標(biāo)題: Titlebook: Diastolic Relaxation of the Heart; The Biology of Diast Beverly H. Lorell (Associate Professor of Medicine Book 1994Latest edition Springer [打印本頁] 作者: polysomnography 時(shí)間: 2025-3-21 18:52
書目名稱Diastolic Relaxation of the Heart影響因子(影響力)
書目名稱Diastolic Relaxation of the Heart影響因子(影響力)學(xué)科排名
書目名稱Diastolic Relaxation of the Heart網(wǎng)絡(luò)公開度
書目名稱Diastolic Relaxation of the Heart網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Diastolic Relaxation of the Heart被引頻次
書目名稱Diastolic Relaxation of the Heart被引頻次學(xué)科排名
書目名稱Diastolic Relaxation of the Heart年度引用
書目名稱Diastolic Relaxation of the Heart年度引用學(xué)科排名
書目名稱Diastolic Relaxation of the Heart讀者反饋
書目名稱Diastolic Relaxation of the Heart讀者反饋學(xué)科排名
作者: 上漲 時(shí)間: 2025-3-21 21:49
Beverly H. Lorell (Associate Professor of Medicine作者: MURKY 時(shí)間: 2025-3-22 03:42 作者: Introvert 時(shí)間: 2025-3-22 08:23
Book 1994Latest editionme subject,published in 1987 - belongs on the shelf of every practisingcardiologist. It will also be an invaluable addition to the library ofscientists researching the effects of diastole on heart function..作者: 哪有黃油 時(shí)間: 2025-3-22 12:21 作者: 受辱 時(shí)間: 2025-3-22 16:07
Human Recognition at a Distance in Videoce. In this chapter we examine the hypothesis that inhibition of ATPase activity in the myofibril, and in the mitochondrial, sarcolemmal, and sarcoplasmic reticular membranes, and alterations in transsarcolemmal cation gradients, singly and in combination, lead to diastolic dysfunction.作者: 受辱 時(shí)間: 2025-3-22 20:05 作者: 鞠躬 時(shí)間: 2025-3-22 22:37 作者: 為寵愛 時(shí)間: 2025-3-23 01:29 作者: ETCH 時(shí)間: 2025-3-23 08:02 作者: Defense 時(shí)間: 2025-3-23 11:46
Es geht um die Führung von Teamsrt, the process of myocardial relaxation is mainly controlled by a powerful calcium pump, the sarco(endo) plasmic reticulum calcium ATPase (SERCA), which pumps calcium back into the sarcoplasmic reticulum. Calcium is then bound to highly charged proteins located in the subsarcolemma cisternae of the作者: 難理解 時(shí)間: 2025-3-23 14:07 作者: 拱形面包 時(shí)間: 2025-3-23 20:25
Chaos im Management — Management im Chaos its ability to regulate intracellular free calcium concentration. Ca. release from the SR increases the intracellular free Ca. concentration (~10–5M), inducing contraction, while Ca. uptake by the SR reduces the calcium concentration (~10–7M), producing muscle relaxation. In cardiac muscle the beat作者: 絆住 時(shí)間: 2025-3-24 00:44
Chaos im Management — Management im Chaosractile proteins {2,3}. However, myocytes form only one third of all cells in myocardium, and the remaining two thirds are nonmyocytes, including fibroblasts, vascular smooth muscle cells, and endothelial cells {1,4}. The remodeling of interstitial components, as well as that of myocytes, has been d作者: pester 時(shí)間: 2025-3-24 03:48
The Problem of Human Re-Identificatione of the pathophysiologic stimulus, the hypertrophic response of the myocardium can assume many forms, shapes, and sizes, leading to concentric hypertrophy, dilated hypertrophy, asymmetric hypertrophy, and enlargement of one to four chambers. While the global structure of the myocardium varies widel作者: Fermentation 時(shí)間: 2025-3-24 07:33
Learning Subject-Discriminative Featurest. As a point of departure for this, we begin with a discussion of the pressure-volume (P-V) relation of the left ventricle of the adult mammal and how diastole is a reflection of the activity and regulation of the myofilaments. The P-V loop, which is shown schematically in Figure 6–1, is rooted in 作者: Latency 時(shí)間: 2025-3-24 14:41 作者: 圍巾 時(shí)間: 2025-3-24 16:47
Human Recognition at a Distance in Videod contraction of the heart muscle with the associated development of force, fiber shortening, and the pumping of blood. Diastole consisted of myocardial relaxation with a decrease in fiber force, fiber lengthening, and the filling of the heart with blood. Diastasis was the period between relaxation 作者: podiatrist 時(shí)間: 2025-3-24 21:46
Human Recognition at a Distance in Videoce. In this chapter we examine the hypothesis that inhibition of ATPase activity in the myofibril, and in the mitochondrial, sarcolemmal, and sarcoplasmic reticular membranes, and alterations in transsarcolemmal cation gradients, singly and in combination, lead to diastolic dysfunction.作者: 身體萌芽 時(shí)間: 2025-3-25 01:56 作者: 轎車 時(shí)間: 2025-3-25 04:46
https://doi.org/10.1007/978-0-85729-124-0walls with minimal enlargement of the left ventricular cavity. In addition to the overall stimulation of cardiac muscle growth, pressure overload induces a fetal pattern of gene programming, including preferential synthesis of β-myosin heavy chain and α-skeletal actin isoforms, which promote a slowe作者: 地牢 時(shí)間: 2025-3-25 11:20
An Overview of Human Reliability Program It was shown that in isolated nonfailing human myocardium, increasing stimulation frequency results in a pronounced augmentation of myocardial performance with regard to isometric force development as well as auxotonic working capacity [1–3]. However, frequency potentiation of contractile force is 作者: 護(hù)航艦 時(shí)間: 2025-3-25 13:35 作者: Isthmus 時(shí)間: 2025-3-25 18:29
https://doi.org/10.1007/978-1-4615-6399-0l Ischemia.” Opie reasoned that “glycolysis may be good for the survival of ischemic myocardium. . . Increasing blood glucose concentrations may benefit the ischemic myocardium by increasing the rate of anaerobic glycolysis, by reversing ion losses, by a direct membrane effect, by altering the extra作者: electrolyte 時(shí)間: 2025-3-25 21:28 作者: FIR 時(shí)間: 2025-3-26 00:29
Uses in Corporate Financial Reportingand myosin, which is regulated in a complex manner by numerous mechanico-chemical factors (Figure 16-1). These factors include the extent of Ca. binding to troponin C, concentrations of ATP, inorganic phosphate, Mg. and H., isoforms of myosin and troponins, the phosphorylation status of some of thes作者: amphibian 時(shí)間: 2025-3-26 07:17
Overview: The Molecular Phenotype of Normal and Impaired Relaxationrt, the process of myocardial relaxation is mainly controlled by a powerful calcium pump, the sarco(endo) plasmic reticulum calcium ATPase (SERCA), which pumps calcium back into the sarcoplasmic reticulum. Calcium is then bound to highly charged proteins located in the subsarcolemma cisternae of the作者: FIN 時(shí)間: 2025-3-26 12:00 作者: Lobotomy 時(shí)間: 2025-3-26 15:22 作者: IDEAS 時(shí)間: 2025-3-26 20:45 作者: 鑲嵌細(xì)工 時(shí)間: 2025-3-27 00:25
Molecular Physiology of Ventricular Hypertrophye of the pathophysiologic stimulus, the hypertrophic response of the myocardium can assume many forms, shapes, and sizes, leading to concentric hypertrophy, dilated hypertrophy, asymmetric hypertrophy, and enlargement of one to four chambers. While the global structure of the myocardium varies widel作者: 敵意 時(shí)間: 2025-3-27 02:45 作者: Lucubrate 時(shí)間: 2025-3-27 07:04 作者: 使成整體 時(shí)間: 2025-3-27 11:44 作者: CLAN 時(shí)間: 2025-3-27 17:25 作者: 思想靈活 時(shí)間: 2025-3-27 21:24 作者: Ventilator 時(shí)間: 2025-3-28 01:37
Cardiac Renin-Angiotensin System in Cardiac Hypertrophy and Failurewalls with minimal enlargement of the left ventricular cavity. In addition to the overall stimulation of cardiac muscle growth, pressure overload induces a fetal pattern of gene programming, including preferential synthesis of β-myosin heavy chain and α-skeletal actin isoforms, which promote a slowe作者: Optometrist 時(shí)間: 2025-3-28 02:37 作者: ORE 時(shí)間: 2025-3-28 09:29
Effects of Hypoxia and Ischemia on Intracellular Calcium and Relaxation: Studies in the Aequorin-Loamic heart disease affects some 7 million Americans, with an estimated annual economic impact of $43 billion, and remains the leading cause of death in the United States [1]. Both hypoxia/reoxygenation and ischemia/ reperfusion are associated with marked abnormalities of cardiac contraction and rhyth作者: 牢騷 時(shí)間: 2025-3-28 13:47
Diastolic Dysfunction During Ischemia: Role of Glycolytic ATP Generationl Ischemia.” Opie reasoned that “glycolysis may be good for the survival of ischemic myocardium. . . Increasing blood glucose concentrations may benefit the ischemic myocardium by increasing the rate of anaerobic glycolysis, by reversing ion losses, by a direct membrane effect, by altering the extra作者: CORD 時(shí)間: 2025-3-28 18:33
Does Calcium Overload Adequately Explain Diastolic Dysfunction During Metabolic Inhibition? cause a transient decrease in end-diastolic distensibility [1,2]. In dogs with a coronary artery constriction, demand ischemia has also been demonstrated to cause an upward shift in the left ventricular pressure-volume curve, consistent with increase in diastolic myocardial stiffness [3,4]. Many fa作者: 環(huán)形 時(shí)間: 2025-3-28 22:39 作者: 傳授知識(shí) 時(shí)間: 2025-3-29 02:02
Molecular Aspects of the Control of Myocardial Relaxationoblasts, vascular smooth muscle cells, and endothelial cells {1,4}. The remodeling of interstitial components, as well as that of myocytes, has been demonstrated to occur during the process of cardiac hypertrophy {5}.作者: fulcrum 時(shí)間: 2025-3-29 05:26
A Molecular Biophysical Approach to Contraction and Relaxational relaxation with a decrease in fiber force, fiber lengthening, and the filling of the heart with blood. Diastasis was the period between relaxation and the next contraction cycle, where the heart undergoes a slight increase in volume. This study is limited to systole and diastole.作者: 偽證 時(shí)間: 2025-3-29 07:27
Cardiac Renin-Angiotensin System in Cardiac Hypertrophy and Failureces a fetal pattern of gene programming, including preferential synthesis of β-myosin heavy chain and α-skeletal actin isoforms, which promote a slower myofilament crossbridge cycling rate [1,2]. Pressure overload is also accompanied by changes in both the distribution and type of collagen in the extracellular connective tissue matrix [3].作者: FOVEA 時(shí)間: 2025-3-29 12:59
Diastolic Dysfunction During Ischemia: Role of Glycolytic ATP Generationit the ischemic myocardium by increasing the rate of anaerobic glycolysis, by reversing ion losses, by a direct membrane effect, by altering the extracellular volume, and by decreasing the circulating free fatty acid concentrations. . . . Each of these aims seems desirable” [1].作者: 組裝 時(shí)間: 2025-3-29 15:47
In Zeiten des Wandels herrscht Chaos1,2]; abnormalities that explain at least in part the insensitivity of the failing myocardium to adrenergic stimulation. At the level of the sarcoplasmic reticulum, investigators have identified a diminution of Ca. uptake sites [3], abnormal Ca. uptake [4], impaired Ca. handling [5], and an impairment in Ca. release [6].作者: Flat-Feet 時(shí)間: 2025-3-29 20:23
The Problem of Human Re-Identificationy, the enlargement of individual cardiac muscle cells invariably accompanies injury from a wide variety of disorders, ranging from postinfarction injury to viral myocarditis and cardiomyopathies (hypertension, diabetes, idiopathic, etc.).作者: coltish 時(shí)間: 2025-3-30 02:49
https://doi.org/10.1007/978-0-85729-124-0, although tonic effects on contractility have been proposed. Alpha-adrenergic transmembrane signaling, with norepinephrine serving as both neurotransmitter and hormone, probably plays an important role in promoting hypertrophy of cardiac myocytes in mammals, and the hypertrophic ventricle clearly has altered diastolic properties.作者: 輕打 時(shí)間: 2025-3-30 06:23
An Overview of Human Reliability Programblunted or even reversed in the failing human heart [1–5]. Since heart rate-dependent regulation of myocardial performance is important for the regulation of cardiovascular function, these experiminental findings may represent a major functional disturbance of the failing human heart.作者: MINT 時(shí)間: 2025-3-30 10:50
Human Reliability and Technology Disruptionm and have been topics of intense laboratory and clinical investigation [2–4]. An increase in the intracellular concentration of calcium ion (Ca..) has been proposed by several investigators as the explanation for the pathophysiologic changes that occur during acute ischemia and reperfusion [5–13].作者: peptic-ulcer 時(shí)間: 2025-3-30 14:18
Developing an Integrated Systemctors might influence left ventricular end-diastolic distensibility in the intact ventricle during ischemia, including a rise in the end-diastolic intracellular free calcium concentration, [Ca.]i, in ventricular myocytes, resulting in increased diastolic force development by the myofilaments, and thus incomplete relaxation [5].作者: 減少 時(shí)間: 2025-3-30 17:46 作者: Expertise 時(shí)間: 2025-3-30 23:58
Learning Subject-Discriminative Featuresonsideration has been the end-diastolic volume (EDV) or end-diastolic stress on the fibers making up the ventricular chambers. It is measured with some fidelity by the end-diastolic pressure, as determined using capillary wedge pressure, and is important in diagnosis and prognosis of cardiovascular disease.作者: Mystic 時(shí)間: 2025-3-31 01:12 作者: tolerance 時(shí)間: 2025-3-31 06:39
Overview: The Molecular Phenotype of Normal and Impaired Relaxationexchange and the sarcolemmal calcium-ATPase pump. Myocardial relaxation is thus an active and energy-requiring process involving proteins of the sarcoplasmic reticulum and the sarcolemma working against a concentration gradient that restores cy-tosolic calcium to its normal end-diastolic concentration of about 10.mol/l.
