標題: Titlebook: Early Life Origins of Ageing and Longevity; Alexander Vaiserman Book 2019 Springer Nature Switzerland AG 2019 Developmental programming.Ag [打印本頁] 作者: eternal 時間: 2025-3-21 16:42
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書目名稱Early Life Origins of Ageing and Longevity影響因子(影響力)學科排名
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書目名稱Early Life Origins of Ageing and Longevity網(wǎng)絡公開度學科排名
書目名稱Early Life Origins of Ageing and Longevity被引頻次
書目名稱Early Life Origins of Ageing and Longevity被引頻次學科排名
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書目名稱Early Life Origins of Ageing and Longevity年度引用學科排名
書目名稱Early Life Origins of Ageing and Longevity讀者反饋
書目名稱Early Life Origins of Ageing and Longevity讀者反饋學科排名
作者: headlong 時間: 2025-3-21 21:41 作者: 使迷醉 時間: 2025-3-22 03:55 作者: Brocas-Area 時間: 2025-3-22 06:24
laboratory stocks of mice, genetic GH deficiency or resistance led to remarkable extension of healthspan and longevity. In GH-deficient Ames dwarf mice a brief period of GH replacement therapy normalizes (“rescues”) many adult aging-related traits and shortens longevity. This indicates that absence 作者: Communicate 時間: 2025-3-22 09:07
https://doi.org/10.1057/9780230390218Dutch famine showed that babies with low birth weights had significantly higher obesity rates as adults. In several other studies, low birth weight has been correlated with development of type 2 diabetes (T2D), cardiovascular disease and obesity. Maternal diseases, such as hypertension and gestation作者: PLAYS 時間: 2025-3-22 15:15
The Making of the Twentieth Centuryhe lifecourse and evidence for transmission of these adverse disease traits to future generations. A key component underpinning this early life developmental programming is that of dysregulation of the growth hormone-insulin-like growth factor (GH-IGF) axis. Phenotypic outcomes in programmed offspri作者: PLAYS 時間: 2025-3-22 18:19 作者: Adenoma 時間: 2025-3-23 00:21 作者: 煩人 時間: 2025-3-23 02:41 作者: CANON 時間: 2025-3-23 06:42 作者: 刻苦讀書 時間: 2025-3-23 11:40 作者: Misnomer 時間: 2025-3-23 14:35
https://doi.org/10.1007/978-3-642-96198-4Multigeneration Study to address this issue. Mortality in G1 (N?=?9,565) was followed 1961–2015 and analysed by their parents’ (G0) age-at-death using Cox regression. For an almost entirely deceased segment of G1 (n?=?1,149), born 1915–1917, we compared exact age-at-death with G0 parents’ age-at-dea作者: 終端 時間: 2025-3-23 18:06
https://doi.org/10.1007/978-94-017-3728-9le of epigenetic mechanisms in mediating these life-long effects, including DNA methylation histone modification and regulation by non-coding RNAs, has been demonstrated. Early-life environmental conditions were repeatedly shown to significantly affect life-course change of epigenetic patterns known作者: Paleontology 時間: 2025-3-24 00:49
https://doi.org/10.1007/978-3-030-24958-8Developmental programming; Aging; Age-related disease; Longevity; Epigenetics; biogerontology; gene expres作者: Celiac-Plexus 時間: 2025-3-24 04:29
978-3-030-24960-1Springer Nature Switzerland AG 2019作者: 使厭惡 時間: 2025-3-24 08:57 作者: 鞭打 時間: 2025-3-24 11:39 作者: 宣傳 時間: 2025-3-24 18:22 作者: 饑荒 時間: 2025-3-24 19:04
Book 2019itionally focused on later stages of the life cycle, growing evidence indicates that both the rate of ageing-associated functional declines, and the risks of later-life chronic pathological conditions, can be traced to origins early in life. .While other books in the field concentrate on the develop作者: pulmonary 時間: 2025-3-25 01:47 作者: 胡言亂語 時間: 2025-3-25 05:23 作者: HAIL 時間: 2025-3-25 07:35 作者: 擦掉 時間: 2025-3-25 13:37 作者: 流動才波動 時間: 2025-3-25 18:57
https://doi.org/10.1007/978-981-10-7311-3eing. Also, understanding the processes by which prenatal undernutrition leads to accelerated ageing may provide clues on how we can detect early ageing and implement interventional approaches when conditions are still reversible.作者: cauda-equina 時間: 2025-3-25 20:48 作者: 諂媚于性 時間: 2025-3-26 01:49
Epidemiology of Early Nutrition and Adult Health: Metabolic Adaptations and Body Compositionery rapid pace. Any perturbation to this process, either through nutritional insufficiency or exposure to endocrine disruptors or toxins, not only interrupts or delays the growth process, but in some cases results in metabolic abnormalities that challenge adult health. In terms of early childhood nu作者: insipid 時間: 2025-3-26 05:41 作者: llibretto 時間: 2025-3-26 09:38 作者: 盡責 時間: 2025-3-26 14:05 作者: 嗎啡 時間: 2025-3-26 18:15
Early Life Developmental Programming of the GH/IGF Axis and Long-Term Healthhe lifecourse and evidence for transmission of these adverse disease traits to future generations. A key component underpinning this early life developmental programming is that of dysregulation of the growth hormone-insulin-like growth factor (GH-IGF) axis. Phenotypic outcomes in programmed offspri作者: 原告 時間: 2025-3-26 23:41 作者: atrophy 時間: 2025-3-27 03:43
The Interplay Between Dopamine and Environment as the Biological Basis for the Early Origins of Mentility to several unfavorable outcomes later in life. It seems that these adversities induce adaptations, altering the metabolism to favor survival at critical periods, but at the expense of the individual’s health as a trade-off. These exposures increase the risk for non-transmittable diseases like 作者: META 時間: 2025-3-27 08:28 作者: 正式演說 時間: 2025-3-27 13:04 作者: GULF 時間: 2025-3-27 16:12 作者: 招人嫉妒 時間: 2025-3-27 20:34 作者: 巧辦法 時間: 2025-3-28 00:13
Early-Life Adjustment of Epigenetic Aging Clockle of epigenetic mechanisms in mediating these life-long effects, including DNA methylation histone modification and regulation by non-coding RNAs, has been demonstrated. Early-life environmental conditions were repeatedly shown to significantly affect life-course change of epigenetic patterns known作者: 易怒 時間: 2025-3-28 04:34 作者: PURG 時間: 2025-3-28 06:27 作者: incisive 時間: 2025-3-28 12:21
General Biology of the Developmental Origins of Healthl regenerative potential mediate healthy aging at the cellular level. This chapter is an overview of fundamental biological platforms for developmental programming of life-course cell level dynamics involved in building the fetal body.作者: dyspareunia 時間: 2025-3-28 16:22
Immunological Basis of In Utero Programming of Adult Diseaseal corticosteroids and intrauterine growth restriction (IUGR), pups are born with low birth weights and develop type 2 diabetes (T2D) as adults. In the IUGR model, development of T2D is dependent on interleukin 4 in the neonatal period. Development of the fetal immune system has the potential to eff作者: 使迷醉 時間: 2025-3-28 19:39 作者: infatuation 時間: 2025-3-29 00:33
Early Life Nutritional Programming of Adult Health Statussk of poor adult cardiovascular and metabolic health. However, in contemporary Western societies, maternal over-nutrition, overweight and obesity are far more common nutritional issues and this has led to an increased focus of the field on the long-term consequences of exposure to these stimuli in e作者: vertebrate 時間: 2025-3-29 03:57
The Interplay Between Dopamine and Environment as the Biological Basis for the Early Origins of Mentrly life adversity. These neurons regulate cognitive-emotional states, notably impulsivity, and reward-based decision-making, thus defining psychiatric phenotypes and contributing to intensify behaviors that in turn promote metabolic disturbances. We will also apply the concept of differential susce作者: facilitate 時間: 2025-3-29 09:30
Nutrigenomics as a Strategy for Neuronal Healthoff of genes. An anti-inflammatory diet during early-life and across the whole life may represent a key strategy for influencing brain plasticity and for building an “epigenetic memory” useful in developing neuronal resilience against early-life stressors and to prevent age-related neurodegeneration作者: Noctambulant 時間: 2025-3-29 11:34
Why Is Parental Lifespan Linked to Children’s Chances of Reaching a High Age? A Transgenerational Hyhters compared to mothers. Parents’ and maternal grandmother’s longevity were associated with resilience in subsequent generations. Resilience scores of G2 men were also associated with those of their G3 sons and with their own mortality in midlife. We conclude that chances of reaching a high age ar作者: sperse 時間: 2025-3-29 16:27 作者: carotenoids 時間: 2025-3-29 20:03
Influence of Maternal Obesity on the Long-Term Health of Offspring作者: 走調(diào) 時間: 2025-3-29 23:57 作者: Mercantile 時間: 2025-3-30 05:30 作者: 增減字母法 時間: 2025-3-30 10:55
l regenerative potential mediate healthy aging at the cellular level. This chapter is an overview of fundamental biological platforms for developmental programming of life-course cell level dynamics involved in building the fetal body.作者: 繁殖 時間: 2025-3-30 14:35
https://doi.org/10.1057/9780230390218al corticosteroids and intrauterine growth restriction (IUGR), pups are born with low birth weights and develop type 2 diabetes (T2D) as adults. In the IUGR model, development of T2D is dependent on interleukin 4 in the neonatal period. Development of the fetal immune system has the potential to eff作者: upstart 時間: 2025-3-30 16:39 作者: Barter 時間: 2025-3-30 23:51
Zeitschriften und Medienunterhaltungsk of poor adult cardiovascular and metabolic health. However, in contemporary Western societies, maternal over-nutrition, overweight and obesity are far more common nutritional issues and this has led to an increased focus of the field on the long-term consequences of exposure to these stimuli in e作者: 繁榮地區(qū) 時間: 2025-3-31 02:57
Zeitstrategien in Innovationsprozessenrly life adversity. These neurons regulate cognitive-emotional states, notably impulsivity, and reward-based decision-making, thus defining psychiatric phenotypes and contributing to intensify behaviors that in turn promote metabolic disturbances. We will also apply the concept of differential susce作者: ASSAY 時間: 2025-3-31 07:42 作者: 不要不誠實 時間: 2025-3-31 11:56
https://doi.org/10.1007/978-3-642-96198-4hters compared to mothers. Parents’ and maternal grandmother’s longevity were associated with resilience in subsequent generations. Resilience scores of G2 men were also associated with those of their G3 sons and with their own mortality in midlife. We conclude that chances of reaching a high age ar
