標(biāo)題: Titlebook: Endothelial Mechanisms of Vasomotor Control; With special Referen H. Drexler,A. M. Zeiher,H. Just Conference proceedings 1991 Dr. Dietrich [打印本頁] 作者: 機會 時間: 2025-3-21 16:09
書目名稱Endothelial Mechanisms of Vasomotor Control影響因子(影響力)
書目名稱Endothelial Mechanisms of Vasomotor Control影響因子(影響力)學(xué)科排名
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書目名稱Endothelial Mechanisms of Vasomotor Control網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Endothelial Mechanisms of Vasomotor Control被引頻次
書目名稱Endothelial Mechanisms of Vasomotor Control被引頻次學(xué)科排名
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書目名稱Endothelial Mechanisms of Vasomotor Control年度引用學(xué)科排名
書目名稱Endothelial Mechanisms of Vasomotor Control讀者反饋
書目名稱Endothelial Mechanisms of Vasomotor Control讀者反饋學(xué)科排名
作者: cuticle 時間: 2025-3-21 23:40 作者: entitle 時間: 2025-3-22 01:24
https://doi.org/10.1007/978-3-531-90867-0d with the maintenance of the liquidity of the blood. Only in 1976, when Moncada et al. discovered prostacyclin (17), and 1980, when Furchgott and Zawadzki (7) described the essential role of the intact endothelium for the vasodilatory action of acetylcholine, was the significance of this organ for vasomotor control recognized.作者: 顯微鏡 時間: 2025-3-22 04:53
Introductiond with the maintenance of the liquidity of the blood. Only in 1976, when Moncada et al. discovered prostacyclin (17), and 1980, when Furchgott and Zawadzki (7) described the essential role of the intact endothelium for the vasodilatory action of acetylcholine, was the significance of this organ for vasomotor control recognized.作者: implore 時間: 2025-3-22 11:33
978-3-642-72463-3Dr. Dietrich Steinkopff Verlag GmbH & Co. KG, Darmstadt 1991作者: cardiac-arrest 時間: 2025-3-22 14:20
https://doi.org/10.1007/978-3-531-90867-0d with the maintenance of the liquidity of the blood. Only in 1976, when Moncada et al. discovered prostacyclin (17), and 1980, when Furchgott and Zawadzki (7) described the essential role of the intact endothelium for the vasodilatory action of acetylcholine, was the significance of this organ for 作者: cardiac-arrest 時間: 2025-3-22 20:17 作者: CHOP 時間: 2025-3-23 00:15
https://doi.org/10.1007/978-3-322-95063-5e nitrosothiol S-nitroso-.-cysteine. EDRF was produced from endothelial cells by basal release or by stimulation with the calcium ionophore A23187. Biological activity was measured as relaxation of porcine coronary arteries preconstricted with prostaglandin F., and chemical analysis was made of the 作者: 勤勞 時間: 2025-3-23 03:05 作者: Mets552 時間: 2025-3-23 07:48 作者: 甜瓜 時間: 2025-3-23 11:11 作者: 雄偉 時間: 2025-3-23 15:34 作者: heterodox 時間: 2025-3-23 20:45 作者: 雜色 時間: 2025-3-24 00:19 作者: 瘙癢 時間: 2025-3-24 06:00 作者: ANTIC 時間: 2025-3-24 09:58
Wilhelm Bleek,Hans J. Lietzmannnd tissue oxygenation were studied. Experiments were performed in isolated perfused rabbit hearts as well as in rabbit hindlimbs in vivo. In isolated hearts, perfusion with hemoglobin (6 μM) or N.-nitro-.-arginine (30 μM) significantly increased vascular resistance. The cGMP level in platelets passi作者: Engulf 時間: 2025-3-24 11:05 作者: 鋼筆尖 時間: 2025-3-24 18:20
Untersuchungsmethoden und -designortic endothelial cells (PAEC) and reperfused ischemic Langendorff hearts of rabbits. Using cGMP as an index of NO generation and 6-oxo-PGF. as an index for PGI. production in endothelial cells, it is demonstrated that the two metabolic pathways for NO and prostacyclin formation act independent of e作者: guzzle 時間: 2025-3-24 20:57
Fallstudien in der Lehrerausbildung — wozu?muscle tone. Native (n) LDL, isolated from fresh human plasma, was oxidized by Cu.-incubation. EDRF released from cultured endothelial cells was inactivated by both n-LDL and ox-LDL (1 mg/ml) as detected in a bioassay system. n-LDL reduced the EDRF-mediated vasodilations of the detector segments by 作者: 伴隨而來 時間: 2025-3-24 23:27 作者: jungle 時間: 2025-3-25 04:12 作者: 浮雕寶石 時間: 2025-3-25 11:32 作者: Servile 時間: 2025-3-25 12:26
https://doi.org/10.1007/978-3-322-95063-5wed more vasodilation per amount of contained NO than did authentic NO. Solutions containing EDRF (basal release or by stimulation with A23187) subjected to the same analysis appeared similar to nitrosocysteine, and were distinct from solutions of NO. These experiments show that nitrosyl compounds o作者: 蚊子 時間: 2025-3-25 18:28 作者: Cloudburst 時間: 2025-3-25 20:00
,überg?nge nach der Berufslehre,of NO-formation correlates with the number of nitrate ester groups and proceeds with a simultaneous nitrite formation (with a ratio of 1: 14 in the presence of cysteine). Nitrosamines such as molsidomine do not need thiol compounds for bioactivation. They directly liberate NO from the ring-open A-fo作者: 缺乏 時間: 2025-3-26 01:06 作者: 商議 時間: 2025-3-26 05:29 作者: BANAL 時間: 2025-3-26 09:25 作者: bleach 時間: 2025-3-26 14:49 作者: 改革運動 時間: 2025-3-26 20:32
Shiva Stucki-Sabeti,Barbara Bonhagesensitivity and circulating levels of endothelin-1, on the other hand, are reduced in this experimental model of hypertension. In the porcine coronary circulation, oxidized low-density lipoproteins selectively reduce endothelium-dependent relaxations to aggregating platelets, serotonin, and thrombin作者: 下級 時間: 2025-3-26 21:26 作者: CHIP 時間: 2025-3-27 04:41 作者: Criteria 時間: 2025-3-27 07:20 作者: Ganglion 時間: 2025-3-27 11:33
Molecular mechanisms of nitrovasodilator bioactivationof NO-formation correlates with the number of nitrate ester groups and proceeds with a simultaneous nitrite formation (with a ratio of 1: 14 in the presence of cysteine). Nitrosamines such as molsidomine do not need thiol compounds for bioactivation. They directly liberate NO from the ring-open A-fo作者: 勛章 時間: 2025-3-27 16:06
Endothelium-mediated regulation of coronary tonethe double product of heart rate and blood pressure dropped by only 8±2% (p = 0.05). The maximal coronary conductance during peak reactive hyperemia (after 20 s ischemia) indicating complete coronary dilation was diminished by 48% after .-NNA..The severe drop in resting myocardial perfusion and O.-s作者: Prologue 時間: 2025-3-27 18:04
Impaired tissue perfusion after inhibition of endothelium-derived nitric oxide vascular resistance and abolition of the increase in platelet cGMP induced by intraarterial infusion of acetylcholine. In addition, the oxygen uptake of the hindlimb (?46%) and the skeletal muscle pO. were significantly reduced. It is concluded that continuously released EDRF has a functional role 作者: insomnia 時間: 2025-3-28 00:52 作者: NAV 時間: 2025-3-28 02:46 作者: ANN 時間: 2025-3-28 10:08 作者: dura-mater 時間: 2025-3-28 11:09
Conference proceedings 1991 as well as the development of new modalities of pharmacotherapy will have to be discussed. The topic addressed by the 1990 Gargellen Conference: Endothelial Mechanisms of Vasomotor Control, clearly is of interest for both basic scientists and clinicians. It has been the aim of the organizers, the S作者: CRACY 時間: 2025-3-28 14:35
on- cepts, as well as the development of new modalities of pharmacotherapy will have to be discussed. The topic addressed by the 1990 Gargellen Conference: Endothelial Mechanisms of Vasomotor Control, clearly is of interest for both basic scientists and clinicians. It has been the aim of the organizers, the S978-3-642-72463-3978-3-642-72461-9作者: 并入 時間: 2025-3-28 18:47
https://doi.org/10.1007/978-3-531-91812-9ted). An important principle in the development of collaterals is the remodeling of pre-existing small vessels into the much larger vascular structure. To accomodate new cells old structures have to be removed by controlled proteolysis (tPA, uPA, elastase).作者: 偶然 時間: 2025-3-29 00:57 作者: 運動的我 時間: 2025-3-29 05:20 作者: 山間窄路 時間: 2025-3-29 09:58
Endothelium and blood flow mediated vasomotion in the conscious dogendothelium, through elaboration of constricting factors, e. g., endothelin, can also induce potent vasoconstriction. In the conscious animal endothelin elicits markedly differing degrees of vasoconstriction among the various regional vascular beds.作者: 分離 時間: 2025-3-29 14:32 作者: Memorial 時間: 2025-3-29 15:40
Schulentwicklung durch Beratungfusion. No significant changes in EDR, MPO or cardiac necrosis occurred during the 90 min of ischemia. Thus, endothelial dysfunction is an early and specific marker of reperfusion injury preceding neutrophil involvement and cardiac necrosis. Superoxide radicals appear to play a key role in the decreased EDR observed early after reperfusion.作者: Fresco 時間: 2025-3-29 22:02
Maximilian Hettmann,Kerstin Tiedemannthat seen in patients with variant angina, and that coronary spasm can produce sudden progression of coronary atherosclerotic obstruction due to intramural hemorrhage. Establishment of various animal models to elucidate mechanisms related to various stages of ischemic heart disease are needed.作者: Offset 時間: 2025-3-30 02:48
Blood flow and optimal vascular topography: role of the endotheliumol value but was restored towards 3, both by basal and by acetylcholine-stimulated EDRF activity. In contrast, endothelin-1 caused a dose-dependent reduction in the junction exponent from its optimal value 3. This suggests that the endothelium helps to optimise microvascular function through EDRF but not endothelin-1 release.作者: 農(nóng)學(xué) 時間: 2025-3-30 07:13
Endothelial dysfunction in myocardial ischemia and reperfusion: role of oxygen-derived free radicalsfusion. No significant changes in EDR, MPO or cardiac necrosis occurred during the 90 min of ischemia. Thus, endothelial dysfunction is an early and specific marker of reperfusion injury preceding neutrophil involvement and cardiac necrosis. Superoxide radicals appear to play a key role in the decreased EDR observed early after reperfusion.作者: Glower 時間: 2025-3-30 08:24 作者: BABY 時間: 2025-3-30 14:51 作者: 失望昨天 時間: 2025-3-30 18:33
Role of EDRF and endothelin in coronary vasomotor controlugh ET is possibly the most potent constrictor of coronary arteries yet described, its role in physiological regulation of coronary vasomotor tone is less certain. Interactions between EDRF and ET in the coronary circulation are highlighted and a schema of regulation of release of endothelium-derived vasoactive factors is proposed.作者: FEMUR 時間: 2025-3-30 23:37 作者: PHON 時間: 2025-3-31 04:24
(and organ perfusion) in health and disease. Physiology, pharmacology, and molecular biology have uncovered a wealth of information on structure and function of this heretofore largely neglected "organ". Clinical medicine is now called upon to define the clinical significance of these observa- tions作者: Anonymous 時間: 2025-3-31 08:28
Introductiond with the maintenance of the liquidity of the blood. Only in 1976, when Moncada et al. discovered prostacyclin (17), and 1980, when Furchgott and Zawadzki (7) described the essential role of the intact endothelium for the vasodilatory action of acetylcholine, was the significance of this organ for 作者: Ibd810 時間: 2025-3-31 09:30 作者: 小官 時間: 2025-3-31 15:59 作者: 草率女 時間: 2025-3-31 18:48
Does EDRF/NO regulate its own release by increasing endothelial cyclic GMP?se cells contain considerable amounts of soluble guanylyl cyclase. The increase in cyclic GMP in these cells provided a sensitive bioassay for EDRF/NO activity. Bovine aortic endothelial cells released a material that markedly enhanced cyclic GMP in RFL-6 cells. The synthesis of this substance could作者: 阻礙 時間: 2025-3-31 22:43
Molecular mechanisms of nitrovasodilator bioactivationprodrugs or carriers of the active principle NO, responsible for endothelial controlled vasodilation. The rate of NO-formation strongly correlates with the activation of the soluble guanylate cyclase in vitro, resulting in a stimulation of cGMP synthesis. Nitrovasodilators thus are therapeutic subst