標(biāo)題: Titlebook: Cellular and Molecular Biology of Atherosclerosis; Antonio M. Gotto Book 1992 Springer-Verlag London Limited 1992 Arterien.Arterienverkalk [打印本頁] 作者: Maudlin 時(shí)間: 2025-3-21 16:37
書目名稱Cellular and Molecular Biology of Atherosclerosis影響因子(影響力)
書目名稱Cellular and Molecular Biology of Atherosclerosis影響因子(影響力)學(xué)科排名
書目名稱Cellular and Molecular Biology of Atherosclerosis網(wǎng)絡(luò)公開度
書目名稱Cellular and Molecular Biology of Atherosclerosis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cellular and Molecular Biology of Atherosclerosis被引頻次
書目名稱Cellular and Molecular Biology of Atherosclerosis被引頻次學(xué)科排名
書目名稱Cellular and Molecular Biology of Atherosclerosis年度引用
書目名稱Cellular and Molecular Biology of Atherosclerosis年度引用學(xué)科排名
書目名稱Cellular and Molecular Biology of Atherosclerosis讀者反饋
書目名稱Cellular and Molecular Biology of Atherosclerosis讀者反饋學(xué)科排名
作者: HIKE 時(shí)間: 2025-3-22 00:12 作者: Acquired 時(shí)間: 2025-3-22 00:58
https://doi.org/10.1007/978-3-540-88399-9se differences are determined within the stem cells, are genetically associated with the development of hypertension, and are similar to those found between the tubular cells of the two strains. This alteration seems to be associated with abnormalities of membrane skeleton (Bianchi et al. 1985; Ferrari et al. 1986, 1987; Bianchi et al. 1990).作者: 脫落 時(shí)間: 2025-3-22 07:19 作者: 皺痕 時(shí)間: 2025-3-22 11:38 作者: BLANK 時(shí)間: 2025-3-22 14:53 作者: BLANK 時(shí)間: 2025-3-22 19:04
978-1-4471-1911-1Springer-Verlag London Limited 1992作者: 老巫婆 時(shí)間: 2025-3-23 00:05 作者: ALLAY 時(shí)間: 2025-3-23 02:44
Open Innovation in the Financial Servicessclerotic CAD. In the USA alone, about 6 million persons have symptomatic myocardial ischemia because of atherosclerotic CAD. About 250000 coronary artery bypass grafting operations were performed in 1990 in the USA and about 300000 coronary angioplasty procedures. The cause of atherosclerosis is no作者: 離開 時(shí)間: 2025-3-23 06:01
Providing a Culture of Open Innovation,t only explains the biological properties of the so-called endothelium-derived relaxing factor (EDRF) but is also the stimulator of the soluble guanylate cyclase in a number of tissues such as the platelet and the brain. Furthermore, NO is a cytotoxic factor released by activated murine macrophages 作者: ARBOR 時(shí)間: 2025-3-23 11:09
Open Innovation in the Financial Servicesollectively called endothelial activation — which may play a role in the pathogenesis of vascular injury, including atherogenesis, the major topic of this book. Endothelial injury is a component of most theories of atherogenesis, and is central to the “response to injury” hypothesis of atheroscleros作者: 協(xié)迫 時(shí)間: 2025-3-23 15:04 作者: 執(zhí)拗 時(shí)間: 2025-3-23 19:38
Matthew J. Linman,Quan Jason Chengblood monocyte in atherosclerosis was first pointed out by Leary as late as 1941. Poole and Florey (1958) later discussed the blood as a source of lesion foam cells in hypercholesterolemic rabbits, and demonstrated a macrophage traversing the endothelium. Still and O’Neal (1962) were the first to de作者: mendacity 時(shí)間: 2025-3-24 00:57 作者: 周興旺 時(shí)間: 2025-3-24 02:41
Katrin Schmitt,Christian Hoffmannerosclerotic plaque (Palinski et al. 1989; Haberland et al. 1988; Yl?-Herttuala et al. 1989; Clevidence et al. 1983). Low-density lipoprotein (LDL) isolated from the vessel wall has been shown to contain oxidized lipids and protein (Yl?-Herttuala et al. 1989; Clevidence et al. 1983) and monoclonal a作者: Coronation 時(shí)間: 2025-3-24 07:00 作者: giggle 時(shí)間: 2025-3-24 14:13
Herpes (Pemphigoid) Gestationis,n, they capture some of the peripherally derived lipids and return them to the liver, where they are metabolized. Lipoprotein lipase (LPL) and hepatic triglyceride Lipase (HL) are enzymes located on the vascular endothelial surface that modify, through lipid hydrolysis, the circulating lipoprotein p作者: needle 時(shí)間: 2025-3-24 15:11
https://doi.org/10.1007/978-3-540-88399-9se differences are determined within the stem cells, are genetically associated with the development of hypertension, and are similar to those found between the tubular cells of the two strains. This alteration seems to be associated with abnormalities of membrane skeleton (Bianchi et al. 1985; Ferr作者: Bernstein-test 時(shí)間: 2025-3-24 22:05
Pruritic Folliculitis of Pregnancy,y lipoprotein (LDL) particles (Whayne et al. 1981; Durrington et al. 1986). The identification of the factors that determine plasma LDL cholesterol (LDL-C) levels is of major public health importance and has been the subject of intense study in recent years. Researchers have used a wide range of app作者: 傲慢物 時(shí)間: 2025-3-24 23:24
Linear IgM Dermatosis of Pregnancy,ide metabolism in the body. In addition fatty acid metabolism is partly dependent on the structure of lipoproteins. For these reasons it is obvious that the regulation of lipoprotein metabolism plays a major role in energy generation in the body. Next to these physiological functions defects in lipo作者: 刀鋒 時(shí)間: 2025-3-25 06:10 作者: barium-study 時(shí)間: 2025-3-25 10:01
Autoimmune Conditions During Pregnancy,interrelated. There exists a strong negative association between levels of HDL cholesterol and CHD risk, a weaker positive relation between TGs and CHD risk, and a strong inverse association between plasma levels of HDL and of the TG-rich lipoproteins. When HDL cholesterol TGs and CHD risk are consi作者: 流動(dòng)性 時(shí)間: 2025-3-25 14:49
https://doi.org/10.1007/978-3-540-88399-9sterol as well as its major protein component apolipoprotein A-I (apo A-I) and the risk of myocardial infarction (reviewed in Gordon and Rifkind 1989). Family and twin studies suggested partial heredity of low HDL cholesterol levels and have put the influence of genes at 35%-50% (Hunt et al. 1989; A作者: laxative 時(shí)間: 2025-3-25 18:43 作者: dendrites 時(shí)間: 2025-3-25 23:45 作者: 心胸狹窄 時(shí)間: 2025-3-26 02:28 作者: 要塞 時(shí)間: 2025-3-26 07:02
1431-004X arch has grown tremendously over the last forty years, shedding a great deal of light on the contributing factors and natural history of the disorder and enabling strategies for its treatment and prevention. Some of the greatest strides in this field in recent years have derived from advances in mol作者: 商業(yè)上 時(shí)間: 2025-3-26 11:54
Providing a Culture of Open Innovation,ate cyclase in a number of tissues such as the platelet and the brain. Furthermore, NO is a cytotoxic factor released by activated murine macrophages and other cells. NO is synthesized by an enzyme, NO synthase, of which two types have been identified, one constitutive and the other induced by cytokines.作者: 浪費(fèi)時(shí)間 時(shí)間: 2025-3-26 15:28 作者: exhibit 時(shí)間: 2025-3-26 17:30 作者: HEPA-filter 時(shí)間: 2025-3-26 23:51
Autoimmune Conditions During Pregnancy,D risk, and a strong inverse association between plasma levels of HDL and of the TG-rich lipoproteins. When HDL cholesterol TGs and CHD risk are considered ., the status of each in the triad becomes less clear.作者: explicit 時(shí)間: 2025-3-27 04:17 作者: 聯(lián)想 時(shí)間: 2025-3-27 07:42
Lp [a]: A Lipoprotein Class with Atherothrombotic Potential,erimental evidence is now permitting to formulate hypotheses on the possible mechanism(s) of this pathogenicity. This experimental evidence will be reviewed and correlated with the current understanding of the structural properties of Lp[a].作者: 瑣事 時(shí)間: 2025-3-27 10:48 作者: 使絕緣 時(shí)間: 2025-3-27 17:24 作者: myelography 時(shí)間: 2025-3-27 21:13 作者: 刪除 時(shí)間: 2025-3-27 22:53 作者: Vsd168 時(shí)間: 2025-3-28 04:08
Book 1992rown tremendously over the last forty years, shedding a great deal of light on the contributing factors and natural history of the disorder and enabling strategies for its treatment and prevention. Some of the greatest strides in this field in recent years have derived from advances in molecular bio作者: Unsaturated-Fat 時(shí)間: 2025-3-28 06:19 作者: Amnesty 時(shí)間: 2025-3-28 11:07 作者: Synchronism 時(shí)間: 2025-3-28 18:19
Genetic Control of Plasma Lipid, Lipoprotein and Apolipoprotein Levels: From Restriction Fragment Lroaches to investigate the problem, including epidemiology, dietary and metabolic studies and more recently the techniques of cellular and molecular biology have been used. This review focuses on the identification of genetic factors that contribute to the between-individual differences in plasma LDL-C levels seen in the general population.作者: 破譯 時(shí)間: 2025-3-28 19:28 作者: FLAIL 時(shí)間: 2025-3-29 00:39
Morphological Findings in the Coronary Arteries in Fatal Coronary Artery Disease,sclerotic CAD. In the USA alone, about 6 million persons have symptomatic myocardial ischemia because of atherosclerotic CAD. About 250000 coronary artery bypass grafting operations were performed in 1990 in the USA and about 300000 coronary angioplasty procedures. The cause of atherosclerosis is no作者: 適宜 時(shí)間: 2025-3-29 04:22
The ,-Arginine: Nitric Oxide Pathway in Physiology and Pathology,t only explains the biological properties of the so-called endothelium-derived relaxing factor (EDRF) but is also the stimulator of the soluble guanylate cyclase in a number of tissues such as the platelet and the brain. Furthermore, NO is a cytotoxic factor released by activated murine macrophages 作者: 人造 時(shí)間: 2025-3-29 09:44 作者: 影響深遠(yuǎn) 時(shí)間: 2025-3-29 15:14
Basic Fibroblast Growth Factor in Vascular Development and Atherogenesis,logies, such as restenosis after angioplasty and intimal proliferation in vessels of transplanted organs. Clearly, the rationale is that the development of these lesions involves the migration and proliferation of smooth muscle cells and monocyte-macrophages, and secretion of large amounts of extrac作者: Resection 時(shí)間: 2025-3-29 17:18
The Monocyte and Endothelial Injury in Atherogenesis,blood monocyte in atherosclerosis was first pointed out by Leary as late as 1941. Poole and Florey (1958) later discussed the blood as a source of lesion foam cells in hypercholesterolemic rabbits, and demonstrated a macrophage traversing the endothelium. Still and O’Neal (1962) were the first to de作者: 相容 時(shí)間: 2025-3-29 22:10
Lp [a]: A Lipoprotein Class with Atherothrombotic Potential,e (ASGVD) (Utermann 1989; Scanu and Fless 1990; Berg 1990). At this time this notion rests predominantly on epidemiological data although emerging experimental evidence is now permitting to formulate hypotheses on the possible mechanism(s) of this pathogenicity. This experimental evidence will be re作者: 裝入膠囊 時(shí)間: 2025-3-30 03:58
Modified Lipoproteins and Atherogenesis,erosclerotic plaque (Palinski et al. 1989; Haberland et al. 1988; Yl?-Herttuala et al. 1989; Clevidence et al. 1983). Low-density lipoprotein (LDL) isolated from the vessel wall has been shown to contain oxidized lipids and protein (Yl?-Herttuala et al. 1989; Clevidence et al. 1983) and monoclonal a作者: 可耕種 時(shí)間: 2025-3-30 07:13
The Molecular Biology of Apolipoprotein B,lipoprotein (LDL). LDL is an oily droplet, containing a core of hydrophobic cholesteryl esters and a surface monolayer of phospholipid and free cholesterol at the surface. The particle is stabilized and made soluble in the water of the blood by the presence at its surface of a huge protein apolipopr作者: frenzy 時(shí)間: 2025-3-30 10:34
Structure and Evolution of the Apolipoprotein and Lipase Gene Families,n, they capture some of the peripherally derived lipids and return them to the liver, where they are metabolized. Lipoprotein lipase (LPL) and hepatic triglyceride Lipase (HL) are enzymes located on the vascular endothelial surface that modify, through lipid hydrolysis, the circulating lipoprotein p作者: STELL 時(shí)間: 2025-3-30 12:28
A Gln to Arg Substitution in the Adducin Family of Proteins is a Necessary but not Sufficient Factose differences are determined within the stem cells, are genetically associated with the development of hypertension, and are similar to those found between the tubular cells of the two strains. This alteration seems to be associated with abnormalities of membrane skeleton (Bianchi et al. 1985; Ferr作者: 箴言 時(shí)間: 2025-3-30 18:30 作者: CRACK 時(shí)間: 2025-3-30 23:34
Receptor Regulation of Lipoprotein Metabolism,ide metabolism in the body. In addition fatty acid metabolism is partly dependent on the structure of lipoproteins. For these reasons it is obvious that the regulation of lipoprotein metabolism plays a major role in energy generation in the body. Next to these physiological functions defects in lipo作者: landmark 時(shí)間: 2025-3-31 02:55
The High-Density Lipoprotein Receptor,prised mostly of cholesteryl esters. The major cells that accumulate cholesterol in these lesions are macrophages which are derived from circulating monocytes that penetrate the endothelial barrier in response to inflammatory signals. Although the precise mechanism for subendothelial recruitment of 作者: CRUE 時(shí)間: 2025-3-31 06:15 作者: 使長胖 時(shí)間: 2025-3-31 12:56 作者: 注意 時(shí)間: 2025-3-31 17:06
Cholesterol-Lowering Clinical Trials: Where Do We Go From Here?,nd long term. The important information that has been gathered from clinical trials and the accomplishments of the late 1980s outline a highly impressive story. We now have firm evidence that the incidence of coronary heart disease (CHD) in the form of myocardial infarction and/or coronary death can作者: 欺騙手段 時(shí)間: 2025-3-31 19:39 作者: Stagger 時(shí)間: 2025-3-31 23:52 作者: 堅(jiān)毅 時(shí)間: 2025-4-1 02:04
Basic Fibroblast Growth Factor in Vascular Development and Atherogenesis, major classes of growth factors can by synthesized by activated macrophages (Ross et al. 1990a; Libby and Hansson 1991) and endothelial cells (Vlodavsky et al. 1987; Hannan et al. 1988; Speir et al. 1991; Baird and Ling 1987; Mansson et al. 1990; Gajdusek et al. 1980; Collins et al. 1987), and an i作者: 書法 時(shí)間: 2025-4-1 08:10
The Monocyte and Endothelial Injury in Atherogenesis,clear whether lipid accumulation in lesion macrophage foam cells occurred due to sequestration of blood lipophages in the arterial wall, or by the accumulation of intimal lipid by monocytes which had previously traversed the endothelium. Furthermore, the “response to injury” hypothesis in its initia作者: Mangle 時(shí)間: 2025-4-1 12:21
