標(biāo)題: Titlebook: Cellular Ageing and Replicative Senescence; Suresh I.S. Rattan,Leonard Hayflick Book 2016 The Editor(s) (if applicable) and The Author(s), [打印本頁(yè)] 作者: Addendum 時(shí)間: 2025-3-21 19:43
書目名稱Cellular Ageing and Replicative Senescence影響因子(影響力)
書目名稱Cellular Ageing and Replicative Senescence影響因子(影響力)學(xué)科排名
書目名稱Cellular Ageing and Replicative Senescence網(wǎng)絡(luò)公開度
書目名稱Cellular Ageing and Replicative Senescence網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cellular Ageing and Replicative Senescence被引頻次
書目名稱Cellular Ageing and Replicative Senescence被引頻次學(xué)科排名
書目名稱Cellular Ageing and Replicative Senescence年度引用
書目名稱Cellular Ageing and Replicative Senescence年度引用學(xué)科排名
書目名稱Cellular Ageing and Replicative Senescence讀者反饋
書目名稱Cellular Ageing and Replicative Senescence讀者反饋學(xué)科排名
作者: 可忽略 時(shí)間: 2025-3-21 23:36
Influence of Donor Age and Species Longevity on Replicative Cellular Senescence when strains are obtained from different individuals. This variability is probably due to both technical issues and heterogeneity inside the tissues. Notwithstanding these limitations, many scientists searched for an inverse relationship of proliferative potential and donor age. Reviewing this lite作者: jarring 時(shí)間: 2025-3-22 01:02
Ageing of the Stem Cells: The Conjoined Twosome Growing Old: Stem Cell and Its Nicheed, the stem cell and the niche are subject to aging. Potent mechanisms of biological aging are control of cellular metabolism, the action of aging genes, epigenetic drift and inflammation, which also modulate the cellular age of stem cells. A common feature of aged stem cells is however hyperactiva作者: GLOSS 時(shí)間: 2025-3-22 07:45 作者: Colonoscopy 時(shí)間: 2025-3-22 08:51 作者: 善辯 時(shí)間: 2025-3-22 12:56
Modeling Cellular Aging: An Introduction – Mathematical and Computational Approachesing. We find that there are a large array of methods from discrete to continuous and from deterministic to stochastic. This chapter is not meant to be a comprehensive coverage of all of the modeling efforts but rather a buffet introduction to what has been done in the field over the last 50–60?years作者: 善辯 時(shí)間: 2025-3-22 18:08
Cell Cycle Checkpoints and Senescenceageing studies. In a sharp contrast to cancer cells, it is firmly regulated by cell cycle checkpoints that ensure evasion of stressed and genetically modified cells, limiting their expansion and serve as an innate check to carcinogenesis. Tumour suppressors and their regulatory proteins play key rol作者: Cumulus 時(shí)間: 2025-3-22 22:50
Mitochondrial Reactive Oxygen Species in Cellular SenescenceMitochondrial dysfunction is the principle cause of increased levels of reactive oxygen species (ROS) and oxidative stress, which is a key mediator of aging. The cell responds to this stressful stimulus by the induction of the cellular aging-stress response, cellular senescence. Here, we discuss the作者: APEX 時(shí)間: 2025-3-23 03:19
Cellular Aging and Tumor Regulationcell, senescence can be one driving force of such diseases. In contrast, cancer is, on the one hand, associated with age as well, but can be seen as a gain of function disease. Cellular senescence can act to prevent cell proliferation and is believed to inhibit cancer growth. Interestingly, this tum作者: 抱狗不敢前 時(shí)間: 2025-3-23 06:11 作者: 使迷醉 時(shí)間: 2025-3-23 11:08 作者: 船員 時(shí)間: 2025-3-23 15:48 作者: JAUNT 時(shí)間: 2025-3-23 22:06 作者: cancellous-bone 時(shí)間: 2025-3-23 22:24
2199-9007 r in cell proliferation regulation.Explains usage of the celThis book covers the origins and subsequent history of research results in which attempts have been made to clarify issues related to cellular ageing, senescence, and age-related pathologies including cancer. .Cellular Ageing and Replicativ作者: 細(xì)微差別 時(shí)間: 2025-3-24 04:57
Cell Cycle Checkpoints and Senescencees, both as molecular sensors and regulators in this process. Aim of the present chapter is to sketch a brief understanding on how cellular senescence is regulated by major tumour suppressor and cell cycle checkpoint proteins as well as by some emerging molecules.作者: nuclear-tests 時(shí)間: 2025-3-24 10:34 作者: 解決 時(shí)間: 2025-3-24 13:17 作者: Herbivorous 時(shí)間: 2025-3-24 17:42 作者: 盲信者 時(shí)間: 2025-3-24 20:51
Who Takes Part in Online Citizen Science?, mechanisms through which mitochondrial ROS promotes senescence. In this context, we will highlight how mitochondrial ROS serves an initiating upstream, or sustaining downstream, role in the induction of senescence. We will also discuss potential interventions to alleviate mitochondrial ROS and delay cellular senescence.作者: 抗體 時(shí)間: 2025-3-25 01:04 作者: 蝕刻術(shù) 時(shí)間: 2025-3-25 06:15
Book 2016 senescence, and age-related pathologies including cancer. .Cellular Ageing and Replicative Senescence. revisits more than fifty-five years of research based on the discovery that cultured normal cells are mortal and the interpretation that this phenomenon is associated with the origins of ageing. T作者: 類人猿 時(shí)間: 2025-3-25 09:32 作者: 失望未來 時(shí)間: 2025-3-25 14:51 作者: badinage 時(shí)間: 2025-3-25 17:34
Telomeres Shortening: A Mere Replicometer?ce suggests that telomeres not only limit the proliferative potential of cells, but also act as highly sensitive sensors of stress in cells. This may be a protective mechanism against cancer, but also contribute to tissue dysfunction observed during ageing.作者: 注入 時(shí)間: 2025-3-25 20:56 作者: 細(xì)微的差異 時(shí)間: 2025-3-26 03:26 作者: 蝕刻 時(shí)間: 2025-3-26 06:51 作者: VAN 時(shí)間: 2025-3-26 12:02
Small Noncoding RNAs in Senescence and Agingie restriction (CR), a reduced caloric intake without malnutrition, protects against senescence and ameliorates the age-related dysfunctions. Since sncRNAs regulate gene expression and control genome stability, it is conceivable that they may contribute to the biological and functional changes that 作者: atopic-rhinitis 時(shí)間: 2025-3-26 15:13
Origins of the Hayflick System, the Phenomenon and the Limit作者: Flu表流動(dòng) 時(shí)間: 2025-3-26 20:14 作者: 割公牛膨脹 時(shí)間: 2025-3-26 22:13 作者: Lasting 時(shí)間: 2025-3-27 02:21
Introduction: On the Politics of Ugliness,trains from several species appear immortals. Moreover, for species that do present cellular senescence, it seems that replicative capacity relates primarily to species adult body mass more than to longevity.作者: 采納 時(shí)間: 2025-3-27 08:43 作者: 違反 時(shí)間: 2025-3-27 13:08
https://doi.org/10.1007/978-3-319-77797-9dual reduction of telomere length and associated damage, and the accompanying changes that take place elicit signals that have an impact on a number of molecules and downstream events. Precise measurements of replicative senescence biomarkers in biological samples from individuals could be clinicall作者: debris 時(shí)間: 2025-3-27 16:34 作者: Genteel 時(shí)間: 2025-3-27 19:00
https://doi.org/10.1007/978-3-319-78583-7egenerative capacity of neural stem and progenitor cells (NSPC). . their neurogenerative capacity is modulated by a variety of external factors, including growth factors, diet, and inflammation. NSPC have been observed to undergo stress-induced senescence . Deregulation of other CNS cell types, incl作者: 缺乏 時(shí)間: 2025-3-27 22:38
Automation and Harassment Detectionie restriction (CR), a reduced caloric intake without malnutrition, protects against senescence and ameliorates the age-related dysfunctions. Since sncRNAs regulate gene expression and control genome stability, it is conceivable that they may contribute to the biological and functional changes that 作者: MOAN 時(shí)間: 2025-3-28 05:01
Book 2016chers in biogerontology and cytogerontology. This collection of articles provides state-of-the-art information, and will encourage students, teachers, health care professionals and others interested in the biology of ageing to explore?the fascinating and challenging question of why and how our cells作者: Pageant 時(shí)間: 2025-3-28 09:19 作者: dagger 時(shí)間: 2025-3-28 14:02 作者: 鳴叫 時(shí)間: 2025-3-28 17:34 作者: Palatial 時(shí)間: 2025-3-28 21:08 作者: 綠州 時(shí)間: 2025-3-29 02:56
https://doi.org/10.1007/978-3-319-76783-3ed, the stem cell and the niche are subject to aging. Potent mechanisms of biological aging are control of cellular metabolism, the action of aging genes, epigenetic drift and inflammation, which also modulate the cellular age of stem cells. A common feature of aged stem cells is however hyperactiva作者: 博識(shí) 時(shí)間: 2025-3-29 06:19 作者: 貨物 時(shí)間: 2025-3-29 08:08
SpringerBriefs in Earth System Sciencesg, which occurs with each round of cell division, leads to a permanent proliferation arrest, also known as replicative senescence. This process has been shown to have important implications in vivo as an increase in the frequency of senescent cells occurs in mammalian tissue with age and in a variet作者: congenial 時(shí)間: 2025-3-29 13:57 作者: WAX 時(shí)間: 2025-3-29 17:03
Palgrave Studies in Alternative Educationageing studies. In a sharp contrast to cancer cells, it is firmly regulated by cell cycle checkpoints that ensure evasion of stressed and genetically modified cells, limiting their expansion and serve as an innate check to carcinogenesis. Tumour suppressors and their regulatory proteins play key rol作者: 有危險(xiǎn) 時(shí)間: 2025-3-29 21:53
Who Takes Part in Online Citizen Science?,Mitochondrial dysfunction is the principle cause of increased levels of reactive oxygen species (ROS) and oxidative stress, which is a key mediator of aging. The cell responds to this stressful stimulus by the induction of the cellular aging-stress response, cellular senescence. Here, we discuss the作者: Intend 時(shí)間: 2025-3-30 00:07
https://doi.org/10.1007/978-3-319-77797-9cell, senescence can be one driving force of such diseases. In contrast, cancer is, on the one hand, associated with age as well, but can be seen as a gain of function disease. Cellular senescence can act to prevent cell proliferation and is believed to inhibit cancer growth. Interestingly, this tum作者: 思想流動(dòng) 時(shí)間: 2025-3-30 07:34 作者: 雪崩 時(shí)間: 2025-3-30 09:43 作者: PURG 時(shí)間: 2025-3-30 15:25
https://doi.org/10.1007/978-3-319-78583-7tance to apoptosis, or detrimental by inducing pro-inflammatory signaling in the microenvironment. Senescent cells have been observed in both aged and diseased tissue, including the brain. The aging brain undergoes changes such as cortical atrophy and increases in inflammatory and oxidative factors,作者: 運(yùn)氣 時(shí)間: 2025-3-30 17:17 作者: Deference 時(shí)間: 2025-3-30 21:13 作者: Affluence 時(shí)間: 2025-3-31 03:10 作者: 有特色 時(shí)間: 2025-3-31 07:04 作者: Gourmet 時(shí)間: 2025-3-31 09:55
