標(biāo)題: Titlebook: Control of Gene Expression by Catecholamines and the Renin-Angiotensin System; Heinz Rupp,Bernard Maisch Book 2000 Springer Science+Busine [打印本頁] 作者: 猛烈抨擊 時(shí)間: 2025-3-21 18:41
書目名稱Control of Gene Expression by Catecholamines and the Renin-Angiotensin System影響因子(影響力)
書目名稱Control of Gene Expression by Catecholamines and the Renin-Angiotensin System影響因子(影響力)學(xué)科排名
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書目名稱Control of Gene Expression by Catecholamines and the Renin-Angiotensin System讀者反饋
書目名稱Control of Gene Expression by Catecholamines and the Renin-Angiotensin System讀者反饋學(xué)科排名
作者: 索賠 時(shí)間: 2025-3-21 20:15
Schlussbetrachtung und Ausblick,transfection of CREB increases the promoter activity of reporter constructs containing human angiotensinogen gene promoter attached to the CAT gene. We also show that co-transfection of DBP (which is a member of C/EBP family of transcription factors) increases promoter activity of these reporter constructs. (Mol Cell Biochem .: 81–90, 2000)作者: Ordnance 時(shí)間: 2025-3-22 03:44 作者: Goblet-Cells 時(shí)間: 2025-3-22 04:53 作者: 使饑餓 時(shí)間: 2025-3-22 12:42
Control of cardiomyocyte gene expression as drug targetdiastolic blood pressure, the diet-induced rise in heart rate was blunted only partially. In addition to drugs interfering with the enhanced catecholamine influence, compounds should be considered that selectively affect cardiomyocyte gene expression via ‘metabolic’ signals. (Mol Cell Biochem .: 135–142, 2000)作者: STRIA 時(shí)間: 2025-3-22 13:02 作者: STRIA 時(shí)間: 2025-3-22 17:06 作者: Feckless 時(shí)間: 2025-3-22 23:06
Transcriptional regulation by cAMP in the heartt mechanism for a cAMP-mediated control of gene expression involved e.g. in spermiogenesis and memory/learning processes. This article discusses recent data leading to the hypothesis that this mechanism also contributes to altered gene regulation in heart failure. (Mol Cell Biochem .: 11–17, 2000)作者: 積習(xí)已深 時(shí)間: 2025-3-23 04:36
Mechanisms of transcriptional activation of cAMP-responsive element-binding protein CREBbiology, review transcriptional activation by CREB proteins through transcription cofactors and present novel insights into the context-and position-specific function of CREB on complex genes. (Mol Cell Biochem .: 5–9, 2000)作者: Diluge 時(shí)間: 2025-3-23 06:19
https://doi.org/10.1007/978-3-658-42876-1t mechanism for a cAMP-mediated control of gene expression involved e.g. in spermiogenesis and memory/learning processes. This article discusses recent data leading to the hypothesis that this mechanism also contributes to altered gene regulation in heart failure. (Mol Cell Biochem .: 11–17, 2000)作者: 音樂會(huì) 時(shí)間: 2025-3-23 11:51 作者: Senescent 時(shí)間: 2025-3-23 16:03
Prof. Dr. Marco Heinrich Inderheesbiology, review transcriptional activation by CREB proteins through transcription cofactors and present novel insights into the context-and position-specific function of CREB on complex genes. (Mol Cell Biochem .: 5–9, 2000)作者: 范例 時(shí)間: 2025-3-23 19:46
,Was verdirbt die Qualit?t der Prognose?,REB-transfected MeWo cells were reduced due to the downregulation of the CRE-dependent expression of the type IV collagenase MMP-2 and the adhesion molecule MCAM/MUC18. In the second mechanism, CREB and its associated proteins act as survival factors for human melanoma cells. Here we demonstrated th作者: minimal 時(shí)間: 2025-3-23 22:54
https://doi.org/10.1007/978-3-658-42882-2ate Ac-DEVD-AMC), and mitochondrial membrane depolarisation (Δψ). Western blots detected accumulation of the tumor suppressor protein p53, relocation of cytochrome c to the cytosol, and expression of the antiapoptotic protein Bcl-x.. Prestimulation with lipophilic cAMP-analogs attenuated apoptosis w作者: landmark 時(shí)間: 2025-3-24 05:18 作者: 表被動(dòng) 時(shí)間: 2025-3-24 06:33 作者: ANTI 時(shí)間: 2025-3-24 11:39
https://doi.org/10.1007/978-3-658-43144-0ine function. In a subsequent stage of development, NE transport regulates differentiation of noradrenergic neurons in the peripheral nervous system and the LC by promoting expression of tyrosine hydroxylase (TH) and dopamine-?-hydroxylase (DBH). Conversely, uptake inhibitors, such as the tricyclic 作者: 繁榮地區(qū) 時(shí)間: 2025-3-24 18:45
Schlussbetrachtung und Ausblick,anolol (β-AR blocker), atenolol (β.-AR blocker), yohimbine (α.-AR blocker), Rp-cAMP (an inhibitor of cAMP-dependent protein kinase AI & AII) and staurosporine (an inhibitor of protein kinase C), but was not blocked by ICI 118, 551 (β.-AR blocker) and prazosin (α.-AR blocker). The addition of a combi作者: 薄膜 時(shí)間: 2025-3-24 21:55
https://doi.org/10.1007/978-3-658-43373-4e found that NaF treatment alone, independent of angiotensin II stimulation, was sufficient to increase the tyrosine phosphorylation levels of paxillin. Furthermore, the ability of either NaF and/or angiotensin II to increase tyrosine phosphorylation levels of paxillin is critically dependent on int作者: 合并 時(shí)間: 2025-3-25 02:11 作者: periodontitis 時(shí)間: 2025-3-25 04:26 作者: 抱狗不敢前 時(shí)間: 2025-3-25 08:33
https://doi.org/10.1007/978-3-658-43404-5 of mechanical steps coupled with the use of a magnet to retain iron-laden vessels. Membranes were prepared and the expressions of G. G. G. and G. were evaluated by Western immunoblotting. Baseline MAP (124 ± 6 mmHg) was only marginally (p > 0.05) higher in SHR when compared with WKY rats (110 ± 4 m作者: 招致 時(shí)間: 2025-3-25 15:38
Regulation of tumor growth and metastasis of human melanoma by the CREB transcription factor familyREB-transfected MeWo cells were reduced due to the downregulation of the CRE-dependent expression of the type IV collagenase MMP-2 and the adhesion molecule MCAM/MUC18. In the second mechanism, CREB and its associated proteins act as survival factors for human melanoma cells. Here we demonstrated th作者: orthopedist 時(shí)間: 2025-3-25 17:49
Attenuation of macrophage apoptosis by the cAMP-signaling systemate Ac-DEVD-AMC), and mitochondrial membrane depolarisation (Δψ). Western blots detected accumulation of the tumor suppressor protein p53, relocation of cytochrome c to the cytosol, and expression of the antiapoptotic protein Bcl-x.. Prestimulation with lipophilic cAMP-analogs attenuated apoptosis w作者: Wordlist 時(shí)間: 2025-3-25 22:00
Catecholamines induce IL-10 release in patients suffering from acute myocardial infarction by transaay in monocytic cells. A cAMP responsive element (CRE) was identified as major target. Here we show that there is no influence of catecholamines on the IL-10 promoter activity in T-cells. In contrast to monocytic cells, in T-cells cAMP-induced PKA-dependent phosphorylation of the CRE-binding protein作者: 漂亮才會(huì)豪華 時(shí)間: 2025-3-26 01:20
Regulation of tyrosine hydroxylase gene transcription by the cAMP-signaling pathway: Involvement of in forming specific DNA/protein complexes with the CRE of the TH gene. To address the transcriptional activation function of individual factors, we replaced the TH CRE with a GAL4-binding site and cotransfected this modified TH promoter-reporter gene with an effector plasmid that encodes GAL4-fused作者: CUMB 時(shí)間: 2025-3-26 06:59 作者: NOVA 時(shí)間: 2025-3-26 09:59 作者: gerontocracy 時(shí)間: 2025-3-26 13:25 作者: 高腳酒杯 時(shí)間: 2025-3-26 20:12
Functional cross-talk between the cyclic AMP and Jak/STAT signaling pathways in vascular smooth musced migration (chemotaxis) of vascular SMC [10, 11], although its effect is less prominent than that of platelet-derived growth factor (PDGF). The cellular mechanisms underlying these diverse actions of Ang II are not clearly understood but are likely to involve the activation of distinct signaling p作者: 植物群 時(shí)間: 2025-3-26 22:09 作者: 憤怒事實(shí) 時(shí)間: 2025-3-27 02:31 作者: 擴(kuò)大 時(shí)間: 2025-3-27 09:09
Control of Gene Expression by Catecholamines and the Renin-Angiotensin System作者: 西瓜 時(shí)間: 2025-3-27 10:38 作者: 扔掉掐死你 時(shí)間: 2025-3-27 15:00 作者: 吸引人的花招 時(shí)間: 2025-3-27 19:18 作者: FIG 時(shí)間: 2025-3-27 22:56
Mechanisms of transcriptional activation of cAMP-responsive element-binding protein CREBiption factors had a profound impact on the understanding of signaling-induced gene transcription. Here we discuss some key aspects of the underlying biology, review transcriptional activation by CREB proteins through transcription cofactors and present novel insights into the context-and position-s作者: Monotonous 時(shí)間: 2025-3-28 05:32
Transcriptional regulation by cAMP in the heartrce in the heart. In addition to this, long-term β-adrenergic stimulation by elevated catecholamines also influences the expressional control of functionally relevant cardiac regulatory proteins in human heart failure. The regulation of transcription by the cAMP-response element (CRE) is an importan作者: 文藝 時(shí)間: 2025-3-28 08:09
Regulation of tumor growth and metastasis of human melanoma by the CREB transcription factor familynsfected with a dominant negative construct of CREB, KCREB. KCREB has a mutation in its DNA-binding domain and can not bind the CRE element. Expression of KCREB yields proper heterodimerization with CREB and its associated proteins, but the proteins associated with KCREB do not confer the same degre作者: arboretum 時(shí)間: 2025-3-28 10:42 作者: Aspiration 時(shí)間: 2025-3-28 16:36 作者: SMART 時(shí)間: 2025-3-28 21:28
Catecholamines induce IL-10 release in patients suffering from acute myocardial infarction by transand systemic IL-10 release in response to acute stress reactions in the absence of any systemic inflammation. . and . studies in experimental models suggest that catecholamines induce IL-10 release via a cyclic adenosine monophosphate/protein kinase A (cAMP/PKA) dependent pathway. Here we studied pat作者: 廢止 時(shí)間: 2025-3-29 02:42
Regulation of tyrosine hydroxylase gene transcription by the cAMP-signaling pathway: Involvement of cholamine neurotransmitters. TH gene expression is regulated in a cell type-specific and cAMP-dependent manner. Evidence from this laboratory and others indicates that the cAMP response element (CRE), residing at —45 to —38 bp upstream of the transcription initiation site, is essential for both basa作者: Nebulous 時(shí)間: 2025-3-29 06:08 作者: Indent 時(shí)間: 2025-3-29 10:30 作者: palliative-care 時(shí)間: 2025-3-29 13:21
cAMP increases the expression of human angiotensinogen gene through a combination of cyclic AMP resption of blood pressure. We show here that the promoter activity of reporter constructs containing human angiotensinogen promoter is increased by cAMP treatment on transient transfection in HepG2 cells. We have identified a composite cAMP responsive element, located around 840 bases upstream from the作者: 蚊子 時(shí)間: 2025-3-29 17:28 作者: 按等級(jí) 時(shí)間: 2025-3-29 21:15 作者: 共同給與 時(shí)間: 2025-3-30 02:14
The inducible cAMP early repressor ICERIIγ inhibits CREB and AP-1 transcription but not AT1 receptoroth muscle cells. To test whether the transcription factor CREB is in the pathway involved in modulation of AT.-receptor gene expression, AT. receptor mRNA levels were measured after stimulation with forskolin, angiotensin II and PDGF-BB in VSMC expressing the inducible cAMP early repressor protein,作者: 說不出 時(shí)間: 2025-3-30 06:31
Angiotensin II-induced changes in G-protein expression and resistance of renal microvessels in youngnomenon may occur in renal preglomerular arterioles and may involve changes in expression of GTP-binding regulatory proteins. We have examined the effects of Ang II on steady state levels of G. G. G. and G. in preglomerular arterioles from young marginally hypertensive SHR and on mean arterial press作者: 使激動(dòng) 時(shí)間: 2025-3-30 11:32
Angiotensin receptor II is present in dopaminergic cell line of rat substantia nigra and it is down ome (150 μM), a metabolite of the dopamine oxidative pathway, was found to down regu-late the expression of angiotensin receptor mRNA in RCSN3 cells by 83% (p < 0.05). (Mol Cell Biochem .: 131–134, 2000)作者: 偽造者 時(shí)間: 2025-3-30 12:55
Control of cardiomyocyte gene expression as drug targeteuro-endocrine activation results in disordered influences of angiotensin II and catecholamines on gene expression. To assess whether angiotensin II type 1 receptor inhibition can also counteract a raised sympathetic nervous system activity, spontaneously hypertensive rats fed a hypercaloric diet we作者: CRAMP 時(shí)間: 2025-3-30 19:23 作者: 宣稱 時(shí)間: 2025-3-30 21:31 作者: 不適當(dāng) 時(shí)間: 2025-3-31 01:18 作者: 走調(diào) 時(shí)間: 2025-3-31 05:41 作者: DENT 時(shí)間: 2025-3-31 10:37 作者: Circumscribe 時(shí)間: 2025-3-31 14:31
Zum Transfer des Erarbeiteten in die Praxis, and alter gene expression. This review discusses a synthetic single-stranded palindromic oligonucleotide, which self-hybridizes to form a duplex/hairpin and competes with cAMP response element (CRE) enhancers for binding transcription factors. This oligonucleotide inhibits CRE- and Ap- 1-directed g作者: BROW 時(shí)間: 2025-3-31 19:53
https://doi.org/10.1007/978-3-658-42882-2uating pro-apoptotic actions of chemotherapeutic agents or programmed cell death as a result of massive nitric oxide (NO) generation. Expression of Cox-2 was achieved by treatment of cells with lipopolysaccharide/interferon-γ or nontoxic doses of NO releasing agents. We reasoned E-type prostanoid fo作者: 槍支 時(shí)間: 2025-4-1 00:19
https://doi.org/10.1007/978-3-658-42934-8nd systemic IL-10 release in response to acute stress reactions in the absence of any systemic inflammation. . and . studies in experimental models suggest that catecholamines induce IL-10 release via a cyclic adenosine monophosphate/protein kinase A (cAMP/PKA) dependent pathway. Here we studied pat作者: Defiance 時(shí)間: 2025-4-1 05:22
Verhandlungstraining im Einkaufcholamine neurotransmitters. TH gene expression is regulated in a cell type-specific and cAMP-dependent manner. Evidence from this laboratory and others indicates that the cAMP response element (CRE), residing at —45 to —38 bp upstream of the transcription initiation site, is essential for both basa
