標題: Titlebook: Cardiac-Vascular Remodeling and Functional Interaction; Yukio Maruyama (Professor and Chairman),Masatsugu Book 1997 Springer Japan 1997 a [打印本頁] 作者: Constrict 時間: 2025-3-21 17:05
書目名稱Cardiac-Vascular Remodeling and Functional Interaction影響因子(影響力)
書目名稱Cardiac-Vascular Remodeling and Functional Interaction影響因子(影響力)學科排名
書目名稱Cardiac-Vascular Remodeling and Functional Interaction網(wǎng)絡(luò)公開度
書目名稱Cardiac-Vascular Remodeling and Functional Interaction網(wǎng)絡(luò)公開度學科排名
書目名稱Cardiac-Vascular Remodeling and Functional Interaction被引頻次
書目名稱Cardiac-Vascular Remodeling and Functional Interaction被引頻次學科排名
書目名稱Cardiac-Vascular Remodeling and Functional Interaction年度引用
書目名稱Cardiac-Vascular Remodeling and Functional Interaction年度引用學科排名
書目名稱Cardiac-Vascular Remodeling and Functional Interaction讀者反饋
書目名稱Cardiac-Vascular Remodeling and Functional Interaction讀者反饋學科排名
作者: STING 時間: 2025-3-21 20:30
Vascular Remodeling During Heart Failurechanges in cardiac muscle function, heart failure is associated with alterations in the vasculature. Functional and structural alterations in the coronary circulation, including a failure of adaptive growth of the coronary circulation, may contribute to the loss of muscle function of the heart and m作者: maroon 時間: 2025-3-22 00:53
Myocardial Interstitial Collagen Matrix Remodeling in Response to a Chronic Elevation in Ventricularindividual myocytes, groups of cardiac myofibrils, muscle fibers, and muscle fiber bundles. Accordingly, it provides for cardiac myocyte and muscle fiber alignment, and in so doing it imparts a mechanical support to the myocardium that assists in the maintenance of ventricular shape, size, and funct作者: paltry 時間: 2025-3-22 05:14 作者: FLIP 時間: 2025-3-22 11:11
Cardiac Hypertrophy and the Renin-Angiotensin Systemx sequence of compensatory events that ultimately result in an adversely remodeled myocardium and a dilated, thin-walled, spherical ventricle. For a period of time, a preclinical heart failure state may exist in which there is ventricular dysfunction caused by myocardial damage but no clinical evide作者: 小鹿 時間: 2025-3-22 14:00
The Role of Manganese Superoxide Dismutase in the Acquisition of Tolerance of the Heart to Ischemia:olerance acquisition from the aspect of oxygen radical metabolism in the heart. Manganese superoxide dismutase (Mn-SOD), which scavenges the initial reductive metabolite of molecular oxygen on ischemia-reperfusion, was induced after various external stimuli including heat shock, α.-adrenergic stimul作者: 小鹿 時間: 2025-3-22 20:56
Remodeling: How Vessels Narrowmarkable ability of normal vessels to maintain the caliber appropriate for the blood flow needed for the subservient organ. Indeed, the ability to adjust caliber must be very primitive. The vessel tree, like any closed circuit, can only continue to conduct blood if each branch has an appropriate res作者: conspicuous 時間: 2025-3-22 22:01
Interaction Between Left-Ventricular Function and Loading Condition in Cardiovascular Diseaseenesis of the progression or amelioration of cardiac dysfunction. A decrease in peripheral resistance increases stroke volume until a certain critical level of afterload reduction; thus, afterload-reducing therapy is frequently performed in patients with heart failure. When afterload reducing is per作者: entitle 時間: 2025-3-23 02:11 作者: 供過于求 時間: 2025-3-23 07:37 作者: malign 時間: 2025-3-23 10:01
Transient Hemodynamic Perturbations: An Approach to the Study of Ventricular Interaction and Restoriem, we have developed approaches using transient (single-beat) hemodynamic perturbations that eliminate the confounding effects of series interactions and filling variations. In experimental studies in open-chest dogs, we have employed rapid changes in right heart volume to quantitate direct right-t作者: 議程 時間: 2025-3-23 16:29
Extrinsic Versus Intrinsic Determinants of the Diastolic Pressure—Volume Relationxternal to the heart, principally resulting from pericardial and right heart loads. Intrinsic factors can be divided into passive and active components. Passive elements include myocardial viscoelasticity, caused by structural proteins within the sarcomere, extra-cellular matrix proteins, and corona作者: 天空 時間: 2025-3-23 20:23
Endocardial—Myocardial Interactionxistence of an endocardium-mediated intracavitary autoregulation of cardiac performance has been postulated. Recent discoveries on morphology and function of the endocardium have substantiated this hypothesis. ., phenotypic expression of several receptors, tight and gap junctions, adhesion molecules作者: Evolve 時間: 2025-3-23 23:39
Arterial Compliance as Load on the Heartperiod), these are the major determinants of systolic and diastolic pressure in the ascending aorta. A decrease in total arterial compliance will, in the nonregulated cardiovascular system, decrease cardiac output and thus mean pressure while increasing pulse pressure. The result is a large decrease作者: Stress-Fracture 時間: 2025-3-24 03:55
Optimal Afterload that Maximizes External Work and Optimal Heart that Minimizes O2 Consumptionmaximizes external work. A theoretical analysis indicated, for a given end-diastolic volume, that external work becomes maximal when effective arterial elastance (..) equals end-systolic elastance (..). The ratio of external work to its maximum value was used to indicate the optimality of the afterl作者: arcane 時間: 2025-3-24 06:46
Influence of Aortic Impedance on the Development of Pressure-Overloaded Left-Ventricular Hypertrophy hypertrophy. To test whether the sustained change in aortic impedance might affect the mode of the development of LV hypertrophy, coarctation of either the ascending (group 1) or abdominal aorta (group 2) was performed in 6-week-old rats. Four weeks after the operation, both groups developed LV hyp作者: 矛盾心理 時間: 2025-3-24 13:32
https://doi.org/10.1007/978-4-431-67041-4adaptation; artery; cardiac function; cardiovascular; circulation; endothelium; heart; heart disease; heart 作者: defendant 時間: 2025-3-24 17:28
Respiratory therapy for children,n-to eightfold. This is accomplished by acute increases in heart rate and stroke volume. The elevations in stroke volume are produced through coordinated adjustments to the cardiac, respiratory, and peripheral circulatory systems which result in 1) enhanced myocardial contractility, 2) decreased sys作者: relieve 時間: 2025-3-24 21:48
,Nursing in Times of Socialism (1970–1973),changes in cardiac muscle function, heart failure is associated with alterations in the vasculature. Functional and structural alterations in the coronary circulation, including a failure of adaptive growth of the coronary circulation, may contribute to the loss of muscle function of the heart and m作者: 侵略者 時間: 2025-3-24 23:36 作者: Mystic 時間: 2025-3-25 04:42 作者: 同步信息 時間: 2025-3-25 11:28
Selective Exercise of Moral Agencyx sequence of compensatory events that ultimately result in an adversely remodeled myocardium and a dilated, thin-walled, spherical ventricle. For a period of time, a preclinical heart failure state may exist in which there is ventricular dysfunction caused by myocardial damage but no clinical evide作者: 集中營 時間: 2025-3-25 12:27
https://doi.org/10.1007/978-1-4757-4163-6olerance acquisition from the aspect of oxygen radical metabolism in the heart. Manganese superoxide dismutase (Mn-SOD), which scavenges the initial reductive metabolite of molecular oxygen on ischemia-reperfusion, was induced after various external stimuli including heat shock, α.-adrenergic stimul作者: Ornithologist 時間: 2025-3-25 17:41
https://doi.org/10.1007/978-1-4757-4163-6markable ability of normal vessels to maintain the caliber appropriate for the blood flow needed for the subservient organ. Indeed, the ability to adjust caliber must be very primitive. The vessel tree, like any closed circuit, can only continue to conduct blood if each branch has an appropriate res作者: 天空 時間: 2025-3-25 22:39 作者: mortuary 時間: 2025-3-26 02:42 作者: 木質(zhì) 時間: 2025-3-26 06:58 作者: 裙帶關(guān)系 時間: 2025-3-26 09:35
https://doi.org/10.1007/978-1-84628-874-6em, we have developed approaches using transient (single-beat) hemodynamic perturbations that eliminate the confounding effects of series interactions and filling variations. In experimental studies in open-chest dogs, we have employed rapid changes in right heart volume to quantitate direct right-t作者: 不可接觸 時間: 2025-3-26 13:48
Nurturing Science-based Venturesxternal to the heart, principally resulting from pericardial and right heart loads. Intrinsic factors can be divided into passive and active components. Passive elements include myocardial viscoelasticity, caused by structural proteins within the sarcomere, extra-cellular matrix proteins, and corona作者: 改正 時間: 2025-3-26 19:41
Nurturing Science-based Venturesxistence of an endocardium-mediated intracavitary autoregulation of cardiac performance has been postulated. Recent discoveries on morphology and function of the endocardium have substantiated this hypothesis. ., phenotypic expression of several receptors, tight and gap junctions, adhesion molecules作者: 阻擋 時間: 2025-3-26 22:49 作者: 單調(diào)女 時間: 2025-3-27 04:41
Nurturing Science-based Venturesmaximizes external work. A theoretical analysis indicated, for a given end-diastolic volume, that external work becomes maximal when effective arterial elastance (..) equals end-systolic elastance (..). The ratio of external work to its maximum value was used to indicate the optimality of the afterl作者: 掙扎 時間: 2025-3-27 08:21
Nurturing Science-based Ventures hypertrophy. To test whether the sustained change in aortic impedance might affect the mode of the development of LV hypertrophy, coarctation of either the ascending (group 1) or abdominal aorta (group 2) was performed in 6-week-old rats. Four weeks after the operation, both groups developed LV hyp作者: Foregery 時間: 2025-3-27 13:20 作者: 混合 時間: 2025-3-27 16:15 作者: 安定 時間: 2025-3-27 19:43
Effects of Promoting Self-Management,, both the myocardium and the ventricle become stiffer. Systolic function is preserved by the accompanying hypertrophy and enhanced contractility unless the increase in myocardial collagen consists of a significant degree of endomyocardial reparative fibrosis. In contrast, chronic volume overload in作者: 繁榮地區(qū) 時間: 2025-3-27 23:54
https://doi.org/10.1007/978-3-319-51769-8rophy of the coronary arterial microvessels (medial thickening and deposition of perivascular collagen) and to endothelial and smooth muscle cell dysfunction. In principle, decreased dilator reserve or impaired coronary autoregulation in cardiac hypertrophy is reversible after relief of the overload作者: foliage 時間: 2025-3-28 03:46 作者: Benign 時間: 2025-3-28 07:10 作者: 得意牛 時間: 2025-3-28 14:03 作者: Mosaic 時間: 2025-3-28 15:27 作者: podiatrist 時間: 2025-3-28 19:29
Nurturing Science-based Venturesof the LV-DPVR appear to be structural, because the relation is generally unaltered by acute pharmacological or physiological manipulations. External influences on the LV-DPVR are primarily direct ventricular interaction and constraining forces from the pericardium. These external loads are more eas作者: 失敗主義者 時間: 2025-3-28 23:33
Nurturing Science-based Venturesteracts with neurohumoral-mediated control. EE cells respond to physical and humoral stimuli with release of several substances that have important inotropic actions on the myocardium. Specific electrophysiological properties determine the intracellular Ca. increase in response to external stimuli a作者: 鍵琴 時間: 2025-3-29 07:04
https://doi.org/10.1007/978-1-84628-874-6 that both diastolic pressure and systolic pressure increase. However, a change in compliance alone can only result in a decrease of diastolic blood pressure. Several methods are available to derive total arterial compliance from aortic pressure and flow. The pulse pressure method, based on the two-作者: 無情 時間: 2025-3-29 10:58
Nurturing Science-based Ventures (0.69 ± 0.26 [SDI]. .. was 0.93 ± 0.08, suggesting that the external work of the left ventricle was nearly maximum at this condition. Similarly, .. was 0.98 ± 0.01, indicating that myocardial oxygen consumption was almost minimum. When dogs ran on a treadmill, their heart rate and cardiac output do作者: 瘙癢 時間: 2025-3-29 15:15 作者: artifice 時間: 2025-3-29 19:15
Vascular Remodeling During Heart Failureof heart failure patients such as exercise intolerance and peripheral muscle fatigue. Potential mechanisms of peripheral vascular alterations are desensitization to neurohormonal activation and vascular remodeling.作者: 帶來墨水 時間: 2025-3-29 20:39
Myocardial Interstitial Collagen Matrix Remodeling in Response to a Chronic Elevation in Ventricular, both the myocardium and the ventricle become stiffer. Systolic function is preserved by the accompanying hypertrophy and enhanced contractility unless the increase in myocardial collagen consists of a significant degree of endomyocardial reparative fibrosis. In contrast, chronic volume overload in作者: Incisor 時間: 2025-3-30 00:50 作者: Anticoagulant 時間: 2025-3-30 04:33 作者: savage 時間: 2025-3-30 09:34
Interaction Between Left-Ventricular Function and Loading Condition in Cardiovascular Diseasediac output is obtained, may not necessarily indicate optimal loading level in this type of cardiac dysfunction. This must be considered clinically when adopting afterload reducing therapy in regional ischemia. Moreover, when intraaortic balloon pumping (IABP) is used for patients with heart failure作者: handle 時間: 2025-3-30 13:50
Adaptation of the Cardiac Cell to Loading: Function in Heart Failureinadequate gene expression. In consequence the amount of Ca. that is released during each heartbeat is less than normal, thus force is reduced. In normal heart muscle, the response of the contractile filaments to Ca. ions depends strongly on sarcomere length, thus explaining Starling’s law of the he作者: BALK 時間: 2025-3-30 16:46 作者: Commonwealth 時間: 2025-3-30 22:44 作者: 存心 時間: 2025-3-31 03:04
Endocardial—Myocardial Interactionteracts with neurohumoral-mediated control. EE cells respond to physical and humoral stimuli with release of several substances that have important inotropic actions on the myocardium. Specific electrophysiological properties determine the intracellular Ca. increase in response to external stimuli a作者: amorphous 時間: 2025-3-31 06:15 作者: Allergic 時間: 2025-3-31 12:32
Optimal Afterload that Maximizes External Work and Optimal Heart that Minimizes O2 Consumption (0.69 ± 0.26 [SDI]. .. was 0.93 ± 0.08, suggesting that the external work of the left ventricle was nearly maximum at this condition. Similarly, .. was 0.98 ± 0.01, indicating that myocardial oxygen consumption was almost minimum. When dogs ran on a treadmill, their heart rate and cardiac output do作者: prosperity 時間: 2025-3-31 13:36 作者: 笨拙的我 時間: 2025-3-31 18:30
