標(biāo)題: Titlebook: Cardiovascular Biology of Purines; Geoffrey Burnstock,James G. Dobson,Joel Linden Book 1998 Springer Science+Business Media New York 1998 [打印本頁(yè)] 作者: 巡洋 時(shí)間: 2025-3-21 19:01
書目名稱Cardiovascular Biology of Purines影響因子(影響力)
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書目名稱Cardiovascular Biology of Purines讀者反饋學(xué)科排名
作者: 鑒賞家 時(shí)間: 2025-3-21 22:30 作者: addict 時(shí)間: 2025-3-22 01:27
Vascular Biology and Pharmacology of Adenosine Receptors,denosine research began 35 years ago, when Berne (1963) and Gerlach et al. (1963) independently proposed that adenosine controls hypoxic coronary vasodilation. The eve of those anniversaries is a fitting time to review the actions of adenosine on blood vessels. Some older reviews (Belardinelli et al作者: Gum-Disease 時(shí)間: 2025-3-22 07:38 作者: Debility 時(shí)間: 2025-3-22 09:27
Adenosine, KATP Channel, and Cardioprotection in the Intact Heart,icacious therapeutics for treating myocardial ischemia. Of particular interest is the adenosine-K. axis. Development of the interest in this area was based on the intersection of several independent lines of investigation. The interest in preconditioning brought the K. and adenosine researchers toge作者: atrophy 時(shí)間: 2025-3-22 13:20
Adenosine Receptor Subtypes and Cardioprotection in Cardiac Myocyte and Transgenic Models,used on improving the blood supply to the ischemic myocardium, recent discoveries of myocardial adaptive protective mechanisms have re-focused therapeutic attention on the heart muscle. Adenosine is released in large amounts during ischemia and has been shown to mediate a number of important cardiop作者: atrophy 時(shí)間: 2025-3-22 17:26
Regulation of Adenosine Receptor Subtypes and Cardiac Dysfunction in Human Heart Failure,e four adenosine receptor subtypes A., A., A., A. and the A.-mediated antiadrenergic signal transduction pathway has been studied in nonfailing and failing human hearts. Gene expression of myocardial A.-adenosine receptors and A.-mediated inhibitory effects on cAMP levels or negative inotropic effec作者: 環(huán)形 時(shí)間: 2025-3-23 01:11
Adenosine and Cardiac Arrhythmias, 1929]. These potent properties and its short half life in vivo have allowed adenosine to establish its place as a unique and invaluable tool for both elucidating the mechanism of cardiac arrhythmias and for treating supraventricular tachycardia.作者: Rankle 時(shí)間: 2025-3-23 04:01
Adenosine and Cardiac Aging, in a greater manifestation of the antiadrenergic action of adenosine reducing ?-adrenergic responsiveness. Results suggest that despite similar adenosine production by young adult and aged myocardium, elevated basal interstitial adenosine levels in aged myocardium result from depressed . activity o作者: deface 時(shí)間: 2025-3-23 05:53 作者: 鬧劇 時(shí)間: 2025-3-23 10:25
The G Protein-Coupled P2Y Receptors,ted the P2Y receptors and are distinguished individually by a numerical subscript. This family includes five phospholipase C-activating mammalian receptors, the P2Y., P2Y. P2Y., P2Y., and P2Y. receptors, that have been cloned and unambiguously shown to be activated by nucleotides after heterologous 作者: MOCK 時(shí)間: 2025-3-23 16:53 作者: Valves 時(shí)間: 2025-3-23 20:03
P2-Purinoceptors and Cardiac Functions,t, the role of adenosine and ATP in the control of cardiovascular function has been widely investigated. Although the determination of ATP actions in many tissues, including the myocardium, is complicated by its rapid catabolism to adenosine, it was already clear in early studies that ATP and adenos作者: 商品 時(shí)間: 2025-3-24 00:10
Neuromodulation in the Cardiovascular System,acetylcholine in sympathetic and parasympathetic nerves, ATP may be differentially released in response to nerve stimulation. ATP released from the nerve terminals of sympathetic neurones may contribute to neurogenic smooth muscle contraction through activation of P2X. receptors, or alternatively in作者: Deadpan 時(shí)間: 2025-3-24 04:32 作者: 搖擺 時(shí)間: 2025-3-24 07:15
P2 Purinoceptors and Regulation of the Function of Platelets, Erythrocytes and Mast Cells,or the development of new pharmacological agonists and antagonists (notably platelets), the molecular cloning of members of the P2Y family, the dissection of the signal transduction pathways (especially erythrocytes), and the identification recently of the elusive permeabilizing ATP receptor (mast c作者: Alveoli 時(shí)間: 2025-3-24 13:41 作者: 粉筆 時(shí)間: 2025-3-24 16:02
Recent Advances in Cardiac Adenosine Metabolism, cardiac adenosine formation is the intracellular dephosphorylation of AMR More than 90% of the adenosine formed is rephosphorylated to AMP via adenosine kinase (1.95 nmol/min/g) and only a small fraction (0.06 nmol/min/g) escapes the metabolic cycle between AMP and adenosine and is released into th作者: 收藏品 時(shí)間: 2025-3-24 22:30 作者: 作嘔 時(shí)間: 2025-3-24 23:17 作者: floaters 時(shí)間: 2025-3-25 05:07 作者: Focus-Words 時(shí)間: 2025-3-25 08:21 作者: GUILE 時(shí)間: 2025-3-25 11:42
Molecular Biology and Pharmacology of Recombinant Adenosine Receptors, but can activate phospholipase C via G protein βγ subunits, but the A. receptor desensitizes much more slowly. The A. and A. receptors both couple to Gs, but theA. receptor also can couple to Gq to mobilize calcium.作者: inconceivable 時(shí)間: 2025-3-25 16:03
Regulation of Adenosine Receptor Subtypes and Cardiac Dysfunction in Human Heart Failure,RNA was not detectable. However, A.-adenosine receptor gene expression was increased by about 60% in patients with dilated cardiomyopathy. Thus, the A.-upregulation may indicate a pathophysiological role for adenosine and adds therefore to the increasingly complex picture of molecular alterations in heart failure.作者: 兇猛 時(shí)間: 2025-3-25 22:31 作者: CT-angiography 時(shí)間: 2025-3-26 02:27 作者: 相反放置 時(shí)間: 2025-3-26 06:23 作者: ANTI 時(shí)間: 2025-3-26 10:43
Robert Boardman,James F. Keeleycellular adenosine concentration is normally very low and adenosine is continuously formed extracellularly from released adenine nucleotides, the concentration gradient for adenosine in the normoxic heart is from extracellular to intracellular. This gradient is rapidly reversed during hypoxia or pharmacological inhibition of adenosine kinase.作者: aristocracy 時(shí)間: 2025-3-26 13:57 作者: 影響深遠(yuǎn) 時(shí)間: 2025-3-26 18:42
Vascular Biology and Pharmacology of Adenosine Receptors,les of adenosine (Tucker and Linden, 1992; Muller et al., 1996; Olsson, 19%; Ongini and Fredholm, 1996; Feoktistov and Biaggioni, 1997) allow the authors to focus this review on important new developments that have occurred over the past two or three years. Readers wanting a broader understanding should consult those earlier articles.作者: 怎樣才咆哮 時(shí)間: 2025-3-27 00:48 作者: Ambulatory 時(shí)間: 2025-3-27 02:09
Recent Advances in Cardiac Adenosine Metabolism,cellular adenosine concentration is normally very low and adenosine is continuously formed extracellularly from released adenine nucleotides, the concentration gradient for adenosine in the normoxic heart is from extracellular to intracellular. This gradient is rapidly reversed during hypoxia or pharmacological inhibition of adenosine kinase.作者: occult 時(shí)間: 2025-3-27 07:37
Roles of Adenosine in Angiogenesis,ch as nitric oxide and vascular endothelial growth factor. Through the interplay of tissue oxygen tension, adenosine, nitric oxide and vascular endothelial growth factor, a complex physiological control system ensures the matching of vascularity and metabolic needs in a variety of organs and circumstances.作者: LAST 時(shí)間: 2025-3-27 10:18 作者: fluoroscopy 時(shí)間: 2025-3-27 17:36
Nuclear Energy and International Cooperationnd it may be mitochondrial. If this is the case, it will be imperative to determine the mechanism by which opening of these channels can exert cardioprotective and energy sparing effects. Determination of these mechanisms may potentially lead to the development of novel and efficacious therapeutics.作者: Celiac-Plexus 時(shí)間: 2025-3-27 17:56 作者: 廚房里面 時(shí)間: 2025-3-28 01:22 作者: Decrepit 時(shí)間: 2025-3-28 04:25 作者: INTER 時(shí)間: 2025-3-28 07:12 作者: Diverticulitis 時(shí)間: 2025-3-28 13:13
Nuclear Export of Herpes Virus RNA,recombinant receptors specific for ATP divided in two families cf ionotropic ligand-gated ion channels P2X-purinoceptors and metabotropic G protein-coupled P2Y-purinoceptors. Molecular characterization and localization of several P2-purinoceptor subtypes in the heart provide functional evidence in f作者: 佛刊 時(shí)間: 2025-3-28 17:18
https://doi.org/10.1007/978-1-349-05984-3bit transmitter release via prejunctional A. receptors thereby diminishing α. adrenoceptor-mediated vasoconstriction. Presumably, not only adenosine but also ATP inhibits transmitter release through activation of presynaptic A. receptors which might explain various observations previously ascribed t作者: 阻撓 時(shí)間: 2025-3-28 20:36 作者: 類人猿 時(shí)間: 2025-3-29 00:11 作者: 帶傷害 時(shí)間: 2025-3-29 03:17 作者: forbid 時(shí)間: 2025-3-29 10:21 作者: 職業(yè)拳擊手 時(shí)間: 2025-3-29 12:07
Adenosine Receptor Subtypes and Cardioprotection in Cardiac Myocyte and Transgenic Models, or “primes” K. channels. The primed channels can be more responsive to adenosine released during the ischemia and thus protect the myocytes. Although unproven, evidence is growing to support this sequence of signaling events in mediating the protective effect of ischemie preconditioning. Activation作者: 世俗 時(shí)間: 2025-3-29 18:17
Molecular Biology of P2X Purinoceptors,terms of receptor stoichiometry, the lining of the ion pore, means by which the ionchannel opens and closes, allosteric modulatory sites, the ligand docking site and reasons for selectivity of nucleotidic analogues. The situation has been compounded by the discovery of splice variants of P2X subunit作者: 摻和 時(shí)間: 2025-3-29 20:18
The G Protein-Coupled P2Y Receptors, pharmacological responses of various tissues with a specific receptor. The most encouraging progress in drug development has been in the synthesis of selective agonists and antagonists for the P2Y. receptor. Mutational and modeling analyses have provided some insight into identification of the bind作者: Metamorphosis 時(shí)間: 2025-3-30 00:33 作者: 不法行為 時(shí)間: 2025-3-30 05:12
P2-Purinoceptors and Cardiac Functions,recombinant receptors specific for ATP divided in two families cf ionotropic ligand-gated ion channels P2X-purinoceptors and metabotropic G protein-coupled P2Y-purinoceptors. Molecular characterization and localization of several P2-purinoceptor subtypes in the heart provide functional evidence in f作者: 人類 時(shí)間: 2025-3-30 09:00
Neuromodulation in the Cardiovascular System,bit transmitter release via prejunctional A. receptors thereby diminishing α. adrenoceptor-mediated vasoconstriction. Presumably, not only adenosine but also ATP inhibits transmitter release through activation of presynaptic A. receptors which might explain various observations previously ascribed t作者: 說(shuō)笑 時(shí)間: 2025-3-30 13:34
Adenosine Receptors, Blood Vessel and Inflammation,-10. These adenosine-mediated changes in PMN and moncyte/macrophage function have the potential to alter the course of an inflammatory response in cardiovascular diseases such as ischemia/reperfusion injury, atherosclerotic plaque development and inflammatory aneurysm formation.作者: 畏縮 時(shí)間: 2025-3-30 19:20
Purinoreceptors and T Cell Function: Regulation of Lymphocyte Activation by Extracellular ATP and Ac T cells (CTL) and T-helper cells. Evidence to support this model is suggested by studies of the contribution of purinergic receptor expression to the . functional responses of T cells. It has been shown that some purinergic receptors in T cells are expressed in a differentiation-dependent manner (作者: 紀(jì)念 時(shí)間: 2025-3-30 23:19
Cardiovascular Biology of Purines978-1-4615-5603-9Series ISSN 0166-9842 作者: 南極 時(shí)間: 2025-3-31 03:53
0166-9842 Overview: 978-1-4613-7563-0978-1-4615-5603-9Series ISSN 0166-9842 作者: 搖曳的微光 時(shí)間: 2025-3-31 08:58
Nuclear Energy and International Cooperation 1929]. These potent properties and its short half life in vivo have allowed adenosine to establish its place as a unique and invaluable tool for both elucidating the mechanism of cardiac arrhythmias and for treating supraventricular tachycardia.作者: 匍匐前進(jìn) 時(shí)間: 2025-3-31 10:20 作者: DRILL 時(shí)間: 2025-3-31 13:50
978-1-4613-7563-0Springer Science+Business Media New York 1998作者: Alopecia-Areata 時(shí)間: 2025-3-31 20:38
Adenosine and Cardiac Arrhythmias, 1929]. These potent properties and its short half life in vivo have allowed adenosine to establish its place as a unique and invaluable tool for both elucidating the mechanism of cardiac arrhythmias and for treating supraventricular tachycardia.作者: Thymus 時(shí)間: 2025-3-31 22:16 作者: 報(bào)復(fù) 時(shí)間: 2025-4-1 04:51
https://doi.org/10.1007/978-3-319-29838-2ne allows the antiadrenergic action resulting from A. stimulation to be modulated by the concentration of interstitial adenosine. The higher the interstitial adenosine level the greater the attenuation of ?-adrenergic-elicited myocardial contractile and metabolic responses. The adenosine A. receptor作者: 芳香一點(diǎn) 時(shí)間: 2025-4-1 06:42
https://doi.org/10.1007/978-3-642-48876-4denosine research began 35 years ago, when Berne (1963) and Gerlach et al. (1963) independently proposed that adenosine controls hypoxic coronary vasodilation. The eve of those anniversaries is a fitting time to review the actions of adenosine on blood vessels. Some older reviews (Belardinelli et al作者: BLINK 時(shí)間: 2025-4-1 10:18 作者: synovial-joint 時(shí)間: 2025-4-1 17:00
Nuclear Energy and International Cooperationicacious therapeutics for treating myocardial ischemia. Of particular interest is the adenosine-K. axis. Development of the interest in this area was based on the intersection of several independent lines of investigation. The interest in preconditioning brought the K. and adenosine researchers toge
