標(biāo)題: Titlebook: Cardiovascular Genomics: New Pathophysiological Concepts; Proceedings of the 2 Pieter A. Doevendans,Stefan K??b Conference proceedings 2002 [打印本頁(yè)] 作者: cerebellum 時(shí)間: 2025-3-21 19:28
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts影響因子(影響力)
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts影響因子(影響力)學(xué)科排名
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts被引頻次
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書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts年度引用學(xué)科排名
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts讀者反饋
書(shū)目名稱Cardiovascular Genomics: New Pathophysiological Concepts讀者反饋學(xué)科排名
作者: BARK 時(shí)間: 2025-3-21 21:01 作者: entrance 時(shí)間: 2025-3-22 01:08 作者: UNT 時(shí)間: 2025-3-22 08:18
1.8 Glossary of general abbreviations,r than single culprit genes form the basis for the widespread penetrance of the disease in the elderly Western population. We are applying functional Genomics to the study of atherosclerosis, with the goal of characterizing healthy and diseased gene expression profiles. While our immediate objective作者: cochlea 時(shí)間: 2025-3-22 09:58
53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,ure and prevention or reversal of end-organ damage with few side effects. This work will review current vascular gene transfer for hypertension with particular emphasis on.gene transfer in models of essential hypertension.作者: RACE 時(shí)間: 2025-3-22 13:17 作者: RACE 時(shí)間: 2025-3-22 18:06
52-220 - 52-261: F6O4SSb2.2H4N - Sb2Se3, these modifier genes have been recalcitrant to direct identification in human populations. In this regard, genetic studies in animals models of disease can identify candidate modifier genes and provide an insight on the genetic interactions that cause phenotypic variation in the human population[..作者: inquisitive 時(shí)間: 2025-3-22 23:02
1.8 Glossary of general abbreviations,oss of myocardial contractile function. The degeneration of structure and function in cardiomyocytes observed in dilation is less amenable to.analysis compared to hypertrophie changes associated with enhanced contractile function and enlarged cardiomyocyte size that are readily apparent by analyses 作者: ARCHE 時(shí)間: 2025-3-23 03:04 作者: Enrage 時(shí)間: 2025-3-23 08:05 作者: Stagger 時(shí)間: 2025-3-23 11:39 作者: capsule 時(shí)間: 2025-3-23 15:37 作者: glucagon 時(shí)間: 2025-3-23 19:03
Cardiovascular Proteomics: Effect of Hypoxic Conditionsmicroarrays, differential display, SAGE) or proteins (2D gel electrophoresis, protein chips) at one time. It leads to the discovery of unexpected genes whose expression is modified but also to the development of new hypotheses concerning the etiology of the disease. It also provides a useful tool to作者: 學(xué)術(shù)討論會(huì) 時(shí)間: 2025-3-23 22:51
Using Comparative Genome Analysis to Find Interaction Partners for Frataxinontext and on the other hand the type of functional interaction shows that conservation of genomic context can indeed be a reliable indication of a functional interaction. The functional interactions that are reflected in the conservation of genomic context include a wide variety of relations betwee作者: defeatist 時(shí)間: 2025-3-24 02:40
In Vitro-In Vivo Gene Expression Analysis in Atherosclerosisr than single culprit genes form the basis for the widespread penetrance of the disease in the elderly Western population. We are applying functional Genomics to the study of atherosclerosis, with the goal of characterizing healthy and diseased gene expression profiles. While our immediate objective作者: Pelvic-Floor 時(shí)間: 2025-3-24 08:29 作者: muster 時(shí)間: 2025-3-24 14:45 作者: 一起 時(shí)間: 2025-3-24 16:48 作者: 漂浮 時(shí)間: 2025-3-24 19:18
Little Mice with Big Hearts: Finding the Molecular Basis for Dilated Cardiomyopathyoss of myocardial contractile function. The degeneration of structure and function in cardiomyocytes observed in dilation is less amenable to.analysis compared to hypertrophie changes associated with enhanced contractile function and enlarged cardiomyocyte size that are readily apparent by analyses 作者: Champion 時(shí)間: 2025-3-25 00:01
Cardiac Hypertrophic Signaling the Good, the Bad and the Uglyignaling malfunctions underly human diseases. For example, cancer evolves following inactivating mutations in growth-inhibitory pathways, resulting in specialized cells with proliferative advantages over its neighbouring cells[.]. Diabetes results from defects in the insulin-signaling pathway used t作者: LVAD360 時(shí)間: 2025-3-25 04:26 作者: Nefarious 時(shí)間: 2025-3-25 09:40
Cardiovascular Genomics: New Pathophysiological Concepts978-1-4615-1005-5Series ISSN 0166-9842 作者: 說(shuō)明 時(shí)間: 2025-3-25 15:15 作者: Mechanics 時(shí)間: 2025-3-25 16:19 作者: 虛度 時(shí)間: 2025-3-25 21:48
53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,esses in heart and/or arteries during these diseases. Moreover, large epidemiological studies have provided insights in environmental as well genetic risk factors and will continue to unravel new interactions. Progress in defining the cellular and molecular interactions involved, however has been hi作者: defile 時(shí)間: 2025-3-26 03:08
53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,o to predict new functional interactions between proteins using comparative genome analysis [.]. Various methods have been proposed and demonstrated to predict functional interaction between proteins based on the genomic context of their genes [....]. These methods are all based on variations of the作者: 中國(guó)紀(jì)念碑 時(shí)間: 2025-3-26 07:12 作者: 網(wǎng)絡(luò)添麻煩 時(shí)間: 2025-3-26 09:09
53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,osis[.]. As our understanding of disease pathology, molecular biology and vectors improves, gene transfer for complex diseases is becoming a realistic prospect. At present, the vast majority of vascular gene transfer studies serve as an investigational, rather than a therapeutic tool. Sizeable obsta作者: Hemiparesis 時(shí)間: 2025-3-26 16:10
1.3 Arrangement of tables and data,lar tone, while the adrenergic system affects vascular tone, cardiac contraction, and heart rate. Thus, it is no surprise that changes in hormonal status can have a strong influence on blood pressure. In fact, most forms of secondary hypertension are determined by an alteration in secretion of speci作者: 似少年 時(shí)間: 2025-3-26 17:04
52-123 - 52-157: Cl3F2Sb.2Cs - Cl3Sb.C14H10,reased peripheral vascular resistance and changes at the endothelial layer of the vessels. The endothelium is a dynamic organ which responds to several stimuli triggered by a wide plethora of effectors in order to restore the loss of function [.]. There is accumulating evidence pointing towards a dy作者: concubine 時(shí)間: 2025-3-26 22:18
53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,rom pump failure. Over the age of 75 years the prevalence rises to 10%. Genetic causes of familial dilated cardiomyopathy, covering about 20% of all patients with dilated cardiomyopathy [.], are currently being resolved at a rapid pace and include cytoskeletal, sarcomeric and metabolic genes [.]. In作者: GULLY 時(shí)間: 2025-3-27 01:27
52-220 - 52-261: F6O4SSb2.2H4N - Sb2Se3,S, heart failure accounts for the majority of the cardiovascular mortality and morbidity in patients over the age of 65 [.]. Most causes of heart failure result from coronary heart disease, but a substantial number are caused by idiopathic dilated cardiomyopathy (DCM). While inherited gene defects h作者: 裹住 時(shí)間: 2025-3-27 07:44
1.8 Glossary of general abbreviations, organization and 13-adrenergic responsiveness. [.] Typical microscopic features include high incidence of myocyte hypertrophy and apoptosis, [.–.] varying degrees of fibrosis and the presence of lymphocytes in the interstitial space [.]. Clinically, the disease displays heart failure symptoms consi作者: gait-cycle 時(shí)間: 2025-3-27 09:54 作者: 絕緣 時(shí)間: 2025-3-27 16:55 作者: 古老 時(shí)間: 2025-3-27 21:45 作者: stress-test 時(shí)間: 2025-3-28 00:55
Human Essential Hypertension: Role of the Genes of the Renin-Aldosterone SystemA number of epidemiologic studies have shown that individual blood pressure levels result from both genetic predisposition and environmental factors. It is generally accepted that approximately 30% of the variance of blood pressure is attributable to genetic heritability and 50% to environmental influences [.].作者: Pulmonary-Veins 時(shí)間: 2025-3-28 04:43 作者: 泥瓦匠 時(shí)間: 2025-3-28 07:22 作者: phase-2-enzyme 時(shí)間: 2025-3-28 12:55
Developments in Cardiovascular Medicinehttp://image.papertrans.cn/c/image/221946.jpg作者: 浪費(fèi)物質(zhì) 時(shí)間: 2025-3-28 16:09
Genomics and Clinical Cardiology: Hype or Hope?rdiography, coronary angiography and programmed electrical stimulation of the heart, to name a few techniques, have enormously advanced our ability to correctly diagnose what is wrong with the heart. Also our non-invasive and invasive therapeutic possibilities have improved to such an extent that de作者: exceed 時(shí)間: 2025-3-28 20:01 作者: 宮殿般 時(shí)間: 2025-3-29 01:41
Cardiovascular Proteomics: Effect of Hypoxic Conditionsesses in heart and/or arteries during these diseases. Moreover, large epidemiological studies have provided insights in environmental as well genetic risk factors and will continue to unravel new interactions. Progress in defining the cellular and molecular interactions involved, however has been hi作者: 萬(wàn)神殿 時(shí)間: 2025-3-29 07:08 作者: 討好美人 時(shí)間: 2025-3-29 07:23 作者: CONE 時(shí)間: 2025-3-29 11:48
Gene Therapy for Hypertension: Future or Fiction?osis[.]. As our understanding of disease pathology, molecular biology and vectors improves, gene transfer for complex diseases is becoming a realistic prospect. At present, the vast majority of vascular gene transfer studies serve as an investigational, rather than a therapeutic tool. Sizeable obsta作者: Suppository 時(shí)間: 2025-3-29 18:48 作者: STIT 時(shí)間: 2025-3-29 22:59 作者: insurrection 時(shí)間: 2025-3-30 03:13 作者: 和平 時(shí)間: 2025-3-30 07:40
Gene Expression in Cardiac Hypertrophy and Failure: Role of G Protein-Coupled ReceptorsS, heart failure accounts for the majority of the cardiovascular mortality and morbidity in patients over the age of 65 [.]. Most causes of heart failure result from coronary heart disease, but a substantial number are caused by idiopathic dilated cardiomyopathy (DCM). While inherited gene defects h作者: 意外的成功 時(shí)間: 2025-3-30 08:23 作者: pineal-gland 時(shí)間: 2025-3-30 13:23 作者: Spartan 時(shí)間: 2025-3-30 17:43
Ion Channel Regulation: From Arrhythmias to Genes to Channels (to Cures?)d, leaving friends and family of a victim in a struggle to make sense of the unfortunate situation. Causes of cardiac arrest are myriad, including obstructive or hypertrophic heart disease, pulmonary embolism, aortic dissection or rupture, and cardiac arrhythmias. In adults in the developed world, t作者: motor-unit 時(shí)間: 2025-3-30 21:26
0166-9842 icht. It was hardly possible to imagine the progress in the field in those four years. In 1997, gene expression was still an art focused on individual genes; in 200 I, many labs have access to micro-array facilities to determine the expression of thousands of genes simultaneously. In 1997, gene expr作者: BULLY 時(shí)間: 2025-3-31 02:52 作者: amphibian 時(shí)間: 2025-3-31 08:35
