標(biāo)題: Titlebook: Complement Activation in Malaria Immunity and Pathogenesis; José A. Stoute Book 2018 Springer International Publishing AG, part of Springe [打印本頁(yè)] 作者: urinary-tract 時(shí)間: 2025-3-21 18:30
書目名稱Complement Activation in Malaria Immunity and Pathogenesis影響因子(影響力)
書目名稱Complement Activation in Malaria Immunity and Pathogenesis影響因子(影響力)學(xué)科排名
書目名稱Complement Activation in Malaria Immunity and Pathogenesis網(wǎng)絡(luò)公開度
書目名稱Complement Activation in Malaria Immunity and Pathogenesis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Complement Activation in Malaria Immunity and Pathogenesis被引頻次
書目名稱Complement Activation in Malaria Immunity and Pathogenesis被引頻次學(xué)科排名
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書目名稱Complement Activation in Malaria Immunity and Pathogenesis讀者反饋
書目名稱Complement Activation in Malaria Immunity and Pathogenesis讀者反饋學(xué)科排名
作者: Lymphocyte 時(shí)間: 2025-3-22 00:03
Christoph Lindinger,Nora Zeiseldown products that are produced upon rupture of malaria-infected erythrocytes, including hemin and the digestive vacuole, activate the alternative pathway of complement. Evidence for complement activation in malaria includes reduction in serum titers of total complement hemolytic activity and of ind作者: vector 時(shí)間: 2025-3-22 04:22 作者: Isolate 時(shí)間: 2025-3-22 08:38 作者: ostrish 時(shí)間: 2025-3-22 12:37 作者: FOIL 時(shí)間: 2025-3-22 14:47
,Kunden: Segmentierung und N?he, Recent studies identifying novel host-pathogen interactions between the complement system and malaria parasites yield insights on the mechanisms of parasite entry into red blood cells, complement evasion strategies and the development of malaria pathogenesis. In particular, human complement regulat作者: FOIL 時(shí)間: 2025-3-22 20:43
,Führung: Die Rolle des Chief Executive, play an important role in suppressing parasitemia and could work by any or a combination of several mechanisms: inhibition of sporozoite invasion of hepatocytes, by blocking merozoite invasion of red blood cells (RBCs), by inducing phagocytosis of merozoites, or by inducing the phagocytosis of infe作者: Intruder 時(shí)間: 2025-3-22 22:35 作者: Interferons 時(shí)間: 2025-3-23 05:18
https://doi.org/10.1007/978-3-319-77258-5malaria; complement; immunity; immune evasion; complement activation; infectious diseases作者: neoplasm 時(shí)間: 2025-3-23 08:25
978-3-030-08412-7Springer International Publishing AG, part of Springer Nature 2018作者: filicide 時(shí)間: 2025-3-23 12:10 作者: 很是迷惑 時(shí)間: 2025-3-23 15:22 作者: Felicitous 時(shí)間: 2025-3-23 18:40
Spitzenkandidaten und Wahlerfolgvation pathways, the reality is that there are multiple activation mechanisms provided by crosstalk with other systems such as the coagulation system. In order to prevent autologous attack, the complement system has many regulatory points that are intended to prevent autologous damage.作者: 一起 時(shí)間: 2025-3-24 01:54 作者: 喃喃而言 時(shí)間: 2025-3-24 02:26 作者: rectocele 時(shí)間: 2025-3-24 09:00
Complement-Like System in the Mosquito Responses Against Malaria Parasites,like thioester-containing protein TEP1 is a major regulator of . development within the mosquito vector. In this chapter, we will discuss biological and chemical properties of TEP1 and the role of its genetic polymorphism in shaping mosquito interactions with the malaria parasites.作者: 易于 時(shí)間: 2025-3-24 13:42
Book 2018r mechanisms of immunity against this pathogen. By contrast, the role that the complement system plays in immunity and pathogenesis in this infection is not very well recognized or understood. Based on the work of a number of research groups, we know that complement plays an important role in these 作者: 只有 時(shí)間: 2025-3-24 17:35
Christoph Lindinger,Nora Zeiselravascular clearance of erythrocytes to the spleen. This erythrocyte opsonization reaction, mediated by both the classical and alternative pathways of complement, may be additionally amplified in severe cases of malaria complicated by acidosis due to increased activation of the alternative pathway of complement.作者: 表臉 時(shí)間: 2025-3-24 22:00
,Kunden: Segmentierung und N?he, provides a brief overview of complement evasion strategies employed by blood-stage malaria parasites and highlights the importance of understanding the complex interplay between complement and parasite infection.作者: 皺痕 時(shí)間: 2025-3-25 02:36 作者: BUDGE 時(shí)間: 2025-3-25 04:50
Complement Evasion Mechanisms of the Human Pathogen ,, provides a brief overview of complement evasion strategies employed by blood-stage malaria parasites and highlights the importance of understanding the complex interplay between complement and parasite infection.作者: 死亡 時(shí)間: 2025-3-25 10:02 作者: Rct393 時(shí)間: 2025-3-25 13:51 作者: extinct 時(shí)間: 2025-3-25 18:42
Role of Complement in Severe Malarial Anemia,netic studies have not revealed any strong associations between CR1 polymorphisms and severe malarial anemia. Further genetic association studies between polymorphisms of the complement genes and susceptibility to SMA are needed to identify additional potential therapeutic targets.作者: Interim 時(shí)間: 2025-3-25 21:06
Role of Complement in Cerebral Malaria,eficient in the receptors for C3a and C5a are fully susceptible to ECM. These results implicated C5b and the membrane attack complex (MAC) as the drivers of ECM pathogenesis. Indeed, treatment with anti-C9 antibody reduced mortality in ECM. The detection of C5a in the serum of C3-deficient mice sugg作者: 谷物 時(shí)間: 2025-3-26 03:45
Complement and Malaria in Pregnancy, low birth weight, and neurodevelopmental deficits. Together, these data indicate a critical role for complement in the pathophysiology of malaria in pregnancy and suggest that it is a target for drugs to reduce malaria-mediated adverse pregnancy outcomes.作者: animated 時(shí)間: 2025-3-26 06:24 作者: euphoria 時(shí)間: 2025-3-26 11:01
Complement Activation in Malaria Immunity and Pathogenesis作者: Robust 時(shí)間: 2025-3-26 16:06 作者: 6Applepolish 時(shí)間: 2025-3-26 17:20
Christoph Lindinger,Nora Zeiselnetic studies have not revealed any strong associations between CR1 polymorphisms and severe malarial anemia. Further genetic association studies between polymorphisms of the complement genes and susceptibility to SMA are needed to identify additional potential therapeutic targets.作者: 皺痕 時(shí)間: 2025-3-26 22:41
Holger Dannenberg,Dirk Zupanciceficient in the receptors for C3a and C5a are fully susceptible to ECM. These results implicated C5b and the membrane attack complex (MAC) as the drivers of ECM pathogenesis. Indeed, treatment with anti-C9 antibody reduced mortality in ECM. The detection of C5a in the serum of C3-deficient mice sugg作者: LOPE 時(shí)間: 2025-3-27 04:01
Holger Dannenberg,Dirk Zupancic low birth weight, and neurodevelopmental deficits. Together, these data indicate a critical role for complement in the pathophysiology of malaria in pregnancy and suggest that it is a target for drugs to reduce malaria-mediated adverse pregnancy outcomes.作者: 寬敞 時(shí)間: 2025-3-27 09:21 作者: Interferons 時(shí)間: 2025-3-27 11:57
nd how these mechanisms may translate into paradoxical enhancement of infection rather than protection. Finally, they also discuss the role of the mosquito complement system on immunity against the parasite.?.978-3-030-08412-7978-3-319-77258-5作者: Oscillate 時(shí)間: 2025-3-27 14:01 作者: ANTI 時(shí)間: 2025-3-27 20:09 作者: HAIL 時(shí)間: 2025-3-27 23:15
Role of Complement in Severe Malarial Anemia,alarial anemia (SMA). There are now considerable data that suggest that complement plays a role in the development of anemia during malaria infection by opsonization of uninfected erythrocytes with C3 fragments which can lead to phagocytosis of erythrocytes. The increased susceptibility to complemen作者: Eclampsia 時(shí)間: 2025-3-28 06:00
Role of Complement in Cerebral Malaria,nt-mediated host defense offers little protection against malaria infection. What role then does complement play in malaria infection, particularly CM? Studies demonstrate that C5-deficient mice are highly resistant to experimental CM, the animal model for CM. Using complement-deficient mice, our la作者: Essential 時(shí)間: 2025-3-28 08:23
Complement and Malaria in Pregnancy, birth outcomes including stillbirth, preterm birth, and fetal growth restriction. Inflammation at the maternal-fetal interface and dysregulated angiogenesis are thought to underlie malaria-associated adverse birth outcomes. Uniquely, the complement system, through its impact on both inflammatory an作者: 不能和解 時(shí)間: 2025-3-28 12:14 作者: 兩棲動(dòng)物 時(shí)間: 2025-3-28 17:54