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標題: Titlebook: DNA Replication, Recombination, and Repair; Molecular Mechanisms Fumio Hanaoka,Kaoru Sugasawa Book 2016 Springer Japan KK 2016 DNA replicat [打印本頁]

作者: palliative    時間: 2025-3-21 16:41
書目名稱DNA Replication, Recombination, and Repair影響因子(影響力)




書目名稱DNA Replication, Recombination, and Repair影響因子(影響力)學科排名




書目名稱DNA Replication, Recombination, and Repair網(wǎng)絡(luò)公開度




書目名稱DNA Replication, Recombination, and Repair網(wǎng)絡(luò)公開度學科排名




書目名稱DNA Replication, Recombination, and Repair被引頻次




書目名稱DNA Replication, Recombination, and Repair被引頻次學科排名




書目名稱DNA Replication, Recombination, and Repair年度引用




書目名稱DNA Replication, Recombination, and Repair年度引用學科排名




書目名稱DNA Replication, Recombination, and Repair讀者反饋




書目名稱DNA Replication, Recombination, and Repair讀者反饋學科排名





作者: exophthalmos    時間: 2025-3-21 21:30
The Fanconi Anemia Pathway and Interstrand Cross-Link Repairion, and the processes of ICL repair in the cell. In addition, we will highlight recent evidence that implicates endogenous aldehydes in creating genomic damage in FA cells, culminating in the FA phenotypes.
作者: 無意    時間: 2025-3-22 00:58

作者: pulse-pressure    時間: 2025-3-22 06:54

作者: 有惡意    時間: 2025-3-22 11:05
https://doi.org/10.1007/978-1-4613-2261-0s stability dominates that of the overall genome and affects cellular functions, such as senescence. In this review, I will introduce the unique mechanisms by which the rDNA repetitive region and its physiological functions are maintained.
作者: PRO    時間: 2025-3-22 16:35

作者: PRO    時間: 2025-3-22 18:34

作者: 十字架    時間: 2025-3-22 22:15
Mismatch Repairritical intermediates. It has been mired by unseemly biochemical conditions and misinterpretation. The contemporary use of real-time single molecule imaging has the potential to finally and fully resolve the mechanics of MMR. This review describes genetic, biochemical, and biophysical studies that contributed to the development of models for MMR.
作者: 欺騙世家    時間: 2025-3-23 03:25

作者: 消極詞匯    時間: 2025-3-23 09:28
Nonhomologous End-Joining accompanied by elucidation of the genetic basis of several types of severe combined immunodeficiency (SCID) and a better understanding of the mechanisms regulating the choice between HR and NHEJ. In this chapter, we will therefore discuss the NHEJ mechanism along with considerations on DSB repair pathway choice and associated disease phenotypes.
作者: 注視    時間: 2025-3-23 13:03

作者: 光亮    時間: 2025-3-23 16:54

作者: 誘騙    時間: 2025-3-23 19:33
https://doi.org/10.1007/978-1-4613-2261-0ion, and the processes of ICL repair in the cell. In addition, we will highlight recent evidence that implicates endogenous aldehydes in creating genomic damage in FA cells, culminating in the FA phenotypes.
作者: Lymphocyte    時間: 2025-3-24 00:48

作者: 擴大    時間: 2025-3-24 03:36

作者: 庇護    時間: 2025-3-24 10:05

作者: Consequence    時間: 2025-3-24 13:34

作者: crescendo    時間: 2025-3-24 17:04
Duo Wang,Qianxia Ma,Ming Zhang,Tao Zhangthousands of origins in animal and plant cells, but without yielding a distinct and universal consensus sequence. However, origin-associated regions with particular base composition features have been identified, such as the G-rich OGRE motif, predicted to form G-quadruplexes. Epigenetic marks such
作者: 溫順    時間: 2025-3-24 19:26

作者: refine    時間: 2025-3-25 02:38

作者: dowagers-hump    時間: 2025-3-25 04:17
https://doi.org/10.1007/978-1-4613-2261-0 influences on DNA and its metabolism. Due to its compactness, chromatin generally represses all DNA-templated reactions by preventing DNA-processing proteins from accessing to and/or functioning at their target sites. On the contrary, there are many cases in which condensed chromatin rather facilit
作者: Estrogen    時間: 2025-3-25 08:19
https://doi.org/10.1007/978-1-4613-2261-0and repair of developmentally programmed DNA double-strand breaks (DSBs). Cytotoxic lesions such as DSBs are hazardous, potentially mutagenic events that can compromise cellular function. However, during meiosis DSBs are required to establish physical connections between homologous maternal and pate
作者: 突變    時間: 2025-3-25 11:40
https://doi.org/10.1007/978-1-4613-2261-0dehyde metabolites or environmental genotoxins such as radiation and chemicals. Unrepaired DNA damage interferes with essential DNA transactions, such as replication and transcription, and eventually gives rise to the alteration of genetic information. Because genomic instability can cause cell deat
作者: Optic-Disk    時間: 2025-3-25 19:42
https://doi.org/10.1007/978-1-4613-2261-0 cross-link (ICL) repair. In this chapter we summarize our current understanding of the function of the FA genes, the mechanism for FA pathway activation, and the processes of ICL repair in the cell. In addition, we will highlight recent evidence that implicates endogenous aldehydes in creating geno
作者: Conserve    時間: 2025-3-25 21:52

作者: 案發(fā)地點    時間: 2025-3-26 03:45
https://doi.org/10.1007/978-1-4613-2261-0ile sites” where DNA damage occurs more frequently than in other regions. In such regions, DNA replication is inhibited and DNA double-strand breaks are induced. Repetitive sequences easily form DNA secondary structures, and during repair of double-strand breaks, a broken end may recombine with a re
作者: 眼界    時間: 2025-3-26 04:48

作者: Fulminate    時間: 2025-3-26 11:44
https://doi.org/10.1007/978-1-4613-2261-0s: homologous recombination (HR) and nonhomologous end-joining (NHEJ). HR is the main repair mode for replication-associated DSBs, while NHEJ deals with most other breaks, especially during the G0/G1 phase of the cell cycle. Characterization of the core NHEJ machinery over the past 20 years has been
作者: 解凍    時間: 2025-3-26 13:03

作者: 牙齒    時間: 2025-3-26 17:31

作者: Painstaking    時間: 2025-3-27 00:16
978-4-431-56717-2Springer Japan KK 2016
作者: Cervical-Spine    時間: 2025-3-27 03:18

作者: 歸功于    時間: 2025-3-27 06:50

作者: 刺耳    時間: 2025-3-27 10:59

作者: 想象    時間: 2025-3-27 14:15
Regulation of Recombination by Chromatinromatin. In this chapter, we focus on three instances, homologous recombination, V(D)J recombination in vertebrates, and mating-type switching in fission yeast, to discuss a wide variety of roles of chromatin in regulating these events.
作者: 控訴    時間: 2025-3-27 18:30

作者: Lament    時間: 2025-3-27 23:21
DNA Damage Recognition and Repair in Mammalian Global Genome Nucleotide Excision Repairys: global genome repair (GGR) and transcription-coupled repair. Both subpathways share common core NER factors but possess unique systems for recognizing DNA damage. Transcription-coupled repair is initiated by the stalling of RNA polymerase II and is responsible for the accelerated repair of DNA d
作者: modest    時間: 2025-3-28 04:08
Centromeric Chromatin and Kinetochore Assembly in Vertebrate Cellsmatin, which is the basis of functional kinetochore assembly. In this review, we introduce recent insights into the centromeric chromatin structure of vertebrate cells and discuss the mechanisms that underlie the formation of this specific chromatin structure.
作者: Perennial長期的    時間: 2025-3-28 06:18

作者: gerontocracy    時間: 2025-3-28 12:54
Translesion DNA Synthesis and Damage Tolerance Pathways
作者: Anthropoid    時間: 2025-3-28 16:19

作者: 地名表    時間: 2025-3-28 19:05
Duo Wang,Qianxia Ma,Ming Zhang,Tao Zhangnized into replicons and replicon clusters, whose firing occurs at discrete subnuclear bodies known as replication foci. A still poorly characterized nuclear matrix structure might be involved in the attachment of replication units and the formation of replication foci. Preferential genomic sequence
作者: 記憶    時間: 2025-3-29 01:31
Duo Wang,Qianxia Ma,Ming Zhang,Tao Zhangfication may affect the firing efficiency. Replication domain is developmentally regulated and thus is a cell type-specific trait. Physiological significance of replication timing regulation will be discussed.
作者: 創(chuàng)作    時間: 2025-3-29 05:39

作者: synovium    時間: 2025-3-29 08:56
https://doi.org/10.1007/978-1-4613-2261-0romes. Here, we review major steps along the recombination pathway, highlighting the mechanisms that regulate DSB formation and repair and the specialized chromosomal structures that are important for this process. For simplicity, we focus primarily on meiotic recombination in the budding yeast, .,
作者: tic-douloureux    時間: 2025-3-29 11:25

作者: 運氣    時間: 2025-3-29 15:56
https://doi.org/10.1007/978-1-4613-2261-0matin, which is the basis of functional kinetochore assembly. In this review, we introduce recent insights into the centromeric chromatin structure of vertebrate cells and discuss the mechanisms that underlie the formation of this specific chromatin structure.
作者: sulcus    時間: 2025-3-29 21:04

作者: NAIVE    時間: 2025-3-30 02:04

作者: 寬宏大量    時間: 2025-3-30 05:06

作者: staging    時間: 2025-3-30 08:49
DNA Replication Timing: Temporal and Spatial Regulation of Eukaryotic DNA Replicationof these origins is regulated by .-acting sequences and factors that may interact with them. Competition for limiting initiation factors also could regulate the order of origin firing in yeasts. In higher eukaryotes, replication timing may be regulated on a larger genome scale. These so-called repli
作者: Incorruptible    時間: 2025-3-30 14:43

作者: 耕種    時間: 2025-3-30 18:24
Regulation of Recombination by Chromatin influences on DNA and its metabolism. Due to its compactness, chromatin generally represses all DNA-templated reactions by preventing DNA-processing proteins from accessing to and/or functioning at their target sites. On the contrary, there are many cases in which condensed chromatin rather facilit
作者: infelicitous    時間: 2025-3-30 21:57
Homologous Recombination During Meiosisand repair of developmentally programmed DNA double-strand breaks (DSBs). Cytotoxic lesions such as DSBs are hazardous, potentially mutagenic events that can compromise cellular function. However, during meiosis DSBs are required to establish physical connections between homologous maternal and pate
作者: recede    時間: 2025-3-31 03:22
DNA Damage Recognition and Repair in Mammalian Global Genome Nucleotide Excision Repairdehyde metabolites or environmental genotoxins such as radiation and chemicals. Unrepaired DNA damage interferes with essential DNA transactions, such as replication and transcription, and eventually gives rise to the alteration of genetic information. Because genomic instability can cause cell deat




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