標(biāo)題: Titlebook: Cellular and Molecular Alterations in the Failing Human Heart; G. Hasenfuss,Ch. Holubarsch,N. R. Alpert Conference proceedings 1992 Dr. Di [打印本頁] 作者: invoke 時間: 2025-3-21 18:45
書目名稱Cellular and Molecular Alterations in the Failing Human Heart影響因子(影響力)
書目名稱Cellular and Molecular Alterations in the Failing Human Heart影響因子(影響力)學(xué)科排名
書目名稱Cellular and Molecular Alterations in the Failing Human Heart網(wǎng)絡(luò)公開度
書目名稱Cellular and Molecular Alterations in the Failing Human Heart網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cellular and Molecular Alterations in the Failing Human Heart被引頻次
書目名稱Cellular and Molecular Alterations in the Failing Human Heart被引頻次學(xué)科排名
書目名稱Cellular and Molecular Alterations in the Failing Human Heart年度引用
書目名稱Cellular and Molecular Alterations in the Failing Human Heart年度引用學(xué)科排名
書目名稱Cellular and Molecular Alterations in the Failing Human Heart讀者反饋
書目名稱Cellular and Molecular Alterations in the Failing Human Heart讀者反饋學(xué)科排名
作者: Flu表流動 時間: 2025-3-21 20:30 作者: 不近人情 時間: 2025-3-22 00:53 作者: 朋黨派系 時間: 2025-3-22 05:13 作者: FLINT 時間: 2025-3-22 11:31 作者: Malfunction 時間: 2025-3-22 16:43
Cardiovascular cyclic nucleotide phosphodiesterases and their role in regulating cardiovascular funcdence for the role that these isozymes have in the regulation of cellular processes has been generated through, or awaits, the identification of selective and potent PDE inhibitors. While selective inhibitors of the cGMP-inhibitable (cGi)-PDE isozyme have been approved for use in the acute treatment作者: Malfunction 時間: 2025-3-22 18:32
Na,K-ATPase expression in normal and failing human left ventriclereparation for orthotopic transplantation or at the time of organ harvest. Abundance of mRNA for all three catalytic α subunits of the Na,K-ATPase was analyzed in samples from patients with end-stage heart failure due to either ischemic or dilated cardiomyopathy, as well as from normal controls. Van作者: CLAM 時間: 2025-3-22 23:33
Structural and functional diversity of human ventricular myosinpressed, possible alterations in the myosin molecule could be of particular relevance. There is increasing evidence that myofibrillar ATPase activity is reduced in congestive heart failure, whereas the findings on myosin ATPase are still controversial. The molecular causes of the reduced activity ar作者: SPASM 時間: 2025-3-23 02:35 作者: Feckless 時間: 2025-3-23 05:44 作者: Flatter 時間: 2025-3-23 10:46 作者: 無能力 時間: 2025-3-23 16:58 作者: FANG 時間: 2025-3-23 19:04 作者: FLASK 時間: 2025-3-23 22:46 作者: 廢除 時間: 2025-3-24 02:43 作者: chuckle 時間: 2025-3-24 09:47
Contraction frequency dependence of twitch and diastolic tension in human dilated cardiomyopathy (Tellel fibered strips of left-ventricular myocardium. Non-failing control tissue (C) was obtained from epicardial biopsies taken during myocardial revascularization surgery on patients with normal ventricular function. End-stage failing tissue was obtained from endocardial and epicardial biopsies from作者: 側(cè)面左右 時間: 2025-3-24 10:41 作者: Carcinoma 時間: 2025-3-24 16:50
Conference proceedings 1992g i. Br., Innere Medizin III - Kardiologie, FRG The syndrome of heart failure continues to be a major challenge to clinicians and scientists. Incidence and mortality of the disease are high, the patient is disabled, and is permanently threatened by the high morbidity and mortality. The clinician fac作者: BROOK 時間: 2025-3-24 20:54 作者: infatuation 時間: 2025-3-24 23:24
Lecture Notes in Computer Science mutations in the cardiac myosin heavy-chain genes. The chromosomal location of other putative FHC loci will also be considered. Finally, the implications of results which demonstrate that cardiac myosin heavy-chain defects produce the pathophysiology of FHC will be considered from both a clinical and basic research perspective.作者: consent 時間: 2025-3-25 05:09
https://doi.org/10.1007/978-3-540-76890-6d cell was less able to buffer the changes in the intracellular calcium, thus providing a biological basis for the arrhythmogenicity of the hypertrophied heart. These various modifications may provide a new key for future pharmaceutical research.作者: BOOST 時間: 2025-3-25 10:09 作者: 蛤肉 時間: 2025-3-25 14:23 作者: 時代錯誤 時間: 2025-3-25 16:56
Cardiovascular cyclic nucleotide phosphodiesterases and their role in regulating cardiovascular func diseases. More potent selective inhibitors of the cGMP-PDE isozyme are needed to determine whether these pharmacological potentiators of EDRF and ANP will be useful in the therapy of angina, hypertension or heart failure.作者: LIMN 時間: 2025-3-25 21:51 作者: 圣歌 時間: 2025-3-26 00:55 作者: Aspirin 時間: 2025-3-26 04:18 作者: 使習(xí)慣于 時間: 2025-3-26 09:37 作者: Mri485 時間: 2025-3-26 16:00
Eine allgemeine Konvergenztheorie,n function may be the basis for .-receptor uncoupling in IDC and ISCDC, whereas in PPH, this phenomenon may result from altered adenylyl cyclase function. Catalytic subunit activity of adenylyl cyclase is decreased in order of increasing pulmonary hypertension in right-ventricular preparations from 作者: HALO 時間: 2025-3-26 18:30
https://doi.org/10.1007/978-3-540-76790-9dilated cardiomyopathy and 48% in ischemic cardiomyopathy, although pertussis toxin substrates were only increased by 40% in dilated cardiomyopathy and no change was observed in ischemic cardiomyopathy. In cardiomyopathy tissue, an inverse relationship was observed between the increase of G. and the作者: ironic 時間: 2025-3-26 23:16 作者: 散布 時間: 2025-3-27 03:10 作者: Affable 時間: 2025-3-27 08:27 作者: 磨碎 時間: 2025-3-27 10:54 作者: Arbitrary 時間: 2025-3-27 17:30
Lecture Notes in Computer Science p < 0.001). Compared to control, in failing myocardium from dilated cardiomyopathic hearts, tension-independent heat (calcium cycling) was significantly reduced. This indicates that in dilated cardiomyopathy reduced peak twitch tension results from decreased calcium activation of contractile protei作者: abracadabra 時間: 2025-3-27 19:56
https://doi.org/10.1007/978-3-540-76848-7ofibrillar percentage of total troponin T comprised of TnT.. In that heart failure in these transplant patients had multiple bases, we propose that rather than a cause of heart failure, the disease-associated changes in troponin T isoform expression are an adaptation to abnormal myocardial function.作者: MAL 時間: 2025-3-28 01:12 作者: concert 時間: 2025-3-28 03:51 作者: AXIS 時間: 2025-3-28 08:42
PerSys 2007 PC Co-chairs’ Messagech stimulates the promoter in a tissue specific and differentiation dependent mode. The presumed signal was located about 210 bp 5′ to the basic promoter which, by itself, is almost inactive, even in muscle cells. The sequence of the signal (CAGCTG) has homology to known E-box sequences. E-boxes (co作者: 宿醉 時間: 2025-3-28 14:06
Image objects and geographic objects 60 min. in most DCM strips (5/9), while in C most of the peaks (8/9) fell between 156 and 180 min.. The peaks from four DCM hearts fell in an intermediate range of frequencies (96–144 min.) which also included one non-failing peak at 132 min.. Diastolic tension declined in both groups as stimulatio作者: Angioplasty 時間: 2025-3-28 17:46
https://doi.org/10.1007/978-3-540-77058-9 whereas at higher stimulation rates force declined again. These results indicate that (1.) alterations of the force-frequency relationship in the failing human heart depend on the underlying cardiac disease and/or the time-course of the disease, and (2.) an increase in the extracellular Ca.-concent作者: aneurysm 時間: 2025-3-28 19:34
Receptor systems in the non-failing human heart the cardiac .-adrenoceptor-G.-protein-adenylate cyclase pathway is the most powerful mechanism to increase heart rate and contractility..On the other hand, at least three receptor systems acting through inhibition of cAMP formation (G.-protein coupled receptors) exist in the human heart: muscarinic作者: 我不重要 時間: 2025-3-29 00:20
Changes in the receptor-G protein-adenylyl cyclase system in heart failure from various types of hean function may be the basis for .-receptor uncoupling in IDC and ISCDC, whereas in PPH, this phenomenon may result from altered adenylyl cyclase function. Catalytic subunit activity of adenylyl cyclase is decreased in order of increasing pulmonary hypertension in right-ventricular preparations from 作者: legitimate 時間: 2025-3-29 04:41
Quantification of Giα-proteins in the failing and nonfailing human myocardiumdilated cardiomyopathy and 48% in ischemic cardiomyopathy, although pertussis toxin substrates were only increased by 40% in dilated cardiomyopathy and no change was observed in ischemic cardiomyopathy. In cardiomyopathy tissue, an inverse relationship was observed between the increase of G. and the作者: 本能 時間: 2025-3-29 10:27 作者: GIDDY 時間: 2025-3-29 13:30 作者: 得罪人 時間: 2025-3-29 17:06 作者: Spina-Bifida 時間: 2025-3-29 20:46
Structural and functional diversity of human ventricular myosin Because the two bands were detected also in normal hearts of large mammals, the existence of V./V. cannot be diagnostic of diseased heart. However, the V./V. ratio was influenced by the hemodynamic load, whereby the fast migrating band (V.) increased with the diastolic and systolic load. Because a 作者: verdict 時間: 2025-3-30 02:49 作者: 傾聽 時間: 2025-3-30 07:50
Troponin T isoform expression in the normal and failing human left ventricle: a correlation with myoofibrillar percentage of total troponin T comprised of TnT.. In that heart failure in these transplant patients had multiple bases, we propose that rather than a cause of heart failure, the disease-associated changes in troponin T isoform expression are an adaptation to abnormal myocardial function.作者: 半導(dǎo)體 時間: 2025-3-30 12:09 作者: 易怒 時間: 2025-3-30 13:45 作者: Bph773 時間: 2025-3-30 17:17 作者: 兇殘 時間: 2025-3-30 22:00 作者: neutralize 時間: 2025-3-31 04:19
Alterations of the force-frequency relationship in the failing human heart depend on the underlying whereas at higher stimulation rates force declined again. These results indicate that (1.) alterations of the force-frequency relationship in the failing human heart depend on the underlying cardiac disease and/or the time-course of the disease, and (2.) an increase in the extracellular Ca.-concent作者: obviate 時間: 2025-3-31 07:14 作者: 并排上下 時間: 2025-3-31 09:14 作者: GRAIN 時間: 2025-3-31 16:31 作者: allergy 時間: 2025-3-31 18:36 作者: FACT 時間: 2025-4-1 00:34
http://image.papertrans.cn/c/image/223080.jpg
