標(biāo)題: Titlebook: Current Concepts in Autoimmunity and Chronic Inflammation; Andreas Radbruch,Peter E. Lipsky Book 2006 The Editor(s) (if applicable) and Th [打印本頁] 作者: 回憶錄 時(shí)間: 2025-3-21 16:30
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation影響因子(影響力)
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation影響因子(影響力)學(xué)科排名
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書目名稱Current Concepts in Autoimmunity and Chronic Inflammation網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation被引頻次
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation被引頻次學(xué)科排名
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation年度引用
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation年度引用學(xué)科排名
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation讀者反饋
書目名稱Current Concepts in Autoimmunity and Chronic Inflammation讀者反饋學(xué)科排名
作者: APEX 時(shí)間: 2025-3-21 21:52
Breaking Ignorance: The Case of the Brain,toantigen in the thymus or in the periphery and active suppression by regulatory cells. A third way to prevent autoimmunity is by hiding self tissues behind a tissue barrier impermeable for circulating immune cells. The latter mechanism has been held responsible for self-tolerance within the nervous作者: 使成波狀 時(shí)間: 2025-3-22 02:18 作者: CAB 時(shí)間: 2025-3-22 07:56 作者: Mutter 時(shí)間: 2025-3-22 11:48 作者: REP 時(shí)間: 2025-3-22 13:55 作者: REP 時(shí)間: 2025-3-22 19:01 作者: 其他 時(shí)間: 2025-3-23 01:01 作者: obsession 時(shí)間: 2025-3-23 03:30 作者: accrete 時(shí)間: 2025-3-23 05:40 作者: Musculoskeletal 時(shí)間: 2025-3-23 13:05
Signalling Pathways in B Cells: Implications for Autoimmunity,of B cells in the maintenance of the disease process beyond just being precursors of (auto)antibody-producing plasma cells. Detailed analyses have implicated a number of surface molecules and subsequent downstream signalling pathways in the regulation of the events induced by BCR engagement. In this作者: Carcinoma 時(shí)間: 2025-3-23 16:22 作者: folliculitis 時(shí)間: 2025-3-23 21:39 作者: 節(jié)省 時(shí)間: 2025-3-24 00:25 作者: Dappled 時(shí)間: 2025-3-24 04:22
Conclusions: Loss, Trauma, Recovery,toantigen in the thymus or in the periphery and active suppression by regulatory cells. A third way to prevent autoimmunity is by hiding self tissues behind a tissue barrier impermeable for circulating immune cells. The latter mechanism has been held responsible for self-tolerance within the nervous作者: 除草劑 時(shí)間: 2025-3-24 10:30 作者: Tremor 時(shí)間: 2025-3-24 12:11
Chi Han Chen,Bo Han Chen,Anthony Y. Changhave been conducted. While it is plausible that some sex-linked genes may contribute to the genetic predisposition for the disease, other likely culprits are the sex hormones estrogen and prolactin. In this chapter we review studies that have addressed the influence of sex hormones in SLE activity a作者: gruelling 時(shí)間: 2025-3-24 18:41
https://doi.org/10.1007/978-3-030-52988-8ue. Here we discuss that autoimmune disease is often preceded by autoreactivity, meaning the priming of autoantigen-specific immune cells without relevant tissue damage. Recent experimental evidence has demonstrated that both the induction of autoreactivity and the conversion into autoimmune disease作者: MORPH 時(shí)間: 2025-3-24 21:39
Chien-Liang Chen,Ding-Jung Chiangcular emphasis on Crohn’s disease and ankylosing spondylitis. Direct proof that infection establishes persistent autoimmunity remains lacking, although it may provoke a prolonged inflammatory response when occurring on a susceptible immunological background. The argument of infective causality is by作者: 野蠻 時(shí)間: 2025-3-25 01:53
Der-Jiunn Deng,Ai-Chun Pang,Chun-Cheng Linacteristic of classic autoimmune disease. Nevertheless, patients with these illnesses experience unprovoked inflammatory disease in the absence of underlying infection. Here we discuss recent advances in eight Mendelian autoinflammatory diseases. The causative genes and the proteins they encode play作者: 愛得痛了 時(shí)間: 2025-3-25 03:42
Weiling Zhou,Bo Li,Zhongjiang Yan,Mao Yangion and uptake of apoptotic and necrotic cells is complex and failures in this process can contribute to the pathogenesis of autoimmune diseases such as systemic lupus erythematosus (SLE). Here, we discuss the recognition and uptake molecules which are involved in an efficient clearance of dying cel作者: outer-ear 時(shí)間: 2025-3-25 09:11
https://doi.org/10.1007/978-3-030-53614-5e relative ease of staining samples, even at the height of the inflammatory process. These studies led to the study of synovial cytokine regulation, and the identification of TNF as a therapeutic target, which has been amply validated in clinical trials and now routine therapy. The next key question作者: 植物群 時(shí)間: 2025-3-25 15:17 作者: 沉著 時(shí)間: 2025-3-25 19:15
Leni Van Goidsenhoven,Anneleen Masscheleinof B cells in the maintenance of the disease process beyond just being precursors of (auto)antibody-producing plasma cells. Detailed analyses have implicated a number of surface molecules and subsequent downstream signalling pathways in the regulation of the events induced by BCR engagement. In this作者: FOIL 時(shí)間: 2025-3-25 22:06 作者: carbohydrate 時(shí)間: 2025-3-26 00:09
S. Rahul,Subin Koshy,R. Sunithape I diabetes (T1D). With proper animal models, some of the complexity behind these diseases can be reduced. The use of linkage analysis and positional cloning of genes in animal models for RA resulted in the identification of one of the genes regulating severity of arthritis in rats and mice, the .作者: Visual-Acuity 時(shí)間: 2025-3-26 04:56
https://doi.org/10.1007/3-540-29714-6T cell; apoptosis; autoimmune disease; autoimmunity; cell; cytokine; diseases; genes; genetics; infection; mac作者: 憤慨一下 時(shí)間: 2025-3-26 10:11
978-3-642-06745-7The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE作者: Allure 時(shí)間: 2025-3-26 15:18
Andreas Radbruch,Peter E. LipskyDiscusses the complex ethiopathologies of autoimmune diseases.Explores the broad variety of cell types and genes involved.Shows how initiating factors in autoimmune disease can possibly be distinct fr作者: 一條卷發(fā) 時(shí)間: 2025-3-26 17:14 作者: 猛烈責(zé)罵 時(shí)間: 2025-3-27 00:43 作者: 吞噬 時(shí)間: 2025-3-27 03:45 作者: 值得贊賞 時(shí)間: 2025-3-27 08:13 作者: CANDY 時(shí)間: 2025-3-27 11:00
T Cell Activation as Starter and Motor of Rheumatic Inflammation,ation and perpetuation of chronic rheumatic inflammation. Consequently, T cell-directed therapies have been employed with substantial clinical success in the treatment of rheumatic diseases. Here, we review current knowledge based on which CD4 T cells can be implicated as the motor of rheumatic inflammation.作者: 條街道往前推 時(shí)間: 2025-3-27 16:58
Chi Han Chen,Bo Han Chen,Anthony Y. Chang following exposure to exogenous sex hormones. We describe how estrogen and prolactin influence B cell maturation and selection, permitting B cells to mature to immunocompetence. Finally, we discuss the relevance and implications of these data for human disease.作者: 輕浮思想 時(shí)間: 2025-3-27 17:48
https://doi.org/10.1007/978-3-030-53614-5tive TNF during an immune response? Are there differences between the induction of the pro-inflammatory TNF and the anti inflammatory IL-10? The analysis of the interaction of the twomost abundant synovial cells, T lymphocytes andmacrophages has provided interesting clues to new therapeutic approaches based on disrupting T-macrophage interaction.作者: 剛毅 時(shí)間: 2025-3-27 21:57
S. Rahul,Subin Koshy,R. Sunithap a new pathway for therapeutic treatment of inflammatory diseases. In most cases, however, a quantitative trait locus (QTL) is the sum effect of several genes within and outside the QTL, which make positional cloning difficult. Here we will discuss the possibilities and difficulties of gene identification in animal models of autoimmune disorders.作者: instate 時(shí)間: 2025-3-28 02:14 作者: 首創(chuàng)精神 時(shí)間: 2025-3-28 08:21 作者: 食道 時(shí)間: 2025-3-28 13:19 作者: 排他 時(shí)間: 2025-3-28 15:55 作者: heirloom 時(shí)間: 2025-3-28 22:02 作者: Tinea-Capitis 時(shí)間: 2025-3-29 00:00 作者: 摸索 時(shí)間: 2025-3-29 04:44 作者: 策略 時(shí)間: 2025-3-29 08:48
Introduction: Writing the Holy Land, a functionally mature T cell subpopulation specialized for suppressive function. Their generation is in part genetically and developmentally controlled. Genetically determined or environmentally induced abnormality in CD25.CD4. regulatory T cell development, maintenance, and function can be a cause of autoimmune disease in humans.作者: 串通 時(shí)間: 2025-3-29 15:01
https://doi.org/10.1007/978-3-030-52988-8vant tissue damage. Recent experimental evidence has demonstrated that both the induction of autoreactivity and the conversion into autoimmune disease is controlled by the activation of the nonspecific innate immune system. Also, the “inflammatory status” of the target organ critically influences the onset of overt autoimmune disease.作者: 小歌劇 時(shí)間: 2025-3-29 18:44
Chien-Liang Chen,Ding-Jung Chiangh it may provoke a prolonged inflammatory response when occurring on a susceptible immunological background. The argument of infective causality is by nomeans trivial, since it carries important consequences for the safety of vaccine development.作者: 斑駁 時(shí)間: 2025-3-29 21:52
Altug Yalcintas,Eylül Seren K?seln lead to the development and propagation of autoimmune diseases such as lupus erythematosus or chronic inflammatory rheumatoid arthritis by the autoantibody BcR-expressing B cells and their secreted autoantibodies.作者: ERUPT 時(shí)間: 2025-3-30 02:30 作者: 性滿足 時(shí)間: 2025-3-30 06:43 作者: 調(diào)情 時(shí)間: 2025-3-30 11:09 作者: 魯莽 時(shí)間: 2025-3-30 12:55
Leni Van Goidsenhoven,Anneleen Masscheleino makes it possible that increased B cell activation can be induced by distortions in the interaction with other cells. Further delineation of these alterations of B cell function in autoimmune conditions will allow development of more precise B cell-directed therapies beyond drastic B cell depletio作者: 熱心助人 時(shí)間: 2025-3-30 20:09 作者: Cocker 時(shí)間: 2025-3-30 21:23
