標(biāo)題: Titlebook: Colon Cancer Prevention; Dietary Modulation o American Institute for Cancer Research Book 1999 The Editor(s) (if applicable) and The Author [打印本頁] 作者: Jurisdiction 時間: 2025-3-21 16:47
書目名稱Colon Cancer Prevention影響因子(影響力)
書目名稱Colon Cancer Prevention影響因子(影響力)學(xué)科排名
書目名稱Colon Cancer Prevention網(wǎng)絡(luò)公開度
書目名稱Colon Cancer Prevention網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Colon Cancer Prevention被引頻次
書目名稱Colon Cancer Prevention被引頻次學(xué)科排名
書目名稱Colon Cancer Prevention年度引用
書目名稱Colon Cancer Prevention年度引用學(xué)科排名
書目名稱Colon Cancer Prevention讀者反饋
書目名稱Colon Cancer Prevention讀者反饋學(xué)科排名
作者: 遺棄 時間: 2025-3-21 23:37 作者: mediocrity 時間: 2025-3-22 03:04 作者: 壯麗的去 時間: 2025-3-22 05:03 作者: 漂浮 時間: 2025-3-22 11:25 作者: artifice 時間: 2025-3-22 15:26 作者: artifice 時間: 2025-3-22 18:47
Digested Fiber from Wheat Bran Induces cdk Inhibitors which Block Colon Epithelial Cells in G1,ive against the development of colon cancer because they cause growth arrest by inducing cyclin-directed kinase (cdk) inhibitors. A more quiescent colonic epithelium is less susceptible to mutagens which can lead to mutations or deletions in the major genes implicated in the progression to colon can作者: ILEUM 時間: 2025-3-22 23:18 作者: abnegate 時間: 2025-3-23 03:26
The Genetics of Hereditary Non-Polyposis Colorectal Cancer and Non-Polypotic Colon Cancer,ence of three affected relatives from two generations is compatible with autosomal dominant inheritance and fulfils the international diagnostic (”Amsterdam”) criteria for hereditary non-polyposis colorectal cancer, HNPCC..作者: 炸壞 時間: 2025-3-23 06:22
Familial Association,of that expected by chance. The genes that are mutated in the rare syndromes are now known and the cellular processes related to these genes are being elucidated. Common familial clustering is also thought to arise from inherited susceptibility, based on segregation studies, but the underlying genes作者: archenemy 時間: 2025-3-23 12:17 作者: jagged 時間: 2025-3-23 15:40 作者: DOTE 時間: 2025-3-23 18:19 作者: 改良 時間: 2025-3-23 23:59 作者: 音的強(qiáng)弱 時間: 2025-3-24 05:49
The fundamental domain of the modular group,condense and the dying cell fragments form membrane bound apoptotic bodies which are subsequently digested by phagocytic cells.. In contrast to necrosis, this process avoids the release of noxious cellular contents, preventing the induction of an inflammatory response.作者: Genetics 時間: 2025-3-24 06:52 作者: 飛來飛去真休 時間: 2025-3-24 12:20
https://doi.org/10.1007/978-1-4615-4149-3apoptosis; cancer prevention; carcinogenesis; cell; genetics; inflammation; prevention作者: Odyssey 時間: 2025-3-24 17:27 作者: alcohol-abuse 時間: 2025-3-24 20:31 作者: 遺留之物 時間: 2025-3-25 01:56
https://doi.org/10.1007/BFb0058625is regulated by a complex interaction of intrinsic genetic signals with external stimuli including growth and dietary factors. Moreover, the interaction of signals maintains not only the quantitative balance between dividing and terminally differentiated cells, but a spatial and temporal coordinatio作者: LAVA 時間: 2025-3-25 04:04
Geometry in the representation space,ncogenic activation of β-catenin. This can come about by the loss of its negative regulator the adenomatous polyposis coli (APC) protein, or by mutations in the β-catenin gene that result in a more stable protein product. The interaction between APC and β-catenin, and additional proteins that affect作者: mettlesome 時間: 2025-3-25 09:11
,Gr??en-characters of quadratic forms, tumors, we must improve our understanding of this tissue’s immune system.. The immune system is believed to play a key role in tissue defense against tumor formation and growth.. Without adequate surveillance and destruction of the abnormal cell, tumors may go undetected and grow. However, hyper-im作者: escalate 時間: 2025-3-25 15:11 作者: 有毛就脫毛 時間: 2025-3-25 18:46
Automorphism groups of bilinear forms,g the progression to cancer, the cells accumulate selective growth advantage that could be manifested by an increase in cell proliferation and/or a decrease in apoptosis and terminal differentiation. Promotion of the progression to cancer could therefore involve agents, growth factors, proto-oncogen作者: Analogy 時間: 2025-3-25 22:59 作者: 小母馬 時間: 2025-3-26 01:59
The fundamental domain of the modular group,the degree of restriction imposed. In an effort to identify the mechanism(s) by which energy restriction exerts this effect, a short term model system of experimentally induced mammary carcinogenesis was used. Given that carcinogenesis is known to involve a dysregulation of tissue size homeostasis i作者: 種類 時間: 2025-3-26 05:55
Non-analytic Eisenstein series,ence of three affected relatives from two generations is compatible with autosomal dominant inheritance and fulfils the international diagnostic (”Amsterdam”) criteria for hereditary non-polyposis colorectal cancer, HNPCC..作者: Rotator-Cuff 時間: 2025-3-26 09:52 作者: 壕溝 時間: 2025-3-26 15:51
The fundamental domain of the modular group,condense and the dying cell fragments form membrane bound apoptotic bodies which are subsequently digested by phagocytic cells.. In contrast to necrosis, this process avoids the release of noxious cellular contents, preventing the induction of an inflammatory response.作者: Genetics 時間: 2025-3-26 20:07 作者: 兇兆 時間: 2025-3-26 23:51
Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/c/image/229659.jpg作者: 使習(xí)慣于 時間: 2025-3-27 01:23 作者: obscurity 時間: 2025-3-27 05:26
Colonic Cell Proliferation and Apoptosis in Rodent Species,condense and the dying cell fragments form membrane bound apoptotic bodies which are subsequently digested by phagocytic cells.. In contrast to necrosis, this process avoids the release of noxious cellular contents, preventing the induction of an inflammatory response.作者: omnibus 時間: 2025-3-27 12:09 作者: GROWL 時間: 2025-3-27 14:16
Non-analytic Eisenstein series,This presentation will address three questions: 1) Why is dietary modification a viable strategy for preventing colorectal cancer? 2) Why are intervention studies an important component of cancer prevention research? 3) What is the current’ state of the evidence’ from dietary intervention studies of colorectal cancer?作者: 蛛絲 時間: 2025-3-27 20:58 作者: reflection 時間: 2025-3-28 01:04 作者: 無關(guān)緊要 時間: 2025-3-28 05:35 作者: languor 時間: 2025-3-28 06:56
,Gr??en-characters of quadratic forms,mune responses and chronic inflammation may actually serve to promote tumor development in this tissue.. Optimal immune function must involve a balance between these two extremes. There is accumulating evidence from both human and animal studies indicating that inflammation may contribute to carcinogenesis in the colon..作者: Nonthreatening 時間: 2025-3-28 11:42
0065-2598 he subject "Colon Cancer Prevention: Dietary Modulation of Cellular and Molecular Mechanisms," with participants representing various disciplines interested in this area. One of the speak- ers provided an appropriate quote from 17th century physician Thomas Adams: "Pre- vention is better than healin作者: Rheumatologist 時間: 2025-3-28 18:31
The fundamental domain of the modular group,t regulate transcription of epithelial-specific genes may aid in understanding the complex cellular transitions that occur during development. The caudal-related homeobox genes will be used as an example of how transcription factors that direct developmental processes may be involved in the pathogenesis of colorectal neoplasia.作者: 放逐某人 時間: 2025-3-28 22:45 作者: GREEN 時間: 2025-3-29 00:32
Geometry in the representation space,ons in the β-catenin gene that result in a more stable protein product. The interaction between APC and β-catenin, and additional proteins that affect assembly and signaling along this pathway, are discussed.作者: 多骨 時間: 2025-3-29 04:15
Siegel‘s Modular Forms and Dirichlet Series elucidated. Common familial clustering is also thought to arise from inherited susceptibility, based on segregation studies, but the underlying genes have not yet been identified. This section will summarize present knowledge of both the rare and more common forms of familial colon cancer.作者: Generosity 時間: 2025-3-29 10:43
Transcriptional Regulation in Intestinal Development,t regulate transcription of epithelial-specific genes may aid in understanding the complex cellular transitions that occur during development. The caudal-related homeobox genes will be used as an example of how transcription factors that direct developmental processes may be involved in the pathogenesis of colorectal neoplasia.作者: 侵略主義 時間: 2025-3-29 12:50 作者: Thyroxine 時間: 2025-3-29 19:27
,Defects in the Regulation of β-Catenin in Colorectal Cancer,ons in the β-catenin gene that result in a more stable protein product. The interaction between APC and β-catenin, and additional proteins that affect assembly and signaling along this pathway, are discussed.作者: CBC471 時間: 2025-3-29 21:31
Familial Association, elucidated. Common familial clustering is also thought to arise from inherited susceptibility, based on segregation studies, but the underlying genes have not yet been identified. This section will summarize present knowledge of both the rare and more common forms of familial colon cancer.作者: Arresting 時間: 2025-3-30 00:55
Book 1999time, observe, and delineate the events associated with colon cancer. In the development colon cancer, the inactivation of the adenomatous polyposis coli (Apc) gene is one of the earliest known events. Normally Apc downregulates the cellular protein beta-catenin, but this is lost during cancer devel作者: maladorit 時間: 2025-3-30 05:03 作者: Crohns-disease 時間: 2025-3-30 11:25 作者: Collision 時間: 2025-3-30 13:55
Automorphism groups of bilinear forms, induces differentiation and growth arrest .. TGFβ1 inhibits colonocyte proliferation by inhibiting phosphorylation of the retinoblastoma protein through blocking the activity of cdk/cyclin complexes. TGFβ1 inhibits cdk kinases by inducing the cdk inhibitors p15 and p21 and causing accumulation of p作者: Optometrist 時間: 2025-3-30 19:55
The fundamental domain of the modular group,tein, one member of the Cip/Kip family of genes that are involved in cell cycle arrest, was increased dose dependently by energy restriction. Based on this and related observations, the hypothesis is advanced that energy restriction inhibits carcinogenesis, at least in part, by delaying cell cycle p
