標(biāo)題: Titlebook: Cell Death and Diseases of the Nervous System; Vassilis E. Koliatsos,Rajiv R. Ratan Book 1999 Humana Press Inc. 1999 Alzheimer.Nervous Sys [打印本頁(yè)] 作者: Sentry 時(shí)間: 2025-3-21 19:29
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System影響因子(影響力)
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System被引頻次
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System被引頻次學(xué)科排名
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System年度引用
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System年度引用學(xué)科排名
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System讀者反饋
書(shū)目名稱(chēng)Cell Death and Diseases of the Nervous System讀者反饋學(xué)科排名
作者: exacerbate 時(shí)間: 2025-3-21 21:46
Advances in 21st Century Human Settlementsidentical signal to die. Nowhere is this more obvious than in the developing nervous system where neurons in the same population often die over several days. In fact, “naturally occurring” cell death in the developing nervous system was overlooked for a long time because of its sporadic asynchronous作者: coddle 時(shí)間: 2025-3-22 00:26 作者: 內(nèi)閣 時(shí)間: 2025-3-22 05:52 作者: 一再困擾 時(shí)間: 2025-3-22 11:00 作者: Induction 時(shí)間: 2025-3-22 16:52 作者: Induction 時(shí)間: 2025-3-22 19:35
https://doi.org/10.1007/978-981-13-3603-4a consequence, powerful genetic approaches to the dissection of the developmental cell death pathway in the nematode . (referred to as programmed cell death) have provided insights into rules that govern cellular life/death decisions, the process by which cells die, and the elimination of cell corps作者: Interlocking 時(shí)間: 2025-3-23 00:33 作者: 跳動(dòng) 時(shí)間: 2025-3-23 05:15 作者: Deceit 時(shí)間: 2025-3-23 08:31 作者: 傀儡 時(shí)間: 2025-3-23 13:18
Orbital Cellulitis: Invasive Fungallustrated by a recently described experimental model of excitotoxicity in cerebellum. In this model of neurotoxicity, administration of certain indole alkaloid derivatives (e.g., harmaline or ibogaine) leads to excitotoxic insult in a subset of neurons in cerebellar cortex. The resulting neuronal in作者: 共同確定為確 時(shí)間: 2025-3-23 14:53 作者: 裝勇敢地做 時(shí)間: 2025-3-23 20:57 作者: 出沒(méi) 時(shí)間: 2025-3-24 01:35
The State of Open and Distance Education,le many thousands of cases of viral encephalitis occur worldwide each year, infections caused by the arthropod-borne viruses (arboviruses) and herpes simplex virus (HSV) are the most life-threatening. The arboviruses that cause encephalitis in humans include members of the flavivirus family, the bun作者: MARS 時(shí)間: 2025-3-24 04:31
Optical and Wireless TechnologiesGene targeting and transgenic strategies have provided strong evidence for the central role of the prion protein in the transmission and pathogenesis of TSEs, now termed prion diseases, and have begun to be used to examine the mechanisms by which the formation of conformationally altered prion prote作者: inculpate 時(shí)間: 2025-3-24 08:50
Anish Sachdeva,Surjit Kumar Gandhins, emphasizing the role of glutamate receptor activation. The focus on intracellular mechanisms has led to an approach of identifying individual mechanisms of injury in simple systems and then examining their role in more intact systems. Rarely, however, are these mechanisms placed into their prope作者: IOTA 時(shí)間: 2025-3-24 10:54 作者: 紀(jì)念 時(shí)間: 2025-3-24 18:33 作者: Crayon 時(shí)間: 2025-3-24 22:25
Optical and Wireless TechnologiesGene targeting and transgenic strategies have provided strong evidence for the central role of the prion protein in the transmission and pathogenesis of TSEs, now termed prion diseases, and have begun to be used to examine the mechanisms by which the formation of conformationally altered prion proteins cause cell dysfunction and death.作者: Extemporize 時(shí)間: 2025-3-24 23:11 作者: SLAG 時(shí)間: 2025-3-25 05:43 作者: 高調(diào) 時(shí)間: 2025-3-25 08:46
Book 1999l nervous system cell death, and has been expertly edited by Vassilis Koliatsos and Rajiv Ratan. That the inap- propriate demise of brain or spinal cord cells is at the root of many neurological diseases has been appreciated since the early days of microscopic neuropathology, but it has only been in作者: 起皺紋 時(shí)間: 2025-3-25 12:18
,From Globalization to China’s Urbanization,rincipal mode of neuronal death has been recognized. Because axotomy can lead to neuronal death via apoptosis, it has emerged as powerful model for studying the molecular regulators and effectors of apoptosis in neurons.作者: 時(shí)間等 時(shí)間: 2025-3-25 16:26
Granulomatosis with Polyangiitis (GPA) neurons can transcribe up to 30% of the expressed genome. Animal studies indicated continued accumulation of DNA damage in neurons and a reduction in repair capacity with advancing age. On the other hand, it was generally believed that the brain was relatively resistant to DNA damage.作者: RAFF 時(shí)間: 2025-3-25 23:14
The State of Open and Distance Education,an acute encephalitis in mice. Since murine Sindbis virus infection exhibits many features that are similar to the human alphavirus-induced encephalitides, it serves as a useful animal model for studying these illnesses.作者: sperse 時(shí)間: 2025-3-26 03:47
Axotomy-Induced Motor Neuron Deathrincipal mode of neuronal death has been recognized. Because axotomy can lead to neuronal death via apoptosis, it has emerged as powerful model for studying the molecular regulators and effectors of apoptosis in neurons.作者: Explicate 時(shí)間: 2025-3-26 05:41
DNA Repair and Neurological Diseases neurons can transcribe up to 30% of the expressed genome. Animal studies indicated continued accumulation of DNA damage in neurons and a reduction in repair capacity with advancing age. On the other hand, it was generally believed that the brain was relatively resistant to DNA damage.作者: 夸張 時(shí)間: 2025-3-26 11:16
Viral Encephalitisan acute encephalitis in mice. Since murine Sindbis virus infection exhibits many features that are similar to the human alphavirus-induced encephalitides, it serves as a useful animal model for studying these illnesses.作者: 獎(jiǎng)牌 時(shí)間: 2025-3-26 12:50 作者: Medicare 時(shí)間: 2025-3-26 17:49 作者: BOON 時(shí)間: 2025-3-26 22:45
Asynchronous Death as a Characteristic Feature of Apoptosisl days. In fact, “naturally occurring” cell death in the developing nervous system was overlooked for a long time because of its sporadic asynchronous nature and the relatively short period of time cells show morphological signs of death.作者: 主動(dòng)脈 時(shí)間: 2025-3-27 04:23 作者: 賠償 時(shí)間: 2025-3-27 05:23 作者: 埋伏 時(shí)間: 2025-3-27 12:16 作者: 躲債 時(shí)間: 2025-3-27 17:15 作者: Subdue 時(shí)間: 2025-3-27 19:55
Dissipative Harmonic Oscillator,80 it has been given a particular, cellular, sense, with the claim that virtually all cell deaths can be classified dichotomously as either apoptosis or necrosis .. Previously, this necrosis had been called “coagulative necrosis” by several authors including Kerr et al. ..作者: 骯臟 時(shí)間: 2025-3-27 23:26
,From Globalization to China’s Urbanization,es mitochondrial energetic and oxidative dysfunction, due to congenital genetic defects, and perhaps to radical-induced mutations and oxidative enzyme impairment, in several neurodegenerative disorders (reviewed by .).作者: Scleroderma 時(shí)間: 2025-3-28 04:38
Advances in 21st Century Human Settlementshologic characteristics of apoptosis are readily recognizable, and internucleosomal DNA degradation is a frequent feature of apoptotic death ., the actual apoptotic mechanism has been difficult to define ..作者: 展覽 時(shí)間: 2025-3-28 06:36
Imaging: Magnetic Resonance Imaginge. The mitochondria, however, contain their own unique DNA (mtDNA), in the form of a 16.6 kB circular molecule that encodes 37 genes, including those for 13 mitochondrial respiratory chain proteins, 22 transfer RNAs, and 2 ribosomal RNAs (.).作者: 鴿子 時(shí)間: 2025-3-28 11:22 作者: SOB 時(shí)間: 2025-3-28 14:43
Neuronal Cell Death in , nt cell death in higher organisms. Here we first review the features of the nematode experimental system and then discuss current understanding of both developmental and degenerative cell death mechanisms in ..作者: Exterior 時(shí)間: 2025-3-28 22:15
Apoptosis Versus Necrosisg as long ago as 1885 .. The word . has been used in English, French (“nécrose”), and German for several centuries to mean the mortification of tissue, and 2000 years ago its Greek equivalent carried a similar meaning. Even today, the word is still frequently used in this general sense, but since 19作者: 脫水 時(shí)間: 2025-3-29 01:51 作者: LUDE 時(shí)間: 2025-3-29 05:44 作者: 精密 時(shí)間: 2025-3-29 07:36
Calcium and Cell Death a plethora of cellular functions have been found to require Ca. signals or simply the maintenance of a set Ca. concentration. The major requirement for the signaling function of Ca. is the existence of a concentration gradient between the interstitial fluid and the interior of the cell. Whereas ext作者: Culpable 時(shí)間: 2025-3-29 15:22 作者: 角斗士 時(shí)間: 2025-3-29 18:39 作者: 撤退 時(shí)間: 2025-3-29 22:02 作者: CIS 時(shí)間: 2025-3-30 01:20
Cellular Interactions that Regulate Programmed Cell Death in the Developing Vertebrate Nervous Systeent of various phenotypes, connectives, and functions. In this context, it is somewhat paradoxical that one of the most fundamental features of the developing nervous system is the widespread death of large numbers of neurons. Characterizing both the cell-cell interactions and molecular mechanisms t作者: 修飾 時(shí)間: 2025-3-30 06:50
Axotomy-Induced Motor Neuron Deathis performed, axonal injury can lead to robust attempts by neurons to regenerate axons and reestablish functional contact with targets, or it can begin a complex process leading to neuronal death. Recent advances have begun to identify genes that execute or regulate a particular phenotype after axon作者: 管理員 時(shí)間: 2025-3-30 12:09 作者: 攀登 時(shí)間: 2025-3-30 14:43
Neurotoxins and Neuronal Deathlustrated by a recently described experimental model of excitotoxicity in cerebellum. In this model of neurotoxicity, administration of certain indole alkaloid derivatives (e.g., harmaline or ibogaine) leads to excitotoxic insult in a subset of neurons in cerebellar cortex. The resulting neuronal in作者: Malfunction 時(shí)間: 2025-3-30 18:15
DNA Repair and Neurological Diseasespproaches to the analysis of DNA repair are beginning to address differences in repair pathways that correlate with patterns of gene expression and DNA replication. Early studies of DNA repair in the nervous system largely ignored the inherent heterogeneity in repair, allowing the assumption that re作者: Talkative 時(shí)間: 2025-3-30 23:41
Cell Death and the Mitochondrial Encephalomyopathies inner mitochondrial membrane where electron transfer through the respiratory chain assemblies leads to efficient production of adenosine triphosphate (ATP), the major currency of energy within the cell (.). The majority of mitochondrial respiratory proteins are encoded by genes in the nuclear genom作者: 暗諷 時(shí)間: 2025-3-31 01:14
Viral Encephalitisle many thousands of cases of viral encephalitis occur worldwide each year, infections caused by the arthropod-borne viruses (arboviruses) and herpes simplex virus (HSV) are the most life-threatening. The arboviruses that cause encephalitis in humans include members of the flavivirus family, the bun作者: 詢問(wèn) 時(shí)間: 2025-3-31 07:42 作者: Perceive 時(shí)間: 2025-3-31 10:21
Blood Flow, Energy Failure, and Vulnerability to Strokens, emphasizing the role of glutamate receptor activation. The focus on intracellular mechanisms has led to an approach of identifying individual mechanisms of injury in simple systems and then examining their role in more intact systems. Rarely, however, are these mechanisms placed into their prope作者: 相符 時(shí)間: 2025-3-31 13:51
