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標題: Titlebook: Endocrinology of the Vasculature; James R. Sowers Book 1996 Humana Press Inc. 1996 Diabetes.Diabetes mellitus.growth factor.insulin.signal [打印本頁]

作者: Scuttle    時間: 2025-3-21 20:07
書目名稱Endocrinology of the Vasculature影響因子(影響力)




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書目名稱Endocrinology of the Vasculature被引頻次學科排名




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書目名稱Endocrinology of the Vasculature讀者反饋




書目名稱Endocrinology of the Vasculature讀者反饋學科排名





作者: Firefly    時間: 2025-3-21 22:21

作者: paragon    時間: 2025-3-22 00:27
Life Annuities: From Immediate to Deferred,cells of the renal afferent arteriole (1). Since the substrate is generally in excess, renin is the rate-limiting step in this cascade. Renin is stored in granules in the JG cells. Its release is stimulated through several pathways. These include:
作者: 短程旅游    時間: 2025-3-22 07:02
https://doi.org/10.1007/978-3-319-30004-7s of transcription (STAT) pathway. These data suggest that AII plays an important role in various cardiovascular diseases associated with VSMC growth and vessel wall inflammation such as hypertension, restenosis following balloon angioplasty, and atherosclerosis.
作者: voluble    時間: 2025-3-22 11:33

作者: AMOR    時間: 2025-3-22 15:07
Angiotensin-Nitric Oxidecells of the renal afferent arteriole (1). Since the substrate is generally in excess, renin is the rate-limiting step in this cascade. Renin is stored in granules in the JG cells. Its release is stimulated through several pathways. These include:
作者: AMOR    時間: 2025-3-22 18:07
Angiotensin II Signal Transduction in Vascular Smooth Muscles of transcription (STAT) pathway. These data suggest that AII plays an important role in various cardiovascular diseases associated with VSMC growth and vessel wall inflammation such as hypertension, restenosis following balloon angioplasty, and atherosclerosis.
作者: vascular    時間: 2025-3-22 23:56

作者: 大猩猩    時間: 2025-3-23 03:54
https://doi.org/10.1007/978-3-642-66435-9 . cGMP, in turn, mediates many of NO-mediated actions in VSMC. However, a number of cGMP-independent effects have been observed following NO stimulation, indicating that activation of guanylyl cyclase accounts for only a portion of the effects of NO.
作者: 刪除    時間: 2025-3-23 08:12

作者: 引水渠    時間: 2025-3-23 11:48

作者: 密碼    時間: 2025-3-23 13:59

作者: negligence    時間: 2025-3-23 22:03
Estrogen and Vascular Reactivityiovascular risk. Conversely, in postmenopausal women taking estrogen replacement therapy, population-based studies indicate there are up to 50% fewer cardiovascular events (4,5). These observations support an important role for estrogen replacement therapy in the prevention of cardiovascular disease.
作者: 開始從未    時間: 2025-3-24 01:52
C-Type Natriuretic Peptideia activation of the same biologically active receptor. ANP and BNP form a dual cardiac peptide endocrine system in which these two peptides function as circulating hormones in the control of intravascular volume and arterial pressure.
作者: multiply    時間: 2025-3-24 06:07
Vascular Actions of Insulin in Health and Diseasen the vascular actions of insulin is also explored. The latter has important clinical consequences given that insulin resistance is a common feature of obesity, of noninsulin-dependent diabetes mellitus (NIDDM), and hypertension, and is associated with an increased risk of macrovascular disease (1,2)
作者: Daily-Value    時間: 2025-3-24 07:23

作者: pester    時間: 2025-3-24 10:43

作者: ANTE    時間: 2025-3-24 17:52
RETRACTED CHAPTER: Thermodynamics of Cycles,ure with a focus on neurovascular tone and endothelium-dependent vasodilation. The chapter closes with thoughts on the implications of increased plasma NEFAs and their effects on neurovascular tone and endothelium-dependent vasodilation to the pathophysiology in the diabetic hypertensive.
作者: 干旱    時間: 2025-3-24 20:50
Laparoscopic Simple Prostatectomy, considering the sodium status .. Studies with angiotensin-converting enzyme (ACE) inhibitors, and more recently with specific AII receptor antagonists, have demonstrated that even when the circulating levels are low, AII remains a major regulator of many biological responses, including blood pressure ..
作者: Mosaic    時間: 2025-3-25 00:42

作者: 牛的細微差別    時間: 2025-3-25 04:26

作者: 出價    時間: 2025-3-25 09:39
The Vascular Renin System considering the sodium status .. Studies with angiotensin-converting enzyme (ACE) inhibitors, and more recently with specific AII receptor antagonists, have demonstrated that even when the circulating levels are low, AII remains a major regulator of many biological responses, including blood pressure ..
作者: BLOT    時間: 2025-3-25 15:33

作者: Postulate    時間: 2025-3-25 18:31
Book 1996oth muscle cells. The distinguished scientists and clinicians open a new area of endocrinology, which sees the vasculature and kidney as endocrine organs and sheds new light on the hormonal regulation of the cardiovascular system. They also add significantly to our current understanding of the autoc
作者: 濃縮    時間: 2025-3-25 23:17
Non-cGMP, Paracrine Effects of Nitric Oxide in the Vasculature . cGMP, in turn, mediates many of NO-mediated actions in VSMC. However, a number of cGMP-independent effects have been observed following NO stimulation, indicating that activation of guanylyl cyclase accounts for only a portion of the effects of NO.
作者: Fracture    時間: 2025-3-26 04:13

作者: 完成才會征服    時間: 2025-3-26 07:57

作者: 解凍    時間: 2025-3-26 10:47
J. G. van der Schroeff,H. J. van der Rheeation of several signaling pathways leads to their mitogenic, vasoconstricting, and developmental actions. In this chapter, the details of ET synthesis and regulation and action and defined physiologic and pathophysiologic roles are examined In particular, there is an emphasis on defining the roles of ET-1 as an important vascular hormone.
作者: 小卷發(fā)    時間: 2025-3-26 13:25

作者: 胖人手藝好    時間: 2025-3-26 18:29

作者: cathartic    時間: 2025-3-27 00:12
John T. Flynn,William S. Tasmann the vascular actions of insulin is also explored. The latter has important clinical consequences given that insulin resistance is a common feature of obesity, of noninsulin-dependent diabetes mellitus (NIDDM), and hypertension, and is associated with an increased risk of macrovascular disease (1,2)
作者: macrophage    時間: 2025-3-27 02:03
The Withdrawal and the End of the Cold War,ulates endothelial cell [Ca.]. (receptor mediated), promotes the release of prostacyclin (PGI), and facilitates inactivation of endothelial-derived relaxing factor (EDRF) or nitric oxide (NO) .,. LDL also stimulates [Ca.]. in vascular smooth muscle cells (VSMC) ., and in cardiac myocytes ..References
作者: Mettle    時間: 2025-3-27 08:44
Study Design/Method/Procedures,mboxane A2, which are vasoconstrictors as well as agonists of platelet aggregation. The endothelium also exerts a profound influence on vasomotion by its ability to metabolize circulating vasoactive substances such as serotonin, epinephrine, bradykinin, and angiotensin I. The endothelium elaborates
作者: faddish    時間: 2025-3-27 10:41
Sidelined: The Millions Who Are Left Out,nd neuropathy in type I diabetic patients. However, the evidence that good glycemic control can prevent or slow the progression ofmacrovascular diseases is not clear . In this chapter, we discuss the cellular and molecular mechanisms that may explain the underlying pathological and clinical abnormal
作者: 闖入    時間: 2025-3-27 17:03
Retroperitoneoscopy in Children, emerging. This chapter reviews effects of sex hormones on the vasculature with emphasis on the processes that influence the progression of atherosclerosis and thus alter cardiovascular risk. Effects of postmenopausal hormone replacement on cardiovascular risk are reviewed.
作者: stressors    時間: 2025-3-27 20:47
Endothelium-Derived Relaxing Factormboxane A2, which are vasoconstrictors as well as agonists of platelet aggregation. The endothelium also exerts a profound influence on vasomotion by its ability to metabolize circulating vasoactive substances such as serotonin, epinephrine, bradykinin, and angiotensin I. The endothelium elaborates
作者: Gratuitous    時間: 2025-3-27 23:17
Vascular Abnormalities in Diabetes Mellitusnd neuropathy in type I diabetic patients. However, the evidence that good glycemic control can prevent or slow the progression ofmacrovascular diseases is not clear . In this chapter, we discuss the cellular and molecular mechanisms that may explain the underlying pathological and clinical abnormal
作者: 量被毀壞    時間: 2025-3-28 03:06
Sex Hormones and the Vasculature emerging. This chapter reviews effects of sex hormones on the vasculature with emphasis on the processes that influence the progression of atherosclerosis and thus alter cardiovascular risk. Effects of postmenopausal hormone replacement on cardiovascular risk are reviewed.
作者: 大方不好    時間: 2025-3-28 08:34

作者: languor    時間: 2025-3-28 10:49

作者: deficiency    時間: 2025-3-28 14:51

作者: 消毒    時間: 2025-3-28 19:04

作者: right-atrium    時間: 2025-3-29 01:07
Estrogen and Vascular Reactivity of myocardial infarction and stroke increases dramatically in women after the menopause . Of women ages 45-64 yr, one of nine have cardiovascular disease; yet after age 65, the ratio increases to one of three . Loss of endogenous estrogens associated with menopause contributes to the increased card
作者: Minatory    時間: 2025-3-29 06:40
C-Type Natriuretic Peptide, vasodilatory, and antimitogenic properties through actions of particulate.guanylyl cyclase and the second messenger guanosine 3’,5’-monophosphate (cGMP).. Brain natriuretic peptide (BNP) shares similar biological actions with ANP, is structurally similar (but genetically distinct), and functions v
作者: choroid    時間: 2025-3-29 07:49
Vascular Actions of Insulin in Health and Diseaselar resistance. In this chapter, the physiologic role of insulin-induced skeletal muscle vasodilation and its mechanism including the interaction between insulin and the endothelium to modulate the endothelium-derived nitric oxide (NO) system are reviewed. Finally, the effect of insulin resistance o
作者: septicemia    時間: 2025-3-29 14:25
Angiotensin-Nitric Oxideular antagonists. The concept that the regulation of blood pressure (and vascular resistance) is the result of a balance between vasoconstriction and vasodilation may be somewhat oversimplified; nevertheless it is a natural place to begin to understand the intricate interaction between these two imp
作者: Presbyopia    時間: 2025-3-29 17:22
Altered Endothelial Function in Diabetes MellitusEarly observations suggested that endothelial injury may be a very early stage in diabetic micro-and macroangiopathy, and subsequent data support this view. A complete review of this rapidly expanding literature is beyond the scope of this chapter; the reader is referred to previous reviews .,. Our
作者: Genome    時間: 2025-3-29 23:44

作者: 名字的誤用    時間: 2025-3-30 00:43
Effects of Hyperglycemia on Vascular Endothelium Nitric Oxide Metabolism effects, but vascular endothelial dysfunction appears to play an extremely important role. Accumulating data suggests that exposure to increased concentrations of glucose leads to endothelial dysfunction. Studies in diabetes have demonstrated elevations in plasma von Willebrand’s factor (1), dimini
作者: 搜尋    時間: 2025-3-30 06:17
Evidence Linking Fatty Fat Acids, the Risk Factor Cluster, and Vascular Pathophysiologyew of the cluster and the possible pathogenetic link to the defect of insulin-mediated glucose disposal. Selected controversies in the insulin resistance—hypertension theory are addressed and another defect of insulin action, namely suppression of lipolysis and plasma NEFAs, is offered as an alterna
作者: PIZZA    時間: 2025-3-30 10:19
Effects of Low Density Lipoproteins on Endothelial Cell Signaling and Adhesion Molecule Expressionlular free calcium concentration [Ca.]. is an important mediator of this activation . Hypercholesterolemia, particularly the low-density lipoprotein (LDL) fraction, is an important risk factor for the development of atherosclerosis, a disease process intimately involving endothelial cells . LDL stim
作者: Intellectual    時間: 2025-3-30 13:00
Angiotensin II Signal Transduction in Vascular Smooth Musclewater balance, blood pressure, and vascular tone. All is produced both systemically and locally in the vessel wall by the actions of renin, which converts angiotensinogen into AI, and angiotensinconverting enzyme (ACE), which cleaves AI to form AII. In addition to its vasoconstrictor activity, AII h
作者: parsimony    時間: 2025-3-30 17:06
The Vascular Renin System a cascade of angiotensin peptides including angiotensin (A) I, II, and III. AII, the most prominent of these peptide hormones, initiates through specific plasma membrane receptors, a host of physiological actions ranging from vasoconstriction to volume and growth regulation .. Circulating plasma le
作者: CAMEO    時間: 2025-3-30 23:39
Effects of Insulin on Vascular Smooth Muscle Contraction in patients with essential hypertension, obesity, and non-insulin-dependent diabetes mellitus . It has been proposed that compensatory hyperinsulinemia increases sympathetic nerve activity and renal Na reabsorption, which could contribute to the genesis ofhypertension . Despite the effects of hyper
作者: Climate    時間: 2025-3-31 03:07
Sex Hormones and the Vasculatureof cardiovascular disease is lower in premenopausal women than in age-matched men, but rises steadily after menopause (2). Postmenopausal women on estrogen replacement therapy have less severe coronary artery disease and a lower risk for cardiovascular mortality than women without hormone treatment
作者: 值得    時間: 2025-3-31 08:45

作者: 外星人    時間: 2025-3-31 12:35

作者: 書法    時間: 2025-3-31 14:11
https://doi.org/10.1007/978-3-642-66435-9 fashion on adjacent vascular smooth muscle cells (VSMC) to activate soluble guanylyl cyclase and generate cyclic guanosine 3’,5’ monophosphate (cGMP) . cGMP, in turn, mediates many of NO-mediated actions in VSMC. However, a number of cGMP-independent effects have been observed following NO stimulat
作者: evince    時間: 2025-3-31 20:39

作者: insolence    時間: 2025-3-31 23:24

作者: irradicable    時間: 2025-4-1 05:18
Andrés Kychenthal B.,Paola Dorta S., vasodilatory, and antimitogenic properties through actions of particulate.guanylyl cyclase and the second messenger guanosine 3’,5’-monophosphate (cGMP).. Brain natriuretic peptide (BNP) shares similar biological actions with ANP, is structurally similar (but genetically distinct), and functions v




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