標題: Titlebook: Cell Death; Mechanism and Diseas Hao Wu Book 2014 Springer Science+Business Media New York 2014 Apoptosis.Cell death.Receptors [打印本頁] 作者: 討論小組 時間: 2025-3-21 17:32
書目名稱Cell Death影響因子(影響力)
書目名稱Cell Death影響因子(影響力)學科排名
書目名稱Cell Death網(wǎng)絡(luò)公開度
書目名稱Cell Death網(wǎng)絡(luò)公開度學科排名
書目名稱Cell Death被引頻次
書目名稱Cell Death被引頻次學科排名
書目名稱Cell Death年度引用
書目名稱Cell Death年度引用學科排名
書目名稱Cell Death讀者反饋
書目名稱Cell Death讀者反饋學科排名
作者: STYX 時間: 2025-3-21 23:03 作者: prick-test 時間: 2025-3-22 03:30
Maria Renata da Cruz Duran,Adnan Qayyumeatment but might be susceptible to a combination of conventional chemotherapy combined with drugs that antagonise cell death inhibitory proteins, such as members of the Bcl-2 and IAP protein families.作者: Extemporize 時間: 2025-3-22 06:52 作者: humectant 時間: 2025-3-22 10:56
Optic Disorders and Visual Fieldn. We will discuss the structural insights for each structural domain of BCL-2 proteins that determine their function in the cytosol and the outer mitochondrial membrane. Furthermore, we will discuss the structural details of the interactions between BCL-2 family members and the various structural p作者: 巨碩 時間: 2025-3-22 13:52 作者: 巨碩 時間: 2025-3-22 18:58
Molecular Basis of Cell Death Programs in Mature T Cell Homeostasis,作者: 切碎 時間: 2025-3-22 22:40 作者: AMEND 時間: 2025-3-23 02:02 作者: Archipelago 時間: 2025-3-23 06:51
Cell Death and Cancer,eatment but might be susceptible to a combination of conventional chemotherapy combined with drugs that antagonise cell death inhibitory proteins, such as members of the Bcl-2 and IAP protein families.作者: 搬運工 時間: 2025-3-23 10:55
Neuronal Death Mechanisms in Development and Disease, such as modulating synaptic plasticity or mediating neurite pruning to ensure the establishment of precise neuronal circuitry. Lastly, we provide a broad overview of the complex and multiple cell death mechanisms seen in neurons after injury and in neurodegenerative diseases.作者: 鐵砧 時間: 2025-3-23 17:26 作者: Sarcoma 時間: 2025-3-23 18:51
Book 2014rgeted therapies aimed at interdicting cell death machineries are also discussed extensively.?The molecular mechanisms that underlie apoptotic cell death are illustrated from the perspectives of both the intrinsic, mitochondrial apoptotic pathway and the extrinsic, death receptor pathway.?Key player作者: 碎石頭 時間: 2025-3-24 01:35 作者: 材料等 時間: 2025-3-24 02:59 作者: 字的誤用 時間: 2025-3-24 08:43
Maria Renata da Cruz Duran,Adnan Qayyum DNA damage and the molecular mechanisms that mediate p53-dependent and -independent apoptosis. Moreover, we discuss the signals that trigger the DDR during malignant propagation and the importance of DNA damage-mediated apoptosis in preventing tumorigenesis.作者: 禍害隱伏 時間: 2025-3-24 12:47 作者: Charade 時間: 2025-3-24 15:47
Historical Perspective: The Seven Ages of Cell Death Research,ement it were elucidated and abnormalities in the physiological cell death process were linked to human disease. The stages of research into the field of cell death can be likened to the ages of man, except in this case there was an extraordinarily long infancy, and despite the current maturity of the field, it continues to rapidly grow.作者: 性滿足 時間: 2025-3-24 20:25 作者: Asparagus 時間: 2025-3-25 01:12 作者: Bureaucracy 時間: 2025-3-25 04:58
Organic Cotton and Environmental Impacts,tides and small-molecule inhibitors suggests that emerging techniques and strategies will be required to identify a new generation of drugs targeting Bcl-2 family proteins. We review some of the more promising therapeutic opportunities for treating diseases that have been revealed by our current mechanistic understanding of these proteins.作者: 狂怒 時間: 2025-3-25 10:20 作者: Insul島 時間: 2025-3-25 13:18
Bcl-2 Family and Their Therapeutic Potential,tides and small-molecule inhibitors suggests that emerging techniques and strategies will be required to identify a new generation of drugs targeting Bcl-2 family proteins. We review some of the more promising therapeutic opportunities for treating diseases that have been revealed by our current mechanistic understanding of these proteins.作者: Yourself 時間: 2025-3-25 16:22 作者: fabricate 時間: 2025-3-25 22:17 作者: Glossy 時間: 2025-3-26 03:04 作者: Patrimony 時間: 2025-3-26 05:41
Structural Basis of Death Receptor Signaling,ric death domain superfamily complexes in caspase activation in apoptosis and the assembly of a functional amyloid complex in kinase activation in necrosis. They open a new vista for our understanding of intracellular signaling in the DR pathway and other innate and adaptive immune pathways.作者: follicular-unit 時間: 2025-3-26 08:34 作者: 進取心 時間: 2025-3-26 15:56
Sarcoptic Skin Disease (or Scabies) and is involved in the pathogenesis of human diseases ranging from immune disorders and neurodegeneration to cancer. Over the last two decades, much has been learned about the molecular basis of this pathway as well as its delicate regulatory mechanisms. The central role of the cellular organelle, 作者: 的染料 時間: 2025-3-26 18:24
Organic Cotton and Environmental Impacts,s develop when cells bypass regulated cell death and grow faster than they die, while in degenerative diseases there is too much cell death. Thus understanding how Bcl-2 proteins modulate cell survival will provide novel important insight into therapeutic targets, as evidenced by recent successes sp作者: 懶鬼才會衰弱 時間: 2025-3-26 21:12
Maria Renata da Cruz Duran,Adnan Qayyumed cell death can lead to developmental defects, neuropathies, infections, and cancer. Inhibitor of apoptosis (IAP) proteins are evolutionarily conserved regulators of cell death with impact on numerous cellular processes. Initially identified as inhibitors of cell death during viral infection, IAP 作者: IRS 時間: 2025-3-27 01:26
Maria Renata da Cruz Duran,Adnan Qayyumment of novel treatments. A cancer is a clonally related population of cells that persists, in part, because cells with altered DNA have been able to survive. It follows that genetic mutations that prevent a cell from killing itself are likely to promote the development of cancer. On the other hand,作者: 倒轉(zhuǎn) 時間: 2025-3-27 07:23
Maria Renata da Cruz Duran,Adnan Qayyumesponse (DDR) is a highly coordinated signaling pathway that functions to detect and repair DNA damage in cells, inhibiting transformation. However, when DNA damage is so severe that it cannot be repaired, the DDR promotes apoptosis, thus preventing the propagation of abnormal cells. The tumor suppr作者: BRUNT 時間: 2025-3-27 10:49
https://doi.org/10.1007/978-981-13-0469-9roperly organized nervous system during development. However, unlike most other cell types, neurons must survive for the lifetime of the organism and therefore possess multiple, unique ways to tightly regulate cell death pathways. Neurons employ several distinct “brakes” within the apoptotic pathway作者: 護航艦 時間: 2025-3-27 16:58
Jyotiranjan Nayak,Sasmita Mishraf building blocks of cellular structures such as lipid membranes, proteins, and nucleic acids. In contrast, nutrient excess can increase ER stress and reactive oxygen species, leading to cellular damage and apoptosis. In this chapter, we detail three important aspects of the interplay between metabo作者: 明確 時間: 2025-3-27 21:42
,Vasoproliferative Tumors of the?Retina,ers necrosis in response to TNF-like death cytokines, certain toll-like receptors, and in response to viral pathogens was described. Signaling adaptors that contain the RIP homotypic interaction motif (RHIM), such as receptor interacting protein kinase (RIPK) 1 and RIPK3, are key inducers for this f作者: 一起平行 時間: 2025-3-27 22:27 作者: 混合 時間: 2025-3-28 03:06
Ning Fan,Xuyang Liu,Jiantao Wangis. Here we review the structural information on DR signaling accumulated in the past decade and more. These studies highlight the assembly of oligomeric death domain superfamily complexes in caspase activation in apoptosis and the assembly of a functional amyloid complex in kinase activation in nec作者: carbohydrate 時間: 2025-3-28 08:59
https://doi.org/10.1007/978-1-4614-9302-0Apoptosis; Cell death; Receptors作者: Archipelago 時間: 2025-3-28 13:34
978-1-4939-4325-8Springer Science+Business Media New York 2014作者: Mnemonics 時間: 2025-3-28 17:33 作者: 撫慰 時間: 2025-3-28 20:58
http://image.papertrans.cn/c/image/222800.jpg作者: 過時 時間: 2025-3-29 00:30
Historical Perspective: The Seven Ages of Cell Death Research,he same terms for publications 25 years ago yields only 42 publications. Although it has been recognised for over 150 years that death of some of an organism’s cells is a normal physiological process, widespread interest in the field has only come about in recent times, once the mechanisms that impl作者: 強制令 時間: 2025-3-29 04:03
The Intrinsic Apoptotic Pathway, and is involved in the pathogenesis of human diseases ranging from immune disorders and neurodegeneration to cancer. Over the last two decades, much has been learned about the molecular basis of this pathway as well as its delicate regulatory mechanisms. The central role of the cellular organelle, 作者: 逃避系列單詞 時間: 2025-3-29 10:37
Bcl-2 Family and Their Therapeutic Potential,s develop when cells bypass regulated cell death and grow faster than they die, while in degenerative diseases there is too much cell death. Thus understanding how Bcl-2 proteins modulate cell survival will provide novel important insight into therapeutic targets, as evidenced by recent successes sp作者: Consensus 時間: 2025-3-29 13:52
IAP Proteins and Their Therapeutic Potential,ed cell death can lead to developmental defects, neuropathies, infections, and cancer. Inhibitor of apoptosis (IAP) proteins are evolutionarily conserved regulators of cell death with impact on numerous cellular processes. Initially identified as inhibitors of cell death during viral infection, IAP
