標(biāo)題: Titlebook: Cardiac Remodeling and Failure; Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc Book 2003 Springer Science+Business Media New York 2003 [打印本頁(yè)] 作者: ATE 時(shí)間: 2025-3-21 17:48
書(shū)目名稱(chēng)Cardiac Remodeling and Failure影響因子(影響力)
書(shū)目名稱(chēng)Cardiac Remodeling and Failure影響因子(影響力)學(xué)科排名
書(shū)目名稱(chēng)Cardiac Remodeling and Failure網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱(chēng)Cardiac Remodeling and Failure網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱(chēng)Cardiac Remodeling and Failure被引頻次
書(shū)目名稱(chēng)Cardiac Remodeling and Failure被引頻次學(xué)科排名
書(shū)目名稱(chēng)Cardiac Remodeling and Failure年度引用
書(shū)目名稱(chēng)Cardiac Remodeling and Failure年度引用學(xué)科排名
書(shū)目名稱(chēng)Cardiac Remodeling and Failure讀者反饋
書(shū)目名稱(chēng)Cardiac Remodeling and Failure讀者反饋學(xué)科排名
作者: defuse 時(shí)間: 2025-3-21 22:49 作者: 流動(dòng)才波動(dòng) 時(shí)間: 2025-3-22 03:46 作者: Camouflage 時(shí)間: 2025-3-22 05:27
978-1-4613-4864-1Springer Science+Business Media New York 2003作者: Creatinine-Test 時(shí)間: 2025-3-22 11:11 作者: 輕觸 時(shí)間: 2025-3-22 15:06 作者: 輕觸 時(shí)間: 2025-3-22 19:34
Theaterarbeit in der Gesundheitsf?rderungin chronic volume overload heart failure might be due to an increased level of OFRs and/or decrease in the antioxidant status. We, therefore, studied the effects of chronic volume overload in the absence and presence of vitamin E (antioxidant) on cardiac function and contractility, cardiac malondial作者: Redundant 時(shí)間: 2025-3-23 00:06 作者: MOAN 時(shí)間: 2025-3-23 02:07 作者: 獨(dú)特性 時(shí)間: 2025-3-23 06:49
Theaterarbeit in der Gesundheitsf?rderunged to the SHR (spontaneously hypertensive rat), but unlike the SHR, all animals, male and female, develop fatal congestive heart failure, most in the second year of life. SHHF rats are now being widely used by the pharmaceutical industry to evaluate potential therapeutic agents for preventing the ma作者: 承認(rèn) 時(shí)間: 2025-3-23 10:50
https://doi.org/10.1007/978-94-009-9260-3y discussed. In isolated muscle strips from end-stage failing human hearts, we have recently demonstrated an inverse correlation of Na.?Ca. exchanger expression with the occurrence of diastolic dysfunction. Moreover, it has been hypothesized that different phenotypes of failing human hearts with res作者: Progesterone 時(shí)間: 2025-3-23 14:56
https://doi.org/10.1007/978-94-009-9260-3A- as well as the protein levels of the sarcoplasmic reticulum (SR) Ca. ATPase (encoded by SERCA2 gene) are reduced in hypertrophied and failing myocardium. On the other hand, investigations on phospholamban (PLB) knock-out and phospholamban transgenic mice have proven that the expression ratio of P作者: STAT 時(shí)間: 2025-3-23 19:15 作者: CLAMP 時(shí)間: 2025-3-23 23:05 作者: 受辱 時(shí)間: 2025-3-24 05:31 作者: 小淡水魚(yú) 時(shí)間: 2025-3-24 09:31 作者: transient-pain 時(shí)間: 2025-3-24 11:53
Normen als Grund bilateraler Au?enpolitikpies that arrest or reverse structural abnormalities produce the most striking improvements in patient outcome. Cardiac myocytes comprise about 75% of the heart mass and provide mechanical force for contraction. Consequently, structural changes in these cells are likely to play a key role in heart f作者: 帶子 時(shí)間: 2025-3-24 17:17
Normen als Grund bilateraler Au?enpolitiko investigate at what level these vascular changes can be recognized, a comparison study of vessel continuity using 3-dimensional reconstruction was performed. The eighteen hearts of acute, chronic myocarditis, and control obtained at autopsy were examined to detect the differences of the microvascu作者: TSH582 時(shí)間: 2025-3-24 20:31
Walter Winkler,Paul Schwarzenbergercise, exacerbation of hemodynamic abnormalities, neurohumoral activation and perturbed NO bioavailability could exhaust coronary flow reserve and thereby impair myocardial O. supply. We tested this hypothesis in swine with a 2–3 week old myocardial infarction (MI) and determined whether a loss of NO作者: decode 時(shí)間: 2025-3-25 02:31 作者: ANA 時(shí)間: 2025-3-25 04:44
MedR Schriftenreihe Medizinrechts and are positioned in the assay selection process by a review of studies from our laboratory that examine LV diastolic function, the impact of strain differences, the influence of anesthesia, and fetal echocardiography.作者: amygdala 時(shí)間: 2025-3-25 10:46 作者: 跟隨 時(shí)間: 2025-3-25 13:16
The Contractile and Neurohormonal Roles of Phospholamban in Heart Failure作者: Heart-Attack 時(shí)間: 2025-3-25 16:42
Pawan K. Singal (Professor),Ian M. C. Dixon (Assoc作者: 周興旺 時(shí)間: 2025-3-25 21:22
Book 2003esearch and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap- tive myocardial as well as humoral responses. When these adaptations or remodel- ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, th作者: hypnotic 時(shí)間: 2025-3-26 03:02
Pathophysiology of Heart Failure: Role of Oxygen Free Radicalswas an increase in pulmonary vascular resistance (PVR) and total systemic vascular resistance (TSVR).The decrease in cardiac contractility was associated with an increase in cardiac MDA and a decrease in cardiac antioxidant reserve and antioxidant enzyme activity. Prevention of volume overload induc作者: Palate 時(shí)間: 2025-3-26 06:40 作者: 松軟 時(shí)間: 2025-3-26 08:55 作者: NOTCH 時(shí)間: 2025-3-26 13:59
The Failing SHHF Rat Heart?-adrenoceptor stimulation is markedly blunted; there is evidence to suggest a decline in excitation-contraction coupling gain; and there is a decline in maximal Ca. activated force per cross sectional area. Isolated perfused failing SHHF hearts exhibit a predisposition to develop mechanical alterna作者: Callus 時(shí)間: 2025-3-26 17:46 作者: Physiatrist 時(shí)間: 2025-3-26 23:28 作者: 噱頭 時(shí)間: 2025-3-27 02:01
Sarcoplasmic Reticulum Gene Expression of Ca2+-Cycling Proteins as a Target for the Treatment of Heave and prevent SR remodeling and improve SR Ca.-release and Ca.-uptake activities in the failing hearts. It appears that gene expression for different SR Ca.-cycling proteins may be one of the most viable targets for the therapy of heart failure.作者: 內(nèi)疚 時(shí)間: 2025-3-27 07:10
Effects of the Beta-Adrenoceptor Blocker Carvedilol in Children with Myocardial Failureweeks and 19 years of age) were enrolled in the study, including 10 patients with dilated cardiomyopathy and six with myocardial failure secondary to congenital heart diseases. All 16 patients tolerated the maximum target dose. After six months of Carvedilol therapy, ejection fraction was increased 作者: 枯萎將要 時(shí)間: 2025-3-27 09:43 作者: expire 時(shí)間: 2025-3-27 15:44 作者: Mirage 時(shí)間: 2025-3-27 21:09
Structural Remodeling of Cardiac Myocytes in Hypertrophy and Progression to Failureemic and hypertensive etiologies, progressive chamber dilatation is due largely to myocyte lengthening from series addition of new sarcomeres. During this critical transition period, myocyte diameter does not change. It is encouraging to know that this maladaptive change in myocyte shape is potentia作者: 演繹 時(shí)間: 2025-3-28 01:47 作者: 拋棄的貨物 時(shí)間: 2025-3-28 05:47 作者: 鋼筆記下懲罰 時(shí)間: 2025-3-28 09:59
Thomas Gerlinger,Klaus Stegmüllerdynamics and clinical status. Yet when applied chronically, treatments based on interfering with these systems improve ventricular function, quality of life, and improve survival. The reasons for the success of this therapy lie more in the cellular and biochemical consequences of reducing adrenergic作者: 不可救藥 時(shí)間: 2025-3-28 11:14
Thomas Gerlinger,Klaus Stegmüllerstence in dilated cardiomyopathy is appropriate..There have been recent breakthroughs in understanding the molecular and genetic mechanisms involved in the dilated cardiomyopathies enabling the improvement of diagnostic strategies and the introduction of new therapies. Ongoing evaluation of antivira作者: instate 時(shí)間: 2025-3-28 17:01 作者: invade 時(shí)間: 2025-3-28 21:23 作者: 扔掉掐死你 時(shí)間: 2025-3-29 00:44
https://doi.org/10.1007/978-94-009-9260-3enotype at the structural, functional and molecular level. The state of knowledge on the molecular aspects and physiological functions of SERCA2a by PLB is briefly reviewed. Alltogether, these observations on PLB function have led us to explore novel strategies of interference by genetic manipulatio作者: 上釉彩 時(shí)間: 2025-3-29 06:31
Metric Geometry in Normed Spaces,ve and prevent SR remodeling and improve SR Ca.-release and Ca.-uptake activities in the failing hearts. It appears that gene expression for different SR Ca.-cycling proteins may be one of the most viable targets for the therapy of heart failure.作者: Critical 時(shí)間: 2025-3-29 09:43
https://doi.org/10.1007/978-3-662-41637-2weeks and 19 years of age) were enrolled in the study, including 10 patients with dilated cardiomyopathy and six with myocardial failure secondary to congenital heart diseases. All 16 patients tolerated the maximum target dose. After six months of Carvedilol therapy, ejection fraction was increased 作者: 泥瓦匠 時(shí)間: 2025-3-29 12:28
Unconditional Convergence and Bases,expression of annexin V is increased and localization becomes heterogenous leading to myocytes devoided of annexin V and interstitial tissue strongly labelled. Annexin VII is known as a skeletal muscle annexin but is also expressed in the heart. It is the third annexin which could be related to regu作者: plasma 時(shí)間: 2025-3-29 19:04
Metric Geometry in Normed Spaces,isometric force, calcium sensitivity, or cooperative activation. Lastly, protein kinase A phosphorylation of troponin I resulted in a large increase in the calcium sensitive activation of the thin filament with no change in maximal activation. These results are interpreted in the context of a molecu作者: Osteoarthritis 時(shí)間: 2025-3-29 21:44 作者: Suggestions 時(shí)間: 2025-3-30 02:44 作者: 劇毒 時(shí)間: 2025-3-30 05:48
1389-1774 s well as humoral responses. When these adaptations or remodel- ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, th978-1-4613-4864-1978-1-4419-9262-8Series ISSN 1389-1774 作者: Promotion 時(shí)間: 2025-3-30 10:16
Pathophysiology of Heart Failure: Role of Oxygen Free Radicalsin chronic volume overload heart failure might be due to an increased level of OFRs and/or decrease in the antioxidant status. We, therefore, studied the effects of chronic volume overload in the absence and presence of vitamin E (antioxidant) on cardiac function and contractility, cardiac malondial作者: 自負(fù)的人 時(shí)間: 2025-3-30 13:23
Therapeutics in Congestive Heart Failure: From Hemodynamics to Neurohormonespheral perfusion, sodium retention, increased ventricular afterload, and progressively deteriorating left ventricular pumping capacity. In new or decompensated heart failure, this hemodynamic paradigm predicts acute clinical improvement. Chronically, however, treatment based fundamentally on inotrop作者: 急急忙忙 時(shí)間: 2025-3-30 16:48
Dilated Cardiomyopathies and Congestive Heart Failurestinguished: dilated, hypertrophic, restrictive, right ventricular, and non-classifiable cardiomyopathies with distinct hemodynamic properties. The new WHO/WHF definition also comprises inflammatory cardiomyopathy, defined as myocarditis in association with cardiac dysfunction. Idiopathic, autoimmun作者: FOLD 時(shí)間: 2025-3-30 22:08
The Failing SHHF Rat Hearted to the SHR (spontaneously hypertensive rat), but unlike the SHR, all animals, male and female, develop fatal congestive heart failure, most in the second year of life. SHHF rats are now being widely used by the pharmaceutical industry to evaluate potential therapeutic agents for preventing the ma作者: sinoatrial-node 時(shí)間: 2025-3-31 03:07 作者: 值得贊賞 時(shí)間: 2025-3-31 06:09 作者: 光亮 時(shí)間: 2025-3-31 10:33
Sarcoplasmic Reticulum Gene Expression of Ca2+-Cycling Proteins as a Target for the Treatment of Heatrin (CQS) are known to determine the function of cardiac sarcoplasmic reticulum (SR). Protein kinase A is known to phosphoryate PLB and RyR whereas Ca./calmodulin kinase phosphorylates PLB, SERCA2a and RyR and thus these regulatory proteins have been shown to alter the activities of SR Ca.-cycling 作者: 彩色 時(shí)間: 2025-3-31 15:44 作者: Mercurial 時(shí)間: 2025-3-31 18:21 作者: musicologist 時(shí)間: 2025-3-31 21:55 作者: 舊石器時(shí)代 時(shí)間: 2025-4-1 04:27
Structural Remodeling of Cardiac Myocytes in Hypertrophy and Progression to Failurepies that arrest or reverse structural abnormalities produce the most striking improvements in patient outcome. Cardiac myocytes comprise about 75% of the heart mass and provide mechanical force for contraction. Consequently, structural changes in these cells are likely to play a key role in heart f
