25 00:33 作者: dainty 時(shí)間: 2025-3-25 04:45 作者: GLARE 時(shí)間: 2025-3-25 08:02
Green Technologies to Combat Air Pollution,iposity rather than general obesity. Echocardiographic EAT measurement in both clinical and research scenarios has several advantages, including its low cost, easy accessibility, rapid applicability, and good reproducibility.作者: Cursory 時(shí)間: 2025-3-25 13:11
Anthropogenic Emissions in Asiauld be used to more accurately measure the different and deeper EAT locations. Proton magnetic resonance spectroscopy (.H-MRS) is the gold standard technique to measure myocardial triglyceride content that is independently correlated with ultrasound measured EAT thickness.作者: 前兆 時(shí)間: 2025-3-25 18:15 作者: 公式 時(shí)間: 2025-3-25 21:26 作者: 沒(méi)有希望 時(shí)間: 2025-3-26 02:40
Magnetic Resonance Imaging of the Epicardial Adipose Tissue,uld be used to more accurately measure the different and deeper EAT locations. Proton magnetic resonance spectroscopy (.H-MRS) is the gold standard technique to measure myocardial triglyceride content that is independently correlated with ultrasound measured EAT thickness.作者: 厭食癥 時(shí)間: 2025-3-26 07:55
Targeting the Epicardial Adipose Tissue,o resume its physiological role. A drug-induced browning effect on EAT suggests the development of pharmacological strategies to increase energy consumption. The potential of modulating the EAT transcriptome with targeted pharmacological agents can open new avenues in the pharmacotherapy of cardiometabolic diseases.作者: 玷污 時(shí)間: 2025-3-26 12:14 作者: Reverie 時(shí)間: 2025-3-26 15:00
978-3-030-40572-4Springer Nature Switzerland AG 2020作者: grovel 時(shí)間: 2025-3-26 20:27 作者: 未成熟 時(shí)間: 2025-3-26 21:39 作者: 儀式 時(shí)間: 2025-3-27 02:00
Gianluca IacobellisFirst authoritative and comprehensive volume dedicated to epicardial fat.Discusses anatomical, biomolecular, genetic, imaging features, and clinical applications of epicardial adipose tissue.Examines 作者: 勤勞 時(shí)間: 2025-3-27 07:44 作者: 的’ 時(shí)間: 2025-3-27 11:29
https://doi.org/10.1007/978-3-662-04956-3ry vessels while the pericardial fat is located externally. Epicardial and intra-abdominal fat both evolve from brown adipose tissue. EAT is supplied by branches of the coronary arteries, whereas pericardial fat is supplied by branches of non-coronary arteries. In the adult human heart, EAT is more 作者: Apogee 時(shí)間: 2025-3-27 14:12
Nikhil Sharma,Avinash Kumar Agarwal the heart is complex and not completely understood. EAT functions can be distinguished in (1) nutritional, (2) metabolic, (3) thermogenic, (4) regulatory, and (5) mechanical. Under normal physiological EAT serves as a buffer, absorbing fatty acids and protecting the heart against high fatty acids l作者: Presbycusis 時(shí)間: 2025-3-27 21:42
https://doi.org/10.1007/978-981-15-3481-2els do not have EAT, leaving its functional role underestimated and poorly elucidated. Recent advances in medicine and science have allowed for better studies that provide a more comprehensive understanding of its physiological role. One way to dissect its function is the study of its gene expressio作者: 修改 時(shí)間: 2025-3-27 22:45 作者: 可卡 時(shí)間: 2025-3-28 02:25 作者: Emg827 時(shí)間: 2025-3-28 08:46
Margaret Burchett,Rachid Mousine,Jane Tarrantomated three-dimensional quantification, non-contrast- or contrast-enhanced cardiac CT imaging allows for reliable assessment of both epicardial adipose tissue volume and attenuation. Once, CT imaging is performed, information of epicardial fat is readily available. There is overwhelming data on th作者: eucalyptus 時(shí)間: 2025-3-28 11:34
Anthropogenic Emissions in Asiagold standard technique to measure the adipose tissue, also because it does not require ionizing radiations. Dedicated cardiac magnetic resonance (CMR) imaging protocols have been developed to assess EAT volume. CMR provides a precise and noninvasive assessment of both EAT thickness and volume. As E作者: LUT 時(shí)間: 2025-3-28 15:17
V. N. Chukanov,B. A. Korobitsintery disease (CAD), as emerged only recently. The mechanisms through which epicardial fat can cause atherosclerosis are complex and multifactorial. Its anatomical proximity to the heart, the unique transcriptome, and intense proteasome are the major atherogenic factors of the epicardial adipose tiss作者: 針葉 時(shí)間: 2025-3-28 22:49 作者: surmount 時(shí)間: 2025-3-29 01:06 作者: 真實(shí)的人 時(shí)間: 2025-3-29 04:26
Budgetary Constraints on Doctrinal Concepts,le, although results are not univocal. Experimental studies suggest a potential protective role of the brown fat features of EAT against the systolic heart failure. A function of EAT p53 and adiponectin has been also suggested in patients with heart failure. A neuromodulatory role of EAT in heart fa作者: 高度表 時(shí)間: 2025-3-29 09:45 作者: interlude 時(shí)間: 2025-3-29 11:29
,Test and Assessment of Air Purifier [1–2],iver infiltration and insulin resistance. EAT has shown to correlate and predict the risk of developing the metabolic syndrome, although there is no consensus on the EAT threshold risk values, yet. Higher EAT is associated with higher cardiometabolic risk, independently of traditional risk markers. 作者: Oligarchy 時(shí)間: 2025-3-29 17:51
https://doi.org/10.1007/978-3-0348-7970-5aldosterone can lead to accumulation of epicardial adipose tissue (EAT), interfering with functional state. In fact, increased mineralocorticoid signaling is critically involved in the secretion of proinflammatory cytokines by EAT with consequent profibrotic cascade activation in the myocardium. In 作者: SPER 時(shí)間: 2025-3-29 19:47 作者: 敏捷 時(shí)間: 2025-3-30 03:39 作者: 險(xiǎn)代理人 時(shí)間: 2025-3-30 06:36 作者: 受傷 時(shí)間: 2025-3-30 10:45
https://doi.org/10.1007/978-981-15-3481-2lites needed by the myocardium. Transcriptomic and proteomic studies show that it is a local source of adipokines with paracrine influence on the myocardium due to the intimate microcirculation shared by both tissues. These analyses also show that it has a role in the immune and endocrine systems af作者: fertilizer 時(shí)間: 2025-3-30 13:28
Margaret Burchett,Rachid Mousine,Jane Tarranws an even more distinct link with disease manifestation, supporting the hypothesis of a local impact of visceral adipose tissue on atherosclerosis development in the underlying vasculature. This book chapter gives an overview over the existing literature, described methods for quantification of EAT作者: 尊敬 時(shí)間: 2025-3-30 16:54
V. N. Chukanov,B. A. Korobitsinrediction of first coronary events. EAT volume is greater in subjects with incident coronary heart disease. The incidence of fatal or nonfatal coronary event significantly increased with higher EAT and remains significant even after adjustment for coronary calcium calcification score and obesity. EA作者: 大暴雨 時(shí)間: 2025-3-30 22:40 作者: 全能 時(shí)間: 2025-3-31 02:57
https://doi.org/10.1007/978-3-030-27783-3inflammation, oxidative stress, atrial remodelling, and genetic factors. EAT has been shown in multiple studies to be a risk factor for development of AF and predictor of recurrence after catheter ablation. The mechanisms directly linking EAT to the pathogenesis of AF also are uncertain. Multiple ph作者: 無(wú)聊點(diǎn)好 時(shí)間: 2025-3-31 08:15
Budgetary Constraints on Doctrinal Concepts,rdial fat was actually lower in subjects with systolic heart failure. Epicardial fat may incur in fibrotic changes during chronic and advanced cardiac failure. Whether EAT plays a role in the long-term prognosis of heart failure requires future investigation.作者: 極力證明 時(shí)間: 2025-3-31 10:57
Air Power in the Nuclear Age, 1945–82ent studies have focused on exploring the autonomic regulation of EAT as it pertains to heart disease pathogenesis and progression. In this chapter, we provide an overview of these studies, immediately following brief introductions on the regulation of global cardiac function by the two branches of 作者: 色情 時(shí)間: 2025-3-31 14:40
https://doi.org/10.1007/978-3-0348-7970-5adrenal gland—paracrine secretion of adipokines from the adipose tissue surrounding the adrenal neoplasia being hormonally active. Considering the EAT characteristics of visceral adipose tissue depot, as well as periadrenal fat, correlation has been found between EAT and cardiovascular changes in ad作者: 祖?zhèn)?nbsp; 時(shí)間: 2025-3-31 17:44
Anatomy of the Epicardial Adipose Tissue,raction and crosstalk between the EAT and the myocardium. Under pathological circumstances, epicardial adipocytes display an intrinsic pro-inflammatory and atherogenic profile. A dense inflammatory infiltrate, mainly represented by macrophages, is commonly detected in epicardial fat of subjects with
